quiz 4 Flashcards

1
Q

CAPE-V

A
  • Consensus Auditory-Perceptual Evaluation of Voice
  • uses visual analog scaling to rate vocal attribute - rater places tick mark along each line - measured and written in column
  • speech tasks - vowel /a/, vowel /i/, 6 sentences, 20-30 secs of spontaneous speech
  • rate vocal attributes as consistent or intermittent
  • vocal attributes: overall severity, roughness, breathiness, strain, pitch, loudness
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

3 vocal registers

A
  • glottal fry
  • modal/chest register
  • falsetto
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

glottal fry

A
  • lowest fundamental frequency
  • limited range, limited intensity/amplitude
  • VF edges are loose and relaxed
  • requires minimal subglottal pressure
  • common at end of sentences
  • not unhealthy bc periodic and low effort
  • can lead to being unhealthy when trying to be louder in glottal fry
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

modal/chest register

A
  • richest and heaviest timbre of registers
  • most common register for conversational speech
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

head voice/falsetto

A
  • highest fundamental frequency range of registers except whistle
  • lightest timbre of registers
  • very little vibration - only outer cover of VF
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

VHI & score interpretation

A
  • Voice Handicap Index
  • used to assess patient’s judgement about relative impact of voice disorder upon daily activities
  • validated, 30 questions asking questions that are functional, physical, and emotional
  • rated 0-4, 0 is never, 4 is always
  • 0-30 = mild/minimal handicap
  • 31-60 = moderate handicap
  • 60-120 = severe handicap
  • if total score changes 18 pts = real change
  • if total score changes 8 points = critical difference
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

VHI 10

A
  • derived from original VHI
  • 10 critical question from 3 domains
  • total of 40
  • greater than 11 = abnormal
  • 6 points is minimal impact difference
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

mean speaking fundamental frequency

A
  • elicit with conversational speech or asking patient to read rainbow passage
  • males 100-120
  • females 200-220
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

maximum phonation time

A
  • tell client to take a deep breath, say /a/ at comfortable pitch and loudness and hold it for as long as they can
  • do 3 trials, take best of 3
  • normative data says 15-25 seconds
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

primary muscle tension dysphonia

A
  • vocal hyperfunction in absence of organic pathology without obvious psychogenic/neurologic etiology
  • behaviorally modifiable, unexplained dysphonia
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

secondary muscle tension dysphonia

A
  • dysphonia occurring as compensatory technique with an organic, psychogenic, or neurologic etiology
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

puberphonia

A
  • failure of voice to reflect development of secondary sex characteristics in puberty
  • usually seen in immediate post-pubescent period
  • elevated speaking pitch, pitch breaks, strained quality
  • patient can produce a normal pitch but doesn’t like it
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

alternative phrasing to “misuse” or “abuse”

A
  • can be rude, as patients are using their voice in a functional way
  • places blame on patient which is not appropriate
  • “caused by high demand for voice”
  • “phonotraumatic behaviors”
  • “irritation from high collision forces of the vocal folds”
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

theoretical framework for vocal hyperfunction

A
  • just because someone uses their voice excessively doesn’t mean it WILL lead to phonotraumatic behavior
  • nonphonotraumatic hyperfunction would be primary muscle tension
  • vocal hyperfunction + precipitating factors = phonotraumatic hyperfunction
  • cycle of developing phonotraumatic lesions, needing more hyperfunction to compensate
  • break cycle with voice therapy, surgical removal, return to normal voice function
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

vocal fold nodules

A
  • most common benign lesions of VF
  • result from continuous voice use
  • generally bilateral, whitish perturbances on striking zone of VF
  • acute nodules are soft and pliable, reddish, mostly vascular, edematous
  • treatment: voice therapy will make person more aware of habits leading to problem
  • medical: surgery may be considered if improvement is not achieved with therapy
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

vocal fold polyps

A
  • benign, phonotraumatic growth that is a fluid-filled sac on VF, usually unilateral
  • anywhere on free edge of VF, usually striking zone
  • may be mistaken for acute nodules, cysts, or papilloma
  • medical: microlaryngeal surgery
  • pre-op counseling/evaluation, post-op rehab
17
Q

