Skin Disorders Flashcards

1
Q

skin

A

plays a major role in protection by acting like the first line of defense, as well as helps regulate body temperature and maintains fluid and electrolyte balance

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2
Q

hair

A

differs in type and function in various body areas

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3
Q

nails

A

are useful for grasping and scraping and have cosmetic value

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4
Q

changes in epidermis related to aging

A

Physical changes: decreased epidermal thickness  skin transparency and fragility
Nursing interventions: handle patients carefully to reduce skin friction and shear; assess for excessive dryness or moisture; avoid taping the skin
Physical changes: decreased cell division  delayed wound healing
Nursing interventions: avoid skin trauma, and protect open areas
Physical changes: decreased epidermal mitotic homeostasis  skin hyperplasia and skin cancers (especially in sun-exposed areas)
Nursing interventions: assess non–sun-exposed areas for baseline skin features; assess exposed skin areas for sun-induced changes

Physical changes: increased epidermal permeability  increased risk for irritation
Nursing interventions: teach patients how to avoid exposure to skin irritants
Physical changes: decreased immune system cells  decreased skin inflammatory response
Nursing interventions: do not rely on degree of redness and swelling to correlate with the severity of skin injury or localized infection
Physical changes: decreased melanocyte activity  increased risk for sunburn
Nursing interventions: teach patients to wear hats, sunscreen, and protective clothing; teach patients to avoid sun exposure from 10amto 4pm

Physical changes: hyperplasia of melanocyte activity (especially in sun-exposed areas)  changes in pigmentation (e.g., liver spots, age spots)
Nursing interventions: teach patients to keep track of pigmented lesions; teach them what changes should be evaluated for malignancy
Physical changes: decreased vitamin D production  increased risk for osteomalacia
Nursing interventions: urge patients to take a multiple vitamin or a calcium supplement with vitamin D
Physical changes: flattening of the dermal-epidermal junction  increased risk for shearing forces, resulting in blisters, purpura, skin tears, and pressure-related problems
Nursing interventions: avoid pulling or dragging patients; assist patients confined to bed or chairs to change positions at least every 2 hours; avoid or use care when removing adhesive wound dressings

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5
Q

changes in the dermis related to aging

A

Physical changes: decreased dermal blood flow  increased susceptibility to dry skin
Nursing interventions: teach patients to apply moisturizers when the skin is still moist and to avoid agents that promote skin dryness
Physical changes: decreased vasomotor responsiveness  increased risk for heat stroke and hypothermia
Nursing interventions: teach patients to dress for the environmental temperatures
Physical changes: decreased dermal thickness  paper-thin, transparent skin with an increased susceptibility to trauma
Nursing interventions: handle patients gently, and avoid the use of tape or tight dressings; use lift sheets when positioning patients

Physical changes: degeneration of elastic fibers  decreased tone and elasticity
Nursing interventions: check skin turgor on the forehead or chest
Physical changes: benign proliferation of capillaries  cherry hemangiomas
Nursing interventions: teach patients that these are benign
Physical changes: reduced number and function of nerve endings  reduced sensory perception
Nursing interventions: tell patients to use bath thermometer and to lower the water heater temperature to prevent scalds

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6
Q

changes in the subcutaneous layer related to aging

A

Physical changes: thinning subcutaneous layer  increased risk for hypothermia and increased risk for pressure injury
Nursing interventions: teach patients to dress warmly in cold weather; assist patients confined to bed or chairs to change positions at least every 2 hours

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7
Q

skin inspection

A

observe and document these features:
Obvious changes in color and vascularity
Presence or absence of moisture
Edema
Skin lesions
Skin integrity

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8
Q

Skin assessment techniques for patients with darker skin

A

Pallorcan be detected in people with dark skin by first inspecting the mucous membranes for an ash-gray color
Cyanosis can be detected in the lips and tongue appearing gray and the palms, soles, conjunctivae, and nail beds have a bluish tinge
Inflammationin dark-skinned patients appears as excessive warmth and changes in skin consistency or texture
Jaundiceis best assessed by inspecting the oral mucosa, especially the hard palate, for yellow discoloration
Ecchymoses appear darker than normal skin; they may be tender and easily palpable

