set 9 - sleeping and eating disorders Flashcards

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1
Q

eating disorders

A

overview:

1) classification of eating disorders

  • binge-eating disorder
  • bulimia nervosa
  • anorexia nervosa

2) biological factors & treatments

3) psychological factors & treatments

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2
Q

eating disorder

defining characteristic

A

Defining characteristic

  • Self-worth is highly influenced by body shape
  • if it is not influenced by this, then they will not meet criteria for ED

Disorders

  • Binge-eating disorder
    Life% = 1%-2% - common to women
  • Bulimia nervosa
    Life% = 1.5%-4% - 10x more common to women
  • Anorexia nervosa
    Life% = 1.5%-2.5% - 10x more common to women
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3
Q

binge-eating disorder

A

Recurrent episodes of binge eating (criteria of bingeing)

  • Eating an amount of food that is far larger than most people would consume in that situation
  • Sense of lack of control over eating

Associated with (symptoms)

  • Eating much more rapidly than normal
  • Eating alone due to embarrassment
  • Bingeing when not hungry

notes:

  • depends on the individual what is excessive
  • lack of control – they can’t stop or choose what food to eat
  • bingeing must occur at least once a week for 3 months
  • they need at least 3 of 5 of the symptoms
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4
Q

bulimia nervosa

A

1) Recurrent episodes of binge eating

2) Recurrent compensatory behaviours to prevent weight gain

  • compensatory behaviours include vomiting, using laxative, and diuretics, some may fast and exercise

3) Excessive influence of weight and shape on self-evaluation

  • how they view themselves changes upon their weight and shape

notes:

  • bingeing should occur at least once a week
  • lots of negative side effects, there is a strong desire to be thin
  • they still stay within 10% of their normal body weight
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5
Q

anorexia nervosa

A

1) Food intake restriction leading to significantly low body weight

  • they eat less food, which leads to significant low body weight (vary within age, gender

2) Intense fear of gaining weight or becoming fat, or persistent engagement in behaviour that interferes with weight gain

  • they deprive themselves of food because they are scared to become fat

3) Distorted body image, self-worth influenced by body weight or shape, or persistent lack of recognition of seriousness of low bodyweight

  • perceived perception of their own body size have exaggerated body views, even if they are dangerously underweight, they still view themselves as overweight

notes:

  • should meet all 3 criteria
  • 25-40% BELOW A LOW BODY WEIGHT

they tend to have more control, for them weight loss must be lost everyday, staying in the same weight is unacceptable

  • 5% will die within 10 years (due to starvation or suicide)
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6
Q

2 subtypes of anorexia nervosa

A

1) restricting

  • Severe restrictions on amount and type of food eaten.

& Restrictive behaviors (counting calories, skipping meals, eliminating certain foods (such as carbohydrates).

  • Often combined with excessive exercise. (dieting, fasting and excessive exercise characterizes that)

2) binge-eating and/or purging

  • Restrict food intake
  • Engage in binge eating and/or purging behaviors (i.e., vomiting, use of
    laxatives or diuretics)

*significantly underweight

  • looks like bulimia but the difference is that people with anorexia they adapt very strict diet whereas ppl with bulimia purge to lose weight
  • people with bulimia tend to hover around normal body weight whereas ppl with anorexia tend to be dangerously underweight weight will differentiate these two
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7
Q

ED onset, course, and gender differences

A

1) Tends to onset in adolescence or young adulthood

  • 90% of cases are diagnosed before 20yrs
  • Onset associated with stressful life event (exposure to violence, family conflict, stress at school can be associated with ED)
  • Chronic unless treated
  • Relapse is common

2) Vast majority are women

  • interestingly, men with ED are more likely to be gay or bisexual. Maybe there is more emphasis on body image and appearance

3) Young athletes are vulnerable

  • young athletes are vulnerable because there is focus on their body weight, shape, strength
  • they are competitive in nature, disorder in eating can be very much reinforced
  • their methods often go unnoticed because restrictive eating is an expected behaviour
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8
Q

