Spastic Dysarthria Flashcards
Hallmarks of spastic dysarthria
Reduced range; reduced spee
Overarching cause of spastic dysarthria
Bilateral UMN damage
Direct and indirect activation pathways
Direct (corticobulbar): facilitate discrete voluntary movements
Indirect (basal ganglia): Regulate reflexes, posture, and tone
UMN damage to direct activation pathway
Loss of skilled movement; hypotonia
UMN damage to indirect activation pathway
Increased muscle tone (hypertonia); spasticity; hyperactive reflexes
Distinguishing signs of UMN damage
Spasticity (spastic weakness)
Normal or increased reflexes
Involves many muscle groups
Lability
Also known as pseudobulbar effect; pt. has random outbursts of crying or laughing
Neuromuscular deficits (direction, range, rhythm, rate, force, tone)
Direction: Normal Range: Reduced Rhythm: Regular for repetitive movements Rate: Reduced Force: Reduced Tone: Excessive
Speech characteristics in spastic dysarthria
Voice: often harsh
Prosody: slow, excess and equal stress
Pt. complaints
Slow speech
Increased effort
Chewing and swallowing problems
Lability
CVA and spastic dysarthria
Bilateral cortical, lacunar infarcts, or single brainstem
Binswanger’s subcortical encephalopathy
UMN disorder caused by hypertensive lacunar strokes
Primary lateral sclerosis
Degenerative corticobulbar and corticospinal disease largely affecting the limbs
Unilateral UNM Dysarthria
Uncommon and rarely worse than moderate; usually resolves. pt’s complain of slurred speech, chewing difficulties, or drooling on the affected side.