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9
Q

Primary lesions

A

are an initial reaction to a problem that alters skin components

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10
Q

Secondary lesions

A

changes in the appearance of the primary lesion; these changes occur with progression of an underlying disease or in response to a topical or systemic therapeutic intervention

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11
Q

Annular

A

circular

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12
Q

Circumscribed

A

well-defined with sharp borders

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13
Q

Clustered

A

several lesions grouped together

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14
Q

Coalesced

A

lesions that merge with one another and appear confluent

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15
Q

Linear

A

occurring in a straight line

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16
Q

Serpiginous

A

with wavy borders, resembling a snake

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17
Q

Universal

A

all areas of the body involved, with no areas of normal-appearing skin

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18
Q

Skin palpation

A

use palpation to gather additional information about skin lesions, moisture, temperature, texture, and turgor
Palpation confirms lesion size and whether they are flat or slightly raised
Consistency of larger lesions can vary from soft and pliable to firm and solid
Subtle changes, such as the difference between a finemacular rash and apapular rash, are best determined by palpating with your eyes closed
Ask the patient whether skin palpation causes pain or tenderness

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19
Q

diagnostic labs/tests

A

cultures
Skin biopsy
Wood’s light examination
Diascopy

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20
Q

pressure ulcer

A

is a loss oftissue integritycaused when the skin and underlying soft tissue are compressed between a bony prominence and an external surface for an extended period
Tissue compression from pressure restricts blood flow to the skin, resulting in reduced tissue perfusion and oxygenation and, eventually, leading to cell death
Complications include sepsis, kidney failure, infectious arthritis, and osteomyelitis

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21
Q

assess for any contributing factors for pressure ulcers

A

Prolonged bedrest
Immobility
Incontinence
Diabetes mellitus
Inadequate nutrition or hydration
Decreased sensory perception or cognitive problems
Peripheral vascular disease
Friction and sheering forces
Inspect the whole body

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22
Q

Incontinence associated dermatitis (IAD)

A

Skin damage associated with exposure to urine or stool
It is a type of irritant contact dermatitis
Once IAD occurs, there is a high risk for pressure ulcer development as well as an increased risk of infection and morbidity

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23
Q

Stage I pressure ulcer

A

Skin is intact, red and does not blanch with external pressure
For patients with darker skin that does not blanch:
Observe for changes compared with an adjacent or opposite area for:
Skin color darker or lighter than the comparison area
Skin temperature (warmth or coolness)
Tissue consistency (firm or boggy)
Sensation (pain, itching)

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24
Q

Stage II pressure ulcer

A

There is partial-thickness skin loss of the epidermis or dermis and skin is not intact
Ulcer is superficial and may be characterized as an abrasion, a blister (open or fluid-filled), or a shallow crater
Bruising isnotpresent

