emergencies Flashcards

1
Q

symptoms that might lead pt to present

A
PAIN
swelling
pus
bleeding
mobility
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2
Q

abscesses

A

periapical
gingival
periodontal
pericoronal

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3
Q

differential diagnoses - irreversible pulpitis

A

poorly localised
constant, dull throbbing
unlikely to see anything radiographically - pulp not necrosed, no bone destruction

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4
Q

differential diagnoses - reversible pulpitis

A

pain triggered by stimulus - hot/cold, not constant - resolves
as it gets more established triggered by sweet
unlikely to see anything radiographically - pulp not necrosed, no bone destruction
heightened response to sensibility testing

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5
Q

differential diagnoses - periapical periodontitis

A

TTP
no response to sensibility testing (if caused by pulp necrosis extension)
pain to biting
might see bone loss radiographically (takes 3m to show)

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6
Q

differential diagnoses - pericoronal abscess

A

PE tooth, usually L8s

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7
Q

PD abscess

A

a localised accumulation of pus within the gingival wall of a PD pocket resulting in the destruction of the collagen fibre attachment and the loss of nearby alveolar bone

active period of PD breakdown which occurs whilst there is marginal closure of the deep PD pocket occluding drainage

non-draining infection of a PD pocket

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8
Q

PD abscess S + S

A
\+ to sensibility testing
mobility
likely to see lat lesion on radiograph (but may not see anything)
pain and tenderness
swelling of gingiva
bleeding
deep PD pocket
may have suppuration
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9
Q

PD abscess - what to do if tooth is unrestorable

A

extract

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10
Q

SDCEP tx of a PD abscess

A

subgingival instrumentation short of base of pocket to avoid iatrogenic damage, LA?
if pus present - drain by incision or through the PD pocket
recommend optimal analgesia
don’t prescribe ABs unless signs of spreading infection or systemic involvement
0.2% CHX MW until acute symptoms subside
review within 10 days and carry out definitive PD instrumentation and recall interval

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11
Q

PD abscesses in non-periodontitis pts

A

impaction of foreign bodies
harmful habits
alteration of root surface e.g. perforation, root fracture etc

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12
Q

what is ANUG now called?

A

necrotising gingivitis
necrotising periodontitis
- (chronically/severely compromised or temporarily/moderately compromised)
necrotising stomatitis

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13
Q

NG presentation

A
acute onset of severe pain
marginal gingival ulceration and craters
loss of ID papilla
slough
rapidly destructive
necrosis locally
may have local lymphadenopathy/systemically unwell
halitosis
bleeding
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14
Q

if NG not treated and controlled rapidly?

A

may leave significant gingival recession

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15
Q

NG microbiology

A

anaerobic bacteria with fusiform and spirochetes in a compromised host

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16
Q

NG risk factors

A
neglected mouth - poor OH
immunocompromised
smoking
stress
malnutrition
lack of sleep
prev NP
younger pts
immunosuppression
17
Q

NG tx SDCEP

A

debridement - LA, US
recommend use of 0.2% CHX MW until acute symptoms subside
review within 10days and do definitive instrumentation as required, recall interval
address risk factors - OH, smoking etc
if no resolution, review general health and consider referral to specialist
ABs if spreading infection/systemic involvement
- metronidazole 200/400mg x3 daily for 3 days

18
Q

secondary herpes

A

often reactivated post-PD surgery esp palatally
often not painful
can be confused clinically with other bullous conditions and if uncertain refer to oral med
symptomatic tx, should resolve 10 days, CHX MW
if doesn’t resolve/recurrent refer to oral med

19
Q

chemical trauma e.g. from etch/bleaching agent

A

acute onset, painful
manage conservatively and prevent infection and tissue loss if possible
irrigate lots
CHX MW and avoid brushing site until it heals
may leave residual gingival recession defect

20
Q

primary herpetic gingivostomatitis spread

A

highly infectious, spread through contaminated saliva

caused by HSV-1, occ HSV-2 in adults

21
Q

primary herpetic gingivostomatitis S + S

A

acute onset usually but not always children
+/- systemic (potentially serious if present) - dehydration/encephalitis
painful ulceration preceded by fragile vesicles which are only rarely visible
affects gingival tissue, tongue, lips, cheeks and roof of mouth

22
Q

primary herpetic gingivostomatitis tx

A

usually managed symptomatically as self-limiting over 5-7 days, attention to fluid intake
consider acciclovir: shortens duration of symptoms, more rapid healing and helps to establish eating/drinking again earlier

23
Q

primary herpetic gingivostomatitis long term effect

A

may result in recurrent lesions at other sites, around 40% of individuals

24
Q

primary herpetic gingivostomatitis SDCEP tx

A
symptomatic relief: local measures
 - avoid dehydration
 - soft diet
 - analgesics
 - antimicrobial MW
if immunocompromised/severe infection 
 - acciclovir 200mg x5 daily for 5 days
25
acute leukaemia
rare presenting as severe hyperplastic gingivitis hyperplastic tissue is full of leukaemic infiltrate acute or slower onset over a few weeks likely systemic S+S to aid diagnosis - bone pain - fever - freq infections - freq or severe nosebleeds - lymphadenopathy - pallor - SOB can also see ulceration of inner surface of lip due to acute neutropenic changes urgent referral via GMP as soon as potential diagnosis is considered
26
gingival abscess
a localised purulent infection that involves the marginal gingiva or ID papillae
27
gingival abscess S + S
localised swelling may have suppuration usually painful and TTP
28
what is a gingival abscess often due to?
subgingivally impacted foreign objects, tend to occur in a prev healthy gingiva e.g. piece of nail due to a nail biting habit
29
gingival abscess management
incise, drain and irrigate may need mechanical debridement to mitigate aetiology 0.2% CHX MW/saline rinse review
30
SDCEP periodontitis associated with endo lesions
clinical AL and also a tooth with necrotic/partially necrotic pulp
31
perio endo lesions S+S
``` swelling suppuration TTP may have generalised PDD with localised pain deep pocketing to root apex with BOP negative response to sensibility tests ```
32
SDCEP perio endo lesion tx
endo tx of tooth (get drainage?) recommend optimal analgesia no ABs unless signs of spreading infection or systemic involvement 0.2% CHX MW until acute symptoms subside review within 10 days and PD instrumentation, recall
33
perio endo lesion
communication between PD lesion and pulp deep pocket around a non-vital tooth the infection may arise primarily from pulpal inflammatory disease expressed through the PDL or the alveolar bone to the oral cavity, or it may be initiated from a PD pocket communicating to the pulp apically or through accessory canals
34
prognosis of primary endo lesions
often respond well to RCT alone
35
prognosis of primary perio/truly combined lesions
often poor
36
prognosis of endo perio lesions associated with trauma and iatrogenic factors e.g. perforation
usually poor as affect tooth structure