Session 6 & 7 Flashcards

1
Q

How many limb leads are there?

A

6

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2
Q

How many electrodes/wires record the six limb leads?

A

4

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3
Q

_________ electrodes and wires that you need to connect to record from all _______ limb leads

A

Four

Six

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4
Q

Name the position/colour of the 4 limb electrodes and a mnemonic

A

Ride Your Green Bike

Starting with the right arm going clockwise

Red - Right Arm
Yellow - Left Arm
Green - Left Foot
Black - Right Foot

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5
Q

Which limb electrode acts as ‘neutral’?

A

Right leg

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6
Q

What is meant by a bipolar electrode?

A

Can be positive or negative depending on configuration

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7
Q

At which angles do leads I, II and III view the heart?

A

0 degrees (pointing horizontally towards the right)
60 degrees
120 degrees

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8
Q

How are the augmented limb leads named?

A

Right arm - aVR
Left arm - aVL
Left foot - aVF

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9
Q

In the augmented limb leads, are the limb electrodes positive or negative?

A

Positive

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10
Q

How are the augmented limb leads measured?

A

With reference to a negative terminal coming from the centre of the heart

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11
Q

What direction does aVR, aVL and aVF view the heart?

A

-150
-30
90

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12
Q

Which limb leads look at the left of the heart?

A

aVL

Lead I

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13
Q

Which limb leads look at the apex of the heart?

A

Lead II

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14
Q

Which limb leads look at the inferior heart?

A

Lead II
aVF
Lead III

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15
Q

Which lead looks at the right of the heart?

A

aVR

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16
Q

Which two limb leads view the heart almost exactly oppositely? What is the significance of this?

A

aVR
Lead II

Inverted images should be seen on the ECG

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17
Q

Describe the position of the six chest leads (C1-C6)

A

C1 - 4th intercostal space, right sternal border
C2 - 4th intercostal space, left sternal border
C3 - midway between C2 and C4
C4 - 5th intercostal space, midclavicular line
C5 - between C4 and C6
C6 - 5th intercostal space, midaxillary line

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18
Q

Before recording an ECG, ____________ must be taken from the patient

What happens to the skin before sticky electrodes are attached in an ECG?

A

Consent

Wiped clean with alcohol wipes

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19
Q

The conducting system of the heart consists of specialised cells that have lost _____________ but have the ability to generate _________ _____________

A

Contractility

Action potentials

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20
Q

In what way do the ventricles depolarise?

A

From endocardium to epicardium

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21
Q

Where is the SA node found?

A

Near the junction of the superior vena cava and right atrium

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22
Q

What is special about the depolarisation that takes place in cells of the SA node?

A

Depolarisation happens the fastest here

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23
Q

Which part of the heart sets the sinus rhythm of the heart? Why this part of the heart?

A

SA node

Cells in the SA node depolarise the fastest

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24
Q

The AV node is continuos with the…

A

Bundle of His

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25
Q

Where is the AV node located?

A

At the interatrial septum just above the tricuspid valve

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26
Q

What is the only conducting pathway from atria to ventricles?

A

The AV node

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27
Q

What structure of the heart prevents direct contact between and conduction impulses from atrial to ventricular myocytes?

So, how do impulses pass from atria to ventricles?

A

Fibrous ring in the heart

Through the AV node

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28
Q

Where are the right and left bundle branches located?

A

Sub-endocardially in the IV septum

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29
Q

Purkinje fibres are fine branches of the…

Purkinje fibres cause the rapid spread of…

A

Bundle of His

Depolarisation throughout the ventricular myocardium

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30
Q

Describe how the ventricles are depolarised and repolarised

A

IV septum depolarised first
Apex, RV and LF free walls depolarised next
Base of the ventricles depolarised last

Repolarisation of ventricles in the reverse order

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31
Q

What does an ECG record?

A

Changes on the extracellular surface of cardiac myocytes during depolarisation and repolarisation

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32
Q

A depolarisation wave from -ve to +ve will result in which complex being seen on an ECG?

What will be seen on repolarisation?

A

Positive complex

Negative complex

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33
Q

A depolarisation wave from +ve to -ve will result in which complex being seen on an ECG?

What will be seen on repolarisation?

A

Negative complex

Positive complex

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34
Q

What ECG complex will be seen if a depolarisation wave travels directly towards a positive electrode?

Obliquely towards?