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25
stage III pressure ulcer
Skin loss is full thickness and damage extends down to but not through the underlying fascia; bone, tendon, and muscle are not exposed Subcutaneous tissues may be damaged or necrotic Undermining and tunneling may or may not be present
26
Stage IV pressure ulcer
Skin loss is full thickness with exposed or palpable muscle, tendon, or bone Often includes undermining and tunneling Slough and eschar are often present on at least part of the wound
27
Deep tissue injury
The intact skin area appears purple or maroon Blood-filled blisters may be present The area may have felt more firm, boggy, mushy, warmer, or cooler than the surrounding tissue before the color change occurred
28
Unstageable pressure ulcer
Skin loss is full thickness, and the base is completely covered with slough or eschar, obscuring the true depth of the wound
29
assessment and document for pressure ulcers
Assess wounds for location, size, color, extent of tissue involvement, cell types in the wound base and margins, exudate, condition of surrounding tissue, tunneling, undermining and presence of foreign bodies Document this initial assessment to serve as a starting point for determining the intervention plan and its effectiveness Assess the wound at each dressing change, comparing the existing wound features with those documented previously to determine the current state of healing or deterioration
30
assess for signs of healing
Beneath the dead tissue, granulation tissue appears Early granulation is pale pink, progressing to a beefy red color as it grows and fills the wound Palpate the wound to determine the granulation texture Healthy granulation tissue is moist and has a slightly spongy texture
31
Nursing interventions for pressure-relieving and pressure reducing techniques
Assist with major position changes every 2 hours in bed, every 1 hour in a chair Pad contact surfaces with foam, silicon gel, air pads, or other pressure-relieving pads Do not keep the head of the bed elevated above 30 degrees to prevent shearing Use a lift sheet to move a patient in the bed. Avoid dragging or sliding him or her When positioning a patient on his or her side, do not position directly on the trochanter Place pillows or foam wedges between two bony surfaces Keep the patient's skin directly off plastic surfaces Help the patient maintain an adequate intake of protein and calories and a fluid intake of 2 – 3L/day Perform a daily inspection of the patient's entire skin and document all sores and signs of infection Use moisturizers daily on dry skin, and apply when skin is damp Keep moisture from prolonged contact with skin Keep areas dry where two skin surfaces touch, such as the axillae and under the breasts Place absorbent pads under areas where perspiration collects Use moisture barriers on skin areas where wound drainage or incontinence occurs Clean the skin as soon as possible after soiling occurs and at routine intervals Use a mild, heavily fatted soap or gentle commercial cleanser for incontinence with tepid rather than hot water While cleaning, use the minimum scrubbing force necessary to remove soil and gently pat dry
32
passive wound dressing
have only a protective function and maintain a moist environment for natural healing; they just cover that area and may remain in place for several days DuoDERM, Tegaderm
33
interactive wound dressing
capable of absorbing wound exudate while maintaining a moist environment in the area of the wound and allowing the surrounding skin to remain dry Hydrocolloids, alginates, hydrogels
34
active wound dressings
improve the healing process and decrease healing time Skin grafts, biologic skin substitutes
35
Surgical management of a pressure ulcer includes
removal of necrotic tissue and skin grafting or use of muscle flaps to close wounds that do not heal by re-epithelialization and contraction Those with poor blood flow are unlikely to have successful graft take and heal The procedures are very similar to the surgical management of burn wounds
36
Negative pressure wound therapy
can reduce or even close chronic ulcers by removing fluids or infectious materials from the wound and enhancing granulation This technique requires that a suction tube be covered by a special sponge and sealed in place Per manufacturer's instructions, the foam dressing is changed every 48 to 72 hours (or at least 3 times weekly). Continuous low-level negative pressure is applied through the suction tube Duration of the treatment is determined by the wound's response
37
Impetigo
