Session 3 Flashcards

1
Q

normal range for WBC?

A

5-10 thousand is the general range.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

normal range for platelets ?

A

150,000-400,000 range cells/mm3

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

what is the range for heemoglobin?

A

12-18 range g/DL
(hematocrit is 3x the amount of hemoglobin. )
(for TKA remeber 10 and 30. hemoglobin below 10 and hematocrit below 30)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

normal range for hematocrit?

A

38%-55%

hemoglobin <8 you should probably wait to ambulate.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

what percentage of effort would be accurate is a person reports a BORG of 14?

A

75%.
(multiply the borg # by 10, should approximate the heart rate)
(9=50%)
(goes up by 5% for each number after)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

a patient reports having tremors, heart arrythmias an cannot sleep at night. ALso reports polyuria and increases hair loss with heat intoleranace. What is the following condition?

A

Hyperthyroidism.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

an individual has increased pigmentation, weakness, weight loss, salt cravings, dizziness with standing, and hypoglycemia. what condition do they have?

A

addisons disease (adrenal hypocortisolism)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

is a person has moonface, buffalo hump, protuberant abodomen, and osteoporosis they have what condition?

A

cushing syndrome (adrenal hypercortisolism)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

what is graves disease?

A

hyperthyroidism

elevated body metbolism
heat intolerance
weight loss
polyuria
tremors/anxiety
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

patient with history of CAD, is taking metoprolol to improve heart function. What would you see on the ECG?

A

increased PR interval.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

what is metoprolol and how does it work?

A

its a beta blocker. given for high blood pressure and angina (chest pain)
- beta blockers block the beta adrenergic receptors of the heart, decreasing sensitivity to floating catecholamines like EPI or NOREPI. thus the PR interval will be increased (meaning a slower heart rate).

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

What is an increased PR interval called?

A

1st degree heart block

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

what is preload?

A

left ventricular end diastolic pressure

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

what is afterload?

A

systemic vascular resistance

how hard the heart has to push to get blood out into the vasculature

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

what is normal ejection fraction?

A

normal is above 55%

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

what condition would an individual be put on warfarin?

A

A-fib. this thins the blood and stops any type of blood clotting in the heart.

17
Q

if an individual with bells palsy has trouble closing their eyes, what muscle could be involved and what nerve?

A

orbicularis oculi and facial nerve CN VII

18
Q

if an individual with bells palsy has trouble opening their eyes what muscles could be involved and what nerve?

A

levator palpebrae, oculomotor nerve CN III

19
Q

what is the main difference between bells palsy (CNVII) and trigeminal neuralgia (CN V)?

A

palsy is about weakness and numbness

neuralgia is more about pain. also jaw and corneal reflex will be absent

neuralgia can be exacerbated with stress or cold while bells palsy will be exacerbated by use of muscles.

20
Q

what is the difference between CVA and bells palsy in terms of

1) onset
2) facial paralysis
3) weakness
4) sensory changes

A

CVA has more acute onset versus bells which is hours to days

2) CVA effects lower facial ms more than upper facial muscles. BElls is full hemi facial paralysis
3) CA will results in extremity weakness versus bells which results in CN VII damage (wont be able to close their eyes)
4) with CVA you will have the 4 D’s (dizziness, diplopia, dysarthia, dysphasia) and with bells you will have limited to no sensation changes

21
Q

Differential diganosis between osgood-schlatter disease and patellofemoral syndrome

A

Osgood-Schlatter disease (OS) is an overuse injury causing pain in the knee area and often a visible growth just below the kneecap to appear. OS occurs when there is irritation to the top, front portion of the shin bone (tibia) where the tendon attached to the kneecap (patella) meets the shin bone. This irritation at the attachment sit of bone and tendon is most often a consequence of excessive stress to the front of the knee during periods of rapid skeletal growth. Adolescents are most commonly affected by OS because they experience the greatest rate of skeletal growth. OS is most frequently experienced in adolescents who regularly participate in sports related activities.

Patellar tendinitis is comparable to Osgood Schlatter’s Disease but occurs in adults and is an injury to the tendon connecting your kneecap (patella) to your shinbone. The patellar tendon works with the muscles at the front of your thigh to extend your knee so you can kick, run and jump. Patellar tendinitis, also known as jumper’s knee, is most common in athletes whose sports involve frequent jumping, such as basketball and volleyball. However, people who don’t participate in jumping sports can get patellar tendinitis as well. For most people, treatment of patellar tendinitis begins with physical therapy to stretch and strengthen the muscles around the knee.

22
Q

what is Sinding Larsen Johansson syndrome (SLJS)

A

osteochrondrosis and tracction epiphysitis affecting the extensor mechanism of the knee which disturbs the patella tendon attachment to the inferior pole o the patella. Tenderness of the inferior pole of the patella is usually accompanied by x-ray evidence of splintering of that pole.

most patient with SLJS also show a calcification at the inferior pole of the patella.

23
Q

A person with weak qauds after back surgery, what would you see during their gait during initial contact through the loading response.

A

Forward trunk flexion. weak quads means that the normal loading response is altered. The patient could not tolerate any knee flexion or they would collapse to the floor. Thus, they will lock the knee, and the easiest way to prevent the quads from firing is to shift the body weight forward onto straight leg, relying on the passive structures of the knee to maintain the extension, rather than relying on quadricep strength.

24
Q

patient fitting for an AFO, upon observation the therapist notes that there is moderate to severe genu recurvatum with stand phase on the affected side. Which of the following will most likely decrease the amount of hyperextension?

A
plantarflexion stop
the combined movements are: 
1) dorsiflexion and knee flexion
2) plantarflexion and knee extension
3) think about the pull on the gastroc muscle.
25
Q

what is the cutoff for blood sugar for the APTA before you discontinue exercise

A

less than 60 no therapy at all.

70 or more you have to be cautious