Shoulder Flashcards
AC joint injury classification scheme
Type I injuries involve purely an AC joint sprain with intact CC ligaments. Type II injuries involve complete tearing of the AC joint and a sprain of the CC ligament with the CC measurement being < 25% greater than of the contralateral shoulder. Type III, IV, V, and VI injuries all involve complete tearing of both the AC and CC ligaments. Type III injuries have a CC distance measurement between 25 and 100% greater than that of the contralateral side (this patient) compared to Type V injuries which have a CC distance > 100% of the contralateral side. Type IV and VI injuries represent posterior and inferior dislocation of the clavicle, respectively. The coracoacromial ligament is generally spared in these injuries
AC joint modified weaver-dunn reconstruction complications
distal clavicle resection, coracoacromial ligament transfer, and augmentation (modified Weaver-Dunn
does not restore native stability to AC joint. **Persistent horizontal (A to P) instability may cause persistent symptoms. **Anatomic repair and recon that preserve distal clavicle offer less risk of horizontal instability
innervation to the AC joint
Supara scapular nerve posteriorly
Lateral pectoral nerve anteriorly
Tx for AC joint injuries
Type: I-II nonop, good functional outcomes. full rehab 6-12 weeks
Type III: controversal, good results and return to sport for athletes
Type IV,V, VI surgery
The importance of the ACLC to joint stability: The ACLC is critical for ? stability. and is compromised with ?
The importance of the ACLC to joint stability has been highlighted in multiple studies. The ACLC is critical for anteroposterior stability and is compromised with distal clavicle excision
Which examination test is most specific for pain related to AC joint osteoarthritis?
Cross body adduction test: pain w/ shoulder in 90 FF and max adduction across body
cross-body adduction test is the most sensitive provocative test for AC joint osteoarthritis at 77%, the O’Brien active compression test has been shown to be most specific at 95%. CSI injection, only 44% accurately enter the joint.
Most common complication following distal clavicle excision? Appropriate amount of distal clavicle excision?
Persistent pain is the most common complication following distal clavicle excision. Although the exact amount of distal clavicle that should be resected is a topic of debate, resection of 10 mm or more of the distal clavicle may lead to instability of the AC joint, especially if the AC capsule is sectioned.
Which of the following is considered an advantage of arthroscopic distal clavicle excision compared with open distal clavicle excision?
Examination of the GH jt allows examination of SLAP tear which could mimic AC jt pain. Arthroscopic DCE sacrifices the inferior AC ligament and preserves the superior AC ligament.
What ligaments provid the most restraint to the AC joint?
Posterior and superior acromioclavicular ligaments provide the most restraint to posterior translation of the acromioclavicular joint and must be preserved during a Mumford procedure. (Distal clavicle excision)
The deltotrapezial fascia becomes confluent with which portion of the AC joint capsule, providing additional stability?
The musculotendinous aponeurosis of the deltotrapezial fascia interdigitates with the superior acromioclavicular joint capsule. The deltotrapezial fascia is believed to have synergistic stabilizing features with the capsule.
Zanca views to evaluate AC joint injuries
Bilateral Zanca views are obtained with 10-15° of cephalad tilt in the AP orientation with 50% penetrance
Risk factors that predispose an individual to rotator cuff tear and affect healing include
age, smoking, sex (female), family history, diabetes mellitus, and high cholesterol
The risk of partial-thickness tears enlarging at 2 and 5 years is ??, respectively, whereas full-thickness tears have a ?? risk of enlarging of at 2 and 5 years, respectively. Fatty infiltration and degeneration?
The risk of partial-thickness tears enlarging at 2 and 5 years is 11% and 35%, respectively, whereas full-thickness tears have a 22% and 50% risk of enlarging of at 2 and 5 years, respectively.
Muscle degeneration is associated with full-thickness tears rather than partial-thickness tears. Fatty degen more common in older pts and larger baseline tears. RCT w/ fatty degen are more prone to enlarge over time.
?% of rotator cuff tendon repairs do not heal; healing is affected by patient age, tendon retraction, and fatty muscle infiltration, with most failure occurring during ?
25% of rotator cuff tendon repairs do not heal; healing is affected by patient age, tendon retraction, and fatty muscle infiltration, with most failure occurring during active rehabilitation (2 to 3 months postoperatively)
clinical findings is commonly associated with symptomatic partial-thickness rotator cuff tears?
a painful arc with active range of motion is common
impingement signs are usually positive,
lift-off test is normal.
