Respiratory Problems Flashcards

1
Q

6 viruses known to cause the common cold:

A
Rhinovirus
Parainfluenza
Adenovirus
Coronavirus
Respiratory syncytial virus (RSV)
Influenza
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2
Q

What is Epiglottitis?

A
  • Serious inflammation of the epiglottis that results in supraglottic obstruction; bacterial in origin
  • Occurs primarily in children between 2 and 7 years, but it can occur from infancy to adulthood.
  • The onset is abrupt and usually preceded by a sore throat, but not by cold clinical manifestations.
  • Considered an emergency situation
  • Epiglottic swelling is diminished after 24 hours of antibiotic therapy and intubated children can usually be extubated by day 3.
  • Bacterial causative agent is usually Haemophilus influenzae.
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3
Q

Symptoms of epiglottitis:

A

a. High fever
b. Irritability and restlessness
c. Red and inflamed throat with cherry edematous epiglottis (Note: Attempt throat inspection ONLY when immediate intubation can be performed if needed.)
d. Difficulty swallowing and drooling
e. Muffled voice
f. Inspiratory and sometimes expiratory stridor
g. Suprasternal and substernal retractions may be visible.
h. Tripod positioning: while supporting the body with the hands, the child thrusts the chin forward and opens the mouth in an attempt to widen the airway.
i. Sallow color of mild hypoxia, possibly progressing to cyanosis

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4
Q

Thumb sign

A

In radiology, the thumbprint sign, or thumbprinting, is a radiologic sign found on a lateral C-spine radiograph that suggests the diagnosis of epiglottitis. The sign is caused by a thickened free edge of the epiglottis, which causes it to appear more radiopaque than normal, resembling the distal thumb.

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5
Q

Nursing interventions for epiglottitis

A

a. Arrange for transport of the child to an emergency facility.
b. Monitor airway status and breath sounds. Assess for signs of respiratory distress: nasal flaring, the use of accessory muscles, stridor, and retractions.
c. Monitor vital signs; take axillary temperature, not oral temperature.
d. Maintain the child in a position that provides the most comfort and security.
e. Do not leave the child unattended and do not allow the child to lie down.
f. Do not restrain the child.
g. Do not attempt to visualize the posterior pharynx using a tongue depressor because this could result in spasm of the epiglottis and airway occlusion.
h. Prepare the child for lateral neck films to confirm the diagnosis.
i. Prepare the child with severe respiratory distress for possible endotracheal intubation or tracheostomy.
j. Establish IV access (any invasive procedure should be done in a setting in which intubation is available if needed).
k. Maintain NPO status.

l. Administer IV fluids and antibiotics as prescribed.,
m. Monitor hydration status and record intake and output.

n. Administer corticosteroids, as prescribed, to reduce edema during the early hours of treatment and for 24 hours after extubation.

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6
Q

What can be done for prevention of epiglottitis?

A

Prevention - Haemophilus influenzae type B conjugate vaccine beginning at age 2 months

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7
Q

What is LARYNGOTRACHEOBRONCHITIS (INFECTIOUS CROUP)?

A
  • Caused by inflammation of the mucosa lining the larynx and trachea, which results in airway narrowing
  • Narrowed airways impede inspiration, resulting in stridor.
  • Viral etiology is most common but may have viral or bacterial etiology.
  • Most common type of croup
  • Primarily affects children less than age 5 years
  • Gradual onset, often preceded by an upper respiratory infection
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8
Q

4 stages of LARYNGOTRACHEOBRONCHITIS

A

Stage I
Anxiousness or fear, Hoarseness (primary complaint),
Croupy cough (like a seal barking),
May demonstrate inspiratory stridor when disturbed
Stage II
Respiratory stridor becomes continuous.
Soft tissue of the neck may retract.
Intercostal retractions of the lower ribs. Accessory muscles.
Stage III
Signs of anoxia and carbon dioxide retention develop such as tachypnea, restlessness, anxiety.
Sweating, Pallor
Labored respiration, mild wheezing may be present.
Stage IV
Intermittent cyanosis progressing to permanent cyanosis
Respiratory failure (cessation of breathing)

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9
Q

ACUTE SPASMODIC LARYNGITIS

A

Brief attacks of laryngeal obstruction that occur suddenly, chiefly at night.
Absent to mild signs of inflammation.
Usually affects children ages 1 to 3 years and may be recurrent.
There is no associated fever.

