Red Book - Abdominal Compartment Syndrome Flashcards

1
Q

Normal IAP

A

5-7 mmHg

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2
Q

Define Abdominal Perfusion Pressure

A

APP = MAP - IAP

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3
Q

Define Intra-abdominal hypertension

A

IAH - sustained or repeated pathologival elevation in IAP above 12 mmHg

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4
Q

Define Abdocompartment syndrome

A

Sustaned IAP >20mmHg

with or without APP<60mmHg

associated with NEW organ dysfunction

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5
Q

Grading of ACS

A

1 - 12-15mmHg
2 - 16-20mmHg
3 - 21-25 mmHg
4 > 26mmHg

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6
Q

How to classify ACS

A

Primary:

Due to pathology of abdomen

Secondary:

Extra-abdominal process

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7
Q

Catergorise the risk factors for ACS:

A

Diminished wall compliance –> surgery, tight closure, trauma, burns, proned

Increased abdominal contents –> Intra-luminal –> ileus, gastroparesis
extraluminal –> ascites, haemoperitoneum/pneumo

Capillary leak –> sepsis, trauma, burns, pancreatisis
hypothermia, acidosis
massive transfusion
positive fluid balance

Other: mechanical vent, PEEP>10, increased head angle, hypotension

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8
Q

Measure IAP

A

Direct or indirect

Direct - Needle into abdomen - trochar at laparoscopy

Indirect - Intra-vesicle pressure - urinary bladder catheter
25mls into catheter, attach drainage bag, cross clamp
16g needle to a transducer is attached to culture port site

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9
Q

Resp issues with IAH

A

Basal atelectasis, collapse, diaphragm splints
Reduced chest wall compliance

V/Q mismatch, hypoxeamia, hypercapnoea

PEEP - worsens venout return and CO

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10
Q

CVS effects of IAH

A

Raised IAP transmits to vasculature
Reduced CO –> reduced venous return
Leads to reduced APP

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11
Q

Neuro effects of IAH

A

Raised ICP as cerebral venous return impeded due to raised thoracic pressure

Hypoxaemia causes cerebral vasodilation and worsens ICP

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12
Q

Renal effects

A

Direct compression of renal vessels, reduces RBF

Pressure on tubules, reduces filtration gradient

Compensatory activates RAAS - worsening insultin

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13
Q

GI/hepatic IAH

A

Hypoperfusion and venous hypertension - bowel oedema

Ischaemia and translocation

Reduced hep art. flow

Bilary stasis

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14
Q

Principles of management

A

ABCDE approach treating specific abnromalities as found:

Principles:
Serial monitoring

Medical:
Improve wall compliance, sedate, paralyse, analgesia, positioning

 Fluid - avoid excess resus, consider RRT

 Evacuate intra-lumen - gastric decompression, laxatives, pro-kinetics

  Evacutae extra lumen - collections drained, paracentesis

  Organ support - APP>60 with vasopressors

Surgical - laparotostomy
negative pressure wound dressing

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15
Q

Complications of IAH

A

Untreated, 100% mortality

Multi organ failure

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16
Q

Define an open abdomen

A

WCACS Consensus - one that requires a temporary abdominal closure due to skin and fascia not being closed after laparotomy

17
Q

Why have an open abdo

A

Traditionally a last resort strategy in abdominal catostrophe where it cant be closed

Preferred now and preventitive in both trauma/non-trauma

18
Q

Indications for an open abdom

A

Severe necrotising pancreatitis

Abdominal sepsis

Damage control surgery (trauma)

Emergency vascular surgery

19
Q

Issues with an open abdomen

A

Nursing - skin care, wound moisture etc
positioning, turning
pain

Significant fluid loss and unrecordable fluid balance

Malnutrition - source protein/nitrogen loss

Infection

Visceral injury/adhesions

Ileus

Wall - hernias, cannot close, enterocutaneous fistula

20
Q

Options for temporary closure

A

1) Bogota bag (3 litre urology irrigation bag)
2) Negative pressure dressings/Vac
3) Synthetic mesh devices
4) Velcro sheath - Whittman pathc

21
Q

Managing the open abdo on ICU

A

General:
Nutrition, fluid balacne, nursing care
Resus to normal physiology

Correct hypothermia/coag/acidosis
Lung protective vent
Sedate, analgesia and blockade
(RASS -4)

Enteral feeding if GI working
Add in extra protein 2g/litre of nitrogen lost

Abx
Washouts

Stoma is soiling