Respiratory - 11/15 Quiz Flashcards

1
Q

Pressure of the air in the pleural space is the same as barometric pressure because the air drawn into the pleural space during inspiration is forced back out during expiration.

A

Communicating pneumothorax

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2
Q

Pressure of the air in the pleural space exceeds barometric pressure because air enters during inspiration but cannot exit during expiration.

A

Tension pneumothorax

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3
Q

Alternating periods of deep and shallow breathing; apnea lasting 15 to 60 seconds, followed by ventilations that increase in volume until a peak is reached, after which ventilation decreases again to apnea (e.g., CHF, sleep apnea)

A

Cheyne-Stokes (breathing pattern)

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4
Q

Increased ventilatory rate, small tidal volume

A

Restricted (breathing pattern)

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5
Q

Rhythmic and effortless with normal tidal volume

A

Eupnea (breathing pattern)

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6
Q

Irregular, quick inspirations with an expiratory pause

A

Gasping (breathing pattern)

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7
Q

Increased ventilatory rate, small tidal volume, increased effort, prolonged expiration, wheezing

A

Obstructed (breathing pattern)

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8
Q

Increased ventilatory rate, very large tidal volume, no expiratory pause

A

Kussmaul (breathing pattern) - DKA DM I & renal failure patients

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9
Q

waking up with dyspnea during the night and needing to sit upright or stand to breathe

A

Paroxysmal nocturnal dyspnea

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10
Q

Rib fractures that disrupt the mechanics of breathing can cause a portion of the chest wall to collapse ________ during inspiration, an acute condition known as _______ chest.

A

inward; flail

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11
Q

A person who has pneumothorax has _____ in the pleural space.

A

air

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12
Q

A person who has _______ has persistent abnormal dilations of the bronchi and a chronic cough that produces large amounts of purulent _______.

A

bronchiectasis; sputum

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13
Q

Pulmonary fibrosis is an excessive amount of ______ tissue in the lungs and causes ______ lung compliance.

A

fibrotic; decreased

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14
Q

In asthma, long-term airway damage that is irreversible is known as airway _________.

A

remodeling

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15
Q

During an acute asthma episode, inflammatory mediators cause inflammation, hypersecretion of ______, and bronchial smooth muscle ________.

A

mucus; constriction

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16
Q

An asthma episode that does not resolve with usual treatment is called ______ ________.

A

status asthmaticus

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17
Q

Genetic deficiency of ____ __________ causes early-onset emphysema because this enzyme normally inhibits the action of _________ _________ that can destroy lung tissue.

A

alpha-1 antitrypsin; proteolytic enzyme

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18
Q

Alveolar ventilation is inadequate in relationship to the metabolic demands; leads to respiratory acidosis

A

hypoventilation

19
Q

Alveolar ventilation exceeds the metabolic demands; leads to respiratory alkalosis from hypocapnia.

A

hyperventilation

20
Q

Head injuries causes…(hypo/hyper-ventilation)

A

hyperventilation

21
Q

Airway obstruction causes… (hypo/hyper-ventilation)

A

hypoventilation

22
Q

Respiratory muscle weakness causes… (hypo/hyper-ventilation)

A

hypoventilation

23
Q

Reduced compliance of chest wall causes… (hypo/hyper-ventilation)

A

hypoventilation

24
Q

Anxiety causes… (hypo/hyper-ventilation)

A

hyperventilation

25
Q

A person who has ….. has persistent abnormal dilations of the bronchi and a chronic cough that produces large amounts of purulent …..

A

bronchiectasis; pus-like sputum

26
Q

Most common cause of pulmonary edema

A

left-sided heart failure

27
Q

Pink frothy sputum, dyspnea, hypoxemia, increased work of breathing, crackles/rales, dullness on percussion, hypoventilation with hypercapnia

A

Pulmonary edema

28
Q

Cor pulmonale …-sided HF

Pulmonary edema …-sided HF

A

Right

Left

29
Q
Conditions related to \_\_\_\_\_ :
lung fluid increases,
systemic inflammatory response occurrence,
lungs dry out,
lung volume decreased
hypoxemia (always at end phase)
A

Acute Respiratory Distress Syndrome (ARDS)

30
Q

RN receives report on pt w/ARDS who has been intubated for 6 days and had progressive hypoxemia that responds poorly to high levels of oxygen. This pt is in which phase of ARDS case management?

A

fibro-proliferative:

fibrotic and proliferative phase at same time

31
Q

Airway obstruction is worse with expiration.
More force/time is required to expire given vol of air; emptying lungs is slowed.
unifying s/sx: wheeze & dyspnea.
Inc. WOB, vent-perf mismatch, dec. FEV1 (in 1 second).

A

Obstructive pulmonary disease

32
Q

Types of obstructive pulmonary disease

A

asthma, chronic bronchitis, emphysema, COPD

33
Q

Life threatening bronchospasms not reversed by usual measures

A

Status asthmaticus

34
Q

Albuterol nebulizer

A

quick-relief agent

beta-adrenergic agonist

35
Q

Oral prednisone

A

long-term control

steroid inflammatory

36
Q

Ipratropium inhaler

A

quick-relief agent

anticholinergic

37
Q

Salmeterol inhaler

A

long-term control

beta-adrenergic agonist

38
Q

The pulmonary vasoconstriction leading to development of cor pulmonale in patient with COPD results from…

A

alveolar hypoxia and hypercapnia

pulmonary HTN -> vasoC

39
Q

Abnormal permanent enlargement of gas-exchange airways accompanied by destruction of alveolar walls w/o obvious fibrosis.

A

Emphysema

40
Q

Inherited deficiency of enzyme α1-antitrypsin;

Involves entire acinus, damage is more randomly distributed, involves LOWER lobes of lung.

A

Primary emphysema;

Panlobular/Panacinar

41
Q

Main cause is cigarette smoke. Possible contributors: air pollution, occupational exposures, childhood resp. infxns.;
Septal destruction occurs in respiratory bronchioles & alveolar ducts, usu in UPPER lobes, alveolar sac remains intact, tends to occur in smokers w/chronic bronchitis.

A

Secondary emphysema;

Centrilobular/Centriacinar

42
Q

Pneumonia acquired <48 hours of hospitalization.

Streptococcus pneumoniae

A

community-acquired

43
Q

Pneumonia acquired >48 hours of hospitalization.

Staph, or commonly Gram-Neg e.coli, pseudo, kleb, enterob.

A

hospital acquired; nosocomial infection