Characterized by red macules that become thin-walled vesicles that rupture and become covered with honey-yellow crust May have formation of bullae (large fluid-filled blisters) from original vesicles, then the bullae rupture leaving red raw areas Exposed areas of the body, face, hands, neck and extremities are most frequently involved Contagious and may spread to over parts of the body or to other people
38
Folliculitis
Isolated erythematous pustules occur singly or in groups; hairs grow from centers of many of the lesions Occasional papules are present There is little or no associated discomfort There is no residual scarring Areas of hair-bearing skin, especially areas of shaving, thighs, buttocks, and axillae are affected
39
Furuncle (boil)
Small, tender, erythematous nodules become pus filled Lesions may be single or multiple and also recurrent May progress and involve the skin and subcutaneous fatty tissue, causing tenderness, pain, and cellulitis Occasional scarring results
40
Cellulitis
Localized area of inflammation may enlarge rapidly if not treated Redness, warmth, edema, tenderness, and pain are present On rare occasions, blisters are present Cellulitis is often accompanied by lymphadenopathy and fever Lower legs, areas of persistent lymphedema, and areas of skin trauma (e.g., leg ulcer, puncture wound) are affected
41
Herpes zoster (varicella zoster)
Lesions are similar in appearance to herpes simplex and also progress with weeping and crusting Grouped lesions present unilaterally along a segment of skin following the pathway of a spinal or cranial nerve (dermatomal distribution) Eruption is preceded by deep pain and itching Postherpetic neuralgia is common in older adults Areas of the anterior or posterior trunk following the involved dermatome; face, sometimes involving trigeminal nerve and eye are affected
42
Herpes simplex
Grouped vesicles are present on an erythematous base Vesicles evolve to pustules, which rupture, weep, and crust Older lesions may appear as punched-out, shallow erosions with well-defined borders Lesions are associated with itching, stinging, burning or pain Type 1 classically occurs on the face and type 2 on the genitalia, but either may develop in any area where inoculation has occurred
43
Candidiasis
The erythematous macular eruption occurs with isolated pustules or papules at the border (satellite lesions) Candidiasis is associated with burning and itching Oral lesions (thrush) appear as creamy white plaques on an inflamed mucous membrane Cracks or fissures at the corners of the mouth may be present Skinfold areas: perineal and perianal region, axillae, beneath breasts, and between the fingers; under wet or occlusive dressings can all be affected along with the oral or vaginal mucous membranes
44
Pediculosis (lice) S/S
Pediculosis capitis (head lice) Pediculosis corporis (body lice) Pediculosis pubis (pubic, or crab, lice) Pruritus and excoriation from scratching Matting and crusting of the scalp and a foul odor indicate a probable secondary infection
45
Scabies
A contagious skin infection caused by mite infestations, transmitted by close contact with an infested person or infested bedding
46
scabies S/S
Intense itching Red pruritic eruptions Small raised burrows created by the mites Usually affects between the fingers, wrists, elbows, knees, edges of feet, axillary folds, under breasts, near the groin, or penis / scrotal areas
47
Bedbugs
This parasite does not live on humans; however, it feeds on human blood The insect bites a human host at night and sucks blood for 3 to 10 minutes The bite area resembles a mosquito or flea bite with a raised bite mark surrounded by a wheat
48
bedbugs S/S
The bite causes an itchy discomfort Clustered bite marks
49
Psoriasis
is a chronic, autoimmune disorder that results from overstimulation of the immune system (Langerhans' cells) in the skin that target keratinocytes, causing increased cell division (because some degree of cellular regulation is lost) and plaque formation Chronic condition with exacerbations and remissions
50
psoriasis S/S
Reddened papules or plaques covered by silvery white scales Bilateral distribution Borders between the lesions and normal skin are sharply defined Lesions thicken during exacerbations and extend to new body areas
51
psoriasis nursing interventions
Nonpharmacological Urge patients and families to consider support groups Pharmacological Topical steroids Phototherapy / ultraviolet light Systemic biologic & immunomodulating agents
52
skin cancer
occurs as a result of failure of cellular regulation over cell division Current estimates are that one in five Americans will develop skin cancer in their lifetime It is estimated that approximately 9,500 people in the U.S. are diagnosed with skin cancer every day Basal cell and squamous cell carcinomas, the two most common forms of skin cancer, are highly curable if detected early and treated properly Five-year survival rates for regional and distant stage melanomas are 63 percent and 20 percent, respectively