A

Tall +ve complex

Smaller +ve complex

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35
Q

What ECG complex will be seen if a depolarisation wave travels at 90 degrees to a positive electrode?

A

No complex seen

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36
Q

What ECG complex will be seen if a wave of depolarisation travels obliquely away from a positive electrode?

Directly away?

A

Small -ve complex

Deep -ve complex

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37
Q

How does SA node depolarisation affect an ECG reading?

A

No effect, insufficient signal to register on surface ECG

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38
Q

How does atrial depolarisation affect an ECG reading? (Viewing from the apex)

A

Causes a small upward deflection of the P wave

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39
Q

What is the cause in the heart of a p wave on an ECG?

A

Atrial depolarisation

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40
Q

How does the delay at the AV node affect an ECG reading?

A

No visible effect - signal very small and isoelectric flat line segment

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41
Q

What is the name given to the segment of the ECG that shows delay at the AV node?

A

Isoelectric flat line segment

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42
Q

The non-conducting fibrous ring found in the heart is only crossed by the…

A

Bundle of His

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43
Q

As well as AV delay what else contributes to the isoelectric flat line segment of the ECG?

A

The spread of depolarisation from atrium to ventricle via the His Purkinje system

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44
Q

What is the cause in the heart of the Q wave of an ECG?

A

Depolarisation of the myocardium - IV septum from left to right

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45
Q

In which way does the IV septum depolarise?

A

From left to right

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46
Q

What effect does depolarisation of the myocardium (IV septum) have on an ECG?

A

A small downward deflection - Q WAVE

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47
Q

What change in the heart causes the R wave of an ECG?

A

Depolarisation of the apex and free ventricular wall

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48
Q

What effect does depolarisation of the apex and free ventricular wall have on the ECG?

A

Produces a large upward deflection called the R wave

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49
Q

Why is the R wave, large and upwards on an ECG?

A

Depolarisation moving directly towards an electrode

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50
Q

How will LV hypertrophy affect the R wave of an ECG?

A

Will result in a taller R wave

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51
Q

Which change in the heart causes the S wave of an ECG?

A

The end of depolarisation, as depolarisation spreads upwards to the base of the ventricles

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52
Q

What effect does the end of depolarisation (depolarisation upwards to the base of the ventricles) have on an ECG?

A

Results in a small downward deflection - S WAVE

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53
Q

What change in the heart causes the T wave of an ECG?

A

Ventricular repolarisation

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54
Q

What effect does ventricular repolarisation have on an ECG?

A

Produces a medium upward deflection - T WAVE

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55
Q

Why is the T wave seen as an upwards deflection?

A

It is as a result of repolarisation and is moving AWAY from the electrode

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56
Q

Where does ventricular repolarisation begin? In which direction does ventricular repolarisation occur?

A

Epicardial surface

Opposite direction to depolarisation

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57
Q

P wave of an ECG is due to __________ _______________
QRS complex of and ECG is due to _____________ _______________
T wave of an ECG is due to _____________ _______________

A

Atrial depolarisation

Ventricular depolarisation

Ventricular repolarisation

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58
Q

Are all the components of the QRS complex seen in all leads when an ECG is carried out?

A

Not always

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59
Q

How many electrodes are used in the recording an ECG? How many chest? How many limb?

A

10

6
4

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60
Q

10 electrodes used in the recording of an ECG give how many leads of the heart?

Leads of the heart are effectively different _________ of the heart

A

12 leads

Views

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61
Q

The limb leads give __ views of the heart from __ electrodes in the ______________ plane

A

6
4

Vertical

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62
Q

Name three limb leads that are best for looking at the inferior surface of the heart

A

II
III
aVF

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63
Q

Name two limb leads that are useful for looking at the left side of the heart

A

I

aV

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64
Q

Chest leads give __ views of the heart from __ chest electrodes in the ____________ plane

A

6

6

Horizontal

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65
Q

What part of the heart can be described as lateral? (ECGs)

A

LV = Lateral Side

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66
Q

Which chest leads give views of the right ventricle and septum?

What is another term for these leads?

A

V1
V2

Septal leads

67
Q

Which chest leads give views of the apex and anterior wall of the ventricles?

What is another term for these leads?

A

V3
V4

Anterior leads

68
Q

Which chest leads give views of the left ventricle?

What is another name for these leads?

