root caries Flashcards

1
Q

What is caries?

A

Reversible but progressive disease on dental hard tissues

Instigated by action of bacteria upon fermentable carbohydrates in the plaque biofilm on tooth surfaces over time

Leading to bacterially generated acid demineralisation and ultimately proteolytic destruction of the organic component of tissues

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2
Q

What bacteria works mostly on root caries?

A

Actinomyces spp (gram positive anaerobes)- due to proteolytic affect

Also, mutans streptococci and lactinobacilli

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3
Q

What are pathological factors?

A

Acid-producing bacteria
Fermentable carbs frequency
Saliva flow/function abnormal

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4
Q

What are protective factors?

A

Saliva
Fluoride
Antibacterials (chlorhexidine, xylitol etc)

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5
Q

Why is saliva important?

A

Mechanical flushing (99% water)
Calcium and phosphate ions- lowers pH
Critical pH- 5.5
Antibacterial effects- fluoride, hydrogen peroxide, thiocyanate, IgA, lysozyme, PRP)
Buffering systems (bicarbonate, phosphate, protein)

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6
Q

What is the epidemiology of root caries?

A

7% of adults

20%- 75-84yrs

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7
Q

What are the risk factors for root caries?

A
Polypharmacy (dry mouth/contain sugar)
Oral hygiene
Institutionalisation
Movement disorders
Diminished cognitive skills
More exposed root surfaces
Drug addiction 
Hyposalivation
Systemic diseases
Decreased saliva quality
Diet (soft and short meals high in carbs)
Poor manual dexterity
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8
Q

Why might someone have hyposalivation?

A
Radiotherapy
Medications
Autoimmune disorders (sjögrens disease)
Smoking
Idiopathic
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9
Q

What is the histopathology?

A

Demineralisation- soft consistency, not always change in colour
Cementum/dentine
Darkening- extrinsic staining into softened more porous dentine
Intact collagen- reversible
Continuing demin- collagen broken down
Necrotic dentine- needs removal

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10
Q

How does the caries spread?

A

Along cementoenamel junction without being too deep
-encircles

Advanced lesions progress to pulp

Can spread coronally to enamel and cause delamination

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11
Q

How can you tell a lesions activity?

A

Active- lighter/matte, softer/rougher, gingival inflam, plaque build up, inaccessible

Inactive/arrested- dark and shiny, harder, clean

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12
Q

How can non operative caries control be undertaken?

A

OHI- modify handles? Carer education
Plaque control- remove plaque retentive factors
Diet advice
Fluoride
Casein phosphopeptide-amorphous calcium phosphate (CPP-ACP)
Antimicrobial agents

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13
Q

How can hyposalivation be managed?

A
Potentially manage cause
OHI
High fluoride toothbrush, mouthwash, varnish
Diet sheet- limit sugar
Sip water freq
Chewing gum- xylitol
Saliva substitute (Biotene)
Extreme caries risk- 2-3 months recall
Salivary stimulant med- pilocarpine
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14
Q

What are the aims of operative management of carious lesions?

A
  1. Aid plaque control and manage caries activity at location
  2. Protect pulp-dentine complex and arrest lesion by sealing
  3. Restore function, form and aesthetics
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15
Q

What do retraction cord?

A

Packed into gingival sulcus to retract gingivae to improve visibility, access and moisture control

-rubber dam not always achievable

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16
Q

How should the caries be managed?

A

Shouldn’t need high speed unless enamel

Rotary instruments
Hand instruments (excavators, hatchets, gingival margin trimmers, chisels)
Air abrasion (aluminium oxide, bio glasses)
Chemo-mechanical methods (Carisolv, Papacarie)
17
Q

What restorative material should be used?

A

Enamel and sound dentine- composite

Caries-affected and sclerotic dentine/cementum- GIC

18
Q

Why would you use GIC?

A

Chemical bond to tooth tissue
Suboptimal isolation (not pools of saliva)
Lesion at gingival margin
Substrate to bond- mainly dentine (w questionable quality)
Lower aesthetic concerns

Little finishing as can dessicate

19
Q

Why should you use composite?

A
Micromechanical bond
Optimal isolation
Lesion above gingival margin
Some enamel to bond/good quality dentine
Higher aesthetic concern