Rheumatoid and Osteoarthritis Flashcards

1
Q

Define osteoarthritis

A
  • Slow progressive non-inflammatory disorder of the diarthrodial joints
  • Not considered a normal part of aging process- but age does influence development
  • Cartilage destruction can begin between ages 20 and 30
  • Symptoms common at ages 50-60
  • “my hips really bad, I need a hip replacement”
  • Can be inflamed at some point
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2
Q

Etiology and Pathophysiology of osteoarthritis

A
  • Single cause for OA has not been identified: has to do with cartilage and cartilage getting damaged
  • OA results from cartilage damage that triggers a metabolic response at level of chondrocytes
  • Cartilage becomes: thick, yellow, and granular; soft and less elastic; less able to resist wear with heavy use
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3
Q

Discuss the osteoarthritis

A
  • Wide space between bones in first picture
  • Space between bones is less in second picture
  • Narrow space between bones, causing pain at this point in 3rd picture, described as bone on bone, hurting even when laying in bed, hurting all the time
  • Steroid injections help
  • Being overweight would put more strain and pressure on the joints
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4
Q

Clinical Manifestations of Osteoarthritis

A

Joint pain:
- Predominant symptom ranging from mild discomfort to significant disability
- Pain worsens with joint use
- Early stages: rest relieves pain
- Later stages: pain with rest and sleep is disturbed because of pain and increased joint discomfort
- Localized
- Be careful throughout the day, do a bit of work then rest the joint, no strenuous work all day
- As cartilage wears more and more, it becomes more painful
- Osteo: localized, in the one joint, other may be impacted but not the exact same, CRP and ESR would be normal levels since they are localized
- Rheumatoid: systemic
- Joint stiffness occurs after periods of rest or statis position
- Early morning stiffness usually resolves within 30 minutes
- Overactivity can cause mild joint effusion (swollen, fluid build up); temporarily increases stiffness

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5
Q

Define Herberden’s and Bouchard’s

A
  • These nodes are associated with OA- not to be confused with rheumatoid nodules or tophi from gout
  • Build up at the joints, almost like scar tissue building up
  • Distal: Heberden’s
  • Middle: bouchard’s
  • Hands in alignment
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6
Q

Define Rheumatoid Arthritis

A
  • Chronic, systemic autoimmune disease
  • Inflammation of connective tissue in diarthrodial (synovial) joints
  • Periods of remission and exacerbation
  • Frequently accompanied by extra-articular manifestations
  • Occurs globally, affecting all ethnic groups—more women than men
  • Something triggered the body to go into immunity overdrive
  • Attacking own body, weakened immune system or overdrive
  • Inflammation in both hands, going to be symmetrical
  • Period of remission, symptoms go away, and exacerbation, symptoms flare up
  • Extra articular manifestations: symptoms outside of the joints because it is attacking the whole body
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7
Q

Etiology and Pathophysiology of RA

A
  • Cause of RA is unknown
  • Two possibilities currently: autoimmune response, genetic factor; patient reports precipitated by an event - infection and stressors (cascade of destruction ensues as body mounts attack)
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8
Q

Discuss the RA

A
  • Synovium is inflamed with oste
  • B. destruction of cartilage, loses stability, causes more swelling, destruction of tissue
  • C. begins to erode the bone and attack, causes more swelling
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9
Q

Extra Articular Manifestations of RA

A
  • keratoconjunctivitis: dry eyes
  • Swollen lymph glands
  • Myositis: sore muscles
  • Raynaud’s syndrome: impaired blood flow in cold weather, immediately turns blue
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10
Q

Complications of RA

A
  • Joint destruction begins as early as first year of disease without treatment
  • Flexion contractures and hand deformities
    • Cause diminished grasp strength
    • Affect patient’s ability to perform self-care tasks
  • Want to start them on medication to prevent destruction of joints by suppressing immune system in some way
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11
Q

Clinical Manifestations of RA

A
  • Onset is typically insidious
  • General stiffness may precede onset of joint symptoms
  • Symptoms occur symmetrically
  • Patient experiences joint stiffness after periods of inactivity
  • Morning stiffness may last from 60 minutes to several hours or longer
  • Increased pain when moving
  • Joints become tender, painful, and warm
  • Joint pain ↑ with motion
    • Varies in intensity
    • May not be proportional to degree of inflammation
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12
Q

Nursing Assessment

A
  • Full hx
  • Physical
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13
Q

Nursing Diagnoses

A
  • chronic pain
  • impaired physical mobility
  • disturbed body image
  • ineffective self-health management
  • self-care deficit
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14
Q

Nursing Goals for RA and OA

A
  • Reduction of inflammation
  • Management of pain
  • Maintenance of joint function
  • Perform self-care
  • Prevention or minimization of joint deformity
  • Maintain a positive self-image
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15
Q

Nursing Interventions for OA and RA

A
  • Rest and Joint Protection
    • Patient must understand importance of balancing rest and activity
    • Maintain good body alignment during rest
      • Encourage positions of extension, avoid positions of flexion
  • Modify activities to put less stress on joints
  • During periods of acute inflammation, affected joint should be:
    • Rested
    • Maintained in a functional position (with splints or braces if necessary)
    • Immobilization time should be as limited as possible
  • Heat and Cold Applications:
    • May help reduce pain and stiffness
    • Heat is used more often than ice
      • Ice appropriate for acute inflammation
    • Heat therapy is especially helpful for stiffness to loosen muscles.
  • Hot packs, whirlpool baths, ultrasound
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16
Q

Nutrition and Exercises for OA and RA

A
  • Balanced Nutrition
  • Nutritional Therapy: No diet will cure RA-avoid processed foods and chemical preservatives, Red meat and dairy, refined sugars
  • Weight-reduction program is critical for overweight patient
  • Exercise is a fundamental part of OA management to tolerance
17
Q

Treatment for RA

A
  • Drugs remain cornerstone of treatment for RA.
  • Disease-modifying antirheumatic drugs (DMARDs) can lessen permanent effects of RA (methotrexate)
  • Corticosteroids
  • NSAIDS
  • Care begins with a comprehensive program of education and drug therapy
  • Chemo attacks the cells that are breaking down the body
  • Not all chemos cause you to lose your hair
  • Want to slow down the immune system, that’s what methotrexate and corticosteroids do
18
Q

Health Educations

A
  • Weight control-both
  • Smoking cessation-RA
  • Limit exposure to environmental pollutants-RA
  • Stress reduction-RA
  • Exercise-strengthening-OA
  • Avoid injury and get injuries treated-OA
  • Omega 3/fish oil and Vitamin D-OA
19
Q

Evaluating Outcomes

A
  • Reduction of inflammation
  • Management of pain
  • Maintenance of joint function
  • Perform self-care
  • Prevention or minimization of joint deformity
  • Maintain a positive self-image
20
Q

Comparison of the two

A