Sinus & Atrial Rhythms Flashcards

0
Q

A P-wave that is uniformly rounded would most likely be coming from the ________ node, but a P-wave that is notched, flattened, or diphasic, would be called would be called an _______ P-wave.

A

1) Sinus

2) Atrial

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1
Q

What are the 5 types of Atrial Arryhthmias discussed in Walraven?

A

1) Wandering Pacemaker
2) Premature Atrial Complex (an ectopic beat)
3) Atrial Tachycardia
4) Atrial Flutter
5) Atrial Fibrillation

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2
Q

What is the pathophysiology causing a wandering pacemaker?

A

A wandering pacemaker is caused when the pacemaker role switches from beat to beat from the SA node to the Atria and back again (sometimes it can wander to the AV junction as well).

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3
Q

What are the rules for a Wandering Pacemaker with regards to the following:

1) Regularity
2) Rate
3) P Wave
4) PRI
5) QRS

A

1) Regularity - Slightly irregular (as the the pacemaker roles wanders between sites)
2) Rate - Normal (60-100)
3) P Wave - Changes with each complex as the pacemaker role changes between sites.
4) PRI - Varies as the the pacemaker site changes but will still be less than 0.20 seconds (may even be less than 0.12 secs)
5) QRS - Normal (less than 0.12 secs)

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4
Q

What is the difference between an ectopic beat caused by irritability and an ectopic beat caused by an escape mechanism? How can you distinguish between the two?

A

1) Irritable Ectopic Beat - Caused when a a site somewhere along the conduction system becomes irritable and overrides the SA node for a single beat.
2) Escape Ectopic Beat - A beat initiated somewhere outside the SA node because the SA node failed to fire.

*You can tell the difference between the two because and irritable ectopic beat will come early in the cardiac cycle, while an escape beat will late in the cardiac cycle and preceded by a prolonged R-R interval.

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5
Q

What are the rules for PACs with regards to the following:

1) Regularity
2) Rate
3) P Wave
4) PRI
5) QRS

A

1) Regularity - Depends on the underlying rhythm, regularity will be interrupted by the PAC.
2) Rate - Depends on the underlying rhythm
3) P Wave - P Wave of early beat differs from the sinus P Waves. Can be flattened or notched or lost inside the preceding T Wave.
4) PRI - 0.12 to 0.20 secs but may exceed 0.20 secs
5) QRS - Normal (less than 0.12 secs)

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6
Q

Identify the pathophysiology behind Atrial Tachycardia (AT).

A

AT is caused by a single focus in the Atria that fires rapidly to override the SA node and thus assumes peacemaking responsibility for the entire rhythm.

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7
Q

What are the rules for Atrial Tachycardia with regards to the following:

1) Regularity
2) Rate
3) P Wave
4) PRI
5) QRS

A

1) Regularity - Regular
2) Rate - 150 to 250
3) P Wave - Peaked, flattened, notched or diphasic, and can be lost in T Wave because the rate is so fast.
4) PRI - Normal (0.12 to 0.20 secs)
5) QRS - Normal (less than 0.12 secs)

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8
Q

Identify the pathophysiology behind Atrial Flutter?

A

Atrial Flutter happens when an area in the atrium initiates an impulse that is conducted in a repetitive, cyclic pattern, creating a series of flutter waves (with sawtooth appearance) at a rate between 150 and 250 bpm.

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9
Q

Explain why there is usually more than one P Wave (Sawtooth) between each QRS complex on and Atrial Flutter strip.

A

To protect the ventricles from receiving too many impulses, the AV node blocks some of the impulses from being conducted through to the ventricles. Rose that do get through are conducted normally.

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10
Q

What are the rules for Atrial Flutter with regards to the following:

1) Regularity
2) Rate
3) P Wave
4) PRI
5) QRS

A

What are the rules for Atrial Flutter with regards to the following:

1) Regularity - Atrial rhythm is regular; ventricular rhythm is usually regular but can be irregular if there is a variable block.
2) Rate - Atrial rate 250 to 350 bpm; ventricular rate varies
3) P Wave - Characteristic sawtooth pattern
4) PRI - Unable to determine
5) QRS - Normal (less than 0.12 secs)

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11
Q

What are the two most characteristic features of Atrial Fibrillation?