Active and passive range of motion measurements are often equal, although active range of motion can be painful.
External rotation lag signs are often seen with larger full-thickness tears.
The usual presentation of traumatic subscapularis tears is most often seen after forced
forced external rotation: The typical mechanism of injury is a fall and the patient grasps something to prevent the fall. This maneuver forces the arm into external rotation against resistance.
What is the most important stabilizing mechanism in the midrange of motion of the glenohumeral joint?
Concavity compression is a stabilizing mechanism by which muscular compression of the humeral head into the glenoid fossa stabilizes the glenohumeral joint against shear forces. This is dependent on the depth of the concavity and the magnitude of the compressive force.
What is the most consistent finding regarding glenohumeral kinematics in patients with symptomatic tears of the rotator cuff?
Superior translation of the humeral head with more than 30 degrees of abduction
Normal glenohumeral kinematics are represented by ball-and-socket modeling when the rotator cuff is intact. This is true for motion that involves more than 30 degrees of abduction. In patients with shoulder pain and symptomatic rotator cuff tears, superior translation occurs with abduction beyond 30 degrees. This is quite evident in massive tears but is seen consistently to a lesser degree with smaller tears.
Patients over 40 with initial anterior dislocation
Patients older than age 40 years at the time of initial anterior dislocation have low rates of redislocation; however, 15% of these patients experience a rotator cuff tear. Moreover, there is a dramatic increase (up to 40%) in the incidence of rotator cuff tears in patients older than age 60 years. Axillary nerve injury may occur but is less common than rotator cuff tear.
A rotator cuff tear is the most common cause of recurrent instability following a first-time dislocation in patients older than age 40 years. Dislocations occur through a posterior mechanism rather than by an isolated labral avulsion or a Bankart lesion as seen in younger patients.
Most common location for cuff tears
The most common location of rotator cuff tears is the junction of the supraspinatus and infraspinatus tendons, approximately 13 to 17 mm posterior to the biceps within the rotator crescent. This area is known to be a critical zone of hypovascularity, predisposing it to degenerative tears. The vascularity of this area also decreases with age, suggesting that the increasing incidence of rotator cuff tears with age is not related to chronic abrasive wear but to decreasing vascularity and degenerative changes.
Impingement syndrome: tx outcomes surgery vs PT
Nonsurgical management also has been shown to be as effective and less expensive than surgical subacromial decompression with acromioplasty. If bony impingement caused by acromial spurring was the cause, physical therapy alone, with no modification of the bony architecture, would not be expected to lead to outcomes similar to those of acromioplasty
impingement syndrome caused by an acromial spur, as proposed by Neer, is not the primary process that explains the origin of most degenerative rotator cuff tears.
Inflammatory markers elevated in subacromial bursitis
Several inflammatory markers have been shown to be elevated in subacromial bursitis. These include metalloproteases, tumor necrosis factors, and cyclooxygenase 1 and 2.
Calcific tendonitis:
what are the 3 stages? What is deposited in the tendon? Symptomatic? How does it appear on imaging? Tx?
- precalcific, calcific (3), and postcalcific
- deposition of calcium phosphate crystals (hydroxyapatite) within the rotator cuff tendon.
- 35% do not have symptoms, so evaluate pts for other symptoms
- 1.5-2 cm from insertion, On MRI, calcium deposits appear hypointense on all sequences.
On ultrasonography, calcium deposits appear hyperechoic.
Tx: Nsaids, PT, CSI. 27 percent failure rate at 6 months associated w/ b disease, larger deposits adn extension of calcification medial to acromial. .
USguided neddling to aspirate and then inject saline or steorid into lesion (UGNL) relief at 1 year, 5 year outcomes showed equal in comparison to CSI in SA alone. Arthroscopic debridment for recalcitrant disease
The precalcific stage generally is pain free; fibrocartilaginous metaplasia of tenocytes into chondrocytes occurs predominantly in less vascular areas of the tendon.
Calcific stage characterized by foci of calcium deposition; often separated by septae.
The resorptive phase typically is the most painful, directed by inflammatory response involving macrophages and multinucleated giant cells.
Pt has right shoulder pain over GT and Neer Hawkins impingement, dx and initial mgmt
Subacromial impingement: Initial management should consist of stretching exercises directed at the posterior capsule and a program of rotator cuff and deltoid strengthening exercises performed below the horizontal in a ‘safe’ plane. The judicious use of subacromial cortisone injections (one or two) may be helpful. Anterior acromioplasty is reserved for patients who have failed to respond to nonsurgical management.