SMT-3 Manual, pgs 28-40 Flashcards

1
Q
  1. According to van Dieen et al 2003, is TrA involved in spine stability?
A

No, if taught to contract the core, they would be contracting above required level for stabilization

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2
Q
  1. Is there a relation between LBP and abdominal atrophy according to Mannion et all 2001
A

No

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3
Q
  1. What are improvements in LBP w/ core strength due to
A
  • Better neural activation

- Overcoming psychological aspects of pain

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4
Q
  1. How did Kavcic et al 2004 relate core and athletes
A
  • Athletes do not specifically activate a group of core muscles
  • They use a natural muscular activation pattern
  • Conscious effort beyond natural activation decreases stability
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5
Q
  1. How did Helewa et al 1999 show asymptomatics with weak core
A

Asymptomatic received back education vs bac education with core strengthening. Found no difference.

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6
Q

546,7. Does poor trunk proprioception predispose athletes to LBP? Author?

A

No. Silfies 2007

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7
Q

548,9. Who did SR on LBP and core stab (2 articles)? What were the conclusions

A

Authors:
1. van Tulder 2000
2. Cairnes 2006
Conclusions:
1. Stab/ Core strength is no more effective than general ex to prevent/manage LBP
2. + Effects are secondary to effects that physical ex gives anyway

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8
Q
  1. According to Lederman 2007 are there studies to date that show that strengthening core muscles (abdominals, TrA) can decrease back pain?
A

No. Exception being O’Sullivan 1997 for spondylolysis and spondylolisthesis.

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9
Q
  1. What is RUSI
A

Rehabilitation Ultrasound Imaging, an alternative method to EMG to indirectly measure the recruitment of ab mm by assessing morphological changes

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10
Q
  1. Who reviewed reliability of RUSI?
  2. What 4 variables were used?
  3. What were the static muscle thickness results?
  4. Changes in muscle thickness?
  5. Thickness changes over time?
A
  1. Costa 2009.
    553.
  2. Thickness (at rest and contracted)
  3. Thickness changes (measure activity by degree of thickness changes between rest and contracted states)
  4. Diff in thickness changes over time (improved or deterioration)
  5. Quality of study (+, -, doubtful, not informative)
    Static muscle thickness results
  6. ICC >80% of ICC values ranging from .8-.0 (Very good)
    • ICC
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11
Q
  1. What type of pts were used in studies by Costa 2009 (RUSI study)
A

Out of 21 studies, only 2 recruited pts with LBP

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12
Q
  1. Are Costa 2009 results clinically relevant?

Results were as follows:

  1. ICC >80% of ICC values ranging from .8-.0 (Very good)
  2. ICC
A

No, you have to use pts actually seeking care for that condition.

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13
Q
  1. What did Vera-Garcia 2007 compare?
  2. Which was most ineffective?
  3. Which improved spinal stability (at the cost of additional spinal compression)
  4. Improved spinal stability and without spinal compression
A
  1. Ab hollowing vs ab bracing vs natural strategy used in conjunction w/ sudden trunk perturbations
  2. Ab hollowing
  3. Ab bracing
  4. Natural strategies
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14
Q
  1. According to Davis 2002, what increased spinal compression
A

Mental processing and stress.

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15
Q
  1. What does Lederman 2010 say about pain and core exercises?
A
  • Core ex could be a constant reminder of pt’s condition, reinforcing a continuous cycle of pain
  • Pt’s should be encouraged to relax their trunk instead of holding it tight
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16
Q
  1. Who found evidence of lumbar multifidus wasting ipsilateral to LBP symptoms
  2. What area was measured?
  3. What subjects were used?
  4. Who had marked asymmetry
  5. Where was LM atrophy found
  6. How soon did atrophy occur?
A
  1. Hides 1996
  2. L2-S1 cross sectional area measured w/ real time ultrasound
  3. 51 asymptomatic. 26 with 1st episode actue and subacute unilateral LBP
  4. LBP pts. 31% change in LBP pts vs 3% asymptomatic
    • Symptomatic segment, ipsilateral to pain; confined predominantly to that segment.
  5. 24hrs within injury in one subject
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17
Q

571.). In Hides et al, 1996, what level was mostly affected in regards to lumbar multifidus muscles atrophy?

A
  1. L5
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18
Q
  1. How did Hides et al 1996 train the lumbar multifidus muscles?
A
  1. Treatment group. Specific Lumbar multifidus exercises vs control non-active standard medical management.
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19
Q
  1. what were the dependent variables for the Hides et al 1996?
A

573: Pain, disability, range of motion, lumbar multifidus cross sectional area (taken weekly over the 4weeks of intervention period and at 10 weeks).

20
Q

574: In Hides et al 1996 what were the results for pain & disability?

A

574: no difference between the 2 groups @ 4 weeks.

21
Q

575: In Hides et al 1996 what were the results of the experimental group?

A

575: LM CSA was 0.7%

22
Q

576: In Hides et al 1996 what were the results of the control group?

A

576: LM CSA remained 16.8%

23
Q

577: In Hides et al 1996 what were the results @ 1 year follow up?

A

577: Only 30% of the LM exercise group suffered recurrence of LBP vs 80% of the control group.