ED biological factors and treatment

A

1) Etiological factors

  • Genetic contribution - family members with individuals with ED are 5x more likely to develop it
  • may be linked to impulsitivity and emotional instability
  • Low level of serotonergic activity - lack or improper diet leading to that
  • The runner’s high/exercise anorexia- can reinforce excessive exercising behaviour

2) Treatment

  • SSRIs - short term
  • Atypical antipsychotics - not a lot of evidence however they have a side effect of weight gain
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9
Q

ED psychological factors

culture

A

(Culture)

  • Significant increase in rates since the 1950s
  • Significantly less common in developing and non-Western countries - there are higher rate in Caucasians
  • Exposure to media - there is a direct connection with media exposure, media influences what an acceptable body type is
  • African-Americans vs. Caucasians
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10
Q

ED psychological factors

views on weight

A

Etiological factors

(Views on weight)

  • Dieting teenagers - 60% of females and a third of men in gr 8 and 9 are dieting / dieting is a risk factor for developing an ED
  • What the other sex wants - women tend to assume that men want thin women and men assumes that women wants a muscular man
  • Choosing friends who reinforce body-image ideas
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11
Q

ED psychological factors

other cognitive factors

A

Etiological factors

(Other cognitive factors)

  • Low sense of control - diminished sense of control in bulimia is low
  • in anorexia there is a low set of control, as the disorder progresses their sense od control increases,
  • High perfectionism - high perfectionism tends to be linked to low self esteem (common to ppl with anorexia)
  • Perceptions of body shape - for them a single snack can cause them to view their body to be fatter
  • Inaccurate beliefs about what is a healthy weight - they often have an unrealistic belief about how low they can go and still be healthy
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12
Q

ED psychological factprs

family influences

A
  • perfectionistic mothers - tend to reinforce cultural opinions on weight and emphasize self control
  • family preoccupation with appearance - perfectionistic parents see children as a reflection of them
  • how they dress their kids and how they present themselves
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13
Q

ED psychological treatment

issues in treatment

A

1) Very unlikely to seek treatment

  • people with anorexia are unlike to seek treatment because they complete deny that they don’t have a problem
  • people with bulimia avoid treatment due to shame (a lot of shame associated with bulimia) / more likely to be forced into treatment

2) Often fake agreement with treatment

  • they may agree and comply so they can be let out sooner, go home and continue

3) If too thin, weight must be restored first

  • if they are dangerously underweight, no 1 priority is to restore weight
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14
Q

ED psychological treatment

CBT

A

CBT is fairly effective 40-60% success rate

1) Psychoeducation around health effects

  • important - what are they doing to their body
  • Help restructure cognitive distortions or views that they may have
    • challenge those dyfucntional patterns

2) Meals controlled by therapist, workers, and family

3) Challenge dysfunctional thoughts regarding body shape, weight, and eating

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15
Q

ED psychological treatment

IPT(interpersonal psychotherapy)

A

Targets dysfunctional relationships

  • work relationships with their mother
  • family therapy is more common in anorexia and acc increases success

May work as well as CBT over the long term

  • in terms of ED, a combination of medications, CBT and IPT may be the best
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16
Q

sleep-wake disorders

A

overview

1) insomnia

2) Hypersomnolence Disorder

3) Narcolepsy
- Biological Factors
- Psychological Factors

4) Circadian Rhythm Disorders
-Biological Factors
-Psychological Factors

17
Q

sleep disorders

what are the two important states

A

1) slow-wave sleep: restorative sleep, when you wake up in the morning, you feel rested

2) rapid eye movement (REM): in some cases lack of REM sleep is indicative of sleeping disorders

  • dreaming is associated with REM sleep
18
Q

sleep in other disorders

A

Anxiety, mood, and psychotic disorders have sleep issues

Sleep issues may be caused by, and/or precipitate, these disorders

  • Sleep problems will often show up with individuals who have anxiety, mood, and psychotic disorders
  • Sleep issues may be caused by them
  • Lack of sleep can precipice other psychological disorders or vice versa
19
Q

sleep disorders

A

1) Insomnia disorder
12mo% = 6% Life% = 10%

  • onset = adulthood

2) Hypersomnolence disorder
Life% = < 1%

  • onset = typically 17-24, but individuals aren’t diagnosed until 10-15 years later

