root resorption Flashcards

1
Q

definition

A

the non-bacterial destruction of the dental hard and soft tissue due to the interaction of clastic cells

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2
Q

stimulation of osteoclast activity

A

signals can up/down regulate osteoclast activity

  • RANKL promotes development so upregulates
  • OPG inhibits RANKL and so the development so down regulates
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3
Q

RANKL stimulation

A

parathyroid hormone, B3 and IL-1B
bacterial lipopolysaccharides
trauma (physical, chemical)
chronic inflammation

  • apart from trauma can be hard to identify cause
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4
Q

the cell

A

multinucleate giant cells
e.g. osteoclast - when attached to dentine sometimes called dentinoclast as attacking dentine
highly specific and effective
key features
- v motile
- ruffled border (large SA so can resorb v quickly)
- in contact with dentine

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5
Q

aspects of OC/dentinoclast

A
sit in Howships lacunae
intracellular vesicles
release proteolytic enzymes
ruffled border
integrins to attach
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6
Q

surfaces which act to prevent resorption

A

PDL (external)
- e.g. trauma - PDL necrosis = intimate contact between OCs and dentine

cementum (external)
- particularly the non-mineralised layer

predentine (internal)
- non-collagenous component, closest to pulp

but when damaged the resorption process can begin

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7
Q

types of internal resorption

A

inflammatory

replacement

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8
Q

types of external resorption

A

inflammatory
replacement
cervical
surface

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9
Q

EO exam - smile line

A

if there is a consideration for endo microsurgery

- post-surgical recession or scarring (aesthetics)

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10
Q

IO exam - tooth in question

A
coronal integrity of remaining tooth and Rx quality
colour
PD pocketing
sinus inc location in relation to mucogingival jct
swelling
apical tenderness
TTP
mobility
occlusal contact in ICP and guidance
integrity of adjacent teeth
sensitivity test
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11
Q

IO exam - coronal integrity of remaining tooth and Rx quality

A

can tooth be predictably restored after tx?

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12
Q

IO exam - colour

A

pink spot

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13
Q

IO exam - PD pocketing

A

with a PCP12 probe both horizontally and vertically (BPE not small enough)
- is there a PD communication with the resorption?

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14
Q

IO exam - sinus inc location in relation to mucogingival jct

A

has the internal resorption perforated the RC?

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15
Q

IO exam - swelling

A

associated with periradicular disease

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16
Q

IO exam - apical tenderness

A

associated with PR disease

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17
Q

IO exam - TTP

A

a test of the PDL not necessarily PR disease

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18
Q

IO exam - mobility

A

no physiological mobility and high pitched percussion

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19
Q

IO exam - occlusal contact in ICP and guidance

A

is the tooth in fct and prudent to retain?

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20
Q

IO exam - integrity of adjacent teeth

A

alternative replacement options e.g. bridge

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21
Q

IO exam - sensitivity test

A

pulp response

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22
Q

radiographic examination

A

absolute minimum is an up to date PA subsequent to any tx
- need 2 from different angles for parallax (SLOB)
- 30 degree mesial or distal beam shift
CBCT (not always needed)

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23
Q

radiographic appearance - internal resorption

A

ballooning out - parallel lines of RC disappear

parallax - doesn’t move - stays centred in canal

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24
Q

radiographic appearance - external resorption

A

can just make out parallel lines of RC system - superimposed on resorption
parallax - appears to change position