53
Actinic keratosis (premalignant) distribution and course
Cheeks, temples, forehead, ears, neck, backs of hands, and forearms May disappear spontaneously or reappear after treatment; slow progression to squamous cell carcinoma is possible
54
Actinic keratosis (premalignant) S/S
Small (1-10 mm) macule or papule with dry, rough, adherent yellow or brown scale Base may be erythematous Associated with yellow, wrinkled, weather-beaten skin Thick, indurated keratoses more likely to be malignant
55
Basal cell carcinoma distribution and course
Sun-exposed areas, especially head, neck, and central portion of face Metastasis is rare; may cause local tissue destruction; 50% recurrence rate related to inadequate treatment
56
Basal cell carcinoma S/S
Pearly papule with a central crater and rolled, waxy borders Telangiectasias and pigment flecks visible on close inspection As it grows, it undergoes central ulceration and sometimes crusting
57
squamous cell carcinoma distribution and course
Sun-exposed areas, especially head, neck, ears, nose and lower lip; sites of chronic irritation or injury (e.g., scars, irradiated skin, burns, leg ulcers) Rapid invasion with metastasis via the lymphatics occurs in 10% of cases; larger tumors are more prone to metastasis
58
spuamous cell carcinoma S/S
Rough, thickened, scaly tumor (may involve bleeding) Indurated margins Fixation to underlying tissue with deep invasion
59
melanoma distribution and course
Can occur anywhere on the body, especially where nevi (moles) or birthmarks are evident; commonly found on upper back and lower legs; soles of feet and palms in dark-skinned people Radial growth phase followed by vertical growth phase; rapid invasion and metastasis with high morbidity and mortality
60
melanoma S/S
Irregularly shaped, pigmented papule or plaque Variegated colors, with red, white, and blue tones
61
Assess for ABCDE features that are associated with skin cancer:
A: asymmetry of shape B: border irregularity C: color variation within one lesion D: diameter greater than 6mm E: evolving or changing in any feature (shape, size, color, elevation, itching, bleeding, or crusting)
62
Prevention techniques for skin cancer
Avoid sun exposure between 11am and 3pm Use sunscreen with the appropriate skin protection factor for your skin type Wear a hat, opaque clothing, and sunglasses when you are out in the sun Keep a “body map” of your skin spots, scars, and lesions to detect when changes have occurred Examine your body monthly for possibly cancerous or precancerous lesions Seek medical advice if you note any of the ABCDE changes
63
skin cancer nursing interventions
Nonsurgical management Topical or systemic chemotherapy, biotherapy or targeted therapy Radiation therapy Surgical management Cryosurgery Curettage and electrodesiccation Excisional biopsy Mohs’ surgery Wide excision
64
The tissue destruction
caused by a burn injury leads to local and systemic problems that affect:  Fluid and electrolyte balance Protein losses Sepsis development Changes in metabolic, endocrine, respiratory, cardiac, hematologic, and immune functioning
65
Superficial-thickness wounds
Have the least damage because the epidermis is the only part of the skin that is injured
66
Superficial-thickness wounds S/S
Redness, pain, tingling and increased sensitivity to heat occurs as a result; desquamation (peeling of dead skin) occurs 2 to 3 days after the burn
67
superficial-thickness wounds heals...
rapidly in 3 to 6 days without a scar or other complication
68
Superficial partial-thickness wounds
Caused by injury to the upper third of the dermis, leaving a good blood supply The small vessels bringing blood to this area are injured, resulting in the leakage of large amounts of plasma, which in turn lifts the heat-destroyed epidermis, causing blister formation
69
Superficial partial-thickness wounds S/S
Pink to red, painful, blisters
70
Superficial partial-thickness wounds heals in...
10 to 21 days with no scar, but some minor pigment changes may occur
71
Deep partial-thickness wounds
Extend deeper into the skin dermis, and fewer healthy cells remain Blisters usually do not form because the dead tissue layer is thick, sticks to the underlying dermis, and does not readily lift off the surface
72
Deep partial-thickness wounds S/S
Red and dry with white areas in deeper parts (dry because fewer blood vessels are patent) with moderate edema
73
Deep partial-thickness wounds heals in...
2 to 6 weeks, but scar formation results
74
Full-thickness wounds
