RECOVER CPR Advanced life support Flashcards
What two monitoring device techniques should be started straight away when performing CPR?
electrocardiogram and end tidal CO2
When should ECG traces be monitored during CPR and why?
During pauses in compressions as it is susceptible to motion artifact
What is end tidal CO2 monitoring useful for?
identification of return of spontaneous circulation
chest compression quality
What are the 3 steps to start advanced life support?
Initiate monitoring (ECG and End Tidal CO2)
Obtain vascular access
Administer reversal agents
When should advanced life support be initiated?
After basic life support (chest compression and ventilation)
Why should alcohol never be used for ECG electrodes during CPR?
Alcohol should NEVER be used as a coupling material during CPR.
Alcohol is highly flammable and may ignite if electrical defibrillation is necessary.
Why should you make sure the ECG monitor is compatible with the electrical defibrillator?
ECG Monitor Compatibility
If using an electrical defibrillator, ensure that either the defibrillator’s ECG monitor is used or that any external ECG unit is safe for defibrillator use.
ECG systems can be damaged by the application of a defibrillator if they are not designed for this use. If you have both a defibrillator and a separate ECG monitor in your practice, contact the manufacturer of the ECG monitor to ensure that it is safe for use with your defibrillator.
Why is it important to carry out uninterrupted cycles when carrying out CPR?
Full 2-minute cycles of chest compressions without interruption are recommended to optimize perfusion to the brain and heart. It takes approximately 1 minute of uninterrupted chest compressions to attain a maximal, steady state blood flow to the heart and other tissues. Prematurely pausing chest compressions prevents development of this maximal, steady state blood flow and results in poor perfusion of the tissues. Chest compressions should never be stopped solely to evaluate the ECG, but instead the ECG should be evaluated during the brief pauses between 2-minute cycles.
Why are ECG signals highly susceptible to artifact?
ECG signals are highly susceptible to artifact due to electrical signals from other equipment in the environment and motion of the patient.
Can ECG readings be evaluated during chest compressions?
While chest compressions are being delivered, the artifact signal is much larger than the ECG signal, and it is generally not possible to accurately interpret the ECG. Attempts to interpret the ECG during chest compressions are likely to lead to erroneous conclusions.
How long should the pause between CPR cycles last?
This pause between cycles should last no more than 2 to 5 seconds and only long enough for a rhythm diagnosis to be made
When should the announcement of the person taking over chest compressions take place?
The recorder or team leader should announce that a change in compressor is about to occur 5 to 10 seconds before the change to allow the new compressor to get into position and to allow any available team members to look at the ECG monitor.
Should a pulse be felt during chest compressions during CPR?
Femoral pulse palpation may be useful during CPR and should be employed if there are enough team members available. Failure to feel a pulse accompanying chest compressions supports a reassessment of technique: and a reconsideration of compression style.
Weak pulses may be appreciated during adequate chest compressions and disappear during compressor rotation
During the pause when the compressor changes, what three major types of rhythms can be seen on the ECG?
The critical diagnostic goal is to determine which of the three major types of rhythms is present: (1) a perfusing rhythm, (2) a non-shockable arrest rhythm, or (3) a shockable arrest rhythm. Only perfusing rhythms are associated with pulses, palpable apex beats, and/or auscultable heart sounds.
How do you detect a perfusing rhythm during CPR?
Perfusing rhythms are repeated rhythms of any shape that generate pulses.
How many non shockable arrest rhythms are there?
There are two non-shockable arrest rhythms, neither of which is associated with effective cardiac output. These are called “non-shockable rhythms” because electrical defibrillation is not effective and may cause additional myocardial injury.
Pulseless electrical activity
Like pulseless electrical activity, asystole is a non-shockable rhythm that should never be defibrillated.
What is pulseless electrical activity characterised by?
Pulseless electrical activity (PEA) is characterized by ECG activity that appears coordinated and repeats at a rate of less than 200 per minute, but is not associated with the generation of a palpable pulse. Most commonly, PEA rhythms have rates of less than 50 per minute and are characterized by narrow QRS complexes. However, PEA can look like a normal sinus rhythm or have wide and bizarre complexes. It is occasionally referred to in older literature as electromechanical dissociation (EMD).
How many shockable arrest rhythms are there?
