Red Eye 2 Flashcards
Degenerative Changes in Conjunctiva”
Pinguecula”
Pinguecula”
! An overgrowth of vascular conjunctiva onto
the cornea associated with destruction of the
superficial layers of the nasal cornea”
! May encroach upon the visual axis or cause
astigmatism “
! No treatment is needed. Lubrication with
artifical tears and occasional mild steroid
drops”
! Cause is unknown, but chronic sunlight
exposure and eye irritation may contribute”
! Welding is a significant occupational risk
Pterygium”
• Non-cancerous growth of the conjunctiva overlying
the sclera”
• Primary symptom is a painless area of elevated white
tissue with blood vessels on the inner or outer edge
of the cornea”
• Treatment – none unless the pterygium begins to
block vision –then Sx removal; wear uva & uvb
blocking sun glasses, wide brim hat”
• Due to collagen degeneration of the subconjunctiva
resulting from exposure! – Sunlight and wind!
– Risk factors are exposure to sunny, dusty, sandy, or
windy areas, i.e., farmers, fisherman, people living near
the equator!
– Rare in children
Sudden painful monocular loss of vision -
DDx
Acute (angle closure) glaucoma" " Acute anterior uveitis (iritis)" " Endophthalmitis" " Temporal arteritis" " Optic neuritis" " Keratitis (corneal inflammation)
Red Eye, pain, maybe change in Va”
Glaucoma”
Glaucoma”
Atrophy of retinal ganglion cell layer in presence of increased
intraocular pressure”
– Increased IOP has not been defined as causative”
– Risk factors”
• Fam Hx glaucoma”
• Increasing age”
• DM”
• Obesity”
• Hx of Ocular trauma”
– Leading cause of blindness among African Americans
• Primary open-angle glaucoma”
– Mc glaucoma”
– Occurs in 15% of over 80 crowd”
– Anterior Chamber (AC) anatomy appears normal, but
aqueous outflow is reduced”
– Progressive visual field loss begins peripherally and
occurs so slowly Pt may not be aware of it until late in
progress of disease”
– Intraocular tension is an effective screen
Glaucoma—Pathophysiology”
• Chronic (Wide-angle) Glaucoma”
– Common and degenerative in the elderly”
– Obstruction of canal of Schlemm “ diminished
outflow of aqueous humor”
– Intraocular pressure increases slowly and
asymptomatically”
– Increased pressure compresses blood flow to retina =
ischemia and damage to retinal cells”
• As pressure increases, more of the retina and optic disc are
damaged”
– Damage is irreversible, eventual blindness”
Types of Glaucoma
• Congenital" • Secondary" • Juvenile" • Chronic open angle" • Acute closed angle" • Many different types"
Acute glaucoma!
• Emergency" • Can be more gradual" • Red eye" • Achy, abdominal pain" • Misty vision" • Go from light into dark" • Small eye, shallow anterior chamber, pupil mid dilated, " • Iris/lens contact" • Push the iris forward" • Eye feels hard"
Acute closed angle glaucoma”
• Often starts in the evening. Especially in
those over 50 years.
• Severe pain first. Impaired vision and
haloes around lights. May have history of
past episodes relieved by going to sleep
(the pupil constricts during sleep).
• Refer even if attack spontaneously
resolves.
Acute Angle Closure Glaucoma (AACG) “
- Diagnosis
History - acute onset, higher risk in farsighted
Symptoms - pain, halos (around lights), visual loss (usually peripheral), nausea/vomiting
signs - conjunctival injection, corneal edema, mid-dilated/fixed pupil, increased IOP (normal: 10-20 mmHg)
Glaucoma”
• Angle-closure glaucoma”
– Ophthalmic emergency”
– Pt presents with red painful eye”
• Nausea and vomitting are common”
• Pupil is usually fixed in a mid-dilated position, and cornea
appears cloudy d/t pressure driven edema”
• The iris is bowed forward by posterior accumulation of
aqueous humor, sealing off the anterior chamber angle”
– Presence of crescent shadow”
• Risk factor: “”
– Narrowed anterior chamber angle”