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Philosophy III midterm palmer > Reflex models > Flashcards

Reflex models Flashcards

1
Q

Reflex models can be understood as different combinations of communications between somatic and visceral structures. What are the 4 models?

A

Somato-somatic
Somato-visceral
Visceral-somatic
Viscero-visceral

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2
Q

Local spinal effects of subluxation causes muscle hypertonicity/ imbalance, fixation etc.

A

Somato-motor/ somato-somatic; proprioceptive insult

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3
Q

Somato-visceral aka

A

Somato-autonomic

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4
Q

Somatic dysafferentation causes

A

Somatic efferent reflex effects

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5
Q

What is an increase in nociceptive afferent impulses combined with diminished proprioceptive impulses primarily from mechanoreceptors?

A

Somatic dysafferentation

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6
Q

Who believed that innervated somatic tissues in spine were a source of bombardment of neurologic signals leading to hyperstimulation or facilitation?

A

Korr

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7
Q

Who believed that nociceptive neurons are the afferents which produce facilitation?

A

Seaman

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8
Q

Facilitation aka

A

Hyperstimulation from bombardment of neurologic signals from spine

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9
Q

What is nociceptive spasm?

A

Isolated segmental spinal muscles which don’t act in coordination with rest of spine

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10
Q

Who proposed nociceptive facilitation?

A

Seaman

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11
Q

Facilitation can result in a?

A

Positive feedback cycle

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12
Q

What concept states that an effect of spinal fixation/hypomobility associated with subluxation may cause diminished afferent signals from somatic structures, primarily mechanoreceptors?

A

Deafferentation concept

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13
Q

What are the most common mechanoreceptors affected in the deafferentation concept?

A

Type I and II

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14
Q

With the diminished afferent signals of the deafferentation concept what happens to the CNS?

A

It is deprived of information needed for balance and coordination- ataxia and dizziness

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15
Q

Who stated that chiropractors don’t take pressure off nerves but put pressure on mechanoreceptors?

A

Carrick

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16
Q

Who stated that 99% of all neurologic syndromes are related to deafferentation?

A

Carrick

17
Q

Decreased mechanoreceptor input associated with decreased or restricted joint mobility causes increased perception of?

A

Pain

18
Q

Increased nociception and or decreased mechanoreception

A

Somatic dysafferentation

19
Q

Increased sympathetic stimulation of target tissues and organs

A

Sympatheticotonia

20
Q

Visceral afferents -> somatic efferents

A

Viscero-somatic reflex model

21
Q

Somatic afferents -> visceral efferents

A

Somato-visceral reflex

22
Q

Anterior horn effects

A

Somato-somatic

23
Q

Lateral horn effects

A

Somato-autonomic

24
Q

Modification of sympathetic nerve activity locally and globally

A

Sympatheticotonia

25
Q

Who stated that subluxation reduces brain/cortical summation?

A

Murphy

26
Q

Who stated that reduced brain summation dis-inhibits SNS?

A

Murphy

27
Q

Who stated that correcting the subluxation will reduce SNS activity, reduce catecholamine release, enhance Th1 response which will improve infection fighting and inhibit Th2 response which will reduce allergic/ atopic disease s&s?

A

Murphy

28
Q

Who stated that the most critical effect of manipulation is the quieting of sympathetic hyperactivity?

A

Korr

29
Q

Subluxation can result in a sustained increase in production of?

A

TNF-a

30
Q

Adjustments do what to TNF-a?

A

Decrease it

31
Q

Who stated that subluxation leads to dysautonomia?

A

Kent

32
Q

Who developed the compensation reaction- concept that hypomobility in a segment leads to hyper mobility elsewhere?

A

Jirout

33
Q

Change in central axis of motion was who?

A

Kapandji

34
Q

Loss of joint end play

A

Mennell

35
Q

Positional dyskinesia

A

Suh

Philosophy III midterm palmer (2 decks)

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