25
internal inflammatory resorption - prevalence
<1%
26
internal inflammatory resorption - vague clinical findings
can be unrestored no PD pocketing unless lesion has perforated root surface colour and mobility normal no sinus unless PR disease sensitivity testing positive = mostly found incidentally on radiograph
27
internal inflammatory resorption - pathogenesis
coronal pulp is necrotic lesion includes inflammatory and vascular tissue - if perforated will communicate with PDL apical pulp is vital (need vital pulp to allow it to progress and keep nibbling) lesion will continue to progress until apical pulp goes completely necrotic - until PA radiolucency - indicates complete necrosis - RR likely stopped by this point can happen in any part of root - in apical region a consequence can be root perforation
28
internal inflammatory resorption - likely cause
trauma
29
internal inflammatory resorption - tx
orthograde endo possible haemorrhage active irrigation and disinfection, internist medicament (disperse and agitate CaOH e.g. endoactivator) - won't be able to mechanically clear all the D of OCs can be hard to find apical canal beyond lesion thermal obturation e.g. warm vertical compaction
30
internal replacement resorption - diagnosis
no clinical indications | incidental finding radiographically - trabecular pattern in pulp - ossified
31
internal replacement resorption - tx
``` RCT nearly impossible: - ossified - thin root dentine in walls - perforation risk if can't RCT often let it run course then when symptomatic look at replacement options ```
32
external surface resorption - clinical features
can be unrestored increased physiological mobility + to sensibility tests
33
external surface resorption - radiographic features
PDL intact | no obvious periapical radiolucency
34
external surface resorption - aetiology
orthodontics - 90% of teeth have some form of ESR - 2-5% severe ESR - 15% moderate - usually teeth for anchorage are the worst affected ectopic teeth - pressure from erupting tooth pathological lesions - pressure from adjacent pathological lesion e.g. OK idiopathic
35
external surface resorption - tx
pulp healthy - DON'T RCT remove the source to stop the resorption splint if mobile
36
external inflammatory resorption - clinical findings
usually restored no PD pocketing, colour normal may have sinus, swelling, apical tenderness, TTP mobility may be increased depending on extent sensitivity negative - pulp is necrotic
37
external inflammatory resorption - radiographic findings
POLL | periapical radiolucency
38
external inflammatory resorption - aetiology
pulp is necrotic - bacterial or dental trauma in origin periapical inflammatory lesion precipitates the resorption process majority (81%) of teeth with periapical lesions will have microscopic areas of RR - only 7% of these are detectable radiographically
39
external inflammatory resorption - tx
remove the cause of the inflammation usually orthograde endo (re) tx, possibly surgical endo, or extraction if apex significantly resorbed may not be able to control GP - need to alter technique arrests RR only external RR that benefits from endo
40
classification: Heithersay and Patel
``` class 1 class 2 class 3 class 4 ``` ``` 3D imaging apico-coronal direction 1 crestal 2 coronal 1/3 3 middle 1/3 4 apical 1/3 circumferential 1 1/4 2 1/2 3 3/4 4 >3/4 ```
41
external cervical resorption - clinical findings
can be unrestored PD pocketing: yes if extensive and profuse BOP - probe may drop into a hard cavity when you probe margin - can misdiagnose as caries - but will be hard to probe pink spot - lesion has good blood supply - don't always see e.g. if small or in particular position may not see it until get to a critical stage normal or no mobility positive to sensitivity testing = won't always have these S and S
42
external cervical resorption - radiographic findings
radiolucency at level of CEJ can still see parallel RC lines, changes in position with parallax CBCT can show apical coronal spread
43
external cervical resorption - aetiology
``` orthodontics trauma - avulsion and luxation - don't traumatise CEJ with forceps when repositioning traumatised tooth historical non-vital whitening when heat was applied - vvv rare with modern bleaching wind instruments viral infection (from cats) systemic disturbance (thyroid) ```
44
external cervical resorption - pathogenesis
portal of entry bone-like tissue PRRS: predentine, also called Pericanalar Resorption Resistant Sheet (PRRSP) protects the pulp
45
external cervical resorption - tx options
``` monitor - the resorption will v likely continue - significant number are progressive extraction and prosthetic replacement surgical repair leave root in situ - preserve ST and bone surgical repair and orthograde endo internal repair and orthograde endo - if can't access surgically ``` need clean and frosted dentine otherwise RR will continue - keep going with chemical disinfection
46
external cervical resorption - tx options - surgical repair
flap to expose NaOCl - coagulation necrosis can't get rid of all lacunae with handpiece as can't see them and would remove too much tooth
47
external cervical resorption - tx options - surgical repair and orthograde endo
1/3 pulp exposed/after pulp becomes necrotic | do endo first to avoid periapical radiolucency
48
external cervical/replacement resorption - healing?
``` v rarely does it cease and allow healing lesion has a trabecular pattern bone rather than ST the PDL is present no tx ```
49
external replacement resorption - clinical findings
``` can be unrestored but infraoccluded - also gingival margin level no PD pocketing, possibly erythematous not TTP but high-pitched note no physiological mobility - ankylosed positive to sensitivity tests ```
50
external replacement resorption - radiographic findings
loss of obvious PDL cotton wool appearance root surface replaced by trabecular pattern - bone affects a lot of root surface
51
external replacement resorption - aetiology
trauma - significant injuries to there periodontium such that bone (OCs) are then in contact with external root dentine to begin resorption e. g. avulsion or lateral luxation
52
external replacement resorption - tx options
decoronation | monitor
53
external replacement resorption - tx options - decoronation
if infra occlusion is >1mm in a growing pt remove crown to alveolar level and allow root to resorb - root replaced by bone this preserves bone volume adjacent teeth and periodontium develop normally tooth replacement with denture or RBB
54
external replacement resorption - who do you need to intervene early in a growing pt?
alveolus growing around it - need height of bone for implant - also adjacent teeth tip into prosthetic space - MD and vertical problem
55
external replacement resorption - tx options - monitor
if pt has stoped growing endo will not stop resorption - no RCT because of infra occlusion can add composite incisally
56
primary tx strategies for resorption - orthograde endo
external inflammatory | internal inflammatory
57
primary tx strategies for resorption - surgical endo
external cervical
58
primary tx strategies for resorption - no endo
external surface | external replacement