A

V5
V6

Lateral leads

69
Q

What are the horizontal and vertical axes of an ECG?

A

Horizontal = Time

Vertical = Voltage

70
Q

What is the normal speed that an ECG runs at?

71
Q

How many small squares make up 1 second on an ECG tracing?

How many large squares make up 1 second on an ECG tracing?

A

25 small squares

5 large squares

72
Q

What is 1 cardiac cycle with regards to the different waves seen on an ECG?

A

Each PQRST complex (one P wave to the next)

73
Q

How many large boxes is equal to 1 minute on an ECG trace?

A

300 large boxes

74
Q

How is the heart rate measured from an ECG?

A

Number of large boxes between complexes (R waves) counted

300/Number of Large Boxes between complexes = HR

75
Q

How is the heart rate measured from an ECG if the heartbeat is irregular?

A

Count the number of QRS complexes in 6 seconds (30 large squares)

Times by 10 = HR

76
Q

How long should a normal PR interval last in…

I) seconds
II) boxes

A

0.12-0.20 seconds

3-5 small boxes

77
Q

When will a PR interval be considered prolonged?

A

Greater than 1 large box

78
Q

A normal QRS interval should be no more than how many…

I) seconds
II) small boxes

A

0.12 seconds

3 small boxes

79
Q

The QT interval varies with…

A

Heart rate

80
Q

What is normal sinus rhythm? Where does it originate?

A

Characteristic rhythm of a healthy heart

Sinoatrial node

81
Q

Name 5 factors that are considered when looking for normal sinus rhythm

A
Heart Rate
Rhythm 
P wave 
PR interval 
QRS
82
Q

What values for heart rate is consistent with normal sinus rhythm?

A

60-100 bpm

83
Q

What is looked for when assessing the P wave to see if someone is in normal sinus rhythm?

A

Whether it is before QRS complex and identical each time

84
Q

What value will PR interval and QRS take in seconds in a person with normal sinus rhythm?

A

PR - 0.12-0.20 seconds

QRS - less than 1.2 seconds

85
Q

What is sinus bradycardia?

A

Sinus rhythm where there is a heart rate less than 60bpm

86
Q

What is sinus tachycardia?

A

Sinus rhythm with a rate of 100 bpm or more

87
Q

Abnormal rhythms in the heart can result from either abnormal… (2)

A

Impulse formation

Conduction

88
Q

What two types of rhythms are seen in the heart?

A

Supraventricular

Ventricular

89
Q

Supraventricular rhythms consist of rhythms from which parts of the heart? (3)

A

SA node
Atrium
AV node

90
Q

Ventricular rhythms consist of rhythms from which part of the heart?

A

Ventricles

91
Q

How will abnormal supraventricular rhythms affect…

I) Ventricular depolarisation
II) Conduction

A

Normal ventricular depolarisation will take place

Conduction will be normal via the His-Purkinje system

92
Q

How will abnormal supraventricular rhythms affect the QRS complex of an ECG?

A

Will be normal (narrow)

93
Q

How do abnormal ventricular rhythms affect…

I) Depolarisation in the ventricles
II) Conduction

A

Depolarisation takes longer

Conduction not via normal His-Purkinje system

94
Q

How do abnormal ventricular rhythms affect the ECG seen?

A

Wide and bizarre QRS complexes

95
Q

A wide QRS complex is typically longer than ___ small boxes

96
Q

Will abnormal ventricular rhythms always produce the same bizarre QRS complexes?

A

No, depends on the foci of the ventricular rhythm

97
Q

Give two example of conditions that can result from abnormal ventricular rhythms

A

Ventricular tachycardia
Ventricular fibrillation
Ventricular premature beats

98
Q

How is the rhythm of the heart interpreted from an ECG?

A

By looking at the rhythm strip at the bottom of the 12 lead ECG

99
Q

What is the rhythm strip of an ECG?

A

A long (approx 10 seconds) recording of limb lead II

100
Q

Describe the impulses/depolarisation seen in atrial fibrillation

A

There are multiple foci throughout the atria

Depolarisation and impulses chaotic

101
Q

How does atrial fibrillation affect the p waves seen on an ECG?

A

No p waves will be seen instead just a wavy baseline

102
Q

How does atrial fibrillation affect the contraction of the atria?

A

They quiver rather than contract

103
Q

What happens to the rapid, irregular impulses that arrive at the AV node in atrial fibrillation?

