Shock Flashcards
1. Explain concepts related to the assessment of an emergency department patient experiencing a shock emergency. 2. Describe various patient presentations related to shock emergencies. 3. List interventions necessary for a patient presenting with a shock emergency.
define shock
Shock is a state of impaired tissue perfusion resulting from circulatory failure brought on by various causes.
shock analogy to car
Gas = blood and plasma
Fuel pump = heart
Fuel line = arteries and veins
how is shock classified?
by the cause of inadequate perfusion
categories of shock
hypovolemic (low on gas)
cardiogenic (bad fuel pump)
distributive (fueling 2 cars with 1 gas line)
obstructive (blockage in fuel line)
cellular cascade in shock
similar for all causes of shock
–initially irreversible but w/o tx becomes irreversible
cell injury -> inflammation -> cell death
anaerobic metabolism -> glucose phosphorylation -> lactic acidosis
microvascular thrombosis -> depletion of clotting factors -> DIC
3 stages of shock
compensated
uncompensated
irreversible
compensated shock overview
Stage I or nonprogressive
compensatory mechanisms initiated
vital organs perfused
full recovery
uncompensated shock overview
Stage II or progressive
compensatory mechanisms inadequate
vital organs hypoperfused
recovery more difficult
irreversible shock overview
Stage III
compensatory mechanisms failed
cellular and organ death
refractory to tx
overview of physiological of compensated shock
SNS response
RAAS
ADH
intracellular fluid shift
SNS response in compensated shock
release of epi/norepi
vasoconstriction
increased HR, contractility, BP
RAAS in compensated shock
increased serum sodium
water reabsorption
decrease urine, increase BP and CO
ADH in compensated shock
renal reabsorption of sodium and water
decrease urine, increase BP and CO
intracellular fluid shift in compensated shock
increased intravascular volume
increase BP and CO
overview of physiology of uncompensated shock
altered capillary permeability respiratory insufficiency cardiac depression tissue hypoperfusion brain hypoperfusion
altered cap permeability in uncompensated shock
leakage into interstitial space
edema, decreased BP and perfusion
respiratory insufficiency in uncompensated shock
pulm edema
VQ mismatch
crackles, dyspnea, increased RR
cardiac depression in uncompensated shock
diminished venous return
ischemia of nonvital organs
decreased urine output increased lactate base deficit mixed venous sat (below 65%) cool skin decreased peripheral pulses
brain hypoperfusion in uncompensated shock
AMS
define hypovolemic shock
blood, plasma, or fluid loos reduces circulating blood volume and cardiac output
most common type of shock
common causes of hypovolemic shock
traumatic hemorrhage (long bone/pelvic fx, solid organ rupture, open wounds)
nontraumatic hemmorrhage
(GI bleed, ruptured AA, posterior epistaxis)
fluid shifts
(peritonitis, massive crush injuries, severe burns)
non-blood fluid loss
(v/d, diaphoresis)
urinary fluid loss
(DKA, diabetes insipidus, diuretic abuse)
hypovolemic shock assessment
ABCs cause labs trend VS MAP (above 60) (SBP + 2DP/3) urine output LOC
parameters of hypovolemic shock
blood loss HR BP RR urine output CNS
Class I of hypovolemic shock
blood loss below 15% HR below 100 BP normal RR 14-20 Urine above 30ml/hr CNS slightly anxious
Class II of hypovolemic shock
blood loss 15-30% HR 100-120 BP normal RR 20-30 Urine 20-30 ml/hr CNS mildly anxious
Class III of hypovolemic shock
blood loss 30-40% HR 120-140 BP decreased RR 30-40 urine output 5-20 ml/hr CNS anxious, confused
Class IV of hypovolemic shock
blood loss > 40% HR > 140 BP decreased RR > 35 urine output negligible CNS confused, lethargic
hypovolemic shock intervention overview
airway
control bleeding, reduce fluid loss
restore volume
blood replacement