There are two shockable arrest rhythms, neither of which is associated with effective cardiac output. These are called “shockable rhythms” because electrical defibrillation may be effective and should be done as soon as possible.
pulseless ventricular tachycardia, ventricular fibrillation (VF) is a “shockable” rhythm because electrical defibrillation is the most effective treatment.
What rate would true ventricular tachycradia be?
over 200
What is the major difference between pulse ventricular tachycardia and pulseless electrical activity?
The major differentiating feature between pulseless VT and PEA is the rapid rate
How is pulseless ventricular tachycardia recognised on an ECG?
Pulseless ventricular tachycardia (pulseless VT) is recognized by organized, repeated, wide QRS complexes at a rate greater than 200 per minute without accompanying pulses. The major differentiating feature between pulseless VT and PEA is the rapid rate
How is ventricular fibrillation recognised on an ECG?
VF may be recognized by a wavy chaotic line on the ECG, and may be characterized as either fine ventricular fibrillation (low amplitude, high frequency) or coarse ventricular fibrillation (high amplitude, low frequency). The major differentiating feature of VF is the lack of a consistent, repeated waveform.
Is this statement true or false?
Pulseless electrical activity often has a faster rate than pulseless ventricular tachycardia
FALSE
VF may be recognized by a wavy chaotic line on the ECG, and may be characterized as either fine ventricular fibrillation (low amplitude, high frequency) or coarse ventricular fibrillation (high amplitude, low frequency). The major differentiating feature of VF is the lack of a consistent, repeated waveform.
What does the end tidal CO2 reflect?
The end tidal CO2 (EtCO2) reflects the amount of CO2 in the exhaled air at the end of breath, and is similar to the partial pressure of CO2 in the arterial blood.
What two factors determine the end tidal CO2 factors?
The EtCO2 value will be determined by two main factors: (1) minute ventilation (the product of respiratory rate and tidal volume), and (2) the amount of blood returning from the tissues to the lungs.
Why can the ETCO2 be used to monitor the quality of chest compressions?
Because it is directly related to cardiac output, the EtCO2 can be used to monitor the quality of chest compressions during CPR.
During CPR, the EtCO2 reflects the amount of blood returning from the body to the lungs. EtCO2 will be directly related to the cardiac output. With higher cardiac output, the EtCO2 will be higher, while with lower cardiac output, the EtCO2 will be lower.
What can a sudden increase in ETCO2 indicate (30mmHg)?
Cardiac output increases dramatically upon return of spontaneous circulation ROSC; therefore, a sudden increase in EtCO2 to > 30 mmHg provides a vital clue that ROSC has occurred
What is the only value that can be accurately monitored during chest compressions?
Unlike the ECG, auscultation, direct pulse palpation or echocardiography, the end tidal CO2 can be evaluated while chest compressions are occurring
If there is asudden increase in ETCO2 that may indicate a return of spontaneous circulation what should be done before stopping chest compressions?
In any patient with a sudden increase in EtCO2 during chest compressions, it is important to assess the presence of a pulse before stopping chest compressions. In addition, you can assess for other signs of ROSC, such as voluntary motor movements, chewing on the endotracheal tube, return of the corneal or palpebral reflex, or signs of consciousness. However, until a spontaneous pulse has been definitively identified, chest compressions should be continued.
How can you accurately verify correct ET tube placement during CPR?
Verify accurate ETT placement during CPA using one of the following approaches:
Direct visualization of chest movements during a positive pressure breath
Direct visualization of placement through the arytenoid cartilages during intubation
What are the two main reasons an ETCO2 might read 0 when performing chest compressions?
There are 2 main reasons the EtCO2 may read zero during CPR:
The ETT is in the esophagus
The cuff on the ETT is not sufficiently inflated
How can ventilation affect ETCO2 in CPR?
If ventilation is provided at a rate faster than 10 bpm, or tidal volumes larger than 10 ml/kg are delivered, the EtCO2 will drop. In this case, the decreased end tidal CO2 is due to increased minute ventilation, not lower cardiac output. In order for EtCO2 to be a useful indicator of cardiac output during CPR, breaths must be delivered consistently at a rate of approximately 10 per minute, as described in the basic life support algorithm.
What is PvCO2?
peripheral venous CO2 partial pressure
Can PvCO2 be substituted for EtCO2 during CPR?