A

Only some are conducted to ventricles when the AV node is not refractory

104
Q

Why are only some of the impulses from the atria conducted to the ventricles at the AV node in atrial fibrillation?

A

Impulses arrive rapidly/irregularly

AV node is in refractory period

105
Q

How does atrial fibrillation affect…

I) Ventricular depolarisation/contraction
II) QRS complex of the ECG
III) Pulse
IIII) Heart rate

A

Normal ventricular depolarisation and contraction

Normal shape (narrow) and irregular

Irregularly irregular

Irregularly irregular

106
Q

What are AV conduction blocks?

A

Where there is delay/failure of conduction of impulses from atrium to ventricles via AV node and the bundle of His

107
Q

What are two causes of AV conduction blocks? Which is more common?

A

Acute Myocardial Infarction (more common)

Degenerative Changes

108
Q

What are the three types of AV conduction blocks?

A

First degree heart block
Second degree heart block
Third degree heart block (complete heart block)

109
Q

What are the two types of second degree heart block?

A

Mobitz type 1

Mobitz type 2

110
Q

What mechanism works in the heart to avoid pacemakers ‘competing’ with each other?

A

In the case of failure of the SA node the NEXT FASTEST to depolarise will take over (AV node)

111
Q

Which part of the heart takes over from the SA node in case of failure?

In this case, ___________ rhythm is replaced by _______________ ___________ rhythm

A

AV node

Sinus
Ventricular escape rhythm

112
Q

What happens in first degree heart block?

A

There is SLOW conduction in the AV node and bundle of His

113
Q

How does first degree heart block affect the ECG?

I) P wave
II) PR interval
II) QRS

A

P wave normal

PR prolonged (>5 small squares)

QRS normal

114
Q

If the PR interval is prolonged in an ECG, it is typically >__ small squares

115
Q

As well as lead II being used in a rhythm strip, what other two leads are commonly used?

116
Q

What happens in Mobitz type 1 second degree heart block?

A

There is progressive lengthening of the PR interval until one P is not CONDUCTED

This allows the AV node time to recover

Cycle begins again

117
Q

How is the PR interval of an ECG affected in Mobitz type 1 second degree heart block?

A

Progressively lengthened

118
Q

What happens in mobitz type 2 second degree heart block?

A

There is sudden non-conduction of a beat resulting in dropped QRS complex

119
Q

Which type of heart block has a high risk of progression to complete heart block?

A

Mobitz type 2 second degree heart block

120
Q

Describe the PR interval and QRS complex seen in mobitz type 2 second degree heart block

A

PR interval normal

QRS complex dropped

121
Q

What happens in third degree heart block?

A

Atrial depolarisation is normal but impulses are not conducted to ventricle

Ventricular pacemaker takes over (ventricular escape rhythm)

122
Q

How does third degree heart block affect…

I) atrial depolarisation
II) conduction to ventricles
III) pacemaker of the heart

A

Normal atrial depolarisation

Not conducted

Ventricular pacemaker takes over (ventricular escape rhythm)

123
Q

What sort of heart rate is seen in third degree heart block?

A

Very slow - ~30-40 bpm

124
Q

What are the implications of the very slow heart rate seen in third degree heart block? What is therefore required for treatment?

A

Heart rate is too slow to maintain BP and perfusion

Urgent pacemaker

125
Q

How does third degree heart block affect the QRS complexes seen on an ECG?

A

Wide QRS complexes

126
Q

Apart from causing a wide QRS complex, how are the P-P intervals and R-R intervals affected on an ECG as a result of third degree heart block?

A

Constant

Constant but much slower

127
Q

Describe the relationship between the P waves and QRS complexes in third degree heart block?

A

There is no relationship between the P waves and QRS complexes

128
Q

What causes ventricular ectopic beats?

A

An ectopic focus in ventricle muscle

129
Q

How does an impulse that originates in the ventricle muscle spread (e.g. In ventricular ectopic beats)?

A

It is not spread rapidly via the His-Purkinje system follows abnormal conducting pathways

130
Q

How do ventricular ectopic beats affect…

I) depolarisation of the ventricles
II) QRS complex

A

Slower depolarisation of the ventricles

Wider QRS complex with different shape

131
Q

What is ventricular tachycardia?

A

A run of 3 or more consecutive ventricular ectopics

132
Q

Is ventricular tachycardia dangerous? What can it lead to?

