Quiz 5 (second half) Flashcards

1
Q

what does Phlegmasia mean?

A

greek word phlegma meaning inflammation

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2
Q

what does PAD stand for?

A

Phlegmasia alba dolens

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3
Q

what is Phlegmasia alba dolens used for?

A

in the medical literature in reference to extreme cases of lower-extremity deep venous thrombosis (DVT) that cause critical limb ischemia and possible limb loss

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4
Q

what does Phlegmasia alba dolens (PAD) describe?

A

the patient with swollen and white leg becuase of early compromise of arterial flow secondarty to extensive DVT

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5
Q

what is Phlegmasia alba dolens also known as?

A

milk leg

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6
Q

who does milk leg effect?

A

women in the third trimester of pregnancy or post partum

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7
Q

what does PCD stand for?

A

Phlegmasia cerulean dolens

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8
Q

when is the diagnosis of phlegmasia alba dolens (PAD) or phlegmasia cerulaea dolens (PCD) made?

A

clinical grounds in patients who have extensive DVT on imaging

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9
Q

is PAD or PCD more advanced?

A

Phlegmasia cerulea dolens (PCD)

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10
Q

what is PCD considered a precursor for?

A

frank venous gangrene

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11
Q

what is PCD characterized by?

A

severe swelling and cyanosis and blue discoloration of the extremity

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12
Q

points to remember about Phlegmasia alba dolens?

A
  • known as milk leg or white leg
  • blanching of extremities, edema, and discomfort
  • leg is pale and cool
  • diminished arterial pulse due to spasm
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13
Q

points to remember about Phlegmasia cerulen dolens?

A
  • painful blue edema
  • pain and cyanosis
  • DVT in deep, superficial and collateral veins
  • extends into capillaries 40-60% of the time
  • irreversible ischemia, necrosis and gangrene
  • surgical emergency
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14
Q

what is Trousseau’s sign?

A

concerns hypercoagulability associated with cancer

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15
Q

what is DVT in occult malignancy based on

A

the finding of spontaneous venous thrombosis in patients with underlying malignancy

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16
Q

what us a concern when patients present with DVT and have no known risk factors?

A

they may have an occult malignancy

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17
Q

is DVT with or without malignancy more extensive and aggresive?

A

DVT associated with malignancy

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18
Q

what does DVT malignancy clinincaly demonstrate?

A

a very swollen and painful extremity

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19
Q

Patients with DVT related to Trousseau’s syndrome usually clinically manifest cancer within ____________

A

1-2 years

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20
Q

where do malignancies associated with venous thrombosis typically arise in?

A
  • breast
  • GI tract
  • GU tract
  • lung
  • brain
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21
Q

why can detection of thrombus in the SVC be difficult?

A

due to the frequency of it occuring centrally in the vein

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22
Q

what makes compression impossible for the SVC?

A

overlying bones (clavicle)

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23
Q

what does diagnosus of subclavian vein obstruction rely on?

A

secondary signs of obstruction

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24
Q

what does the SCV look like on spectral?

A

close to the right atrium, so pressure flucuations in the atrium are readily tramsitted into the vein, producing a pulsatile waveform

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25
Q

when is the normal pulsatility of subclavian vein obstruction blunted?

A

when there is a venous obstruction between the heart and the site where doppler waveform is obtained

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26
Q

what is diagnostic for subclavian vein obstruction?

A

demonstrating the asymmetry between the right and left SVC waveform

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27
Q

what should be apart of the SVC thrombosis examination?

A

many involve IJV thrombosis

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28
Q

what should be a potential cause in subclavian vein obstructon?

A

extrinsic compression as in large lymoh nodes due to breast cancer

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29
Q

if the SVC is the site of obstruction, what may occur?

A

collateral flow in the internal mammary veins may occur (flow appears reversed)

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30
Q

what is Lemierre’s syndrome?

A

jugular vein thrombosis

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31
Q

where do we see Lemierre’s syndrome?

A

in a patient with a neck infection and enlarged lymph nodes

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32
Q

what does IJV compression result in?

A

thrombus formation due to stasis

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33
Q

what is behcet’s disease/syndrome?

A

inflammation in blood vessels (veins and arteries)

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34
Q

what is the etiology of behcet’s disease/syndrome?

A

unknown or is autoimmune (bodys immune system attacks healthy cells)

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35
Q

what does behcet’s disease/syndrome cause in the arms and legs?

A

redness, pain, and swelling in the arms and legs

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36
Q

what can behcet’s disease/syndrome cause in the vessels?

A

blood clot to form

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37
Q

what can imflammation in large arteries lead to?

A

complications such as aneurysms or blockage of a vessel

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38
Q

what else does behcet’s disease/syndrome affect?

A

mouth, skin, genitals, eyes, joints, digestive system and the brain (causeing a stroke)

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39
Q

The etiology of IVC thrombus mirrors what?

A

that of DVT in general

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40
Q

what does IVC thrombus relate to?

A

specific situations relate to the IVC only but the wide variety of these situations all relate in one or more ways to VIRCHOWS CLASSIC DESCRIPTION

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41
Q

Virchow’s classic description

A
  • wall damage
  • hypercoagulability
  • venous stasis
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42
Q

what are numerous malignancies associated witha?