Reinke’s Edema/Smoker’s Polyps

A
  • chronic, diffuse swelling of superficial lamina propria
  • usually bilateral, may be worse on one side
  • strongly associated with smoking
  • treatment: therapy can optimize vocal function but will not eliminate edema; smoking will prevent lesions from worsening
  • medical: can zap them with a laser
18
Q

laryngomalacia

A
  • inward collapse of subglottic structures of larynx during inhalation
  • 75% of congenital anomalies of larynx
  • most prevalent cause of inspiratory stridor in neonate
19
Q

subglottic stenosis

A
  • narrowing of space below glottis and above first tracheal ring
  • congenital or acquired
  • rare, but one of most common cuases of upper airway instruction in pediatrics
20
Q

esophageal atresia

A
  • failure of esophagus to develop as a continuous passage
21
Q

tracheoesophageal fistula (TEF)

A
  • abnormal opening between trachea and esophagus
  • can occur with esophageal atresia
22
Q

laryngopharyngeal reflux disease (LPRD)

A
  • gastric juices move through UES and spill into pharynx
  • causes hoarseness, cough, throat clearing, globus sensation, sore throat, dysphagia
  • laryngeal signs: hypertrophy of interarytenoids, erythema/edema, irritation, ulceration, granulation near vocal process
  • treatment: SLP, ENT, patient work to plan successful reflux management and voice therapy program
23
Q

lifestyle/dietary modifications to manage reflux

A
  • lose weight
  • limit alcohol/caffeine
  • limit mint/peppermint
  • don’t eat 3hr prior to bedtime
  • limit intake of carbonation
  • cessation of smoking
  • sleep on left side with head elevated
  • reduce intake of fatty foods
  • reduce intake of chocolate
24
Q

reflux medications/alignates

A
  • medications are either H2 blockers or proton pump inhibitors
  • alignates: OTC product made from algae; ingest it, turns to gel, floats to top of stomach, acts as raft blocking reflux
25
Q

vocal process contact ulcers/granulomas

A
  • benign ulcerations developing of medial aspect of vocal processes of arytenoid cartilages due to irritation
  • granulated tissue forms over ulcers as a protective mechanism, turning into contact ulcer granulomas
  • etiologies: hard glottal attacks with throat clearing, LPRD, intubation
  • treatment: reflux meds, lifestyle modifications for reflux, behavioral voice therapy, surgery can remove granulation tissue but may return
26
Q

leukoplakia

A
  • white patch; can be precancerous
  • refer out for biopsy, closely monitor
27
Q

erythroplakia

A
  • red patch; can be pre-cancerous
  • refer out for biopsy, closely monitor
28
Q

laryngeal cysts

A
  • closed sac filled with fluid or semi-solid substance
  • often soft and pliable
  • usually benign, unilateral, anywhere on VF
  • often caused by abnormal blockage of ductal system of mucous glands
  • may be phonotraumatic lesion
  • treatment: surgical removal with superficial lesion on superior edge of VF
  • behavioral voice therapy to optimize healing and help efficient voice production
29
Q

recurrent respiratory papilloma

A
  • wart-like growths, viral in origin, growing in dark, moist caverns in airway
  • seen more commonly in children, usually benign
  • treatment is aggressive surgical management to assure airway
30
Q

juvenile onset recurrent respiratory papilloma (JORRP)

A
  • wart-like growths, viral in origin, occurring in dark, moist caverns of airway; usually benign
  • associated with mothers who have genital HPV
  • most common cause of pediatric hoarseness
  • treatment is aggressive surgical management
31
Q

sulcus vocalis

A
  • grooved medial edge of VF, usually bilaterally symmetrical
  • may in superficial lamina or may tether vocal ligament
  • either congenital or acquired
32
Q

laryngeal webbing

A
  • abnormal tissue forms between VF
  • can be congenital or acquired
  • treatment: if compromising more than 50% of glottis, treatment is required to establish airway