Duration of the attack is a few hours and the child feels well the next morning

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10
Q

Treatment for ACUTE SPASMODIC LARYNGITIS

A

Administer steam from a hot bath or shower or cold steam from a humidifier in the bedroom to relieve mild clinical manifestations.

Attempt exposure to night air to terminate laryngeal spasm.

Administer cough and cold medicines as prescribed if the child has accompanying upper respiratory infection manifestations.
Administer a bronchodilator such as albuterol, as prescribed, if bronchospasm is suspected.

If moderately severe clinical manifestations, child may need to be hospitalized for observation and administration of cool mist and racemic epinephrine therapy.

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11
Q

BACTERIAL TRACHEITIS

What is it and what are symptoms

A

• Bacterial infection involving the mucosa of the upper trachea
• Common pathogens include Staphylococcus aureus, group A betahemolytic streptococcus, and Haemophilus influenzae
• Exhibits features of both croup and epiglottitis
Affects children ages 1 month to 6 years
• Usually preceded by upper respiratory infection with croupy cough
• May be a complication of laryngotracheobronchitis
• Early identification is essential to prevent airway obstruction.
• Usually follows a slower course than epiglottitis.

Symptoms:
Fever, Brassy cough, Inspiratory stridor
Hallmark: production of thick purulent tracheal secretions which can result in airway obstruction and even respiratory arrest

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12
Q

Sleep apnea risk factors

A

Risk factors include a family history of sleep apnea, hypothyroidism, recessed chin, large neck, cigarette smoking, and alcohol or sedative use.

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13
Q

Name and describe three types of sleep apnea:

A
  • -Obstructive sleep apnea (OSA) is the most common type and involves the upper respiratory tract. With obstructive sleep apnea, obstruction of the airway occurs at either the soft palate (nasopharynx) or at the level of the tongue (oropharynx).
  • -central sleep apnea, the brain fails to trigger the muscles of respiration.
  • -Mixed sleep apnea is a combination of these two types.

Blood oxygen levels drop with each apneic episode in all types of sleep apnea.

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14
Q

Without treatment, individuals with OSA are at risk for severe and life-threatening complications of the heart and lungs.
What are some complications of sleep apnea?

A

Secondary health problems include hypertension, at dysrhythmias, myocardial infarction, stroke, hypoxia (reduced oxygen content or tension), hypercapnia (abnormally increased arterial carbon dioxide tension), pulmonary hypertension, or cor pulmonale (hypertrophy or failure of the right ventricle resulting from disorders of the lungs, pulmonary vessels, or chest wall).
It has been estimated that up to 50% of people with sleep apnea have hypertension.

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15
Q

Symptoms of OSA

A

presence of a narrowed oropharynx, enlarged tonsils or uvula, or prominent tongue.

Nasal obstruction by a deviated septum may be present.

loud snoring or snorting. Bed partners report this sign because the patient may be unaware.

morning headaches, daytime somnolence (prolonged drowsiness or sleepiness), memory problems, personality changes, gastric reflux, sore throat, impotence, fatigue.

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16
Q

Other than foreign body obstruction, name 3 causes of swelling of the mucous membranes within the larynx that can close off the opening and lead to suffocation.

A

Swelling of the larynx rarely occurs with acute laryngitis but is more often seen with:
1-epiglottitis,
2-inflammation of the throat, such as in scarlet fever.
3-In cases of a severe allergic reaction (anaphylaxis), the edema can close off the airway, leading to death if not reversed.