A

1) No discernible P Waves

2) Grossly irregular rhythm

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12
Q

What are the rules for Atrial Fibrillation with regards to the following:

1) Regularity
2) Rate
3) P Wave
4) PRI
5) QRS

A

1) Regularity - Grossly irregular
2) Rate - Atrial rate greater than 350 bpm; ventricular rate varies greatly.
3) P Wave - not discernible; atrial activity is referred to as fibrillatory waves (f waves).
4) PRI - Unable to measure
5) QRS - Normal (less than 0.12 secs)

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13
Q

Is the ventricular rhythm regular or irregular in Atrial Flutter?

A

This depends on how the AV node is blocking the impulses. If the atrial impulses are being conducted in a regular pattern (i.e., 2:1 or 4:1), the ventricular rhythm would be regular. But if the conduction ration varied (i.e., 2:1 then 3:1then 4:1), the ventricular rhythm would be irregular.

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14
Q

Explain the following terms:

1) Atrial Fibrillation with Controlled Ventricular Response
2) Rapid Ventricular Response (Atrial Fibrillation Uncontrolled)

A

1) Atrial Fibrillation with Controlled Ventricular Response - Afib with a ventricular rate of 100 bpm or less.
2) Rapid Ventricular Response (Atrial Fibrillation Uncontrolled) - Afib with a ventricular rate greater than 100.

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15
Q

What are some of the common causes of PACs?

A

1) Caffeine
2) Alcohol
3) Nicotine
4) Stretched Atrial Myocardium (i.e., Hypervolemia )
5) Hypokalemia
6) Hypoxemia
7) Early indicator of CHF and PEs

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16
Q

What are some of the common causes of Atrial Flutter?

A

1) CAD
2) Hypertension
3) Mitral Valve Disease
4) Hyperthyroidism
5) Chronic Lung Disease
6) Cor Pulmonale (right ventricular failure)
7) Cardiomyopathy

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17
Q

What is the medical management for a patient with Atrial Flutter?

A

If the PT is unstable, urgent electrical Cardioversion is indicated. If the patient is stable, the QRS is narrow, and RR is regular, Adenosine (6mg) may be rapidly administered via IV to slow the conduction through the AV node, if ineffective, 12mg may be administered via rapid IV bolus.
If Adenosine fails to convert the rhythm, cardioversion is indicated.

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18
Q

What is the medical management for a patient with Atrial Fibrillation?

A

Electrical cardioversion is indicated for an unstable patient unless the patient has been in A-fib > 48hrs, this is because they are at greater risk for embolization.
IV Adenosine is indicated for the stable patient in A-fib < 48hrs.

19
Q

Describe Wolf-Parkinson’s Syndrome.

A

WPW Syndrome is caused by the presence of an abnormal accessory electrical conduction pathway between the atria and the ventricles. Impulses traveling down this abnormal pathway may stimulate the ventricles to contract prematurely, resulting in a SVT. WPW Syndrome has a characteristic delta wave that makes the QRS wider than usual.

20
Q

Why should medications that block AV conduction be avoided for patients in A-fib who also have a wide QRS and irregular ventricular rhythm?

A

Because an accessory pathway might be present (as in WPW Syndrome) and these meds will actually increase the ventricular rate thought that accessory pathway.

21
Q

What are some examples of meds that slow the conduction of impulses through the AV node and are indicated for A-flutter and A-fib?

A

1) Adenosine
2) Diltiazem
3) Verapamil
4) Digoxin

22
Q

Warfarin is medicated for patients who have A-fib along with which other comorbidities?

A

1) > 75 yrs old
2) Hypertension
3) Heart Failure
4) Hx of stroke

23
Q

What Tx option is available to A-fib patients who are unresponsive to meds?

A

Ablation therapy or pacemaker implantation

24
Q

Describe the difference between Coarse A-fib and Fine A-fib.

A

Course A-fib has a bumpy baseline, while fine A-fib has a fine, almost smooth, discernible baseline.

25
Q

Which electrolyte is responsible for depolarization? Repolarization?

A

1) Depolarization - Sodium

2) Repolarization - Potassium

26
Q

What is the clinical significance of Hypokalemia in heart patients?

A

Hypokalemia can lead to irritability and excitation (the opposite of polarized/resting)

27
Q

What is the clinical significance of Calcium and Magnesium on heart patients?.

A

1) Calcium - Responsible for cardiac contractility, vascular tone and blood clotting.
2) Magnesium - Activates enzymes involved in the breakdown of ATP for energy, which is responsible for neuromuscular function and myocardial contraction/irritability.