24
Q

578: What did Hides et al 1996 say contributed to the decrease in LM?

A

578:

  1. Reflex inhibition
  2. Pain inhibition
  3. Disuse atrophy
    - But since reduced mm size was seen after resolution of pain; pain inhibition was not the cause.
    - Since the onset was rapid and localized, the decrease in mm suggested disuse atrophy was not the cause.
    * * Reflex inhibition is most likely the cause.
25
Q

579: What was the conclusion for Hides et al 1996?

A

579: Sensory input from the joint is processed & modulated in the SC to produce an effect in the specific muscles which act on the joint in question (Richardson et al 1999, Hodges 2013)

26
Q

580: What did Kader et al 2000 investigate regarding LM atrophy?

A

580: Side and vertebral level of LM degeneration (decrease in mm size) in pts. with LBP.

27
Q

581: In Kader et al 2000 how was LM atrophy grade?

A

581:

Mild 50% loss

28
Q

582: In Kader et al 2000 what % of pts. presented with LM atrophy?

A

582: 80% of pts. Majority had atrophy BL @ both L4-L5 & L5-S1 (most frequent)

29
Q

583: In Kader 2000 et al what else did 35% of pts. present with?

A

583: Disc degeneration. Most common @ L4-L5 & L5-S1

30
Q

584: What correlation in Kader et al WAS found to be significant?

A

584: LM atrophy was significantly correlated with Leg pain.

31
Q

585: What correlation in Kader et al was NOT found to be significant?

A

585: The following were NOT found to be significantly correlated…
1. LM atrophy & LBP
2. LM atrophy & Radic. symptoms (.08)
3. LM atrophy & N. root compression (.14)
4. LM atrophy & HNP (.10)
5. LM atrophy & # of degenerative discs (.08)

32
Q

586: In Kader et al 2000, in 18/38 pts. w/ root pain & 25/57 w/ leg pain (radic or non radic) what was the only MRI explanation/finding?

A

586: Only LM atrophy was present. No HNP, stenosis, N. root compression, etc. present.

33
Q

587: Bogduk 1989 believes what causes LM atrophy?

A

587: Lumbar Dorsal Root Syndrome

34
Q

588: According to Bogduk 1989, how does Lumbar Dorsal Ramus Syndrome contribute to leg pain referred from LBP?

A

588: LBP with referred leg pain is induced by the irritation of structures innervated by the dorsal ramus nerve; medial branch of the posterior primary ramus. Such as facets, LM, interspinous lig, or myofascial pain/spasm/ischemia. Resulting in a self-sustained cycle promoting LM atrophy.

35
Q

589: Who measured fat infiltration of LM in LBP for 854 13 y.o. (442) and 40 y.o. (412) Danish participants?

A

589: Kjaer et al 2007

36
Q

590: In Kjaer et al 2007, what population was fat infiltration more common?

A

590: Adults (81%) vs adolescents (14%). with a (+) correlation between LM fat infiltration & LBP.

37
Q

591: In Kjaer et al 2007, what was the most common segment for fat infiltration?

A

591: L5 with no sig. diff. between sides.

38
Q

592: In Kjaer et al 2007, who had sig. less severe fat infiltrations?

A

592: Active adults.

39
Q

593: In Kjaer et al 2007, how was fat infiltration severity ranked?

A

593:
Normal = 0-10% fat w/ mm
Slight = 10-50% fat w/ mm
Severe = > 50% fat w/ mm

40
Q

594: In Kjaer et al 2007, what were their conclusions?

A

594: This is the 1st convincing evidence that fat infiltration in LM is strongly associated w/ LBP in adults.

41
Q

595: In Kjaer et al 2007, was there a difference in BMI levels & fat infiltration?

A

595: No, association was not affected by BMI.

42
Q

596: In Kjaer et al 2007, what were the two main “trouble area” segments where fat infiltration was found?

A

596: L4 and L5. Suggesting that LBP initiates the mm changes and not vice versa. However it remains unclear.

43
Q

597: In Kjaer et al 2007, what remains unclear in regards to fat infiltration of LM?

A

597:
1. If LM fat infiltration is the cause of LBP or vice versa…
2. If LM fat infiltration is reversible…
3. If LM fat infiltration reversibility coincides w/ improved symptoms…

44
Q

598: Who attempted to determine the most effective exercise for LM by comparing (10 weeks) of Ab Hollowing vs. Stabilization + dynamic concentric-eccentric resistance training vs. Stabilization + dynamic concentric-isometric-eccentric resistance training?

A

598: Danneels et al 2001

45
Q

599: In Danneels et al 2001, what was the Group 1 Tx?

A

599: Abdominal hollowing to co-contract LM & TrA in supine, sitting, standing, and functional activities. Done at a level = 30% MVIC (Jull & Richardson 1994, Richardson et al 1999)

46
Q

600: In Danneels et al 2001, what was the Group 2 Tx?

A

600: Stabilization + progressive concentric-eccentric resistance training;
1. Leg ext in 4 point kneeling
2. Trunk lifting from prone
3. BL leg lifting in prone
* ** 3 sets each ex. @ 70% 1 RM (15-18 reps or until fatigue) w/ 2 sec concentric and 2 second eccentric.