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17
Q

Ineffective Ciliary Clearance - Normal cilia beat in a coordinated unidirectional fashion to mobilize mucus and clear particulate matter from the airways. cilia perform this function inadequately or not at all. Ciliary impairment is associated with conditions including:

A
  • Cystic fibrosis
  • Primary ciliary dyskinesia
  • Kartagener syndrome
  • Status post heart-lung or lung transplantation
  • Smoking or exposure to second hand smoke
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18
Q

Abnormal amounts of mucus with altered physical properties, inhibiting normal airway clearance, may be present in:

A
  • Cystic fibrosis
  • Chronic obstructive pulmonary disease
  • Bronchiectasis (COPD), emphysema, chronic bronchitis
  • Asthma
  • Mechanical ventilation
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19
Q

What is atelectasis?

A

Atelectasis means “incomplete expansion” and is defined as collapse of lung tissue because of airway obstruction, an abnormal breathing pattern, or compression of the lung tissue. Obstructive atelectasis is the most common type.

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20
Q

What is obstructive atelectasis ?

A

When the airway becomes completely obstructed, the gas distal to the obstruction becomes absorbed into the pulmonary circulation and the lung collapses.
can be caused by a foreign body, tumor, or mucous plugging.

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21
Q

What is Nonobstructive atelectasis?

A

caused by loss of contact between parietal and visceral pleurae, as well as compression, loss of surfactant replacement of parenchymal tissue by scarring or infiltrative disease.
Abnormal breathing patterns, such as hypoventilation and a slow respiratory rate, can also lead to atelectasis. In such cases, the lung does not fully expand, which causes the lower airways to collapse.

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22
Q

It is common after surgery, or in patients who were in the hospital. Risk factors for developing atelectasis include:

A

• Anesthesia
• Mucus that plugs the airway
• Foreign object in the airway (most common in children)
• Prolonged bed rest with few changes in position
• Shallow breathing (may be caused by painful breathing)
• Tumors that block an airway
• Lung diseases
–Pleural effusion

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23
Q

PNEUMOTHORAX is positive pressure (air) within chest cavity resulting from accumulated air that cannot escape during expiration. It leads to collapse of lung, mediastinal shift, and compression of the heart and vessels. When pressure builds up in the pleural space, the lung on the affected side collapses, and the heart and mediastinum shift toward the unaffected lung.
There are 3 types:

A
  1. Closed (spontaneous): rupture of a subpleural bulla, tuberculous focus, carcinoma, lung abscess, pulmonary infarction, severe coughing attack, or blunt trauma.
  2. Open (traumatic): communication between atmosphere & pleural space because of opening in chest wall.
  3. Tension: one-way leak; may occur during mechanical ventilation or CPR, or as a complication of any type of spontaneous or traumatic pneumothorax.
24
Q

Symptoms of pneumothorax

A
  • Pain – May be sharp, aggravated by activity. Location—chest; may be referred to shoulder, arm on affected side.
  • Restlessness, anxiety
  • Dyspnea
  • Cough.
  • Cessation of normal movements on affected side.
  • Absence of breath sounds on affected side.
  • Pallor, cyanosis.
  • Shock.
  • Tracheal deviation to unaffected side.
  • X-ray: air in pleural space.
25
Q

Nursing interventions for pneumothorax

A
  • Place sterile occlusive gauze dressing over wound.
  • Tape dressing on three sides to allow air to escape during expiration.
  • Place client on affected side to diminish possibility of tension pneumothorax.
  • Careful administration of narcotics to prevent respiratory depression (avoid morphine}.
26
Q

Interventions for closed chest drainage

And the purposes of the chest drainage

A

client in sitting position, ensuring safety by having locked over-the-bed table for client to lean on, or have a nurse stay with client so appropriate position is maintained throughout the procedure.

Explain purpose of the procedure—
to provide means for evacuation of air and fluid from pleural cavity;
to reestablish negative pressure in pleural space;
to promote lung reexpansion.

27
Q

What is pleurisy.
Causes?
Symptoms?

A

Pleurisy, an inflammation of the pleural sac, can be associated with upper respiratory infection, pulmonary embolus, thoracotomy, chest trauma, or cancer.
Chest x-ray reveals the presence of air or fluid in the pleural sac.
Symptoms include
Sharp pain on inspiration, dyspnea,
Cough,
Chills and fever

28
Q

Management of the client with pleurisy includes:

A

analgesics, antitussives, antibiotics, and oxygen.