A

Persistent ventricular tachycardia is dangerous and requires urgent treatment

Ventricular fibrillation

133
Q

How will ventricular tachycardia appear on an ECG?

A

With rapid, regular and broad beats

134
Q

What is ventricular fibrillation?

A

Abnormal, chaotic and fast ventricular depolarisation

135
Q

Describe the impulses seen in ventricular fibrillation

A

There are numerous impulses from numerous ectopic sites in ventricular muscle

136
Q

Describe the contraction seen in ventricular fibrillation

A

No coordinated contraction of the ventricles - ventricles quiver

137
Q

How can ventricular fibrillation affect cardiac output? What can this cause?

A

No cardiac output

Cardiac arrest

138
Q

In case of cardiac arrest as result of ventricular fibrillation, what must be done to restore rhythm in the heart?

A

CPR and immediate defibrillation

139
Q

Describe the pulse and heart rate seen in ventricular fibrillation

A

No pulse or heart beat

140
Q

What causes cardiac arrest, atrial fibrillation or ventricular fibrillation?

A

Ventricular fibrillation

141
Q

Ventricular fibrillation causes cardiac arrest. Name 4 other differences between atrial and ventricular fibrillation.

A

AF - ventricular depolarisation normal (chaotic in VF)
AF - normal ventricular contraction (no coordinated contraction in VF)
AF - irregularly irregular HB and pulse (no HB/pulse in VF)
AF - cardiac output present (no cardiac output in VF)

142
Q

Ischaemia and therefore myocardial infarction is due to…

A

Reduced perfusion of the myocardium

143
Q

Why can myocardial infarction cause changes in different ECG leads in different cases?

A

MI doesn’t affect all parts of the heart, changes will be seen in the leads facing the affected area

144
Q

Why is it important to look at the PQRST complexes in all 12 leads when investigating an MI?

A

MI doesn’t affect all parts of the heart - changes will only be seen in the leads facing the affected area

145
Q

What parts of the heart do the six chest leads look at?

A

V1, V2 - Septal Leads
V3, V4 - Anterior Leads
V5, V6 - Lateral Leads

146
Q

What parts of the heart of do the limb leads (excluding aVR) look at?

A

I, aVL - Lateral Leads

II, III and AVF - Inferior Leads

147
Q

Where do major coronary arteries lie in the heart?

A

On the epicardial surface of the heart

148
Q

Which muscle in the heart is furthest away from major coronary arteries on the epicardial surface? What is the implication of this?

A

Subendocardial muscle

Most vulnerable to MI

149
Q

What ECG changes can be seen in leads facing the affected area of an MI? (2)

A

ST segment depression

T wave inversion (-ve instead of +ve)

150
Q

What causes the ECG changes (ST segment depression and T wave inversion) seen in MI?

A

Abnormal current during repolarisation

151
Q

If ischaemia/MI is less severe, ischaemic ECG changes may only be seen as a result of…

A

Exertion - e.g. Exercise

152
Q

When will ischaemic ECG changes be seen at rest in an MI?

A

When there is a severe reduction in the size of the lumen of the coronary artery

153
Q

What causes STEMI to occur?

A

Complete occlusion of the lumen of a coronary artery

154
Q

Describe the extent of the muscle injury seen in a STEMI

A

Muscle injury extends full thickness - endocardium to epicardium

155
Q

Injury to the epicardium in a STEMI causes which change in the ECG?

A

ST elevation in leads facing the area

156
Q

If perfusion is not re-established in a STEMI what will happen?

A

Muscle necrosis

157
Q

What feature of an ECG can indicate that muscle necrosis has taken place as a result of a STEMI?

A

Developed Q waves that persist after the acute STEMI

158
Q

Q waves are usually narrow and caused by…

A

Depolarisation of the septum

159
Q

What appearance do pathological q waves take?

A

Wider/deeper than normal

> 1 small square wide
2 small squares deep

160
Q

How does hyperkalaemia affect the resting membrane potential?

A

Less negative RMP

161
Q

How does hypokalaemia affect the resting membrane potential?

A

More negative RMP

162
Q

How does the excitability of the heart change as a result of hyperkalaemia?

A

Becomes less excitable

163
Q

What effect does the less negative RMP seen in hyperkalaemia have on the cell during an action potential?

A

Inactivates voltage gated Na+ channels, heart becomes less excitable causing conduction problems