Destruction of the entire epidermis and dermis, leaving no skin cells to repopulate This wound does not regrow, and areas not closed by wound contraction require grafting
75
Full-thickness wounds S/S
Hard, dry, leathery eschar  forms from coagulated particles of destroyed skin; with severe edema May be waxy white, deep red, yellow, brown, or black
76
Full-thickness wounds healing time
can range from weeks to months depending on establishing a good blood supply
77
Deep full-thickness wounds
Extend beyond the skin and damages muscle, bone, and tendons All full-thickness burns need early excision and grafting
78
Deep full-thickness wounds S/S
Charred, blackened and depressed, with sensation completely absent
79
Deep full-thickness wounds healing?
Grafting may not be successful. Amputation may be needed when an extremity is involved
80
Vascular changes resulting from burn injuries
Circulation to the burned skin is disrupted immediately after injury by blood vessel occlusion Fluid shift occurs after initial vasoconstriction as a result of blood vessels near the burn dilating and leaking fluids into the interstitial space Leakage of fluid and electrolytes from the vascular space continues, causing extensive edema, even in areas that were not burned, leading to weight gain Hypovolemia, metabolic acidosis, hyperkalemia, and hyponatremia occur in the first 24-36 hours Fluid remobilization starts at about 24 hours after injury, when the capillary leak stops. The diuretic stage begins at about 48 to 72 hours after the burn injury
81
For patients in the emergent/resuscitative phase
nurses should do a primary survey and monitor circulation. As the taut, burned tissue becomes unyielding to the edema underneath its surface, it begins to act like a tourniquet, especially if the burn is circumferential. As edema increases, pressure on small blood vessels in the distal extremities causes an obstruction of blood flow and consequent tissue ischemia and compartment syndrome
82
Pulmonary changes resulting from burn injuries
Respiratory problems are caused by superheated air, steam, toxic fumes, or smoke Respiratory damage from an inhalation injury can occur in the upper and major airways and the lung tissue and can cause edema that leads to obstruction The lining of the trachea and bronchi may slough 48 to 72 hours after injury and obstruct the lower airways Leaking capillaries cause alveolar edema, which can occur immediately or up to a week after the injury
83
Cardiac changes resulting from burn injuries
Heart rate increases and cardiac output decreases because of the initial fluid shifts and hypovolemia that occur after a burn injury Workload of the heart and oxygen demands increase with decreased perfusion, oxygen delivery and BP  shock Cardiac output may remain low until 18 to 36 hours after the burn injury Cardiac output improves with fluid resuscitation and reaches normal levels before plasma volume is restored completely Proper fluid resuscitation and support with oxygen prevent further complications
84
Gastrointestinal and renal changes resulting from burn injuries
The sympathetic nervous system stress response increases secretion of epinephrine and norepinephrine, which inhibit GI motility and further reduce blood flow to the area Secretions and gases collect in the GI tract, causing abdominal distention Peristalsis decreases, and a paralytic ileus may develop The increased production (and loss) of heat breaks down protein and fat (catabolism), rapidly uses glucose and calories, and increases the metabolic rate and calorie needs If there is inadequate blood flow, acute kidney injury can occur
85
Age related changes increasing complications from burn injuries
Thinner skin, sensory impairment, decreased mobility Slower healing time More likely to have cardiac impairments Reduced inflammatory and immune responses Reduced thoracic and pulmonary compliance More likely to have pre-existing medical conditions such as diabetes mellitus, kidney impairment, or pulmonary impairment
86
Emergency management of burn
Assess the airway for patency Administer oxygen as needed Cover the patient with a blanket and cover the wound with a clean dry cloth Keep the patient on NPO status Elevate the extremities if no fractures are obvious Obtain vital signs Initiate an IV line and begin fluid replacement Administer tetanus toxoid for prophylaxis Perform a head-to-toe assessment
87
Respiratory assessment inspection for burns
Inspect the mouth, nose, and pharynx Burns of the lips, face, ears, neck, eyelids, eyebrows, and eyelashes are strong indicators that an inhalation injury may be present Burns inside the mouth and singed nasal hairs also indicate possible inhalation injury Black particles of carbon in the nose, mouth, and sputum; edema of the nasal septum; and a “smoky” smell to the patient's breath indicate smoke inhalation
