skin integrity and wound care Flashcards

1
Q

Promoting healthy skin

A
  • Self assessment
  • Bathing
  • Nutrition
  • Hydration
  • Sunscreen
  • Moisturizing
  • Perfusion
  • Mobility
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2
Q

Skin care strategies for infants

A
  • gentle bathing
  • avoid alcohol based products
  • no direct adhesives
  • limit topical agents
  • maintain adequate nutrition
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3
Q

Skin care strategies for adults

A
  • complete bath every other day
  • tepid water temperature
  • pat dry
  • no tight or rubbing clothes
  • maintain adequate nutrition
  • avoid caffeine & alcohol
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4
Q

Cultural Considerations

A
  • Darker skinned individuals may be subject to drier hair, scalps, and skin
  • Use of skin moisturizers, hair oils and different cleansers may be necessary
  • Some products contain shea butter, olive oil, petrolatum and more
  • caution products that are oil or alcohol based may also increase damage to skin
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5
Q

Types of impaired skin integrity

A

Disorders (inflammatory, papulosquamous, vesiculobullous, vascular), lesions, insect bites, infections, tumors (begin, malignant), burns, pressure ulcer, surgery & trauma

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6
Q

Wound classifications

A
  • Acute wound- occurred suddenly, trauma, heal in orderly sequence of events
  • Chronic wound- caused by chronic condition & don’t heal in orderly manner
  • Partial thickness wound- involves partial loss of skin layers, not deeper tissues, superficial & painful due to exposed nerve endings, scarped knee, heals quickly
  • Full thickness wound- involves total loss of epidermis & dermis, plus extends into subcutaneous & occasional muscle, heals by complex process of scar formation
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7
Q

Normal wound healing

A

Intentions; primary, secondary, tertiary
Regeneration
Scarring

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8
Q

Phases of wound healing

A
  1. Inflammatory (control bleeding, establish clean wound bed, clot dissolution releases growth factors, release vasoactive substances, 1-5 days)
  2. Proliferative (rebuilding phase, days 6-21, granulation- tissue fills the wound with vessels & connective tissue, epithelialization, contraction- wound edges pull together to reduce the size of the wound opening)
  3. Remodeling (final phase pf fun thickness repair 3 weeks up to 2 full years, replacing connective tissue- strength is acquired within 3 months- but tissue is never as strong as original tissue 80% optimal, fill thickness repair, keloid scar- tissue extending beyond boundaries of the original wound heal)
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9
Q

Factors affecting wound healing

A

tissue perfusion, nutritional status, diabetes mellitus, corticosteroids, age

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10
Q

Factors affecting wound healing (tissue perfusion & nutritional status & Nutritional Status & Corticosteroids)

A
  • Tissue perfusion: fibroblast proliferation, collagen synthesis, leukocyte activity, phagopcytosis, reepithelialization
  • Nutritional status: connective tissue needs- proteins, vitamins, minerals
  • Diabetes mellitus: leukocytosis, growth factor, collagen synthesis, tensile strength
  • Corticosteroids: inhibits repair, 20-40 mg/day
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11
Q

Factors affecting healing- older adult

A
  • Reduced macrophage functioning
  • Delayed inflammatory response
  • Delayed collagen synthesis
  • Slower epithelialization
  • Increased incidence of chronic illnesses that compromise circulation & tissue oxygenation
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12
Q

Wound healing complications

A
  • infection
  • hemorrhage
  • dehiscence
  • evisceration
  • fistula
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13
Q

S/S of infection

A

Drainage (purulent, increasing in quantity), pain, redness & swelling, elevated temperature, increased WBC

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14
Q

Hemorrhage

A
1st 48-hours (occurs most in this period); check wound frequently 
Excessive bleeding (pressure with sterile dressing, sterile packing, fluid replacement, surgical repair)
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15
Q

Dehiscence & Evisceration

A

Dehiscence (disrupted wound layers, increased serousanguinous drainage, “something given way”)
Evisceratin (protrusion of viscera)

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16
Q

Fistula development

A

Abnormal passage- internal organ to skin, one organ to another

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17
Q

Wound assessment

A

Location- where on the body
Dimensions & depth- length, width, depth, tunneling
Stage- what layers of tissue are involved
Status of wound bed- presence of eschar (dry brownish or black tissue), slough
Exudate- fluid how much, what does it look like,
Status of wound edges- open, proliferative, closed
Status of surrounding skin- redness, warmth, is it inflamed
Pain- intense pain is associated with wound care, treat aggressively

18
Q

Types of dressings

A

Xeroform; gauze; tegasorb; tegaderm; bioclusive transport dressing; montgomery straps; ABD binder

19
Q

Wound drainage

A
  • Serous; clear/ watery plasma
  • Purulent; thick, yellow, green, tan, brown
  • Serousanguinous; pale, red, watery, mixture of clear and red fluid
  • Sanguinous; bright red, indicates active bleeding
20
Q

Wound management

A

Abrasions- non adherent, absorptive dressing
Lacerations- clean (closed primarily), contaminated (open to heal)
Surgical incisions- closed by sutures or staples, heal quickly
Skin tears- viable flap (cleanse wound & roll flap back into place & secure with adhesive strips), no flap (cleanse & apply non adherent, adsorptive dressing)

21
Q

Wound care facts

A

Wound care should be performed:

