Shock Flashcards
What is shock?
(A severe imbalance between oxygen supply and demand lead to inadequate cellular energy production)
What are the two characteristics of the cardiovascular system that are pertinent to oxygen delivery?
(Cardiac output and arterial content of O2)
What are the two characteristics of the cardiovascular system that are pertinent to cardiac output?
(Heart rate (which is determined by the sympathetic vs. parasympathetic NS) and stroke volume (which is determined by preload, afterload, and contractility))
What is hypovolemic shock and how does it impact oxygen delivery?
(A decreased intravascular volume which = decreased preload which = decreased CO which = decreased oxygen delivery)
What are the etiologies for hypovolemic shock?
(Hemorrhage, severe dehydration, third space fluid loss, and severe burns)
What is distributive shock?
(A maldistribution of fluid d/t changes in vascular tone and increased vascular permeability = leaky vessels)
What are etiologies for distributive shock?
(Histamine induced vasodilation = anaphylactic shock, cytokine-mediated endothelial dysfunction = septic shock, neurogenic shock, and pheochromocytomas or extreme fear = catecholamine release)
What type of shock is associated with GDV, vena cava obstruction, tension pneumothorax, cardiac tamponade, and positive pressure ventilation?
(Obstructive shock → compression of heart or great vessel that interferes with venous return = decreased diastolic filling and preload = decreased cardiac output)
What is cardiogenic shock?
(A decrease in forward flow from the heart failing to pump = a primary decrease in cardiac output)
What are etiologies for cardiogenic shock?
(Systolic failure, diastolic failure, AV valve degeneration/defects, and brady or tachyarrhythmias)
What type of shock is associated with severe pulmonary dz, anemia, and dyshemoglobinemias (CO toxicity, methemoglobinemia, etc.)?
(Hypoxic shock = decreased arterial oxygen content = decreased tissue oxygen delivery)
What is metabolic shock?
(Deranged cellular metabolism leading to inappropriate O2 use in the tissues (tissues have plenty of O2 they just aren’t using it), etiologies can be severe hypoglycemia and mitochondrial dysfunction)
What effect will the baroreceptors sensing decreased stretch have on the body?
(Will trigger release of catecholamines from the adrenals → increased heart rate, increased cardiac contractility, and peripheral vasoconstriction)
What triggers increased respiratory rate and tidal volume as a compensatory mechanism in the face of shock?
(Chemoreceptors sensing changes in pH, CO2/O2, etc.)
What is the purpose of activating the RAAS as a compensatory mechanism in the face of shock?
(Angiotensin II triggers peripheral vasoconstriction and renal reabsorption of sodium (water follows) and ADH triggers increased renal water absorption)