Brainscape's Knowledge GenomeTM

Browse over 1 million classes created by top students, professors, publishers, and experts.


See full index

Y4 Cardiology > Shock > Flashcards

Shock Flashcards

1
Q

Define shock and how quickly can it kill someone ?

A
  • Shock is inadequate organ perfusion leading to inadequate oxygen delivery to tissues and eventually organ failure due to circulatory failure i.e. problem with the heart, blood vessels or flow of blood
  • Can lead to death in seconds to hours

SBP <90

MAP <50-60

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

What is MAP and how can it be calculated ?

A

Mean arterial pressure - is defined as the average pressure in a patient’s arteries during one cardiac cycle

MAP = CO x TPR

MAP = (2 x DBP) + SBP all divided by 3

can be rewrote as 2DBP/3 + SBP/3

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

What is someones MAP a good measure of and what level or MAP would indicate shock?

A

MAP is a good measure of tissue organ perfusion i.e. the oxygen reaching the organs

For normal tissue perfusion you need a MAP of 50-60mmHg (note if someone has atherosclerosis then this would need to be higher to adequately perfuse there tissues)

So MAP <50-60 would indicate shock

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

What does the inadequate tissue organ perfusion in shock lead to ?

A
  1. Metabolic Acidosis (pH < 7.35) due to ATP hydrolysis and lactic acid formation (due to anaerobic metabolism) further worsening global enzyme function and cellular performance
  2. Thrombus formation can occur at a capillary level resulting in patchy tissue injury, or in large vessels, leading to infarction.
  3. Eventual cellular necrosis results in mortality

In survivors, a degree of tissue injury may be irreversible, contributing to chronic morbidity

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

What are the 3 main immediate signs of tissue hypoperfusion (shock)?

A
  1. Skin: mottling
  2. Brain: confusion, agitation, reduced GCS (<15)
  3. Kidneys: oliguria (<0.5ml/kg/h)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

What 2 things can shock essentially result from ?

A

Think back to the MAP = CO x TPR (SVR)

CO = HR x SV

So shock can essentially result from inadequate CO or a loss of systemic vascular resistance (TPR), or both

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

After seeing the immediate signs of tissue hypoperfusion (shock) what test is then diagnostic that someone is in shock?

A

Measure someones arterial blood gases:

  • see a metabolic acidosis
  • lactate levels >2mmol/L
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

What are the 2 main types of shock which primarily result in inadequate cardiac output?

A
  1. Hypovolaemic shock
  2. Cardiogenic shock (has a subtype known as obstructive shock)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

What are the main types of shock which result in peripheral circulatory failure (decreased SVR)

A
  1. Septic shock
  2. Anaphylactic shock
  3. Neurogenic shock

These are all grouped as disruptive shock

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Define stroke volume

A

The amount of blood pumped by the left ventricle of the heart in one contraction.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Describe the mechanism behind cardiogenic shock

A
  • There is sustained decreased cardiac contractility
  • This results in reduced force of cardiac contraction and ==> decreased stroke volume
  • This reduced SV results in decreased CO and ==> decreased MAP (below 50-60 resulting in inadequate tissue organ perfusion and ==> shock)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

How do the body try to compensate for the decreased CO in cardiogenic shock and what sign does this result in ?

A

MAP = CO x TPR

The body tries to compensate by increasing TPR this results in COOL, CLAMY peripheries

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

List the main causes of cardiogenic shock

A

Pump failure:

  • ACS - e.g. MI (most common)
  • Arrhythmias
  • Aortic dissection
  • Acute valve failure e.g. mitral regurg
  • Cardiomyopathy
  • Myocarditis/pericarditis

Obstruction shock (subtype of cardiogenic):

  • PE
  • Tension penumothorax
  • Cardiac tamponade
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Along with the 3 general signs of shock what would make you think cardiogenic shock ?

A
  • Cool, clamy peripheries
  • Tachycardia
  • Normal temp
  • Hypoxia
  • Hypotension
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

What is the treatment of cardiogenic shock ?