May need thoracentesis.

The nurse should also observe the client for signs of a pneumothorax.

29
Q

What is pleural effusion and what are its causes?

A

accumulation of fluid in the pleural space.

rarely a primary disease process but is usually secondary to other diseases, such as:
heart failure, TB, pneumonia, nephritic syndrome, connective tissue disease, pulmonary embolism, neoplastic tumors.

30
Q

What is hemothorax?

A

Hemothorax is bloody fluid in the pleural space due to trauma or, rarely, as a result of coagulopathy or after rupture of a major blood vessel, such as the aorta or pulmonary artery.

31
Q

What is Empyema?

A

Empyema is pus in the pleural space. It can occur as a complication of pneumonia, thoracotomy, abscesses (lung, hepatic, or subdiaphragmatic), or penetrating trauma with secondary infection.

32
Q

What is acute respiratory distress syndrome (ARDS),

also called shock lung or adult respiratory distress syndrome

A

results from increased permeability of the alveolocapillary membrane. Fluid accumulates in the lung interstitium, alveolar spaces, and small airways, causing the lung to stiffen. Prohibits adequate oxygenation of pulmonary capillary blood.
Severe ARDS can cause intractable and fatal hypoxemia. However, patients who recover may have little or no permanent lung damage

33
Q

Symptoms of ARDS

A
  • Apprehension, confusion, restlessness
  • Crackles, rhonchi
  • Dyspnea, rapid shallow breathing
  • Hypoxemia
  • Intercostal & suprasternal retractions
  • Cough
  • Hypotension
  • Motor dysfunction
  • Tachycardia
34
Q

What is a symptom that could indicate pulmonary edema?

A

clear, frothy sputum

35
Q

What is COPD

A

A group of disorders associated with persistent or recurrent obstruction of airflow; includes:
chronic bronchitis,
pulmonary emphysema, and
bronchial asthma

36
Q

What is Pulmonary emphysema:

A

1) Destruction of alveoli, narrowing of small airways (bronchioles), and the trapping of air results in loss of lung elasticity.
2) Primary contributing factor is smoking cigarettes
3) Develop a deficiency of alpha antitrypsin (enzyme that blocks the action of proteolytic enzymes that are destructive to elastin and other substances in the alveolar walls)

37
Q

What is chronic bronchitis?

A

1) Excessive mucus secretions within the airways and recurrent cough
2) Contributing factors include heavy cigarette smoking, pollution, and infection.
3) Copious sputum production
4) Hypoxemia resulting in polycythemic: ruddy look to the skin, compensation
5) Pulmonary hypertension leading to cor pulmonale

38
Q

What is bronchial asthma?

A

1) Abnormal bronchial hyperreactivity to certain substances
2) Extrinsic: antigen-antibody reaction triggered by food, drugs, or inhaled particles
3) Intrinsic: pathophysiological conditions within the respiratory tract
4) Status asthmaticus
a) An asthma attack lasting more than 24 hours
b) A medical emergency
c) Usually responds to epinephrine hydrochloride (adrenalin) s.q. and aminophylline, theophylline, and ethylenediamine (Phyllocontin)

39
Q

What is cor pulmonale?

A

“Heart of the lungs”
Right ventricle enlarges as result of disease that affects structure of lungs.
Most common cause is COPD.

Occurs when there is increased pressure and pulmonary hypertension

There is destruction of the pulmonary capillaries, increased resistance of the pulmonary capillary bed, and shunting of unaerated blood across the collapsed alveoli.

• Initially, the right heart fails, then the left heart fails because of decreased cardiac output

40
Q

Symptoms of cor pulmonale

A
Paroxysmal nocturnal dyspnea (PND) and orthopnea
Right heart failure orthopnea
Peripheral edema (dependent)
Jugular vein distension 
Left heart failure 
Dyspnea 
Cyanosis
Cough
Substernal pain
Syncope on exertion 

Treatment: diuretics, calcium channel blockers, bronchodilator.
Treat underlying disease.