A

IVCT (IVC thrombus)

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43
Q

what is the most familar malignancy associated with IVCT?

A

renal cell carcinoma

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44
Q

what is the path of spread of RCC?

A

the intravascular tumor extends from the renal vein and can propagate as far as the heart

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45
Q

what are Other genitourinary tumors that reportedly cause IVCT?

A

seminomas and teratomas

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46
Q

what can any structure that is anatomically related to the IVC generate?

A

either a direct compression or vascular invasion

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47
Q

what have all presented in association with IVCT?

A
  • retroperitoneal leiomyosarcoma
  • adrenal cortical carcinoma
  • renal angiomyolipooma
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48
Q

hepatic hemagioma has caused IVCT from __________

A

extrinsic compression

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49
Q

what is a risk factor for DVT and the extension into the IVC?

A

malignancy itself

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50
Q

what may extrinis compression result from?

A

nontumoral sources and increase the likelihood of IVCT

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51
Q

what generates both venous stasis and turbulent flow?

A

the distortion of the normal caval anatomy

52
Q

what is an uncommon clinical situation that can compress the IVC?

A

abdominal aortic aneurysms

53
Q

what can hepatic abscesses form?

A

amebae or echinococci and generate thrombosis of the IVC from compression

54
Q

what disease has been reportadly clinnically associated with thrombosis of the IVC?

A

polycystic disease of the right kidney

55
Q

what with the pancreas may cause thrombosis of the IVC?

A

pancreatic pseudocysts and acute pancreatitis

56
Q

what should not be confused with thrombus in the IVC?

A

enlarged retroperitoneal lymoh nodes impressing the IVC in a patient with chronic lymphatic leukemia

57
Q

dealing with blood, what can cause IVCT?

A

psoas hematomas and other hematomas of the retroperitoneim

58
Q

what does trauma include?

A

sequlae of virchows triad

  • vessel injury
  • hypercoagulability
  • stasis
59
Q

what may direct trauma of the IVC be the result of?

A

either penetrating or blunt trauma

60
Q

the balance between what is both delicate and dynamic?

A

balance between coagulation system and the fibrinolytic system

61
Q

disorders that disrupt the balance in the coagulation sysetm can cause what?

A

IVC thrombus formation may occur (nephrotic system is a classic example)

62
Q

what do people with nephrotic system have?

A

massive urinary protein losses and diminished levels of antithrombin 3

63
Q

what have been describes with nephrotic syndrome?

A

both renal vein thrombosis and IVCT

64
Q

patients with what may present with iatrogenic IVCT?

A

recent history of medical care

65
Q

the expansion of what has led to increased recognition of iatrogenic IVCT?

A

endovascular techonolgy

66
Q

what are interventions that reportedly been identifiable rates of IVCT include?

A
  • hepatic transplantation
  • dialysis access
  • femoral venous cathedars
  • pacemaker wires
  • vena caval filters
67
Q

what is an inferior vena cava filter?

A

small cone-shaped device that is implanted in the IVC just below the kidneys

68
Q

what is the vena cava filter designed to capture?

A

am embolism, a blood clot that has broken loose from one of the deep veins in the legs on its way to the heart and lungs

69
Q

what are numerous other clinical situations that have been associated with IVCT?

A
  • developmental anomalies of the IVC
  • retroperitoneal fibrosis
  • pregnancy
  • OC
70
Q

Is May Thurner syndrome a common or rare condition?

A

rare

71
Q

what may compression of the common venous outflow tract of the left lower extremity may cause?

A

discomfort, swelling, pain or blood clots

72
Q

what may be the cause of the May-Thurner syndrome?

A

DVT in the iliofemoral vein

73
Q

what is the specific problem of May-Thurner syndrome?

A

compression of the left common iliac vein by the overlying right common iliac artery

74
Q

what does compression of the common illiac veins lead to?

A

pooling or stasis of blood, predisposing the individual to the formation of blood clots

75
Q

where is May-thurner syndrome more common?

A

in the left leg as the artery acultely overlaps the left iliac vein

76
Q

wha does May-Thurner syndrome frequently manifests as?

A

pain when the limb is dependant and/or significant swelling of the whole limb

77
Q

what are other causes of leg swelling, pain or tenderness?

A
Popliteal(Baker’s) cysts-ruptured
Hematoma/muscle injury
Superficial thrombophlebitis
Iliac nodes/pelvic masses
Arteriovenous fistula
Lymphedema
Knee joint effusion
Rt heart failure
78
Q

wat does a hematoma/muscle tear look like?

A

an anechoic structure between the medial gastrocnemius and the soleus muscle

79
Q

what are the differential diagnosis of hematoma/muscle tear?

A
  • hematoma caused by a gastrocnemius muscle rupture
  • a hematoma caused by a plantaris tendon rupture
  • ruptured bakers cyst
80
Q

how do we differentiate a hematoma/muscle tear?

A

it is important to examine the fluid collection in its full length and a coexistent DVT must be ruled out

81
Q

if a ruptured bakers cyst is seen, what must be assessed?