PvCO2 (peripheral venous CO2 partial pressure) should not be used as a substitute for EtCO2, as there is often very little association between PvCO2 and EtCO2 during CPA. During CPR, the PvCO2 may be much higher than the EtCO2 because of poor blood flow in the periphery and buildup of CO2 in these peripheral tissues. Using PvCO2 rather than the EtCO2 may lead the rescuer to believe that compression quality is good when, in fact, the EtCO2 may actually be low due to poor blood flow to the lungs
What two main purposes are changes in ETCO2 useful for?
Changes in EtCO2 will reflect changes in blood flow, and are therefore useful for two main purposes during CPR: (1) evaluating the efficacy of chest compressions, and (2) identifying ROSC.
How does compression quality need to be improved if the ETCO2 is below 15 mmHg during CPR?
Compression quality (rate, depth, chest recoil) should be improved during CPR in any patient with EtCO2 values less than 15 mmHg.
How can an improvement in rate, depth and chest recoil be achieved during CPR when the ETCO2 in low?
Increasing compression depth up to one-half the width of the chest
Verifying that the compression rate is between 100 and 120 compressions per minute
Ensuring that compressions are being performed at the correct location on the chest based on the patient’s conformation and size
Taking care not to lean on the chest between compressions to allow for full elastic recoil and filling of the heart
IF chest compressions during CPR are being performed correctly, what elese needs to be assessed if ETCO2 is low?
Remember that increased minute ventilation (respiratory rate greater than 10 per minute and/or large tidal volumes) will decrease EtCO2, even if chest compressions are being done correctly. Therefore, in order to use EtCO2 as a measure of chest compression efficacy, ventilation must be delivered consistently at a rate of 10 breaths per minute, as described in the BLS algorithm.
Other monitoring devices, such as indirect blood pressure monitors and pulse oximeters, are often desirable in emergency and critical care medicine, but during CPR, these devices become less useful.
Why do you think that is?
During CPA, indirect blood pressure monitors and pulse oximeters are not useful due to the lack of adequate pulse pressure.
These devices rely upon pulsatile arterial blood flow that is detectable by the machine, and during CPR, there is typically inadequate pulsatile flow for accurate readings. Attempts to determine either indirect blood pressure or oxygen saturation will result in inaccurate or undetectable results. In addition, like the ECG monitor, both indirect blood pressure monitors and pulse oximeters are highly susceptible to the motion artifact present during chest compressions.
Finally, these monitors may distract resuscitative efforts by providing extraneous or inaccurate information to the team leader.
Which of the following monitoring devices is LEAST likely to be helpful during CPR?
pulse oximeter
capnograph
ECG
Because of motion artifact and lack of a consistent peripheral pulse, the pulse oximeter is unlikely to be of value during CPR. In addition, attempts to get a good signal from a pulse oximeter during CPR can distract team members from more important tasks. On the following screen, watch the video for an example of how focusing on the pulse oximeter can distract the team from doing effective CPR.
IF a central line is not available to use, in what order should peripheral veins be used?
If a patent catheter is available, it should be used. If more than one catheter is present, the catheter closest to the heart should be used in order to improve drug delivery time. A central line will deliver drugs into the venous system closest to the heart and should be used to administer CPR drugs, if available. If only peripheral catheters are available, they should be prioritized in the following order:
Jugular
Cephalic
Saphenous
IF there are several catheters which one should be used for drug delivery during CPR and why?
The catheter closest to the heart should be used in order to improve drug delivery.
dog with veins outlined
In animals without a pre-existing catheter, BLS should begin promptly, and the team leader should provide direction as to the type of vascular access to be obtained. In cases with adequate team members, more than one individual may work on IV catheter placement at separate sites.
How much flush should follow drug administrationin CPR?
Regardless of the location of the catheter, following drug administration the catheter should be flushed with an isotonic crystalloid solution to facilitate flow to the heart and ultimately to the tissues. While there is no specific evidence regarding the volume of flush, in general 3-5 cc in cats, 5-10 cc in small to medium sized dogs, and 10-15 cc in large and giant breed dogs is reasonable.
What are the options for vascular access for drug administration in CPR?
Options for vascular access include:
Percutaneous peripheral venous
Venous cutdown
Intraosseous (IO)
What size catheter can be used for drug administration with CPR?
A smaller than average catheter (e.g., 20-22 G) may be placed more easily than a larger catheter. The initial small-bore catheter can be used during CPR for drug administration. Following ROSC, a larger bore catheter may be placed if needed.
