Quiz 3 Flashcards
Cerebrovascular Accident
sudden loss of blood supply resulting in loss of oxygen supply to the brain
damages kills brain cells
neurological deficits related to areas affected
affects opposite side of the body to the hemisphere of the brain affected
Ischemic stroke
87%
caused by thrombus- blood clot or embolus traveling blood clot
Hemorrhagic stroke- 10%
caused by rupture of blood vessel with bleeding into the brain
blood is leaked into adjacent brain tissue
often more severe than ischemic strokes
anurism is a type
usually die of complications if live after stroke
Risk Factors for CVA
Risk Factors: controllable: hypertension cigarette smoking excessive alcohol intake high cholesterol intake obesity
uncontrollable: increasing age male sex black race history of DM previous CVA or TIA family history
Effects
mental functions sensory functions neuromuscular movement-related functions voice speech functions
Dysfunction:
flaccid paralysis or hypotonicity absent or reduced reflexes impaired posture sensory deficits visual impairments perceptual dysfunction cognitive dysfunction behavioral and personality changes impaired speech and language skills
Early Treatment:-Warning Signs
warning signs:
early treatment can reduce progression and residual effects if within 4 hours of start treatment
use of clot buster drugs
FAST- face, arm, speech, time
Medical management:
emergency treatment: open airway, establish fluid balance, and treat medical problems
give medicine
surgery may be indicated
*Hemispheric Lateralization:
Left cerebral hemisphere- Right hemipharesis
responsible for language, time concept, and analytic thinking
often have aphasia- partial or total loss of language communication and apraxia- motor planning problems
Right cerebral hemisphere- left hemipharesis
controls visual-perceptual function and perception of the whole
neglect/inattention more common
may retain good verbal but poor functional performance- dysarthria
Cerebellum Stroke:
abnormal reflexes of the head and torso
coordination and balance problems
ataxia
dizziness
problems with swallowing and articulation
cranial nerve deficits- vertigo, nausea, vomitting, headaches, nystagmus, slurred speech
Brainstem Stroke
controls primary functions- breathing, heart rate, blood pressure and arousal
dizziness
problems with swallowing and articulation
cranial nerve deficits
paralysis
likely to be critically ill- to need mechanical ventilation
MCA- Middle Cerebral Artery
most common
largest vessel branching off the internal carotid artery
most common cerebral occlusion site
feeds- femoral, temporal, parietal lobes of brain and basal ganglia and internal capsule
MCA- Effects
Effects of complete MCA CVA
facial asymmetry, arm weakness, and speech deficits
hemiplegia or hemiparesis of contralateral side
affecting lower part of face arm and hand
sensory loss in same areas
homonymous hemianopsia- visual field deficits affecting the same half of the visual field in both eyes
ACA- Anterior Cerebral Artery
least common- frontal and parietal lobes classic signs: contralateral leg weakness and sensory loss behavioral abnormalities incontinence may occur
PCA- Posterior Cerebral Artery
feeds the medial occipital lobe and inferior and medial temporal lobes
vision- contralateral homonymous hemianopsia
larger strokes- aphasia and neglect
Cognitive Dysfunction:
Inatention and memory deficits attention and concentration deficits disorientation insight deficits judgement and safety awareness deficits
Transient Ischemic Attack: TIA’s
incomplete stroke with symptoms lasting from a few minutes to 24 hours
Role of OT: Tx of CVA
improve motor function
integrate sensory-perceptual and cognitive functions
facilitate maximum level of functional independence
encourage resumption of life roles
promote health management and maintanence behaviors to prevent recurrent stroke
Grading of treatment:
increase length and complexity of activity
consider time for completetiong, extend of steps, number of steps, amount of physical assistance, verbal cues used and adaptive equipment
Remedial vs Compensatory Treatment:
remediation- restoring function
compensation- adaptation of task or environment
usually use a combination of both
Abnormal Reflexes/ Postural Mechanism post CVA
delayed righting, equilibrium, and protective response
address balance and trunk control of head and trunk-normal postural mechanisms
Positioning Techniques:
reduce abnormal tone promote alignment prevent contracture & skin breakdown Bed-- alternative positions AE use as needed prevent decupitus ulcers affected arm is supported resting hand splint in functional position
Balance Impairments:
poor automatic & postural adjustments against gravity
decreased weight bearing on affected side
Positioning Treatment
NDT- handling techniques- plevis, shoulders, chest & head) use wedges for anterior pelvic tilt encourage crossing midline co-treat with PT for balance sit on PB to improve righting reactions
Sensoriomotor Interventions-
Occupation-based Treatment Ideas
Weight shifting —- prepare for wheelchair transfer by weight shifting to front of seat
Weight bearing prone, side lying, then progress to sitting— watch a movie or read a book on a wedge in prone or side lying, in sitting rest arm on lap tray
Practice sit<>stand transfers - toilet transfers
segmental rolling/trunk rotation- in side lying, reach for clock
pelvic tilt- in sitting reach for snacks forward and overhead and place in bowl at elbow height
Abnormal Muscle Tone:
Flaccid paralysis: often replaced by spastic paralysis
more affective
synergies- want to break out to prevent contractors choose activities move away from synergies
Spasticity is described as minimal, moderate, or severe
can fluctuate through out the day
Motor Control Deficits:
Voluntary muscle control absent immediately after stroke function returns in stages and may stop at any stage return proximal to distal