Respiratory Surgery Flashcards

1
Q

Laryngeal collapse: Cx

A

Stertor (expiratory); Stridor (Inspiratory); Exercise intolerance; Coughing; Regurg and vomiting

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2
Q

Tracheal obstruction: Cx

A

Cough; Dyspnea; Cyanosis; Collapse

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3
Q

Tracheal obstruction: Tx

A

Resection and anastomosis; Tracheostomy

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4
Q

What is the main surgical treatment for lung lobe consolidation, bronchiectasis; lung lobe torsion or pulmonary neoplasia?

A

Partial or complete lung lobectomies

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5
Q

What are the four main components of BOAS?

A

Stenotic nares; Elongated, thickened soft palate; Everted laryngeal saccules; Hypoplastic trachea; (also can have abherent turbinates; lorg tongue; tracheal collapse)

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6
Q

List at least 3 clinical signs of BOAS

A

Stertor; Obstructive sleep apnea; Heat intolerance; Cyanosis and collapse; GI Signs (difficult swallowing, regurg, reflux, hiatal hernia)

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7
Q

How do we diagnose BOAS?

A

Typical way is to evaluate the dog’s breathing at rest and exercise (walk), and do a sedated oral and laryngeal exam, based on these parameters we can assign a severity score; CT?radiology can be nice for issues like tracheal/bronchial hypoplasia, collapse, hiatal hernias, aspiration pneumonia

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8
Q

Medical management of BOAS

A

Weight management; Avoid overheating

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9
Q

List 5 surgical procedures we can do to manage BOAS

A

Rhinoplasty; Endoscope-guided turbinectomy; Shorten soft palate (e.g. Staphylectomy); Partial tonsillectomy; Laryngeal sacculectomy

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10
Q

Prognosis of BOAS

A

The more severe, the worse the prognosis

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11
Q

What does post-operative management look like for dogs with BOAS?

A

Susceptible to post-operative airway inflammation or pneumonia so address w/ supplemental oxygen, anti-inflammatories and possible a tracheostomy

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12
Q

Pathomechanism of Laryngeal Paralysis (LarPar)

A

Idiopathic disorder typically in older large breed dogs (e.g. Labs and Goldens) where there is damage to the recurrent laryngeal nerve/caudal laryngeal nerve leading to failure of the cricoarytenoideus dorsalis muscle to abduct the arytenoids. Typically bilateral

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13
Q

GOLPP

A

Geriatric Onset Laryngeal Paralysis and Polyneuropathy

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14
Q

Three components of GOLPP

A

Laryngeal paralysis; Radial nerve dysfunction; Tibial nerve dysfunction

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15
Q

Clinical signs of Laryngeal Paralysis

A

Change in phonation; Gagging; Exercise intolerance; Laryngeal stridor (during inspiration the arytenoids and vocal folds get pulled into the larynx closing the airway); Cyanosis; Dyspnea

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16
Q

How is laryngeal paralysis diagnosed?

A

Radiographs to rule out other causes and look for supportive pathology such as aspiration pneumonia, non-cardiogenic pulmonary edema and megaesophagus); Laryngoscopy under light anaesthesia

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17
Q

How would you address an emergency case of LarPar?

A

Cooling, oxygen therapy and anxiolytics +/- emergency intubation or tracheostomy

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18
Q

For mild cases of LarPar, medical management solely tends to be appropriate. Describe medical management of this condition

A

Weight management, stress reduction, exercise restriction and avoiding heat. Warn owners that it will progress and can cause an emergency

19
Q

For moderate to severe cases of LarPar, how can we surgically manage the condition? What is a drawback?

A

Unilateral cricoarytenoid lateralization (“tie-back”); risk of aspiration (no swimmy :( )

20
Q

Prognosis of LarPar

A

Fair to poor (worsens as disease progresses)

21
Q

For tracheal collapse, which type of stenting is preferred, intraluminal or extraluminal?

A

Intraluminal (easier, and less severe complications especially in dogs where sx approach could be difficult)

22
Q

Does stenting a tracheal collapse stop the dog from coughing?

A

Naur

23
Q

Provide at least three causes of a spontaneous pneumothorax

A

Pulmonary emphysema; Pulmonary neoplasia; Chronic pneumonia; Migrating plant material; Asthma; Lungworm; Heartworm

24
Q

Clinical signs of spontaneous pneumothorax

A

In moderate to severe cases, we get restrictive breathing, hypoventilation, diminished lung sounds and respiratory distress

25
Q

Diagnosis of pneumothorax

A

US and PE to confirm presence of pneumothorax

26
Q

Four treatments for spontaneous pneumothorax

A

Thoracocentesis; Thoracotomy + continuous suction; Autologous blood patch (basically using animal’s own blood, injecting it into the pleural space, and incuding the formation of a fibrin clot); Surgery

27
Q

Pathomechanism of pyothorax

A

Accumulation of septic purulent fluid in the pleural space

28
Q

Clinical signs of pyothorax

A

(May not be evident): tachypnea; dyspnea; cough; lethargy; weight loss; anorexia; restrictive breathing pattern

29
Q

Treatment of pyothorax

A

Typically, thoracocentesis or chest tube to drain +/- lavage + antibiotics (Fluoroquinolone and penicillin or clindamycin)

30
Q

Diagnosis of a pyothorax

A

Thoracocentesis; Cytology and culture

31
Q

When would surgical management of a pyothorax be indicated? What does this involve?

A

If you have failure of medical management (3-7d) or complications with the thoracostomy tube; remove inciting cause of pyothorax

32
Q

Prognosis of pyothorax

A

Guarded to good; Dogs tend to do better than cats with surgery

33
Q

Pathomechanism of a diaphragmatic hernia

A

Trauma leads to deatchment/rupture of part of the diaphragm from the body wall, allowing abdominal organs (liver most often) to enter the thoracic cavity

34
Q

Clinical signs of a Diaphragmatic hernia

A

Respiratory distress; Exercise intoelrance; Muffled heart and lung sounds: +/- borborygmi

35
Q

Diagnosis of a diaphragmatic hernia

A

radiographs; Ultrasound

36
Q

How are patients with diaphragmatic hernias treated?

A

Taken into surgery ASAP once stabilized; hernia is reduced via an abdominal approach while patient is on ventilatory support

37
Q

Briefly describe diaphragmatic hernia reduction surgery

A

Gently reduce the abdominal contents back into the abdomen, breaking adhesions where possible; Close opening dorsal to ventral using a continuous pattern +/- surgical mesh if chronic; Place thoracostomy tubes

38
Q

Prognosis of diaphragmatic hernia patients that receive surgeru

A

80-90% survival to discharge

39
Q

True or false. Death w/in 24 h of DH reduction is typically due to issues related to the GI tract.

A

False. Death w/in 24 h of DH reduction tends to be due to either pulmonary or cardiac dysfunction. Following 24hj, death typically due to GI damage or an unrelated issue

40
Q

True or false. Diaphragmatic hernias and peritoneopericardial diaphragmatic hernia are acquired.

A

False. PPDH are congenital while DH are acquired

41
Q

PPDH

A

Opening in the ventral diaphragm which allows the pericardial sac and peritoneal cavity to interact

42
Q

Clinical signs of PPDH

A

Muffled heart sounds, Boroborygmi in the thoracic cavity; Dyspnea; Cardiac dysnfunction

43
Q

Diagnosis of PPDH

A

radiographs; Ultrasound

44
Q

Surgery used to correct PPDH

A

Midline celliotomy (may have to enter pleural space and pericardium)