Respiratory pt. 1 Flashcards
review: purpose of URT
- delivers O2 and expel CO2
- warms, filters, and humidify air
structures of URT
- nose
- sinuses/nasal passages
- pharynx
- tonsils + adenoids
- larynx - epiglottis, glottis, vocal cords, adjacent cartilage
- esophagus
- trachea - surrounded by carriage
structures of LRT
- main stem bronchi
- lobar and segmental bronchi
- bronchioles
- lungs: left (2 lobes), right (3 lobes)
- alveoli (gas exchange, produces surfactant)
- pleura (fibrous membrane, cavity contains fluid that allows fluidity between structures w/o friction)
- diaphragm (assessor muscles that pulls everything down -> air to throat) (negative pressure in lung fields)
alveoli surfactant function
- protein lipid
- coats alveoli to keep lungs open
- patient and inspiratory effect
types of pleura (2)
- visceral pleura (lungs)
- parietal pleura (chest wall)
clicker: what is the purpose of cilia
move mucus upwards towards the larynx (up and out of airway)
describe inspiration
muscle activity required
- diaphragm: descent of diaphragm -> enlarge thoracic space -> intrathoracic pressure falls -> air enters thoracic cavity (negative pressure)
- accessory muscles: intercostal muscles helps with inspiration
describe expiration
passive process normally with elastic recoil
- counterbalanced by the chest wall coupled to pleural membrane/space
oxygen pressure in the alveoli is ________ than in the capillaries. why?
- higher
- O2 diffuses (high solute to low solute) into bloodstream, binds to hemoglobin
- O2 pumped throughout body via perfusion
describe V/Q
- ventilation (oxygen) & perfusion (blood)
- normally 1:1 match
- diseases can lead to mismatch
iclicker: if a person comes in w/ chronic GIB and a hemoglobin reflecting 6.4 mg/dL, what changes would this cause to O2 sat?
none, no correlation
review: ABG levels
PaO2: 80-100 mmHg (blood gas)
PaCO2: 35-45 mmHg (CO2)
PaHCO3: 22-26 mmHg (bicarb)
pH: 7.35-7.45 mmHg (pH)
describe oxyhemoglobin dissociation curve
- shows relationship between PaO2 and CO2
- shifts to the right and left
- significance of the shape to clinical situation
- PaO2 =/= SpO2
- tip: pulse ox (spO2) can be very unreliable, ABGs are more important
assessment for respiratory tract
- age and related changes
- hx (medical (HF), family, smoking)
- meds
- ax
- occupation/exposure hx (industrial)
- current symptoms/complaints
why is smoking history important to note
direct correlation -> lung ds.
what are symptoms/complaints of respiratory
- cough (productive/non-productive)
- sputum production (yellow = pneumonia nclex)
- chest pain (pattern w/ deep inspiration = normal r/t pneumonia BUT deep pain r/t COPD = more serious)
- dyspnea (impacts on ADL, paroxysmal nocturnal dyspnea), orthopnea (lay flat, SOB)
physical assessment for repisratiry
- IPPA
- lab tests
- radiograph
- other: invasive tests, pulse, pulmonary function test, exercise testing
what is included in IPPA assessment ( normal vs. abnormal)
auscultation - normal (tone differs, top = high pitch, low = low pitch)
- bronchial (Upper)
- bronchovesicular (upper)
- vesicular (lower alveoli)
abnormal
- wheezing (airway constricting -> asthma)
- crackles (fluid in lungs -> atelectasis)
- friction rub ( shuffling gravel -> pleural infiltrates)
- dull resonance (fluid in lungs) vs. hyper resonance (air in lungs -> COPD)
what is included in lab tests for resp
- ABGs
- CBC (wbc - infection, increased RBC/HgB)
- sputum (important to get deep sputum out for culture)
what is included in radiograph assessment in resp
- CXR (AP/Lateral, portable)
- CT/MRI (underlying issues with lungs, MRI - cancer, metastatic)
- angiography (dye in veins courses through body for PE assess and blood flow)
what are the 2 types of invasive tests
bronchoscopy, thoracentesis
describe bronchoscopy (uses, nurse interventions)
uses
- camera inserted to see bronchioles
- used for pt. with chronic cough
- diagnosis/biopsy
- removal of secretions (mucus plugs developed on ventilator)
nursing interventions
- teaching about anesthesia inhibiting ability to cough
- maintain NPO status
- monitoring patient resp. function
describe thoracentesis
uses
- removes fluid from sac
- diagnosis/biopsy (cancer, pleural fluid)
- decompression (allow for pulmonary expansion)
nursing interventions
- teaching: what to expect
- positioning: (sit at side of bed, leaning over bedside table)
- monitoring: lidocaine, post (chest pain - normal)
describe pulmonary function test
measures lung capacity
- tidal volume: inspiratory vol. during quiet resp.