28
Q

What is the significance of Phosphorous on heart patients?

A

Phosphorous is essential for ADP/ATP, which are esponsible for energy inside the cell.
Hypophosphatemia can cause muscular weakness, including respiratory and cardiac arrest.

29
Q

What does the QT interval represent and what’s the normal range?

A

The QT interval represents the total time for ventricular depolarization and repolarization, and is measured form the beginning of the QRS complex to the end of the T-wave.
The normal range is 0.36 to 0.44 seconds.

30
Q

What does the TP segment represent, how is it measured and which 2 significant events happen during this segment?

A

The TP segment represents diastole. It is measured from the end of the T-wave to the beginning of the P-wave. Two significant events that happen during diastole are:

1) Ventricular Diastolic Filling
2) Coronary Artery Filling

31
Q

What are the mentioned med classes that can prolong the QT interval?

A

1) Asthma inhalers - Albutrol and Salmeterol
2) Antihistamines - Benadryl
3) Antiarrhytmics - Quinidine
4) Antipsychotics - Amitryptyline and Perphanazine

32
Q

Name 6 common causes of Sinus Bradycardia.

A

1) Damage to SA Node (i.e., inferior wall MI)
2) Parasympathetic stimulation (i.e., Vagal stimulation)
3) Hypoxemia (i.e., during suction)
4) Athletic hearts
5) Pacemaker failure
6) Meds (i.e., Beta-blockers)

33
Q

Explain the difference between PVCs and Bradycardia with ventricular escape beats.

A

1) Premature Ventricular Contractions - When the ventricles contract first, before the atria have optimally filled up. The heart beat is initiated by the Purkinje Fibers.
2) Bradycardia with Ventricular Escape Beats - Happens when the ventricles initiate a heart beat because the SA node’s rate is too slow.

34
Q

Which class of medication is indicated for patients experiencing Bradycardia with Ventricular Escape Beats?

A

Atropine - Stimulates the SNS

35
Q

(T/F) During sinus arrhythmias, the heart rate increases with inspiration and decreases with expiration.

A

True

36
Q

Define Paroxysmal Atrial Tachycardia.

A

PAT is an acute onset of Atrial Tachycardia

37
Q

What are the 3 mentioned clinical significances of PAT?

A

1) Coronary arteries are unable to fill due to short diastolic time
2) Shortened ejection time with decreased cardiac output may progress to heart failure, shock or death.
3) Increased HR causes increased MVO2, especially with MI

38
Q

What is MVO2 and how is it calculated?

A

MVO2 - Myocardial Volume Oxygen Consumption

MVO2 = SBP x HR

39
Q

Describe Ablative Therapy used to treat Atrial Tachycardia.

A

A special catheter is placed against the area of the heart responsible for the problem. Radio-frequency energy is then passed to the tip of the catheter, so that it heats up and destroys the target area. Considered a non-surgical procedure.

40
Q

What is the standard Tx for the following types of patients experiencing Atrial Tachycardia:

1) Stable PT
2) Symptomatic PT
3) Life-threatening/Unstable PT

A

1) Stable PT - Vagotonic maneuvers (Valsalva maneuver, carotid sinus massage, dig, pacemaker override.
2) Symptomatic PT - Adenosine (may cause brief arrest), Verapamil (titrate and give slowly).
3) Life-threatening/Unstable PT - Synchronized cardioversion.

41
Q

What’s the difference between controlled and uncontrolled Atrial Fibrillation?.

A

1) Controlled - Atrial Fibrillation with a ventricular rate of 100 or less.
2) Uncontrolled - Atrial Fibrillation with a ventricular rate greater than 100.

42
Q

How does a patient with Atrial Fibrillation usually present?

A

Weak, SOB, and syncope is usually common. All are as a result of lack of cardiac output.

43
Q

What are the 5 mentioned treatments for acute atrial fibrillation?

A

1) Diltiazem (CCB) - IV bolus then drip
2) Procainamide or Ibutilide drip (Antiarrhytmics)
3) Cardioversion
4) Ablative Therapy
5) Anticoagulants - To prevent clots and resulting CVA

44
Q

What are the 4 mentioned Txs for Chronic Atrial Fibrillation?

A

1) Diltiazem (CCB) - oral
2) Procainamide or Amiodorone (Antiarrhytmics) - Oral
3) Digoxin
4) Anticoagulants