Arterial Ulcers vs. Venous Ulcers of the LE Flashcards
1
Q
Describe peripheral arterial disease
A
- Involves progressive narrowing and degeneration of arteries of upper and lower extremities
- Atherosclerosis is the leading cause in majority of cases
- PAD is strongly related to other types of cardiovascular disease (CVD) and their risk factors.
- Patients with PAD have a significantly higher risk of mortality (in general), CVD mortality, and major coronary events.
- Thus PAD is a marker of advanced systemic atherosclerosis.
- Arteries transport oxygenated blood but may develop atherosclerosis (narrowing of arteries due to build up of plaque) leading cells become ischemic because there is no oxygen
- Cold temp causes them to become pale, blue
2
Q
Arterial Clinical Manifestations of PAD
A
- Paresthesia: Numbness or tingling in the toes or feet; Produces loss of pressure and deep pain sensations; Injuries often go unnoticed by patient
- Thin, shiny, and taut skin
- Loss of hair on the lower legs
- Diminished or absent pedal, popliteal, or femoral pulses
- Intermittent claudication
- Pallor of foot with leg elevation: (blanching of the foot) develops in response to leg elevation (elevation pallor).
- Reactive hyperemia of foot with dependent position
- Rest pain
- Conversely, reactive hyperemia (redness of the foot) develops when the limb is in a dependent position (dependent rubor).
- Tell diabetics to check feet once a day because they can lose their sensation
- Taut – tight skin
Intermittent claudication: not enough oxygen reaching muscles; pain in calf induced by exercise that is relieved by rest, when increasing oxygen demand; really bad when client is always in pain - Arterial = Allow extremities to go down
3
Q
Recognizing clinical manifestations
A
- Tips of toes, foot, or lateral malleolus
- Rounded, smooth, looks “punched out”
- Minimal drainage
- Black eschar or pale pink granulation
- Arterial is black, dead tissue because oxygenated blood isn’t reaching the area
4
Q
Complications of PAD ulcers
A
- Atrophy of the skin and underlying muscles
- Delayed healing
- Wound infection
- Non-healing arterial ulcers and gangrene are most serious complications
- Tissue necrosis
- Amputation: If adequate blood flow is not restored; If severe infection occurs
- Non healing ulcers that can become gangrene (infection) or necrosis
- More ampt to have amputations since everything dies off; amputate limb or live with infection that can spread
5
Q
PAD ulcer nursing diagnoses
A
- Ineffective tissue perfusion (peripheral)
- Impaired skin integrity
- Activity intolerance
- Ineffective self-health management
6
Q
PAD ulcer collaborative care
A
- Cessation of risk factor habits: Diet modification, smoking, weight management, hygiene
- Medication Management: BP control, BG regulation, coagulation, cardiovascular pulmonary support and care
- Surgical/Radiographic interventions: debridement, angioplasty, bypass
- Diagnostic needs: monitoring labs, ultrasound (US), cultures, ankle-brachial index (ABI)
- Surgery is indicated in patients with long areas of stenosis or severely calcified arteries.
- Be empathetic with clients
- Synthetic grafts typically are used for long bypasses, such as an axillary-femoral bypass. When a person’s own vein is not available, human umbilical vein or a composite sequential bypass graft (native vein plus synthetic graft) is an alternative.
7
Q
Describe venous insufficiency
A
- veins and valves fail to move; calf muscle pump; incompetent valves
- venous HTN, varicosities venous thromboembolism (VTE)
- painful/debilitating ulcers medial
- Calves fail to move blood up towards head
- Have problems with veins getting blood back to the body
- More at risk with venous problems because of blood clots
- More painful and debilitating than ulcers since you have a lot of pressure and fluid that is collecting
- Ulcers tend to be on medial malleolus - venous
- Lateral malleolus – arterial
- Likely related to where vessels are
8
Q
Venous insufficiency clinical manifestations
A
- Edema present, unilateral or bilateral
- Hemosiderin staining: life of a RBC is done so it releases iron causing the staining to ooze to the skin
- Thick hardened skin “leathery” appearance
- Wounds “weep”
- Skin becomes friable
- Near medial malleolus
- Irregularly shaped
- Moderate to large amount of drainage
- Yellow slough or dark red “ruddy” granulation
Venous = eleVate
Arterial = Allow extremities to go down
9
Q
Complications of Venous insuffiency
A
- Pain worse when dependent
- Non-treatment leads to increase in wound size
- Acute leads to chronic if continued treatment not adhered to
- Wound(s) reopening in same location common
- Rarity of amputation
- Putting legs above level of heart is helpful because youre helping blood go back to the heart
- V goes up, legs go up, venous
- A goes down, arterial problem, legs go down
10
Q
Venous insufficiency nursing diagnoses
A
- acute pain
- ineffective health maintenance
- risk of impaired skin integrity
11
Q
Venous insufficiency collab care
A
- Compression (Thromboembolic deterrent (TED) hose)
- Moist environment dressings
- High protein diet
- Elevation
- Hyperbaric support: angiogenesis; Hyperbaric pushes oxygen under pressure to put oxygen into body and directed into wound to help healing
- Underlying medical management support
- Drug Therapy: anticoagulants
- Cessation of risk factors (as with PAD)