How is an intravenous catheter secured when placed using a cut down?
The catheter is then secured with sutures to avoid dislodgment.
When should intraosseous catheter placement be considered during CPR?
Placement of an intraosseous catheter may be considered when prompt IV access is not practical or possible, or when venous catheterization has been unsuccessful. IO catheters facilitate rapid drug delivery into the central circulation.
What are the common sites for intraosseous catheter placement? What is the preferred site and why?
Many sites are possible for placement of IO catheters. Common sites include the humerus, the femur, and the tibia. Because the humerus is close to the heart, IO catheters in this bone will provide the fastest CPR drug delivery to the sites of action.
Due to the proximity of the humerus to the thorax, where chest compressions are occurring, it is often easier to use the hindlimb during CPR. However, recall that the humerus is closest to the heart, so if it is accessible, it is the preferred site of IO catheter placement.
What size needle can be used for an intraosseous catheter for puppies and kittens?
For kittens and puppies, an 18 or 20 gauge standard needle without a stylet may be used. Alternatively, the use of a spinal needle with stylet will reduce the risk of plugging the needle with bone during placement.
When would intratracheal drug administration be indicated?
In general, IV or IO administration is preferred due to the variable absorption of drugs administered into the trachea. However, intratracheal drug administration may be considered when there is no IV or IO access readily available.
What emergency drugs can be given intratracheal?
Drugs that may be given IT include epinephrine, atropine, and vasopressin.
What emergency drugs should never be given intratracheal?
However, sodium bicarbonate should never be administered IT.
If drugs have been given intratracheal should you stop attempting to get intravenous access?
Recall that IT administration of drugs is a stopgap measure as you continue to attempt to obtain direct vascular access via IV or IO catheterization. As soon as IV or IO access is obtained, drugs should be re-dosed via the IV or IO route.
Is the same amount drug doses given intratracheal compared to other routes?
There is limited experimental evidence to guide IT dosing, but in general, doses of two to ten times IV/IO doses are recommended
how many ml of saline should emergency drugs be diluted with when given intratracheal?
IT drugs should be diluted in 5 to 10 ml of either sterile water or saline.
Why should drugs that are given intratracheal be diluted?
In order for IT drugs to be effective, they must be absorbed across the tracheal/bronchial epithelium and reach the bronchial circulation.
How do you administer drugs intratracheal?
A long catheter, such as a 5 French red rubber catheter, is fed through the endotracheal tube.
The diluted drug is administrated through the red rubber catheter. The syringe delivers a pulse of 5 to 10 ml of air into the red rubber catheter to blow as much of the fluid out of the tube as possible.
The catheter should then be withdrawn from the endotracheal tube and the ventilation bag re-connected. Two breaths should be administered rapidly to help disperse the medication throughout the pulmonary tree.
Which of the following drugs should never be administered IT?
Drugs that may be given IT include epinephrine, atropine, and vasopressin. However, sodium bicarbonate should never be administered IT. Recall that IT administration of drugs is a stopgap measure as you continue to attempt to obtain direct vascular access via IV or IO catheterization. As soon as IV or IO access is obtained, drugs should be re-dosed via the IV or IO route.
Intracardiac administration of emergency drugs is preferred over intratracheal administration.
True or false?
Given the evidence that intratracheal (IT) drug administration leads to therapeutic plasma concentrations of most emergency drugs and the risks associated with blind intracardiac (IC) injections (such as laceration of coronary vessels), the IT route is preferred over the IC route during CPR.
The next card is a summary on access on drug administration
Prompt vascular access is essential to successful outcomes in CPR. Multiple options are available, including percutaneous peripheral catheterization, venous cutdown, and intraosseous catheterization.
If multiple catheters are available, the catheter that is closest to the heart and therefore provides the most rapid delivery of drugs to the central circulation should be used.
If percutaneous catheterization is not immediately successful, a cutdown procedure may be performed to achieve venous access.
Rapid vascular access may be obtained using intraosseous (IO) catheters as well.
If venous or IO catheterization is not possible, certain drugs may be administered intratracheally. In this scenario, attempts at IV or IO catheterization should continue and drugs re-dosed as soon as vascular access is achieved.
Why do you think it is important to administer reversal agents intravenously?