  • From an area that is the least contaminated to the most contaminated area
  • With a new gauze pad for each wipe
  • This means that you should start from top to bottom, inner to outer, using a new pad for each wipe
22
Q

Wound irrigation

A

steady flow of a solution across an open wound surfaceto achieve wound hydration, to remove deeper debris, and to assist with the visual examination

23
Q

Wound culture

A

a test to find germs (such as bacteria or a fungus) that can cause an infection. A sample of skin, tissue, or fluid is added to a substance that promotes the growth of germs. If no germs grow, the culture is negative. If germs that can cause an infection grow, the culture is positive

24
Q

Pressure Ulcers stage 1

A
  • pressure-related skin alteration
  • changes may include: skin temperature, tissue consistency, sensation, color
  • intact skin
  • non-blanchable
  • affects epidermis
  • redness
  • warm skin
  • softer than normal
25
Q

Pressure Ulcers stage 2

A
  • partial thickness skin loss: epidermis, dermis
  • presents clinically as: abrasion, blister, shallow crater
  • open epidermis & may include the dermal layer
  • looks like blister
  • blanchabe
26
Q

Pressure Ulcers stage 3

A
  • full thickness skin loss: subcutaneous tissue, fascia
  • clinically presents as: deep crater, undermining
  • down to the fascia w/ tunneling
27
Q

Pressure Ulcers stage 4

A
  • full thickness skin loss: tissue necrosis, muscle, bone, supporting structures, sinus tracts
  • through to the bone, may be necrotic- big infection risk
  • looks like holes
28
Q

Pressure ulcers

A

Etiology- shearing & friction: layers slide against one another, pressure: reduces or eliminates blood flow (prolonged, high intensity)
Location- over bony protection
Results in subcutaneous compressions- blood vessel compression, ulcer development, tunneling extension up under top layer

29
Q

Pressure ulcer risk factors

A

Altered LOC, immobility, maceration, pain, malnourished, debilitation, impaired sensory perception, shear & friction

30
Q

Pressure ulcer risk assessment

A

Physical assessment
Wound & ostomy care nurse- guidelines
Standard-risk assessment tools- Braden scale for predicting pressure sore risk, Norton scale for prediction of pressure ulcer risk

31
Q

Barden Scale

A
19-23= no risk
15-18= low risk
13-14= mod. Risk
10-25= high risk 
< or equal to= very high risk
32
Q

Norton Scale

A

< 14 is at risk

  • Assesses client risk factors for pressure ulcer formation
  • Clients are scored 1-4 points for: physical condition, mental condition, activity, mobility, incontinence
  • Higher the score, the better the patient is and the lower their risk factor is for developing a pressure ulcer
33
Q

Pressure ulcer assessment

A
  • location
  • stage & tissue involvement
  • drainage
  • measurements: size, depth, undermining & tunnels
34
Q

Consequences of pressure ulcer

A

Pain (provide analgesics prior to dressing changes)
Anxiety (over change in body image)
Economics (increases cost of care & length of hospitalization)
Quality of care (used as a quality or lack of quality indictor for facilities)
Use of resources (increase in nursing, costs, & support service care)

35
Q

Prevent pressure ulcer

A

Identify high-risk clients, identify contributing factors, ongoing assessment, hygiene & skin care, repositioning with support, reduce shear & friction, control moisture, nutrition & fluid support

36
Q

Independent nursing interventions for pressure ulcer care

A

Skin care, teaching, pressure relief, reduction of shear & friction, don’t massage over bony prominences, nutrition & hydration

37
Q

Dependent & independent nursing interventions

A

Cleaning of wound bed, dressing type & frequency per order, pressure relief specialized mattresses, surgical procedures debridement/reconstruction/skin flaps, controlling infection with antibiotics, nutrition & fluid support enteral or parenteral

38
Q

Lower extremity ulcer

A

Arterial (caused by arterial insufficiency, results in ischemic tissue, heals by improving tissue perfusion, lifestyle changes, baby aspirin & avoiding the cold)

Venous (cause by venous insufficiency, one-way vale damage, dark red ulcer bed, edema, ulceration, exudate, DVT’s, treatment is compression wraps)

Neuropathic ulcer (found in pt w/ diabetes or neurological damage; treatment is keep blood glucose normal, foot care, & routine exams)

39
Q

Nursing process for wound management

A

Assessment, nursing diagnosis, goal & outcomes, establish plan, implement plan, evaluation

40
Q

Wound care plan

A

Nursing diagnosis- impaired skin integrity R/T immobility and poor nutrition AEB: 4cm reddened area on patient’s left hip
Goal (opposite of diagnosis)- skin integrity will improve by time of discharge from facility
Outcomes- patient will exhibit improved or healed left hip, patient will have minimal complications, patient will verbalized precipitating factors and appropriate skin care measures

41
Q

Nursing interventions & rationales

A

Assess: Inspect skin q-shift, document findings. Provides evidence of effectiveness of skin care regimen and signs of further complications.

Do: Provide wound care as prescribed and report adverse response. Maintain or modify treatment plan. Turn/reposition patient every two hours. Provides uniform pressure over bony prominences and promotes uniform blood flow to tissues.

Teach: Explain wound and skin care to client and caregiver. Promotes compliance with treatment regimen.

Evaluate (review the outcomes): the outcomes): what worked? Changes? Patient will exhibit improved or healed left hip wound. Patient will have minimal complications. Patient will verbalize precipitating factors as well as appropriate skin care measures.