Regional Flashcards
What are the 5 divisions of the spinal column, and how many vertebrae are present in each?
The vertebral column is made up of 33 vertebrae. Cervical- 7, Thoracic -12, Lumbar-5, sacrum- 5 fused, coccyx- 4 fused
What are the anatomic borders of the facet joint?
The facet joint is formed by the superior articular process of one vertebra and the inferior articular process of the vertebra directly above.
Injury to the facet joint can compress the spinal nerve that exits the respective intervertebral foramina, causing pain and muscle spasm along the associated dermatome
What ligament covers the sacral hiatus? what is the significance of this?
The sacral hiatus is covered by the sacrococcygeal ligament.
The ligament is punctured during the caudal approach to the epidural space
order the 5 ligaments of the spinal column from posterior to anterior.
Supraspinous ligament
interspinous ligament
ligamentum flavum
posterior longitudinal ligament
anterior longitudinal ligament
What ligaments are penetrated during the midline approach to the epidural space? how about the paramedian approach?
midline approach: the needle will pass through 3 ligaments
*supraspinous ligament
* interspinous ligament
* ligamentum flavum
Paramedian approach: the needle will pass through 1 ligament
* ligamentum flavum
The paramedian approach bypasses the other ligaments. With either approach the needle should NEVER pass through the anterior or posterior longitudinal ligaments.
List all the structures and spaces between the skin and the spinal cord as they would be encountered during a subarachnoid block.
Skin
subcutaneous tissue
muscle
supraspinous ligament
interspinous ligament
ligament flavum
epidural space
dura mater
subdural space
arachnoid mater
subarachnoid space
pia mater
spinal cord
what happens when you accidently inject local anesthetic into the the subdural space during a SAB? How about during an epidural?
The subdural space is a potential space between the dura mater and the arachnoid mater.
Inadvertent injection of local anesthetic into the subdural space yields the following outcomes:
*Epidural dose-> high spinal with delayed onset (15-20 min)
*spinal dose-> failed spinal
What is Batson’s plexus? and what is its significance?
The epidural veins (Batson’s plexus) drain venous blood from the spinal cord. These valveless veins pass through the anterior and lateral regions of the epidural space.
Obesity and pregnancy increase intra-abdominal pressure, causing engorgement of the plexus. This is associated with an increased risk of needle injury or cannulation during neuraxial techniques.
What is plica mediana dorsalis, and what is its significance?
While its existence remains controversial, many speculate that a band of connective tissue courses between the ligamentum flavum and the dura mater. If it does exist, it could conceivably create a barrier that would impact the spread of medications within the epidural space. The plica mediana dorsalis has long been considered the culprit for difficult epidural catheter insertion as well as unilateral epidural blocks.
What is a dermatome, and which ones are important as you assess neuraxial anesthetic?
the dermatome relates to an area of skin that is innervated by a spinal nerve- it is not necessarily the area of skin that is the same plane as the spinal nerve.
C6- 1st digit (thumb)
C7- 2nd and 3rd digits
C8- 4th and 5th digits
T4- Nipple line
T6- xyphoid process
T10- umbilicus
T12- Pubic symphysis
L4- anterior knee
Compare and contrast the site of action for spinal vs epidural anesthesia.
site of action: Spinal anesthesia
* The primary site of local anesthetic action is on the myelinated preganglionic fibers of the spinal nerve roots.
* Local anesthetics also inhibit neural transmission in the superficial layers of the spinal cord
Site of action : Epidural anesthesia
* Local anesthetics in the epidural space must first diffuse through the dural cuff before they can block the nerve roots
* local anesthetics also leak through the intervertebral foramen to enter the paravertebral area. Here, local anesthetics cause multiple paravertebral blocks
What factors do and do NOT contribute to the spread of local anesthetics in the subarachnoid space?
Significantly affect spread:
Controllable factors:
* Baricity of local anesthetic
* Patient position during and after block placement
* dose
* site of injection
Non-controllable factors:
* VOlume of CSF
* Density of CSF
Does NOT significantly affect spread
* Barbotage
* Increase intra-abdominal pressure (coughing, labor)
* speed of injection
* orientation of bevel
*addition of vasoconstrictor
* weight
*gender
What is the primary determinant of spread for epidural anesthesia?
Volume
Discuss the differential blockade of spinal anesthesia.
Different types of nerves have different sensitivities to the local anesthetic blockade.
* Autonomic fivers are blocked first
* Sensory fibers are blocked second
* Motor neurons are blocked last
Why is this important?
* Autonomic blockade is 2-6 dermatomes higher than sensory block
* sensory block is 2 dermatomes higher than motor block
How is differential blockade different with epidural anesthesia?
There is no autonomic differential blockade with epidural anesthesia
Sensory block is 2-4 dermatomes higher than motor block
Compare and contrast nerve fibers in terms of subtype, myelination, function, size, conduction velocity, and block onset.
Discuss the cardiovascular effects of neuraxial anesthesia.
Sympathectomy vasodilates the arterial and venous circulations, although it predominantly affects the venous capacitance vessels. Consequently, there is a reduction in venous return, cardiac output, and blood pressure. Volume loading with around 15mL/kg and vasopressors will minimize HoTN.
bradycardia is caused by:
* blockade of the preganglionic cardioaccelerator fibers at T1-T4. This promotes a relative increase of parasympathetic tone.
* unloading of cardiac mechanoreceptors (bezold- jarisch reflex)
*unloading of the stretch receptors in the SA node.
Discuss the respiratory effects of neuraxial anesthesia.
In healthy patients, neuraxial anesthesia has negligible effects on minute ventilation, tidal volume, respiratory rate, dead space, and arterial blood gas tensions.
Accessory muscle function is reduced. Impariment of the intercostal muscles (inspiration and expiration) as well as the abdominal muscles (ability to cough and clear secretions) will decrease pulmonary reserve. This is particularly important for the patient with severe COPD.
Apnea is usually the result of brainstem hypoperfusion (decreased blood flow to the ventilatory centers in the medulla).
Apnea is usually NOT the result of phrenic nerve paralysis or high concentrations of local anesthetic in the CSF.
How does neuraxial anesthesia affect the neuroendocrine response to stress?
By inhibiting afferent traffic originating from the surgical site, neuraxial anesthesia diminishes the surgical stress response. This reduces circulating levels of catecholamines, renin, angiotensin, glucose, thyroid-stimulating hormone, and growth hormone
How does neuraxial affect GI function?
The gut receives parasympathetic innervation from the vagus nerve (CN 10) and sympathetic innervation from the sympathetic chain (T5-L2)
Neuraxial local anesthetics inhibit the sympathetic tone, which increases the parasympathetic tone to the gut. This relaxes the sphincters in the gut and increases peristalsis.
How does neuraxial anesthesia affect renal and hepatic blood flow?
So long as systemic blood pressure is maintained, hepatic and renal blood flow and function are unchanged
What is the risk of neuraxial anesthesia in the patient with coagulopathy? What lab values are considered contraindications to a neuraxial technique?
Risk of spinal or epidural hematoma.
Neuraxial blocks are contraindicated in significant pathologic or therapeutic coagulopathic states.
* platelet count <100,00mm
* PT, aPTT, and/or bleeding time twice the normal value
what cardiac pathologies present a risk of hemodynamic collapse with neuraxial anesthesia?
Valve lesions with fixed stroke volume.
* severe aortic stenosis
* severe mitral stenosis
* Hypertrophic cardiomyopathy
what is the risk of a neuraxial technique in the patient with intracranial HTN?
There is an increased chance of brain herniation with sudden change in CSF pressure