A

deep veins as coexistant DVT may be overlooked

82
Q

what does a bakers cyst look like?

A

A cyst with a ‘neck’ at its deepest extent, extending into the joint space between the semimembranosus tendon and the medial head of the gastrocnemius muscle

83
Q

what identification makes a definitive diagnosis for a bakers cyst?

A

neck between the tendons

84
Q

when may lymph nodes in the groin swell?

A

injury or infection in the foot, leg, groin, or genitals

85
Q

when may a lump form in the groin?

A

testicular cancer, lymphoma, or melanoma

86
Q

what is superficial thrombophleitis?

A

inflammation of the vein with thrombus

87
Q

what is considered threatening for PE?

A

thrombus at the SFJ and SPJ

88
Q

who presents with knee joint effusion?

A

patients with arthritis, recent trauma, or knee surgery and infection

89
Q

what wil fluid in the joint cause?

A

pain and swelling

90
Q

what is presented with Rt heart failure?

A
  • bilateral leg edema

- fluid may back up into your abdomen, legs and feet, causing swelling

91
Q

what is lymphedema?

A

swelling in one or more extremities that results from impaired flow of the lymphatic system

92
Q

in lymphedema, where is it most effected?

A

single arm or leg

93
Q

what can the sign and symptoms of lymphedema mimic?

A

DVT

94
Q

who has a higher risk of developing lymphedema?

A

patients with cardiovascular diseases, such as those with DVT venous leg ulcers and varicose veins

95
Q

what is a complication of lymphedem?

A

cellulitis

96
Q

what is considered a risk factor for developing secondary deep vein thrombosis of the upper extremity?

A

injecting drug use

97
Q

what are the main causes of superficial and deep venous thrombosis?

A

the repeated trauma of venapuncture, local infections, and the irritating qualities of the drugs

98
Q

what is responsible for bacteremia?

A

septic thrombosis

99
Q

what is the most common bacteremia with septic thrombosis?

A

aureus

100
Q

what are high risk locations for septic thrombosis?

A

upper limb deep veins

101
Q

how is primary deep vein thrombosis caused?

A

idipoathic or caused by effort induced activity

102
Q

what is cellulits?

A

infectous process in the soft tissues

103
Q

what are clues to the diagnosis of cellultis?

A

tenderness and erythema

104
Q

what may be identified with cellultis?

A

skin thickening, edema, and swelling of subcutaneous tissues

105
Q

what is cellulits caused by?

A

bacterial infection

106
Q

what does cellulitis result from?

A

break in the skin from trauma, surgery, or bite

107
Q

what is a abscess?

A

Well-circumscribed fluid collections within the soft tissues

108
Q

what does abscess look like?

A
  • Well defined walls,simple or complex fluid
  • Gas bubbles appear as bright reflectors
  • May be increased vascularity within the walls of the abscess
109
Q

how do soft tissue tumors appear?

A

isoechoic to hyperechoic with scant vascularity

110
Q

what are benign soft tissue tumors?

A
Lipoma-MOST COMMON
Fibroma
Leiomyoma
Desmoid tumor
Neurofibroma 
Hemangioma
111
Q

what are malignant soft tissue tumors-most common?

A
  • sarcoma and lymphoma

- melanoma

112
Q

what are malignant metastitic soft tissue tumors?

A
  • lymphoma

- leukemia

113
Q

how do malignant soft tissue tumors appear?

A

hypervascular

114
Q

where are most major collateral routes?

A

in the abdomen and pelvis

115
Q

what is an interesting long, collateral route?

A

flow may bypass an aortoiliac obstruction utilizing the subclavian artery as a source of flow

116
Q

what gives rise to the superior epigastric arteries?

A

blood from the SCA courses distally through the internal mammary artery

117
Q

what do the superior epigastric arteries communicate with?

A
  • external iliac artery

- inferior epigastric branches

118
Q

when does the superior epigastric arteries not communicate????

A

if the obstruction is acute

119
Q

when the infrarenal portion of the abdominal aorta or common illiac arteries are involved in atherosclerotic disease where may flow originate from?

A

the SMA via the colic arteries to the rectal, vesical and hemorrhoidal arteries via the IMA

120
Q

how may the lumbar arteries provide flow to the external iliac system?

A

via deep iliac circumflex artery or from the iliolumbar arteries of the internal iliac system

121
Q

when the external iliac or common femoral arteries are blocked, where may flow be reconstructed through?

A

arising from the internal iliac (hypogastric) artery

122
Q

an obstruction of the deep femoral artery may result in collateral flow from where?

A

developing from the common femoral artery via the superfical iliac circumflex artery or the superficil femoral artery

123
Q

what happens if the superficial femoral artery becomes occluded at the adductor canal?

A

the deep femoral artery may provide flow through the descending lateral circumflex artery to the popliteal artery

124
Q

what is flow below the knee common through?

A

collateral circulation is common through an extensive network of muscualr and genicular branches

125
Q

when do collateral flows occur?

A

in the presence of DVT in chronic phase

126
Q

what dilate to take flow up to the groin?

A

superficial vessels - long and short saphenos as well as their branches