3) Narcolepsy
Life% = < 0.05%

  • more prominent in men / typically occurs between 15-25 or 30-35 yrs old

—- for the first 3, a 3 month time frame criterion for diagnosis, symptoms must be present for at least 3x a week

4) Circadian rhythm disorders

  • prevalance rates are unknown becayse many of them are very short duration
20
Q

insomnia disorder

A

A predominant complaint of dissatisfaction with sleep quantity or quality, with at least 1 of the following:

1) Difficulty initiating sleep

2) Difficulty maintaining sleep

3) Early-morning awakening with inability to return to sleep

Specifiers:

-episodic insomnia: symptoms for at least a month but less than 3 months, more acute

  • persistent insomnia: surpassing 3 months, more than 3 months
  • recurrent insomnia: 2 or more episodes in a year
21
Q

insomnia biological factors

A

Etiological factors

1) Delayed body temperature rhythm

  • At night our body typically cools down in preparation of sleep, which increases our drowsiness
  • a delayed body temperature rhythm can be a cause of insomnia disorder – we get drowsy late

2) Drug use - stimulants, adhd meds

3) Hyperthyroidism - rare

22
Q

insomnia biological treatment

A

Sleeping pills work short term and not work in the long term (they may have rebound insomnia)

Melatonin – can induce sleep in the short term but no strong evidence that it works necessarily as a vitamin, moves ahead that “drowsy time”

Antithyroid medications – for hyperthyroidism

23
Q

insomnia psychological factors

A

1) Environmental issues, recurrent napping (noise, lights, recurrent napping can affect how you sleep at night)

2) Association between bed and wakefulness cues - Our bodies forms association with our bed as well, when you watch tv at work, call people then ur body learns to associate your bed not just for sleep, your body wants to be awake now

3) Dysfunctional beliefs regarding sleep - me time, i do not need to sleep cos this is the only time i can get me time (having these beliefs can affect sleep)

24
Q

insomnia psychological treatments

A

1) Elimination of environmental issues/naps

  • behavioural therapy helps explain how we form association to things, now we break those, “im only in bed when I’m sleeping” / retraining your system for associating bed with sleeping, do not use phone while in bed

2) Behavioural therapy for extinction of wakefulness cues

3) Cognitive therapy for beliefs

    • – examine the beliefs we have and challenge those beliefs / worry schedule, we do not worry in bed

exercise and relaxation training is also helpful

25
Q

hypersomnolence disorder

A

Excessive sleepiness despite sleeping for at least 7 hours, with at least 1 of the following:

1) Recurrent periods of sleep or lapses into sleep in the same day

  • sleep or naps aren’t restorative, naps don’t help
  • they may fall asleep lots, get lots of sleep at night, hard to stay awake
  • sleep for 9.5 hours at night

2) Prolonged sleep of more than 9 hours that is unrefreshing

3) Difficulty being fully awake after abrupt awakening - sleep inertia

acute = less than a month

26
Q

insomnia vs hypersomnolence

A

Insomnia – struggle to fall / stay asleep

Hypersomnolence – too much sleep, but it doesn’t help / excessive sleepiness, they can sleep but still feel tired

27
Q

narcolepys

A

Recurrent periods of irrepressible need to sleep, lapsing into sleep, or napping occurring in the same day, with at least 1 of the following:

1) Episodes of cataplexy

  • : episodes of sudden lost of muscle tone with maintained consciousness, looks like they fainted, but they are fully alert, there’s a strong emotion (anger or sadness) that can cause cataplexy
  • cataplexy during REM sleep is what keeps us from getting up and acting our dreams
  • cataplexy only shows up with narcolepsy

2) Hypocretin/orexin deficiency

  • unique to narcolepsy too
  • hormone in the brain that regulates wakefulness, so individuals may have deficiency in this

3) Nocturnal REM sleep latency less than 15 minutes, or a mean sleep latency test of less than 8 minutes and two or more sleep onset REM periods