Reversal Agents for Sedatives: Intravenous Drug Delivery
Intramuscular or subcutaneous administration of reversal agents is not recommended in patients with CPA due to slow, unpredictable uptake. Therefore, intravenous or intraosseous administration of reversal agents is recommended in patients with CPA.
You sedated a dog to repair a laceration with hydromorphone and dexmedetomidine. After the dog was placed in a cage, he is found in CPA.
Because the drugs were administered over 1 hour ago, there is no reason to administer the reversals.
True or False?
In general, if a reversible sedative or anesthetic agent has been administered to an animal with CPA, early intravenous treatment with a reversal agent is indicated. Even if the drug was administered several hours before the CPA occurred, reversal is generally safe and may still have some positive effects.
A patient with a GDV presents to your clinic. You sedate with hydromorphone and trocharize the stomach to stabilize him. You are reassessing him after trocharization and he begins taking agonal breaths and then stops breathing. After initiating basic life support, your next step is to administer naloxone to reverse the hydromorphone.
True or False?
Remember from the RECOVER CPR algorithm that the first step in initiating Advanced Life Support is to connect the ECG and EtCO2 monitors. Next we should verify that our vascular access is present and patent. Only after those 2 steps are completed should reversal agents be administered. Reversal agents do not directly treat CPA, but are adjunctive therapies that may improve the chances of achieving ROSC. Always remember to follow the steps of the algorithm in order.
For patients in CPA, IV fluid therapy should be reserved only for those patients with documented or suspected hypovolemia.
Why do you think that is?
IV fluid therapy is helpful for improving cardiac output in patients with documented or suspected hypovolemia, but may be detrimental in euvolemic patients with CPA due to reduced blood flow and oxygen delivery to core organs.
What is the amount of blood flow and oxygen delivered to a tissue bed determined by?
The amount of blood flow, and hence oxygen delivered, to a tissue bed is determined by the force driving blood into the tissue bed, the resistance to flow offered by the vasculature in the tissue bed, and the force pushing back at the outflow of the tissue bed
What is the driving force for blood flow to most tissue?
The driving force for blood flow to most tissues is the mean arterial blood pressure (MAP), and the outflow force pushing back is the central venous blood pressure (CVP)
How does intravenous fluids help in a patient that is hypovolaemic with spontaneous circulation?
In hypovolemic patients with spontaneous circulation, IV fluid therapy can improve cardiac output and increase MAP because of the relationship between stretch of the ventricles and the subsequent ability of the ventricle to contract more fully due to that stretch. With increased intravascular volume provided by IV fluid therapy, the ventricles experience greater stretch and hence can contract more and increase cardiac output.
Explain how intravenous fluid therapy can be detrimental in patient in cardiopulmonary arrest that is euvolaemic?
If a patient in CPA is euvolemic and fluids are administered intravenously, it is unlikely that cardiac output will increase significantly because the ventricles are not spontaneously contracting. Therefore, fluids administered IV will accumulate in the venous circuit, which is more distensible than the arterial circuit, resulting in increased central venous pressure (CVP) rather than increased MAP. This increase in CVP leads to higher pressure on the outflow side of the tissue bed, resulting in decreased blood flow and oxygen delivery to the tissues. Therefore, in patients in CPA that are euvolemic, fluids should be administered conservatively if at all, until ROSC is achieved and the heart is again contracting spontaneously.
How does intravenous fluids help with hypovolaemia in patients in cardiopulmonary arrest?
For patients in CPA with known or suspected hypovolemia, boluses of IV fluids may be beneficial to provide additional circulating volume. Although CVP will increase, the increased stretch of the ventricles will lead to improved cardiac output, increasing MAP and improving blood flow to the tissues. Patients with trauma, severe vomiting and/or diarrhea, or hemorrhage are likely to benefit from fluids.
Can glucocorticoids be used in patients with cardio pulmonary arrest?
Potentially Detrimental Side Effects of Glucocorticoids
There is a growing body of evidence that corticosteroids may have detrimental effects in many cases, and given the weak evidence supporting their use, glucocorticoids should be avoided unless there is a clear indication. Glucocorticoids have many potentially detrimental side effects that may be particularly dangerous in patients with CPA. Because they are counter-regulatory hormones in glucose metabolism, glucocorticoids commonly cause hyperglycemia, which can perpetuate brain injury in patients with brain ischemia, common in CPA. Glucocorticoids also very commonly lead to gastrointestinal ulceration in dogs, causing significant blood loss and/or bacterial translocation leading to septicemia. In both dogs and cats, they decrease prostaglandin production in the kidney, compromising renal perfusion and potentially leading to ischemic acute kidney injury.