A

Careful fluid management

Ionotropes e.g. dobutamine or vassopressors e.g. noradrenaline for increasing BP

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

What key sign would make you think of obstructive shock (subtype of cardiogenic)?

A

Evidence of raised Jugular Venous Pressure (JVP) and Distended Neck Veins may

Note the rest of its symptoms are the same as cardiogenic shock

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

Define what cardiac tamponade is

A

This is the accumulation of pericardial fluid under pressure.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

What is the classical traid of cardiac tamponade ?

A

Classical features - Beck’s triad:

  1. Hypotension
  2. Raised JVP
  3. Muffled heart sounds
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

What are the additional features of cardiac tamponade other than becks triad ?

A
  • Dyspnoea
  • Tachycardia
  • An absent Y descent on the JVP - this is due to the limited right ventricular filling
  • Pulsus paradoxus - an abnormally large drop in BP during inspiration
  • Kussmaul’s sign (raised JVP on inspiration)
  • ECG: electrical alternans
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

What is the management of cardiac tamponade ?

A

Urgent pericardiocentesis

21
Q

What is the treatment of obstructive shock ?

A
22
Q

Describe frank-starling mechanism

A

The law states that the stroke volume of the heart increases in response to an increase in the volume of blood in the ventricles, before contraction (the end diastolic volume)

23
Q

Describe the mechanism of hypovolaemic shock

A
  1. Reduced blood volume
  2. Lower venous return to the heart (ventricular filling = end-diastolic volume)
  3. Reduced force of cardiac contraction and Cardiac Output (Frank-Starling law)
  4. Therefore reduced BP (MAP) due to reduced CO which results in inadequate tissue perfusion (shock)
24
Q

What are the causes of hypovolaemic shock ?

A
  • Bleeding - trauma, ruptured aortic aneurysm, GI bleed, ruptured ectopic, femoral pseudoaneurysm rupture
  • Fluid loss - D&V, burns, heat exhaustion, pancreatitis
25
Q

What are the main signs/symptoms of hypovolaemic shock along with the 3 initial signs of shock ?

A
  • Tachyponea
  • Tachycardia
  • BP decreased but may be compensated for by SVR in smaller haemorrhage
  • Cool clamy peripheries - due to increased SVR
26
Q

What is the treatment of hypovolaemic shock?

A
27
Q

What are the 4 classifications of hypovolaemic haemorrhagic shock?

A

Note - BP normal until > 30% of blood lost

28
Q

What types of shock are strongly suggested by a patient presenting with the immediate signs of shock plus cold and clamy peripheries ?

A

Cardiogenic (including obstructive) and hypovolaemic shock is suggested

29
Q

Describe the mechanism behind distributive shock

A
  1. Reduced Systemic Vascular Resistance due to Vasodilatation with WARM, RED PERIPHERIES
  2. Reduced Mean Arterial Pressure (MAP = CO x TPR (SVR))
  3. Compensatory increase in Cardiac Output

There is pooling of blood in the tissues due to the vasodilation ==> warm, red peripheries

30
Q

What is the key feature which suggests septic shock ?

A

Warm and well perfused, with bounding pulse (due to increased CO)

Note the other causes of distributive shock also cause this its just sepsis is so much more common

31
Q

What are the main subtypes of distributive shock and which is the most common ?

A
  • Septic - the most common
  • Anaphylactic
  • Neurogenic

Note - burns can also cause distributive shock

32
Q

What are the causes of neurogenic shock ?

A
  • Spinal cord injury
  • Epidural or spinal anaesthesia
33
Q

What are the causes of septic shock

A

Infection

34
Q

what are the causes of anaphlatic shock

A
  • Drugs e.g. penicillin and contrast media in radiology
  • Latex
  • Stings e.g. bees, eggs, fish, peanuts, strawberries

It is an IgE mediated hypersensitivity reaction caused by mast cell (essentially a basophil) degranulation

35
Q

What signs and symptoms along with the 3 immediate shock signs and patient having an acidosis which suggest septic shock?

