Resp Therapy Equipment Flashcards

1
Q

What are the two types of pulse oximetry?

A

transmission- light passes through tissue to the sensor

reflectance- sensor and source are side by side

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2
Q

How does pulse oximetry work?

A

Based on the Beer-lambert law (concentration of absorbing substance in a solution is related to the intensity of light transmitted through that solution)

  • 2 diodes emit alternating light wavelengths (red light which is absorbed by deoxygenated blood and infrared light which is absorbed by oxygenated blood)
  • ratio of absorption is measured and the O2 saturation of hb is derived
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3
Q

Factors that affect pulse ox results

A
  • dark skin pigment
  • nail polish
  • acrylic nail
  • motion
  • ambient or excessive light
  • hypoperfusion (may overestimate)
  • hypoxia (less reliable below 80%)
  • dyshemoglobinemias (COHb absorbs light in the red wavelength similar to oxyhemoglobin, falsely elevated)
  • methemoglobin (Fe3+ vs normal Fe2+, absorbs red and infrared light)
  • IV dyes
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4
Q

Peak Flow Meters
Indications
what is it

A

Indications

  • asthma
  • some pts with COPD who have a component of reactive airway disease

What is it

  • an objective measurement of the severity of airway obstruction
  • uses best out of 3 measurements
  • may help identify triggers
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5
Q

Metered dose inhaler

-recommdations for use

A

recommdations for use

  • if on multiple inhalers, administer bronchdilator first
  • recommend use of a spacer
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6
Q

Why use a spacer?

A
  • improve coordination between delivery of the medication from the inhaler and breathing it into the bronchial tubes
  • reduce the amount of medication that settles in the mouth and throat
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7
Q

Types of nebulizers

A
hand held
hand held with mask
blow by
home neb unit
compact neb
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8
Q

Goals of oxygen therapy

A
  • improve oxygenation

- long term O2 therapy in COPD if needed improves survival, quality of life and decreases hospitalizations

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9
Q

O2 delivery systems

A
nasal cannula
simple face mask
venturi mask
bag-valve mask
bi-PAP or CPAP
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10
Q

What are the methods used to deliver home oxygen?

A
  • compressed O2 (tanks)
  • liquid O2
  • O2 concentrators

*all inhaled through mask or cannula

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11
Q

A nasal cannula provides 6L of O2.

What percent oxygen does nasal cannula provide?

A

6L=44% oxygen

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12
Q

How much oxygen does 1L/min flow rate provide? what is the rate of increase?

A

1 L/min=21-24%

-increasing the O2 flow rate by 1 L/min equates to an increase of about 4% FIO2

2 L/min= 25-28%
3=29-32%
4=33-36%
5=37-40%

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13
Q

How much O2 does a simple face mask provide?

A

40-60% (5-10L)

*minimum flow of 5L/min needed

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14
Q

What is a venturi mask? Who is it good for?

A
  • mask that accurately controls the proportions of inspired oxygen
  • good for patients who retain CO2 and pts who have moderate to severe hypoxemia
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15
Q

How much O2 does a non-rebreather mask provide?
How much is required?
When to use

A

Provides up to 90% O2

Need flow rates of 8-10 L/min

Used for seriously ill, responsive and spontaneously breathing who require high O2 content. Also used to avoid tracheal intubation if acute intervention produced a rapid change.

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16
Q

Bag valve mask/ambu bag
what is it?
disadvantages

A

one way valve mask/bag with an O2 for supplemental O2

disadvantages

  • mask must be sealed tightly over mouth and nose
  • need to coordinate breaths with any spontaneous respiratory effort
17
Q

Oxygen Toxicity
what is it?
possible adverse effects

A

What is it?

  • parenchymal lung injury due to supplemental O2
  • thought to be secondary to the production of oxygen free radicals that result in tissue destruction

Possible adverse effects

  • absorptive atelectasis (washout of alveolar N2 and atelectasis may occur if O2 diffuses out of the capillaries faster than entering
  • extrapulmonary toxicity (seizures, retinopathy, coronary vasoconstriction, decreased stroke volume and cardiac output, bradycardia and increased systemic vascular resistance)
  • accentuation of preexisting hypercapnia (increased CO2 associated with supplemental O2 in pts with chronic compensated resp acidosis
  • airway injury (larger airway inflammation, erythema, and edema)
  • parenchymal lung injury (destruction of alveolar tissue likely secondary to O2 free radicals causing inflammation and cellular death)
18
Q

Oxygen Toxicity

Prevention

A
  • FIO2 less than 60% likely safe without toxicites

- aim for PaO2 60-70 mmHg or SpO2 90-93% with therapies

19
Q

Hypoxic drive pathophys

A
  • chemoreceptors normally rely on CO2 levels to regulate ventilation
  • if chronic elevated CO2 levels occur the body resets and becomes less sensitive to elevated CO2 and the drive to breathe shifts over to rely on lower levels of O2
  • in these patients the addition of supplemental O2 can decrease the drive to breathe and maintain their minute ventilation leading to worsening respiratory failure.