What patients may benefit from glucocorticoid administration in cardiopulmonary arrest?
Patients That May Benefit from Glucocorticoids
However, glucocorticoids may be warranted in certain patient populations. For instance, animals that arrest in association with anaphylactic shock may benefit from anti-inflammatory doses of glucocorticoids (0.1 mg/kg dexamethasone SP IV). Patients with concurrent hypoadrenocorticism (i.e., Addison’s disease) will also benefit from glucocorticoids. Finally, animals that develop critical illness-related corticosteroid insufficiency (CIRCI) after ROSC may also benefit from physiologic steroid therapy. Corticosteroids should only be used during CPR if the patient is known to have or is highly suspected of having one of these disorders. For the vast majority of patients, corticosteroids should not be administered during CPR.
What are the potential benefits of open chest cardiopulmonary resuscitation?
Potential benefits of open-chest CPR (OCCPR):
Better cardiac output
Higher rates of ROSC
Direct assessment of the heart
What may be some of the specific indications for open chest CPR?
Specific indications for OCCPR include:
Pleural space disease such as large volume pleural effusion or pneumothorax
Pericardial effusion
Patients already under anesthesia for thoracic or abdominal surgery
Giant breed dogs with round chest conformations, such as Mastiffs, Saint Bernards, and some Great Danes
What needs to be carried out to perform an open chest CPR?
OCCPR requires:
A rapid emergency thoracotomy for direct cardiac massage
Closure of the thorax
Post-cardiac arrest care
Why is pleural space disease an indication for open chest CPR?
Pleural space disease, such as large volume pleural effusion or pneumothorax, is an indication for OCCPR. Because of the markedly increased intrathoracic pressure in patients with severe pleural space disease, venous return to the heart is compromised and external chest compressions are unlikely to result in forward flow out of the heart. Opening the chest relieves the pressure exerted by the pleural space disease and allows the heart to fill so that chest compressions are effective.
Why is pericardial effusion an indication for open chest CPR?
Pericardial effusion is an indication for OCCPR because cardiac tamponade impedes return of blood to the atria, resulting in minimal cardiac output during external chest compressions. OCCPR allows the rescuer to open the pericardium and remove the pericardial effusion.
Why is thoracic or abdominal surgery an indication for open chest CPR?
OCCPR should be initiated in patients under anesthesia for thoracic or abdominal surgery. For patients undergoing thoracotomy, easy access to the heart for direct cardiac compressions is already available. For patients having abdominal surgery, rolling them into lateral recumbency for chest compressions is not feasible, and direct cardiac compressions can easily be achieved by incising through the diaphragm and compressing the heart through the diaphragmatic incision.
Why is round chest comformations in dogs an indication for open chest CPR?
Giant breed dogs with round chest conformations
In giant breed dogs with round chest conformations, OCCPR is more likely to be of benefit than closed-chest CPR. This is because they have poorly compliant chests that are difficult to compress adequately to generate enough intrathoracic pressure to provide substantial blood flow using the thoracic pump approach.
On the other hand, in cats or in small dogs (<10 kg), in which external chest compressions are more effective due to higher chest compliance, OCCPR is not indicated; in these animals, internal cardiac massage is more challenging due to the small size of their thoracic cavities.
You’re seeing a Gordon Setter with a large volume pyothorax. The owner has authorized any life-saving treatment you feel is in the dog’s best interest.
The dog develops CPA during IV catheter placement. What should you do?
immediate open chest CPR? or close chested CPR?
If OCCPR is to be pursued, it is best to begin the procedure as early in the course of a resuscitative effort as possible. This is why it is important to obtain a CPR code (red, yellow, or green) in any patient admitted to the hospital as discussed in the BLS course. In this case, because the dog has pleural space disease, OCCPR should be pursued.
What is the first step of advanced life support?
ECG
ECG monitoring is an essential part of ALS. The ECG rhythm diagnosis is the primary decision point in the ALS portion of the CPR algorithm. It is therefore important that attachment of ECG leads be a priority after BLS has been started.
Tom presented on emergency for lethargy.
