SMALL B Flashcards
Small bowel obstruction: surgical treatment goals
(a) distinguish mechanical obstruction from ileus (b) determine the etiology of the obstruction (c) discriminate partial from complete obstruction, and (d) discriminate simple from strangulating obstruction
Intraluminal
Foreign bodies, gallstones, meconium
Intramural
Tumors, Crohn’s disease– associated inflammatory strictures
Extrinsic
Adhesions, hernias, carcinomatosis Most common etiology is adhesions
Intestinal obstruction sx
Crampy intermittent abdominal pain and distension No BM or flatus Previous surgery Hypoactive/hyperactive bowel sounds Metallic quality Ascites– intravascular volume depleted Severe, persistent pain suggests strangulation or perforation
Lab results
BUN/CR elevated– >20:1 Hypernatremia Urine electrolytes Hemoconcentration Acidosis Leukocytosis
Imaging
Supine and erect abdominal x-ray Erect CXR –> Free air Single-contrast barium enema Small bowel series CT
What is the best imaging for SBO and what are the findings?
Abdominal x-ray series -Dilated small bowel loops (> 3 cm) -Air-fluid levels seen on upright films -A paucity of air in colon
CT findings
PO contrast, water soluble -Transition zone: dilation proximally, decompression distally (contrast proximally, no contrast distally) -Colon has little gas or fluid -CT shows strangulation
CT findings suggestive of strangulation
thickening of the bowel wall pneumatosis intestinalis (air in the bowel wall) portal venous gas mesenteric haziness poor uptake of intravenous contrast into the wall of the affected bowel -IV contrast if compromise of vasculature is suspected
Small Bowel Series
Detects low-grade or partial small bowel obstruction Water soluble PO contrast (or in NG tube if vomiting) Sequential x-rays to follow where contrast is traveling
Approach to SBO
Management based on: location of obstruction, proximal, mid-gut, distal Level or severity: partial, complete, strangulated, closed-loop
Strangulated bowel obstruction
If the intramural pressure becomes high enough, intestinal microvascular perfusion is impaired, leading to intestinal ischemia and, ultimately, necrosis.
Partial SBO
only a portion of the intestinal lumen is occluded, allowing passage of some gas and fluid. Continued passage of flatus and/or stool beyond 6 to 12 hours after onset of symptoms is characteristic.
Complete SBO
lumen becomes obstructed, gas and fluid accumulate proximally, the bowel distends and intraluminal and intramural pressures rise, and the development of strangulation is more likely.