sleep Flashcards

1
Q

what is a circadian rhythm?

A

24hr cycle in physiological process of living beings; internally generated & externally moderated

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2
Q

3 main measures of sleep

A

EEG, EOG,EMG

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3
Q

4 stages of sleep

A

stage 1: low voltage high frequency
by stage 4: voltage increase but frequency decrease
IS A CYCLE so people travel through them repeatedly

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4
Q

alpha waves

A

weaker bursts of eeg waves

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5
Q

delta waves

A

largest and slowest

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6
Q

REM sleep

what increases?

A

o2 consumption, cerebral firing & blood flow
Muscle contraction
Range of ANS functions i.e. blood pressure

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7
Q

REM and dreaming arguments

A

FOR: Desseilles et al. 2001 (80% of awakenings during REM can report dream)
BUT:
- Mcnamara et al 2010 (review of REM & dreams)
- Siegel 2011 “ (ALSO says about REM & health/memory)
- Oudiette et al 2012 (antidepressants reduce REM but not dreaming)
- Solms, (1997) lesions can affect dreams but not REM

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8
Q

Hobson & Mcarley 1989 theory

A

activation synthesis theory:

  • various waves activate regions (pons)
  • cerebral cortex tries to make sense
  • we dont act out our dreams bc of muscle atonia
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9
Q

Recoperation theories of sleep

A
  • awake disrupts homestasis
  • need sleep to restore
  • also conserves energy: smaller animals sleep more
  • BUT isnt affected by sport
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10
Q

Ecological Niche

A

we’ve adapted this way to avoid preds at night time (Siegel 2009)

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11
Q

memory consolidation: FOR (4)

SWS: NREM

A

Dikelmann & born (2010): sleep helps remember material
NREM: seems to aid consolidation: ESP DECLARATIVE MEMORY (facts) Marshal et al. 2006
REM: “ REM may aid consolidation of NON DEC memory (unconscious)
4) PATTERNS of neuro activity occur during SWS; brain rehearsing? (Dikelmann & born 2010) & (Euston et al 2007)

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12
Q

memory consolidation: AGAINST

A

No correlation between species and time spent asleep and learning capacity - we sleep less than some species but are more intelligent (Siegel, 2001)
NO correlation between time in REM sleep and !) (Borrow et al 1980)

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13
Q

what is the problem with interpreting results from sleep deprivation studies?

A

May be confoudning variables i.e. stress is often the cause of sleep loss - reason for seeing what we are seeing?

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14
Q

Sleep deprivation & humans

A

Durmer & Dinges (2005) - moderate deprivation -moderate effects

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15
Q

deprivation on complex cognition

A

less consistent results - seems that a substantial amount of deprivation is needed for a consistent disruption (Drummond et al 2004) (alexander & home, 2006)

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16
Q

ONLY SOME COG FUNCTIONS ARE SUSCEPTIBLE

A

executive functioning: creative thinking > logical deduction (Nilsson et al 2005)

17
Q

Rechtshaffen & Bergmann (1995)

A

sleep deprivation on animals: carousel

18
Q

Limitations with

Rechtshaffen & Bergmann (1995)

A
  • humans and rats have been kept awake longer than that before w.out dying
    Siegel (2009) & Rial et al. (2007)
  • it is prob stress that killing them: all have enlarged adrenal gland (HPA axis)
19
Q

REM sleep & deprivation: 2 main effects

A

1) REM rebound - more REM in following few nights (Brunner et al, 1990)
2) more deprivation the more likely REM will occur (i.e. frequency)
~~ REM must be separate from SWS

20
Q

Sleep recovery

A

SWS sleep becomes more efficient DOESNT INCREASE SLEEP ( Elmenhorst et al 2008)

21
Q

Evidence in support of the efficient SWS

A
  • waking ppts in REM doesnt interfere with their performance during the day but SWS interruption does (Nykamp et al. 1998)
  • if sleep time is reduced, then time in SWS stays the same but REM time decreases
22
Q

reticular formation WHAT

A

In the brain stem (in the little bulge at front)

intricate network of neurons extending from spinal cord to thalamus

23
Q

reticular formation & sleep

A

Bremer 1936: severed cat brains
disconnect forebrain & sensory input coming in
Cutting the forebrain off - contin sleep
cutting lower than Pons - no effect
~~~ reticular formation affects wakefulness

24
Q

Sleep nuclei

A

reticular REM:controls REM sleep
each site (nuclei) –> controls a particular characteristic of REM sleep
TOGETHER THEY MAKE UP REM SLEEP when all activated

25
Q

2 main elements of a biological clock

A

1) FREE RUNNING: maintains own cycle & doesnt need input from external cues - not totally in line with outside world tho
2) ENTRAINMENT: we can train our bio clock to be in synchro with an external stimulus

26
Q

name for a stimulus that trains our biological clock

A

Zeitgeber

27
Q

where is the biological clock

A

suprachiasmatic nucleus (tiny pair of neuron clusters in the hypothamalmus

28
Q

lesions to this area cause disruption to circadian cycles i.e. sleep - waking cycle - EVIDENCE

A

1) Ralph & menaker (1990) hamster experiment

2) the nuclei displays cycles that are entrained by light /dark cycle

29
Q

suprachaismatic nucleus may not be the only biological clock though - evidence (3)

A

1) bilateral lesions can leave SOME circadian rhythms unaffected
2) bilateral SCN lesions dont elimainate ability of all enviro stimuli to entrain circadian rhythms (i.e. can be others that are entrained)
3) other tissue cells display free running circadian rhythms when maintained in tissue culture

30
Q

neural mechanisms of environment

~ how does light/dark entrain the sleep wake cycle

A

Morin & allen (2006):
~ cutting optic tracts BEFORE chaism - eliminates pathway (after, DOES NOT)
1) Retinohypothalamic tracts leave the chaism & project to the SCN (biological clock)
2) this pathway leads to pineal gland (& other areas)
3) pineal gland secretes MELATONIN

31
Q

Melatonin

A

If light is present: melatonin secretion is inhibited

if darkness: melatonin is released –> melatonin involved with decrease in temp –> leading to sleep?