On physical exam, he had pale oral mucous membranes, poor pulse quality, and was assessed to be 8-10% dehydrated. Additionally, he had a large firm bladder. Immediately following palpation of the bladder, Tom screamed and collapsed in CPA.
What is your first step?
start one-handed compressions, intubate and ventilate
While Tom is likely suffering from a urethral obstruction, CPA requires immediate support of the cardiovascular system. Additionally, while intravenous fluids will likely be useful in this case, the first priority must be restoration of circulation via chest compressions in the patient with CPA. Finally, while determining the arrest rhythm will help guide ALS therapy, the first priority in any patient in CPA is initiation of BLS.
Now that you have started BLS, which of the following monitoring devices would be high priorities?
ECG
While indirect blood pressure monitors and pulse oximeters are often desirable in emergency and critical care medicine, during CPR, these devices become less useful due to the lack of adequate pulse pressure. These devices rely upon pulsatile arterial blood flow, and during CPR, adequate pulsatile flow for device function is typically absent. Attempts to determine either indirect blood pressure or oxygen saturation will result in inaccurate or undetectable results. Equally importantly, attempts to use these monitors may distract the people available for assisting in resuscitative efforts, and provide extraneous and inaccurate recommendations for the team leader.
In placing an IV catheter in Tom (a cat), what is your top priority in cardiopulmonary arrest?
Prompt vascular access is essential for increasing the likelihood of a successful outcome of CPR. Immediate venous cutdown or placement of an IO catheter should be considered when prompt percutaneous access is not practical or possible, or when venous catheterization has been unsuccessful.
Tom’s (a cat) owner is insistent upon the best possible outcome and asks you to perform open-chest CPR. How would you respond?
Explain that open chest CPR is not warranted in cats
Open-chest CPR (OCCPR) has only been associated with a higher rate of ROSC than closed-chest CPR in experimental studies in dogs with VF. It is unlikely to be of benefit over closed-chest CPR in cats or in small dogs (<20 kg) because these smaller animals have greater chest compliance and the thoracic cavity is very small, making internal chest compressions very difficult.
What would be a reasonable initial IV fluid plan for Tom (a cat) during CPR? 8-10% dehydrated
Tom’s initial physical exam was consistent with hypovolemic shock (pale mucous membranes, prolonged CRT, poor pulse quality) and he would likely benefit from an initial fluid bolus. 20-30 ml/kg represents 1/3-1/2 of his total blood volume (60 ml/kg in a cat) and would be a reasonable fluid bolus in this case. 90 ml/kg is an excessive amount for an initial bolus, and a fluid rate of 2 ml/kg is unlikely to address his hypovolemia rapidly enough. If Tom had been euvolemic, withholding fluids during CPR would have been most appropriate, but given the evidence of hypovolemia, a 20-30 ml/kg bolus over 15 minutes is the best choice.
Ruby has been progressively lethargic and anorexic for several days. On initial examination, she is weak with bounding pulses and icterus. An IV catheter is placed and point-of-care blood work shows that she has a hematocrit of 14%, total solids of 7.2 gm/dl, icteric serum and autoagglutination. As you are finishing placing an IV catheter, Ruby vomits and collapses in cardiopulmonary arrest. What is your first step?
Start chest compressions, intubate and ventilate
While Ruby is likely suffering from an immune-mediated hemolytic anemia, CPA requires immediate support of the cardiovascular system. While steroids, blood products, and a fluid bolus may all be part of her initial therapy, the first priority in any patient in CPA is initiation of BLS.
Explaijn what pulseless electrical activity would look like on a ECG monitor?
There are consistent, repeated QRS complexes that are narrow at a rate of less than 200 per minute with no associated pulses, so this is most likely pulseless electrical activity. Had the rate been greater than 200 per minute with wide complexes, pulseless VT would have been more likely.
According to the RECOVER CPR algorithm, which of the following would be the first step in initiating advanced life support in a patient with a patent intravenous catheter in place that arrested due to an inadvertent overdose of hydromorphone?
attach ecg leads
During CPR, what is the minimum EtCO2 associated with good quality chest compressions?
15 mm Hg
Which of the following is the best choice to improve contact between an ECG alligator clip and a patient’s skin during CPR?
Electrode gel
A patient arrests during induction for a spay surgery. After the first cycle of BLS, you diagnose asystole and decide to administer epinephrine. Which of the following would be the best route of administration?
