Section 3 Flashcards
Dehiscence
The wound splits or bursts open
Complications of healing
IDKAU - I don’t keep assholes up
- infection
- dehiscence
- keloid development
- adhesions
- ulceration
Keloid development
Excess collagen
Adhesions
Fibrous connections with nearby tissues which limit movement
Ulceration
Circumscribed, open lesions (sores)
Burns
Caused by- thermal injury
- electrical energy - caustic chemical injury - radiation exposure - inhalation of noxious fumes
- The injury triggers an acute inflammatory response
Superficial partial thickness burns
1st degree burns
- these burns damage the epidermis
- there is erythema (redness), pain and swelling of the affected area
- these Burns do not result in cell necrosis or scarring
Deep partial thickness burns
2nd degree burns
- These burns damage epidermal skin layers and penetrate some dermal skin layers
- scarring can occur in some cases, healing takes 10 to 30 days
Full thickness burns
3rd degree burns
- these damage the epidermis and dermis and can penetrate subcutaneous layer’s as well
- healing is challenged by loss of elastin, replacement of skin cells with collagen, an invasion of microorganisms
Contractures
- Sometimes scarring after burns is extensive. lost of elasticity is evidenced by contractures, areas of thick, shortened, and rigid tissue.
- contractors limit movement
Burn complications
- widespread inflammatory response
- stress hypermetabolism
- impaired defense mechanisms
widespread inflammatory response
Permeable capillaries allow fluid to shift out of blood vessels and it can lead to shock, dead tissue, and Escher (which is exudate that converted into a thick coagulated crust)
stress hypermetabolism
Burns cause higher metabolic demands
impaired defense mechanisms
Greater risk for sepsis (bacterial infection of the blood)