Respiratory failure and Intubation (1B) Flashcards

1
Q

Why would somebody go to ICU?

A
  • Haemodynamic/respiratory/neurological monitoring
  • Support of normal body functions (Intubation and mechanical ventilation. Cardiovascular support eg inotropes)
  • Stabilisation of life threatening injuries
  • Treatment of multi organ failure
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2
Q

What is the definition of respiratory failure?

A

When the patient loses the ability to ventilate adequately (CO2) or to provide sufficient oxygen to the blood and systemic organs

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3
Q

What is hypoxaemic respiratory failure?

A

Long, o2 gas movement, regional ventilation)
PaO2 < 60 mm Hg
PaCO2 < 42 mm Hg

  • Lung disease is severe enough to interfere with O2 exchange
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4
Q

What is hypercapnic respiratory failure?

A

Pump, CO2 gas movement, effective minute ventilation, overall ventilation

PaCO2 > 50 mm Hg

The respiratory system pump is inadequate and cannot maintain ventilation to eliminate the CO2 produced by metabolism

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5
Q

What is acute respiratory failure?

A

Rapid onset, short course and pronounced symptoms

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6
Q

What is chronic respiratory failure?

A

Long duration of poor ABG values with (metabolic) compensation

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7
Q

VE =

A

minute ventilation

RR x VT (tidal volume)

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8
Q

Vd =

A

dead space (non gas exchange area -> upper airways)

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9
Q

VD=

A

dead space ventilation

= RR x Vd

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10
Q

VA=

A

alveolar ventilation

= (VT - Vd) x rr
= VE- VD

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11
Q
Normal male
Vd =
VT =
RR
VE =
VA=
CO2 =
A
Vd = 100mL
VT =500 mL
RR = 12
VE = 61/min
VA= 4.81/min
CO2 = normal
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12
Q
Post abdominal surgery
Vd =
VT =
RR
VE =
VA=
CO2 =
A
Vd = 100 mL
VT = 250 mL
RR = 24
VE = 61/min
VA= 3.61/min
CO2 = increased
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13
Q
DBE with physiotherapist
Vd =
VT =
RR=
VE =
VA=
CO2 =
A
Vd = 100 mL
VT = 750 mL
RR= 8
VE = 61/min
VA= 5.21/min
CO2 = decreased.
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14
Q
PE 2 weeks post discharge
Vd =
VT =
RR=
VE =
VA=
CO2 =
A
Vd = 200 mL
VT = 500 mL
RR= 12
VE = 61/min
VA= 3.61/min
CO2 = increase
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15
Q

Mechanisms & causes

• Hypoxaemic respiratory failure

A

– Reduced gas going to areas with perfusion
• Low lung volume (O2 movement problem)
– No gas going to areas with perfusion
• Acute lobar collapse (O2 movement problem)
– Diffusion impairment

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16
Q

Mechanisms & causes

• Hypercapnic respiratory failure

A
Mechanisms & causes
• Hypercapnic respiratory failure
(CO2 movement problem)
– Depressed drive
• Opiate overdose
– Impaired neuromuscular function
• Cervical spinal cord injury
• Guillain Barré syndrome
• Respiratory muscle dysfunction
– Weakness
– Fatigue
17
Q

Mechanisms & causes

– Increased respiratory load

A
– Increased respiratory load
• Increased airway resistance
– asthma
• altered chest wall compliance
– kyphoscoliosis
– # ribs
• Decreased lung compliance
18
Q

Clinical manifestations

• Hypoxaemia

A

– Decreased mental acuity (PaO2 < 40‐50 mm Hg)
– Agitation followed by somnolence
– Dyspnoea
– Increased RR, change in pattern of breathing
– (Organ failure – renal failure, brain injury)

19
Q

Clinical manifestations

• Hypercapnia

A

– Depends on rate of rise of CO2 and metabolic
compensation
– Dyspnoea
– Increased RR, change in pattern of breathing
• COPD – accessory muscle use, paradoxical breathing,
intercostal space or rib indrawing, pursed lips breathing
– Agitation, tremor
– Confusion to coma
– Increased ICP, headache

20
Q

Implications respiratory failure for physiotherapy

A
• Watch for signs & symptoms of respiratory
failure
• Review medical assessment & management
• Determine type of respiratory failure
– hypoxaemic, hypercapnic
• Determine cause of respiratory failure
– problem list in terms of impairments
• Choose appropriate treatment interventions
21
Q

Reasons for intubation

A
• Maintain patent upper airway
• (suffocation)
• Protect lower respiratory tract
• (aspiration)
• Enable adequate tracheobronchial toilet
• (suctioning)
• Allow ventilatory support
– Mechanical ventilation
• during paralysis & sedation
• rest respiratory muscles
– Oxygen therapy/CPAP/PEEP
22
Q

What is a Tracheostomy?

A

• A tracheostomy is an artificial airway that is shorter
than an endotracheal tube
• Inserted surgically or percutaneously through the
neck into the trachea between 2nd and 3rd tracheal
rings
• Bypasses the upper airway and all air intake occurs
via the tracheostomy tube when cuff is inflated
• Can be temporary or permanent

23
Q

Why is an ETT not done long term?

A

unstable, pressure on tracheal wall

24
Q

Indications for Tracheostomy

A

• The same as for endotracheal intubation but……
• When intubation will be required for a longer period
– As the ETT passes through the vocal cords these can be
damaged
• Voice problems
• Swallowing problems (aspiration)
• Prolonged mechanical ventilation (> 10‐14 days)
• To facilitate weaning from mechanical ventilation
• Tracheomalacia, tracheal stenosis

25
Q

• Compared to an ETT a tracheostomy:

A

– reduces dead space compared to an ETT, easier to wean
from mechanical ventilation
– allows reduction in level of sedation as it is more
comfortable (less gag stimulation)
– patient can communicate more easily ‐ mouth words, or
specialised tubes may allow use of a speaking valve

26
Q

Tracheostomy management

A
• Will vary between facilities
• Multidisciplinary teams
• Physiotherapist
– Checking patency
– Tube changes
– Decannulation (removal)