Cephalic intravenous catheter
Via a red rubber catheter through the endotracheal tube
Intracardiac injection
Lateral saphenous intravenous catheter
Cephalic intravenous catheter
A patient was premedicated for an enucleation and spay with dexmedetomidine and methadone. Surgery was complicated and took 3 hours. One hour after extubation, the patient experienced CPA. Which of the following statements is true?
Because it has been 4 hours since administration of the premedication, reversal agents are unlikely to be effective
Flumazenil and naloxone are indicated
Atipamezole and naloxone are indicated
Atipamezole and flumazenil are indicated
Atipamezole and naloxone are indicated
The ECG is the most useful monitoring device for diagnosing a return of spontaneous circulation during CPR. true or false?
false
Because there is stronger evidence of harm than of benefit in patients with CPA, the use of corticosteroids during CPR is not recommended. true or false?
true
Which is the preferred approach for open-chest CPR in most patients?
Left lateral thoracotomy (right lateral recumbency)
Right lateral thoracotomy (left lateral recumbency)
Median sternotomy (dorsal recumbency)
Right lateral thoracotomy (left lateral recumbency)
You admit a 40kg greyhound to the hospital for a GI foreign body surgery. When discussing the option of open-chest CPR, the owner asks if it would be reasonable to try closed-chest CPR and convert to open-chest CPR if initial attempts are unsuccessful. Which of the following is the correct response to that question?
Because of the morbidity associated with open-chest CPR, it is reasonable to start with closed-chest CPR and convert to open-chest if there is no response
Because her dog is keel-chested, better cardiac output can be achieved with closed-chest CPR than with open-chest CPR
Her dog is more likely to have a good outcome with immediate open-chest CPR than with closed-chest CPR
Her dog is more likely to have a good outcome with immediate open-chest CPR than with closed-chest CPR
In euvolemic patients in cardiopulmonary arrest, a fluid bolus should be given as soon as possible after CPR is initiated to improve perfusion to the core organs.
false
Are shockable rhythms common in dogs and cats?
Remember that non-shockable rhythms are the most common arrest rhythms in dogs and cats.
What 3 different types of rhythms are you focusing on when looking at the ECG
ECG analysis during CPR is focused on differentiating three major types of arrest rhythms:
Perfusing rhythms associated with pulses
Non-shockable arrest rhythms that do not require electrical defibrillation
Shockable arrest rhythms that require electrical defibrillation
When should the ECG be reviewed?.
During the brief pause in compressions between cycles of BLS, the team leader should interpret the ECG and announce the rhythm diagnosis. Agreement should be reached among all team members as to the ECG rhythm diagnosis; if consensus is not reached immediately, chest compressions should be re-started and the discussion about rhythm diagnosis can continue during the next cycle of compressions. Ultimately, if consensus cannot be reached, the team leader should make the diagnosis after considering input from all team members. Chest compressions should never be stopped during a cycle of BLS strictly to evaluate the ECG. However, if there is a sudden rise in EtCO2, and a strong, palpable pulse is noted during a cycle of BLS, it is reasonable to briefly pause chest compressions to review the ECG and palpate the pulse to determine if ROSC has truly been achieved.
What should you do if there are repeated complexes and a palpable pulse? What does this mean?
If repeated complexes are present, quickly palpate the pulse or apex beat to determine if there is blood flow associated with the complexes.
If there are pulses, this is a perfusing rhythm and the patient has achieved a return of spontaneous circulation, so chest compressions can be discontinued.
How long should pulse assessment take? What should you do if you have doubt there is a pulse?
Pulse assessment should take no more than five seconds. If there is any doubt as to whether a pulse is present, assume that one is not present and continue BLS.
If a patient receiving CPR has repeated ECG complexes on the ECG but no palpable pulse and a HR of less than 200 what is this called?
A rate of less than 200 per minute is most consistent with pulseless electrical activity (PEA), a non-shockable rhythm.
In this case, the PEA/Asystole ALS algorithm should be followed. We will discuss treatment of PEA/Asystole in detail later in the module.
Is pulseless electrical activity shockable or not?
not shockable
If a patient receiving CPR has repeated ECG complexes on the ECG but no palpable pulse and a HR over 200 what is this called?
pulseless ventricular tachycardia
Is ventricular tachycardia shockable or not?
shockable