- vital capacity: max amount inhaled/exhaled
- functional residual capacity: amt. of air in lungs after normal expiration
- residual volume: amount of air remaining in lung after max expiration
- total lung capacity: sum of VC + RV
- forced vital capacity: max amt. air exhaled as quickly as possible after max inspiration
- FEV1/FVC ratio: 70% (lower = obstructive issue (COPD), pulmonary fibrosis (can’t expand lungs) = restrictive issue)
clinical applications for pulmonary function tests
- preoperative screening ( returning to baseline)
- disability screening
- defining obstructive/restrictive diseases
- assessing effectiveness of treatment
what is considered obstructive disease
emphysema
- increased airway resistance
- decreased FVC, decreases FEV1 and FEV1/FVC and increased FRC
- basically, retaining air in lungs
what is considered restrictive disease
- restriction in the expansion of the thorax or lung tissue
- reduction in lung volumes but normal flows
what are nursing implications for pulmonary function test
patient preparation (education)
follow up with pulmonologist
which organ is pivotal in acid-base balance. why?
- lungs d/t fastest to respond to alterations in acid/base
- hydrogen ions (H+) are formed by from acids -> bound to CO2 in blood
what is the relationship between pH and hydrogen ions
- pH is measure of the body’s hydrogen ion concentration and is inversely relation
- high H+ = low pH
- low H+ = high pH
- normal pH = 7.35-7.45
- Important: small changes in pH lead to big physiological changes and even death!
what abg is the only one indicator of effective respirations
PaO2 d/t oxygen being carried on hub molecule as oxyhemoglobin
what are the sources of acid
- glucose metabolism
- fat/protein metabolism
- anaerobic metabolism (drives pH, running on treadmill increases acid, decrease pH)
- cell destruction (apoptosis = acid release in blood stream when dead)
what are the sources of bases (HCO3)
- absorption of bicarb by intestines and reabsorption by kidneys (release bicarb into bloodstream to increase pH)
what are the 2 buffers for acid base balance
lungs and kidneys
what are the respiratory mechanisms of acid base
- sensitive to CO2 levels (drive for inspiration/expiration)
- hyperventiliation: “blow it off” to bring pH up
- hypoventilation: retain CO2 to bring pH down
- rapid response: can change RR in minutes
what are the renal mechanisms of acid base
- slower to take effect
- absorption or excretion of HCO3 (buffer acid)
- formation of acids
- formation of ammonium -> loss of hydrogen and drop in pH
acidosis manifestations
- CNS depression (fatigue, flaccid)
- decrease in muscle tone
- increased resp. rate/depth (increase base to bring pH up, compensating)
- myocardial irritability (heart will stop working if pH increases)
- DKA, kausmal breathing
alkalosis manifestations
- CNS excitement/seizures
- tetany, cramps, twitches but weakness
- myocardial irritability
how to evaluate ABGs
henderson-hassalbach equation
- pH will represent primary cause of imbalance
if an oxygen level is less than 80 mmHg (hypoxemia), what to do?
- independent ABG
- correct underlying problem (improves perfusion) or increase oxygen delivery (improve diffusion)
- deceased PaO2 = anaerobic metabolism -> respiratory issues
what are some O2 toxicities
- CO2 retention (lots of CO2 in blood for long time, causes body to lose drive to breathe and get O2 in, COPD)
- washout atelectasis (nonrebreather displaces O2, causing it to go to blood stream, leading to atelectasis)
- free radicals can damage end-organ cells (can’t always give O2, increased oxygen given leads to damaged free radical) ?
what percent is considered RA
21%
what is hypoexmia
decrease in arterial O2
what is hypoxia
decrease in O2 in tissue
goal of oxygen therapy
use least amount of supplemental oxygen necessary to achieve acceptable oxygenation of the patient (improves hypoxemia, subsequent hypoxia, reduces myocardial demand)
additional complications of oxygen
- combustion
- pressure injuries
- dryness of mucous membranes (subsequent infection) (cracking of skin, should humidify always to prevent dryness)
total concentration of oxygen received by patient is dependent on?
patient’s own rr and tidal volume (patient effort)
- part of tidal volume inspired by patient is room air therefore achieved concentration of oxygen delivered to a patient is variable
- low flow: inexpensive, easy to use, fairly comfortable
types of low flow delivery system
nasal cannula
simple face mask
partial rebreather mask
non-rebreather mask
describe nasal cannula
2-6 L
24-44% oxygen delivered
ineffective if mouth breathing
describe simple face mask
5-8 L
40-60% oxygen delivered
don’t let fall to chin, difficult to eat with
describe partial rebreather mask
6-11 L
50-75% oxygen delivered
flaps “off” - air from wall + atmosphere (RA 21%), flap is open
describe non rebreather mask
10-15 L
80-100% oxygen delivered
flaps “on” - exhale (CO2 leaves vent holes), inhale (flaps close, air from wall only)
what are the safety concerns with non-rebreather mask
- atelectasis
- free radical damage
- delirium if hypoxic (will decompensated quickly)