Sleep Apnea Flashcards

1
Q

OSA

A

Obstructive sleep apnea (OSA) is defined by repeated episodes of upper airway closure during sleep that result in recurrent oxyhemoglobin desaturation and sleep fragmentation

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2
Q

Health Consequences of Untreated OSA

A
  • daytime sleepiness –> MVAs, occupational accidents, reduced quality of life
  • oxyhemoglobin desaturations and physiologic stresses from repetitive upper airway obstruction –> increased BP and cardiovascular disease
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3
Q

Apnea

A

Breathing cessation for ≥ 10 seconds

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4
Q

Hypopnea

A

Breathing flow reduction for ≥ 10 seconds accompanied by either a ≥3% or ≥4% oxyhemoglobin desaturation or by arousal from sleep

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5
Q

Risk Factors for OSA

A
  • Obesity, especially with body mass index > 35 kg/m2
  • Family history of obstructive sleep apnea •Retrognathia
  • Treatment-resistant hypertension
  • Congestive heart failure
  • Atrial fibrillation
  • Stroke
  • Type 2 diabetes
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6
Q

Symptoms of OSA

A
  • Witnessed episodes of apnea
  • Loud, frequent, bothersome snoring
  • Choking/gasping during sleep
  • Excessive daytime sleepiness
  • Drowsy driving (recent sleepinessassociated motor vehicle accident or near-miss)
  • Unrefreshing sleep
  • Sleep fragmentation
  • Insomnia
  • Nocturia
  • Morning headaches
  • Decreased concentration
  • Memory loss
  • Decreased libido
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7
Q
A
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8
Q

Central Sleep Apnea Syndrome

A

Central sleep apnea is a disorder in which your breathing repeatedly stops and starts during sleep. Central sleep apnea occurs because your brain doesn’t send proper signals to the muscles that control your breathing.

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9
Q

OSA and Children

A

(1) All children/adolescents should be screened for snoring.
(2) Polysomnography should be performed in children/adolescents with snoring and symptoms/signs of OSAS; if polysomnography is not available, then alternative diagnostic tests or referral to a specialist for more extensive evaluation may be considered.
(3) Adenotonsillectomy is recommended as the first-line treatment of patients with adenotonsillar hypertrophy.
(4) High-risk patients should be monitored as inpatients postoperatively.
(5) Patients should be reevaluated postoperatively to determine whether further treatment is required. Objective testing should be performed in patients who are high risk or have persistent symptoms/signs of OSAS after therapy.
(6) Continuous positive airway pressure is recommended as treatment if adenotonsillectomy is not performed or if OSAS persists postoperatively.
(7) Weight loss is recommended in addition to other therapy in patients who are overweight or obese.
(8) Intranasal corticosteroids are an option for children with mild OSAS in whom adenotonsillectomy is contraindicated or for mild postoperative OSAS.

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10
Q

Treatment for OSA in Adults

A
  • weight loss
  • CPAP
  • manibular advancemnt devices
  • change in sleep position
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