Arterial Ulcers vs. Venous Ulcers of the LE Flashcards

1
Q

Describe peripheral arterial disease

A
  • Involves progressive narrowing and degeneration of arteries of upper and lower extremities
  • Atherosclerosis is the leading cause in majority of cases
  • PAD is strongly related to other types of cardiovascular disease (CVD) and their risk factors.
  • Patients with PAD have a significantly higher risk of mortality (in general), CVD mortality, and major coronary events.
  • Thus PAD is a marker of advanced systemic atherosclerosis.
  • Arteries transport oxygenated blood but may develop atherosclerosis (narrowing of arteries due to build up of plaque) leading cells become ischemic because there is no oxygen
  • Cold temp causes them to become pale, blue
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2
Q

Arterial Clinical Manifestations of PAD

A
  • Paresthesia: Numbness or tingling in the toes or feet; Produces loss of pressure and deep pain sensations; Injuries often go unnoticed by patient
  • Thin, shiny, and taut skin
  • Loss of hair on the lower legs
  • Diminished or absent pedal, popliteal, or femoral pulses
  • Intermittent claudication
  • Pallor of foot with leg elevation: (blanching of the foot) develops in response to leg elevation (elevation pallor).
  • Reactive hyperemia of foot with dependent position
  • Rest pain
  • Conversely, reactive hyperemia (redness of the foot) develops when the limb is in a dependent position (dependent rubor).
  • Tell diabetics to check feet once a day because they can lose their sensation
  • Taut – tight skin
    Intermittent claudication: not enough oxygen reaching muscles; pain in calf induced by exercise that is relieved by rest, when increasing oxygen demand; really bad when client is always in pain
  • Arterial = Allow extremities to go down
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3
Q

Recognizing clinical manifestations

A
  • Tips of toes, foot, or lateral malleolus
  • Rounded, smooth, looks “punched out”
  • Minimal drainage
  • Black eschar or pale pink granulation
  • Arterial is black, dead tissue because oxygenated blood isn’t reaching the area
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4
Q

Complications of PAD ulcers

A
  • Atrophy of the skin and underlying muscles
  • Delayed healing
  • Wound infection
  • Non-healing arterial ulcers and gangrene are most serious complications
  • Tissue necrosis
  • Amputation: If adequate blood flow is not restored; If severe infection occurs
  • Non healing ulcers that can become gangrene (infection) or necrosis
  • More ampt to have amputations since everything dies off; amputate limb or live with infection that can spread
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5
Q

PAD ulcer nursing diagnoses

A
  • Ineffective tissue perfusion (peripheral)
  • Impaired skin integrity
  • Activity intolerance
  • Ineffective self-health management
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6
Q

PAD ulcer collaborative care

A
  • Cessation of risk factor habits: Diet modification, smoking, weight management, hygiene
  • Medication Management: BP control, BG regulation, coagulation, cardiovascular pulmonary support and care
  • Surgical/Radiographic interventions: debridement, angioplasty, bypass
  • Diagnostic needs: monitoring labs, ultrasound (US), cultures, ankle-brachial index (ABI)
  • Surgery is indicated in patients with long areas of stenosis or severely calcified arteries.
  • Be empathetic with clients
  • Synthetic grafts typically are used for long bypasses, such as an axillary-femoral bypass. When a person’s own vein is not available, human umbilical vein or a composite sequential bypass graft (native vein plus synthetic graft) is an alternative.
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7
Q

Describe venous insufficiency

A
  • veins and valves fail to move; calf muscle pump; incompetent valves
  • venous HTN, varicosities venous thromboembolism (VTE)
  • painful/debilitating ulcers medial
  • Calves fail to move blood up towards head
  • Have problems with veins getting blood back to the body
  • More at risk with venous problems because of blood clots
  • More painful and debilitating than ulcers since you have a lot of pressure and fluid that is collecting
  • Ulcers tend to be on medial malleolus - venous
  • Lateral malleolus – arterial
  • Likely related to where vessels are
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8
Q

Venous insufficiency clinical manifestations

A
  • Edema present, unilateral or bilateral
  • Hemosiderin staining: life of a RBC is done so it releases iron causing the staining to ooze to the skin
  • Thick hardened skin “leathery” appearance
  • Wounds “weep”
  • Skin becomes friable
  • Near medial malleolus
  • Irregularly shaped
  • Moderate to large amount of drainage
  • Yellow slough or dark red “ruddy” granulation
    Venous = eleVate
    Arterial = Allow extremities to go down
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9
Q

Complications of Venous insuffiency

A
  • Pain worse when dependent
  • Non-treatment leads to increase in wound size
  • Acute leads to chronic if continued treatment not adhered to
  • Wound(s) reopening in same location common
  • Rarity of amputation
  • Putting legs above level of heart is helpful because youre helping blood go back to the heart
  • V goes up, legs go up, venous
  • A goes down, arterial problem, legs go down
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10
Q

Venous insufficiency nursing diagnoses

A
  • acute pain
  • ineffective health maintenance
  • risk of impaired skin integrity
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11
Q

Venous insufficiency collab care

A
  • Compression (Thromboembolic deterrent (TED) hose)
  • Moist environment dressings
  • High protein diet
  • Elevation
  • Hyperbaric support: angiogenesis; Hyperbaric pushes oxygen under pressure to put oxygen into body and directed into wound to help healing
  • Underlying medical management support
  • Drug Therapy: anticoagulants
  • Cessation of risk factors (as with PAD)
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