  • less than 15 mins: if they fall asleep and they immediately go to rem sleep within 15 minutes, they meet criteria for narcolepsy
  • a mean sleep: if they fall asleep within 8 minutes and they have 2 rem sleep within 5 naps lets say then they can meet critera
  • individuals with narcolepsy often sleepy, and getting excessive amount of REM sleep, they do not get a lot of slow wave sleep
28
Q

other features of narcolepsy

A

1) Sleep paralysis (the old hag)

  • they feel paralyzed but alert
  • decrease with age but sleepiness remains

2) Hypnagogic hallucinations

  • Hallucinations: very intense dreams / response for UFO sightings and alien sightings?
  • decrease with age

3) Restorative naps

  • naps help for ppl with narcolepsy
  • they feel rested after napping
  • in hypersomnia – it is not restorative, no REM onset, not associated with cataplexy
29
Q

hypersomnia and narcolepsy biological factors

A

Orexin deficiency

  • Orexin, also known as hypocretin, is a neuropeptide produced by neurons in the hypothalamus. It plays a crucial role in regulating wakefulness, arousal, and the sleep-wake cycle. In individuals with narcolepsy type 1 (formerly known as narcolepsy with cataplexy), there is a significant deficiency or absence of orexin due to autoimmune destruction of the orexin-producing neurons.

*The loss of orexin leads to dysregulation of sleep-wake mechanisms, resulting in the hallmark symptoms of narcolepsy, such as excessive daytime sleepiness and disrupted REM sleep patterns. Without the stabilizing influence of orexin, individuals with narcolepsy experience sudden transitions between wakefulness and REM sleep, leading to fragmented sleep and symptoms like cataplexy, sleep paralysis, and hallucinations.

Brain damage: autoimmune issues, genetic mutations

Histamine: a wake promoting system in our body, it promotes wakefulness (antihistamine -> drowsiness)

Hypothyroidism: linked to narcolepsy

Hyper- insomnia
Hypo – sleep more

30
Q

hypersomnia and narcolepsy biological treatment

A

Modafinil (Provigil) and amphetamines - can be very predictable, they may work great one day and not tomorrow

Antidepressants for cataplexy

Thyroid medications

  • they are trying to sleep less
31
Q

hypersomnia and narcolepsy psychological factors and treatments

A

Hypersomnia & Narcolepsy

(Etiological Factors)

Sleep used as a coping mechanism ?

  • to avoid stress or distract us
  • narcolepsy seems to be related to medical issues, so a coping mechanism might not explain it
  • very much biologically bases so we would lean more to medications

(Treatment)

Psychoeducation regarding disorder

Development of coping strategies

Adjustment to life circumstances - people may not be able to drive

32
Q

circadian rhythm disorders

A

no 3 month time frame criterion, can be short term

Sleep disruption due to alteration of the circadian system, or misalignment between endogenous circadian system and the individual’s required sleep-wake schedule

Leads to excessive sleepiness, insomnia, or both

  • Delayed sleep type: individuals is consistently going to bed later than desired / onset: adolescent or early adulthood
  • Advanced sleep type: keep going to bed earlier than desired / onset: older adulthood
  • Non 24 hour type: they go to bed later and later because their body is on a 25 / 26 hour clock, not on a 24 hour schedule / adolescent and early adulthood
  • Shift work type: awake or sleepy at the wrong time due to challenging work schedules / we can see this in older adulthood
  • Jet lag type: misalignment in circadian rhythm
33
Q

circadian rhythm biological factors and treatment

A

Circadian Rhythm Disorders

(Etiology)

Suprachiasmatic nucleus (SCN)

Melatonin

SCN: in the hypothalamus tells us our body to change our sleep wake schedules depending on when light is entering our retinas,, what helps us readjust to what time zones we are in

  • safer to be awake during the day

(Treatment)

Melatonin

  • hormone that naturally increases when it is time to be sleepy based on your circadian rhythm
  • treatment, we take it when we would ideally be going to sleep
34
Q

circadian rhythm disorders

A

Circadian Rhythm Disorders

(Etiological Factors)

Changing work schedules - If its problematic, you may have to look at adjusting your schedule is

(Treatment)

Phase delaying - easier to get a person to sleep later than earlier

Exposure to sunlight/bright light therapy - can help you get on with the schedule you want to be on

Changing expectations regarding employment

35
Q
A