Pulpal Irritants Flashcards

1
Q

Irritants can be what three kinds

A

Microbial
Mechanical
Chemical

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2
Q

What is the goal in vital cases

A

Asepsis; to prevent infection, this includes use of a dam to keep bugs out.

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3
Q

What is the goal in non-vital cases

A

Antisepsis; to remove all microorganisms

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4
Q

What organisms are easily eradicated during conventional RCT

A

Obligate anaerobes

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5
Q

What are some examples of facultative bacteria

A

Streptococci
Enterococci
Lactobacilli

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6
Q

What organisms can survive chemo-mechanical RCT

A

Facultative bacteria

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7
Q

What one bacteria is frequently associated with failed RCTs

A

E. faecalis

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8
Q

What are some examples of mechanical irritants:

A
Deep cavity prep
Lack of cooling
Impact trauma
Occlusal trauma
Deep perio curettage 
Ortho movement (too far or too fast)
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9
Q

What are some example of chemical irritants

A

Dentin: cleaning, sterilizing and desensitizing agents.

Some temp and permanent filling materials

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10
Q

Which fibers are myelinated

A

A-fibers; myelinAted, fAst, fAt

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11
Q

A-delta fibers are for

A

Paint
temperature
Touch

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12
Q

C-fibers are for

A

Pain; dorsal root, deep inside the pulp

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13
Q

Where are A-delta fibers principally located

A

Pulp-dentin junction

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14
Q

What type of pain comes from A-delta fibers

A

Sharp
Pricking
Relative low threshold

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15
Q

Where a C fibers principally located

A

Throughout the pulp

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16
Q

What type of pain comes from C fibers

A

Burning
Aching
Less bearable than A-delta fiber sensation
High threshold usually associated with tissue injury

17
Q

What is another name for the cell free zone

A

Zone of Weil

18
Q

Where is the zone of Weil

A

Immediately adjacent to odontoblastic layer in coronal pulp

19
Q

When is the zone of Weil mostly found

A

During middle age pulps

20
Q

What is the cell rich zone

A

Subodontoblastic area
High proportion of fibroblasts
Immune cells: macros, dendritic cells, undif mesenchymal cells

21
Q

Eventual or rapid necrosis depends on?

A
The virulence of bacteria
Ability to release inflammatory fluid to prevent increase in intrapulpal pressure.
Host resistance
Amount of circulation
LYMPH DRAINAGE
22
Q

SAP lesions are more likely to have what two viruses

A

Cytomegalovirus

Epstein barr Virus

23
Q

What substance participates in the inflammatory reaction of the dental pulp

A

metabolites of arachidonic acid

24
Q

Metabolism of arachidonic acid results in the formation of what metabolites

A

Prostaglandins
Thromboxanes
Leukotrines

25
What is the clinical sign of advanced internal resorption
Pink spots in the crown | but have normal pulpal and periapical tests
26
What is the cause of ischemic necrosis
Traumatic injury from disruption of the blood supply
27
What are MMPs
Matrix Metalloproteinases | endopeptidases that significantly contribute to the degradation of extracellular matrix components.
28
What is AAA?
Localized or diffuse liquefaction lesion of pulpal origin. Destroys PR tissues Severe inflammatory response.
29
What are the s/s of AAA
``` Acute Apical Abscess Rapid onset Spontaneous pain Moderate to sev discomfort and swelling Occasional systemic manifestations: temp, malaise, leukocytosis. Painful to percussion/palpation ```
30
What are the s/s of CAA
Chronic Apical Abscess: Long standing lesion. Abscess that drains through a sinus tract.
31
What are lessons that have similar appearance to PR lesions
``` Periapical cemental dysplasia Fibrous dysplasia Ossifying fibroma Primordial cyst lateral periodontal cyst Dentigerous cyst Median cyst Central giant cell granuloma ameloblastoma ```
32
What are the four pulpal diagnosis
Normal Reversible pulpitis Irreversible pulpitis Necrosis
33
What are the six periapical diagnosis
``` Normal SAP AAP Acute apical abscess Chronic apical abscess Condensing osteitis ```