pulpal and periapical disease Flashcards

1
Q

pulpitis

A
  • Most common cause of dental pain /
    toothache
  • Usually caused by caries penetrating the
    dentine
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2
Q

what happens when pulp necrosis is left untreated?

A

pulp necrosis
leads to the death of the pulp

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3
Q

aetiology of pulpits

A

mechanical causes
thermal causes
chemical causes
bacterial causes

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4
Q

mechanical causes

A
  • Trauma
  • Iatrogenic damage during a dental procedure
  • Attrition
  • Abrasion
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5
Q

thermal causes

A
  • Uninsulated metallic restoration
  • During cavity preparation
  • Polishing
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6
Q

chemical causes

A

arise from erosion

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7
Q

what are the usual causes of pulpitis?

A

caries penetrating the dentine
leads to inflammation of the pulp

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8
Q

classification of pulpits

A

reversible pulpitis
irreversible pulpitis

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9
Q

reversible pulpits: clinical features

A
  • Mild to moderate inflammation of the pulp
  • Cold stimuli are common triggers
  • Pain is not spontaneous
  • Relief occurs once the stimulus is removed
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10
Q

reversible pulpitis histopathology

A

pulp hyperaemia
cellular response
formation of tertiary dentine

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11
Q

pulp hyperaemia

A

– Mild to moderate
inflammatory reaction
– Vital pulp remains intact

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12
Q

cellular response

A

– Specialised cells in the pulp
migrate
– Cells proliferate and
differentiate into
odontoblasts

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13
Q

formation of tertiary dentine

A

– Occurs at the dentine-pulp
complex
– Acts as a protective
response to injurious
stimulus

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14
Q

treatment of reversible pulpits

A
  • Placement of Restoration
    – Used if a cavity is present
  • Removal of Noxious Stimuli
    – Seal exposed dentine in
    cases of abrasion
  • Prevention and Periodic Care
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15
Q

clinical features of irreversible pulpits

A
  • Paroxysms of Pain
    – Triggered by sudden temperature changes, especially heat
    – Pain persists even after the stimulus is removed
  • Spontaneous Pain
    – Pain may occur without any apparent trigger
  • Exacerbation of Pain
    – Bending over and lying down increases pain due to
    intrapulpal pressure changes
  • Electric Pulp Vitality Test
    – Affected tooth reacts at a lower current level compared to
    normal teeth
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16
Q

clinical and pathological features of irreversible pulpits

A
  • Nighttime Pain
    – Patient often awake at night due to
    severe pain
  • Difficulty Identifying Affected Tooth
    – Pain may be referred from maxilla to
    mandible or vice versa
    – Referred pain to ipsilateral ear or sinus
    – Pain does not cross the midline
  • Increased Pulp Pressure
    – Lack of escape of inflammatory
    exudate
    – Severe inflammation of the pulp
  • Compression of Pulpal Blood Vessels
    – Results in hypoxic cell injury
    – Leads to pulp necrosis
17
Q

treatment of irreversible pulpits

A

root canal therapy
extraction of affected tooth

18
Q

severe pulpits symptoms

A
  • may cause dull pain
  • can be relatively asymptomatic
19
Q

mild pulpits symtoms

A

may cause excruciating pain

20
Q

chronic hyperplastic pulpitis

A
  • Represents an excessive, exuberant,
    proliferation of chronically inflamed dental
    pulp tissue.
  • Arises due to an extensive carious exposure
    of a young pulp with subsequent
    development of granulation tissue, covered
    at times by epithelium.
  • Results from long-standing, low-grade
    pulpitis.
  • Most commonly involves deciduous molars
    and first permanent molar due to rich blood
    supply and large root opening in a young
    otherwise healthy patient.
21
Q

periapical inflammatory disease: pathogenesis

A

Periapical inflammatory disease: pathogenesis
* A common sequelae of an untreated or
inadequately treated irreversible pulpitis
with pulp necrosis.
* Spread of infection occurs through the
apical foramen into the periapical tissues.
* Occasionally the infection spreads via an
accessory root canal and then becomes
located on the lateral aspect of the root of
the affected tooth.
* Both microbial and host factors play a role
in determining whether the periapical
inflammation manifests as an acute
inflammatory process, known as a
periapical abscess or as a chronic
inflammatory process, known as a
periapical granuloma.

22
Q

periscopical abscess definition

A

refers to the accumulation of acute
inflammatory cells at the apex of a non-vital
tooth.

23
Q

pariapical abscess clinical features

A

the associated tooth is non-vital,
severe constant pain, extreme sensitivity to
percussion, tooth may be extruded. Headache,
fever and malaise may be present.

24
Q

periapical abscess radiographic features

A

may show widening of the
apical periodontal ligament, often no periapical
lesion visible on the radiograph because a de
novo periapical abscess is an acute process and
insufficient time has occurred for significant bone
destruction.

25
periapical abscess histological features
Aggregation of neutrophils often intermixed with inflammatory exudates, cellular debris and bacterial colonies.
26
27
periapcial abscess treatment
Drainage and elimination of the focus of infection.
28
complications of a untreated periapcial abscess
* Spread into the medullary bone resulting in osteomyelitis. * The purulent material may drain through a fistulous tract intraorally forming a parulis (gum boil) or extraorally via a cutaneous sinus through the overlying skin. * Perforation of the cortex and diffuse spread into the overlying soft tissue resulting in a cellulitis. * The purulent material may drain into the blood stream resulting in a bacteremia - which in turn can lead to fatal consequences such as cerebral abscess, cavernous sinus thrombosis or mediastinitis.
29
periapical granuloma definition
refers to a mass of chronically inflamed granulation tissue at the apex of a non-vital tooth.
30
periapical granuloma clinical features
most lesions are assymptomatic
31
radiographic features of periapical granuloma
presents as a periapical radiolucency of varying size. Affected tooth typically shows loss of the apical lamina dura.
32
periapical granuloma - histological features
Composed of a proliferation of fibroblasts and capillaries that forms a mass of granulation tissue (hence the term granuloma). There is an accompanying dense chronic inflammatory cell infiltrate that is predominated by lymphocytes, plasma cells and foamy macrophages.
33
periapical granuloma treatment
Root canal therapy or extraction followed by curettage of the apical soft tissue or apicectomy with retrograde root canal filling.
34
possible consequences of untreated periapical granuloma
Periapical granulomas are not static lesions. They may undergo acute exacerbation and present as a so-called “chronic abscess” which becomes clinically symptomatic or they may progress to form periapical / radicular cysts.
35
periapical abscess - acute inflammation
* Intense pain * Extremely tender to percussion * No radiographic changes
36
periapcial granuloma - chronic inflammation
* Dull / no pain * Slightly tender to percussion * Periapical radiolucency