41
Q

What are bronchodilators and examples

A

Examples include albuterol, epinephrine, salmeterol…

act by relaxing the smooth muscles of the bronchial tree, thereby relieving bronchospasm and decreasing the work of breathing. Bronchodilators are used in the symptomatic treatment of acute respiratory conditions such as asthma, as well as many forms of COPD.

42
Q

Beta 2 agonists are what kind of drug?
Action? Examples?
(The text calls them adrenergics)

A

bronchodilators that increase vital capacity and decrease airway resistance. They work on the smooth muscle in the lungs to cause relaxation. However, they also can affect the entire sympathetic nervous system.
Albuterol, epinephrine, salmeterol, levalbuterol, formoterol, vilanterol

43
Q

Muscarinic antagonists (Text calls them Anticholinergics)are what kind of drug?
Action?
Examples

A

Muscarinic antagonists (Anticholinergics), for example Atrovent, achieve bronchodilation by decreasing the chemical that promotes bronchospasm. Anticholinergics block the parasympathetic nervous system and can cause drying of pulmonary secretions. Adequate hydration should be encouraged to avoid mucus plugging. Inhaled anticholinergics are first-line therapy for COPD once symptoms become persistent.

44
Q

What are xanthines (methylxanthines)
Action?
Example?

A

xanthine derivative: theophylline relaxes the smooth muscle of the bronchial airways and pulmonary blood vessels. Theophylline is generally reserved for patients with COPD who do not respond or cannot take inhaled long-acting bronchodilators.

45
Q

Corticosteroids
Action?
Examples

A

Synthetic corticosteroids are used to relieve inflammation, reduce swelling, and suppress symptoms in acute and chronic reactive airway disease (asthma and some COPDs)

beclomethasone, budesonide, fluticasone, mometasone

46
Q

Leukotriene receptor antagonists

A

Leukotriene receptor antagonists primarily help to control the inflammatory process of asthma caused by leukotriene production

47
Q

Short acting beta 2 agonists SABA
Vs
Long acting beta 2 agonists LABA

Both are bronchodilators

A

Short-acting beta-agonists (SABAs) provide quick relief of asthma symptoms. (Albuterol, levalbuterol)

Long-acting beta-agonists (LABAs) are taken on a daily basis to relax the muscles lining the airways. LABAs are often taken with inhaled corticosteroid. (Salmeterol, formoterol)

48
Q

Examples of muscarinic antagonist?

A

Can referred to as LAMA- long acting muscarinic antagonists.

Tiopropium (spiriva)

49
Q

How is empyema diagnosed?

How is it treated?

A

Diagnosed with chest CT and diagnostic thoracentesis.

Treated by draining the plural cavity by thoracotomy or needle aspiration. May go home with drainage system.
Usually large doses of antibiotics for 4-6 weeks.

50
Q

How is sarcoidosis treated?

A

Corticosteroids
Cytotoxic agents
Immunosuppressive agents

51
Q

How is vital capacity measured?

A

Take maximum breath and exhale fully through a spirometer

52
Q

Nursing interventions for bronchoscopy:

A

NPO 6 hours prior

After procedure, NPO until gag reflex returns.

53
Q

Tachypnea can be caused by:

A
Pneumonia, 
pulmonary edema, 
metabolic acidosis, 
septicemia, 
severe pain, 
rib fracture
54
Q

Symptoms of PE:

A

Depends on size and location.
Dyspnea, tachypnea,
sudden chest pain, anxiety, fever, tachycardia,
anxiety, cough, diaphoresis, hemoptysis, syncope

55
Q

What determines cyanosis?

A

Lake indicator of hypoxia.

Amount of unoxygenated hemoglobin determines cyanosis.

56
Q

Nursing interventions for pleurisy

A

Turn frequently on to affected side to splint Chest wall and reduce stretching the pleurae.
Use hands of pillow to splint when coughing.

57
Q

What kind of ph imbalance does pulmonary embolism cause?

A

Respiratory alkalosis