A
  • Warm and vasodilated
  • Bounding pulse
  • Increased temp >38
  • Cardiac output initially increased
  • tachycardia and tachyponea
  • hypotension
36
Q

What is important to remember about the signs and symptoms of anaphylactic & septic shock?

A

The initial CVS problem is vasodilatation i.e. loss of TPR, but hypovolaemia & cardiac problems will develop rapidly

37
Q

What features would make you think anaphylactic shock ?

A
  • Normal temp
  • Tachycardia and tachyponea
  • Hypotension
  • Urticarial rash
  • Itching, oedema, erythema
  • Diarrhoea and vomiting
  • Sweating
  • Wheeze, laryngeal obstruction, cyanosis
38
Q

What is the 1st line treatment in anphylactic shock ?

A

Give adrenaline

39
Q

What is the management of sepsis ?

A

BUFALO

  • B take bloods,
  • Measure urine output
  • Give fluids
  • Give antibiotics
  • Measure lactate
  • Give oxygen
40
Q

What is the treatment of distributive shock ?

A
41
Q

Describe the initial approach to the management of shock

A

ABCDE

42
Q

In the treatment of shock when someone is hypotensive what is the rate at which they would be fluid challenged ?

A

Fluid challenge ¼ or ½ L in less than half an hour

43
Q
A
  • Dx = anaphylactic shock
  • Tx = as per anaphylaxis guidelines
44
Q

Case:

  • 82 year old man driven in by wife with central crushing chest pain for last 6 hours
  • Clammy, sweaty, cool peripherally, grey in colour, agitated
  • BP 79/45, HR 72, RR 28, SPO2 poor trace
  • Audible wheeze and crackles in chest bilaterally

What is the Dx, initial Mx and Tx?

A
  • STEMI – ST elevation evident in I, aVL and V2-V5, reciprocal changes in II and III
  • ABCDE approach
  • Main treatment for this patient is PPCI, may go on to need IABP – they need early discussion and involvement with cardiology team
  • Cardiogenic shock
45
Q

Case:

  • 23yo stab wound to right side of chest, jumped over fence while running away
  • HR 138, BP 82/55, RR 28, SpO2 94% on 15L O2
  • Swollen and deformed left thigh, pain left groin
  • No significant PMH

Interpret the images, what is the Dx, Mx and Tx ?

A
  1. CXR = right sided hemothorax
  2. Pelvic X-ray = fractures pelvis, Mx = pelvic binder
  3. X-ray = mid-shaft femur fracture, Mx = kendrick splint
  • ABCDE approach – c with haemorrhage control
  • Senior doctor involvement and major trauma team activation
  • Early identification of bleeding site – now with CT
  • Role of interventional radiology
  • Massive haemorrhage protocol
46
Q

Case:

  • Collapsed pale and sweaty 24 yo female, BP 67/35, peripherally shut down, taking no medication.

What should you beware of? obv there is many potential other causes tho

A

Beware of a ruptured ectopic pregnancy causing lots of bleeding

47
Q

Case:

  • 73 yo male sudden onset severe back pain while waving the grandkids off to nursery then collapses on way into house.

What should you beware of ? obv serveral other potential causes

A

Beware of ruptured AAA

48
Q

Case:

  • 39yo female has had 24 hour history of sharp left sided chest pain worse on inspiration
  • This morning collapsed after getting out of bed
  • Just arrived back from Australia, thought her leg was maybe swollen yesterday
  • Reg meds: COCP
  • HR 140, BP 70/36, RR 32, SpO2 88% 15 L

What is the Dx, Ix and Mx ?

A

ECG = Sinus tachycardia, atrial flutter or fibrillation, S1Q3T3 pattern, RBBB incomplete or complete, T wave inversion in the right precordial leads, P pulmonale, Right axis deviation

  • Dx = obstructive shock caused by PE
  • Ix = PE Ix’s
  • Tx = Pe tx’s

Y4 Cardiology (27 decks)

Brainscape helps you reach your goals faster, through stronger study habits.
© 2024 Bold Learning Solutions. Terms and Conditions