88
respiratory assessment for burns signs and symptoms
Progressive hoarseness or brassy cough; drool or difficulty swallowing; audible wheezing, crowing or stridor, dyspnea in supine position
89
Cardiovascular assessment for burns
Changes in the cardiovascular system begin immediately after the burn injury and include shock as a result of disrupted fluid and electrolyte balance At first, cardiac manifestations are from hypovolemia and decreased cardiac output Monitor the degree of edema, and assess cardiac status by measuring central and peripheral pulses, blood pressure, capillary refill, and pulse oximetry Obtain baseline ECG tracings at the time of admission, and continue the ECG monitoring throughout the resuscitation phase
90
Gastrointestinal assessment for burns
Although the GI tract usually is not directly injured, changes in function occur in all burn patients The decreased blood flow and sympathetic stimulation reduce GI motility and promote development of a paralytic ileus Bowel sounds are usually reduced or absent in a patient with severe burns; nausea, vomiting, and abdominal distention may also be present if there is an ileus Patients with burns of 25% TBSA or who are intubated generally require a nasogastric (NG) tube inserted to prevent aspiration and remove gastric secretions
91
Genitourinary assessment for burns
Changes in kidney function with burn injury are related to decreased blood flow and cellular debris. During the fluid shift, blood flow to the kidney may not be adequate for filtration When muscle damage occurs from a major burn or electrical injury, myoglobin is released from damaged muscle and circulates to the kidney, along with other proteins from damaged blood cells; which can contribute and cause kidney failure Assess kidney function and monitor intake and urine output hourly
92
Nursing interventions related to respiratory issues
Nonsurgical management Airway maintenance Promotion of ventilation, gas exchange and oxygen therapy Drug therapy Positioning and deep breathing Surgical management Tracheostomy Escharotomy Chest tubes
93
Nursing interventions related to cardiovascular issues
Nonpharmacological Priority nursing interventions are carrying out fluid resuscitation and monitoring for indications of effectiveness or complications Monitor any invasive cardiac monitoring devices Pharmacological IV fluid resuscitation Drug therapy Surgical Escharotomy
94
Fluid resuscitation (The Parkland Formula)
Initiate and maintain at least one large-bore IV in an area of intact skin (if possible) Coordinate with physicians to determine the appropriate fluid type and total volume to be infused during the first 24 hours postburn Administer one half of the total 24-hour prescribed volume within the first 8 hours postburn and the remaining volume over the next 16 hours 2mL NS or LR x patients weight in kg x % TBSA Assess IV access site, infusion rate, and infused volume at least hourly Monitor vital signs at least hourly Assess for fluid overload Measure and assess strict intake and output at least hourly
95
Nursing interventions related to pain
Nonsurgical Drug therapy with analgesics and opioids Complementary therapy Environmental manipulation Surgical Early excision
96
The acute phase of burn injury begins
about 36 to 48 hours after injury, when the fluid shift resolves, and lasts until wound closure is complete
97
During this phase, the nurse coordinates interdisciplinary care that is directed toward continued assessment and maintenance of the:
Cardiovascular and respiratory systems GI and nutrition status Burn wound care and infection control Pain control Musculoskeletal contractions and promoting mobility Psychosocial interventions
98
acute phase of burn injuries nursing interventions
Provide a safe environment Use of asepsis, ensure daily cleaning of the room and unit Do not share equipment among patients; use disposable items as much as possible Detect problems early Continually assess and monitor these patients carefully Perform dressing changes as ordered Avoid musculoskeletal problems, such as contractions, with proper positioning Ensure adequate diet and fluids Tetanus prophylaxis given
99
Although rehabilitation efforts are started at the time of admission, the technical rehabilitative phase begins with wound closure and ends when the patient achieves his or her highest level of functioning The emphasis is on:
The psychosocial adjustment of the patient The prevention of scars and contractures The resumption of preburn activity, including resuming work, family, and social roles This phase may take years or even last a lifetime as patients adjust to permanent limitations