Brainscape's Knowledge GenomeTM

Browse over 1 million classes created by top students, professors, publishers, and experts.


See full index

Skin > Skin Lesions/Skin Conditions > Flashcards

Skin Lesions/Skin Conditions Flashcards

1
Q

Treatment of mild comedonal acne vulgaris

A

Topical retinoid or benzoyl peroxide

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Treatment of mild to moderate inflammatory acne

A

Topical retinoid, benzoyl peroxide, or antibiotic

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Treatment of moderate to severe inflammatory acne

A

Topical retinoid or benzoyl peroxide or oral antibiotics

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Treatment of severe nodulocystic acne

A

Oral retinoid (derm referral), topical benzoyl peroxide, or oral antibiotic

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

If limited response after 3 months antibiotic therapy….

A

Increase does, change medication, or refer to derm

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Follicular plug

A

Excessive keratin in an inactive hair follicle»resulting in rough, cone-shaped elevated papules

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Teratogenic

A

Can disturb the development of the fetus; increased risk of spontaneous abortion, birth defects

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Adverse effects of oral isotetinoin

A

dry skin, lips, nose, mouth, pruritus, epitaxis, conjunctivitis; increased intracranial pressure, pancreatitis, IBS, premature epiphyseal closure, hyperlipidemia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Telangiectasia

A

Condition in which widened venules cause threadlike red lines or patterns on the skin (spider veins)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Differential diagnoses of acne

A

Peri-oral dermatitis, keratosis pilaris, rosacea, steroid folliculitis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Rosacea clinical presentation

A

Skin: papules and pustules, erythema, telangiectasia
Nonskin: mild conjunctivitis, redness of sclrea, blepharitis, telangiectasia of lid

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Differential diagnoses of rosacea

A

Staphylococcal folliculitis, pustular tinea, systemic lupus erythematous, perioral dermatitis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Classification of rosacea

A

Erythematotelangiectatic (erythema and telangiectasia on nose and cheeks)
Papulopustular (erythema with papules and pustules on nose and chin, erythematous patches on cheeks)
Phymatous (facial erythema, scattered pustules and papules on forehead, nose, cheeks, chin; thick, oily skin; rhinophyma)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Stage 1 pressure ulcer

A

Nonblanchable erythema

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Stage 2 pressure ulcer

A

Loss of epidermis w/ or w/o true ulceration; ulcer shallow with a pink to red wound be without slough

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Stage 3 pressure ulcer

A

Full-thickness (into subcutaneous or beyond) loss w/o underlying muscle, tendon, or bone exposure

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

Stage 4 pressure ulcer

A

Full thickness with underlying bone, tendon, or muscle exposure

18
Q

Unstageable pressure ulcer

A

Covered with debris or eschar which does not allow depth assessment

19
Q

Diffuse hair loss (telogen effluvium)

A

Reversible; caused by chemo or other meds (lithium, warfarin, beta-blockers, retinoids, interferon, amphetamine); initial labs - TSH, hemoglobin/hematocrit, iron

20
Q

Patterened hair loss

A

Miniaturization of individual hair follicles/shaft due to effects of dihydrotestosterone, hyperandrogenism; topical minoxidil, oral finasteride, hair transplant

21
Q

Circumscribed (non-scarring) alopecia

A

Autoimmune process in which T-lymphocytes surround hair bulb deep in dermis; no inflammation

22
Q

Scarring alopecia

A

Irreversible; active lesions with scalp inflammation and eventually scalp scarring; lichen planus and discoid lupus cause

23
Q

Characteristics of psoriasis

A
  • Occurs at any age
  • chronic relapsing skin disease
  • sharply demarcated plaques
  • silvery scales
    • Auspitz sign
  • extensor surfaces (may be on palms and soles)
24
Q

Symptoms and associated disease with psoriasis

A
  • Guttate psoriasis can be triggered by streptococcal infection
  • onycholysis and pitting of nails
  • Koebner
  • psoriatic arthritis
25
Q

Pathogenesis of psoriasis

A

Increased proliferation and shedding of keratinocytes

26
Q

Treatment of psoriasis

A

Topical retinoids and steroids

UV light

27
Q

Characteristics of lichen planus

A
  • skin, hair, nails and mucous membranes (especially oral mucosa) may be affected
  • insatiable itching
  • can occur abruptly or secondary to drug
  • flexor surfaces
28
Q

Symptoms and associated disease of lichen planus

A
  • violaceous and hyperpigmented polygonal papules and plaques
  • Wickham striae (thick hyperkeratotic plaque with white lacelike pattern)
  • Koebner
  • oral squamous carcinoma rare
29
Q

5 Ps of lichen planus

A 
Pruritic
Planar
Polygonal
Purple
Papules/plaques
30
Q

Treatment for lichen planus

A
  • Self-limited resolves in 8-12 months

- first line is topical steroids

31
Q

Seborrheic dermatitis characteristics

A
  • Common, chronic inflammatory papulosquamous disease
  • any age; peaks in adolescence
  • linked to Pityrosporun(Malassezia) yeast
  • favors areas with sebaceous glands (scalp margins, central face, presternal areas)
32
Q

Seborrheic dermatits symptoms and associated diseases

A
  • moist, transparent to yellow, greasy and scaling with coalescing red patches and plaques
  • locations: eyebrows, base of eyelashes, nasolabial folds and paranasal skin, external ear canals
  • flexural skin (skin on skin)
  • seborrheic blepharitis
  • cradle cap
33
Q

Seborrheic dermatitis treatment

A
  • topical antifungals

- antidandruff shampoo or soaps containing zinc pyrithione (ZNP)

34
Q

Acute atopic dermatitis

A

Poorly defined erythematous patches, papules, and plaques with or without scale. Skin may be extremely dry, cracked, and scaly.

35
Q

Chronic atopic dermatitis

A

Lichenification results from repeated rubbing or scratching; Fissures

36
Q

Atopic dermatitis characteristics

A
  • Usually begins in infancy
  • Characterized by dry skin and pruritus; consequent rubbing leads to increased inflammation and lichenification and to further itching and scratching: itch-scratch cycle
  • skin barrier dysfunction
  • Often associated with a family history of AD, allergic rhinitis, and asthma
  • Most experts believe there is a genetic basis for AD (80% concordance in monozygotic twins), influenced by environmental factors
  • Staph aureus exotoxins may trigger
37
Q

Location of atopic dermatitis

A

Predilection for the flexures, front and sides of the neck, eyelids, forehead, face, wrists, and dorsa of the feet and hands. Generalized in severe disease.

38
Q

Exacerbations of atopic dermatitis

A
  • Skin Barrier Disruption: decrease of barrier function and increased transepidermal water loss.
  • Infections: S. aureus secondary infection is often present in severe case
39
Q

Special populations of atopic dermatitis

A
  • Children: affects the cubital and popliteal fossae, neck, and face
  • Adults: lichenification and exoriations
40
Q

Atopic dermatitis management

A
  • optimal skin care (hydration, topical, unscented moisturizers)
  • NO ANTIHISTAMINES
  • Antibiotics if secondary infection suspected
  • wet-wrap therapy

Skin (9 decks)

Brainscape helps you reach your goals faster, through stronger study habits.
© 2024 Bold Learning Solutions. Terms and Conditions