Pulmonary Rehabilitation Flashcards

1
Q

COPD

A

respiratory disorder caused largely by smoking, characterized by PROGRESSIVE partially reversible airway obstruction and lung hyperinflation, systematic manifestations, and increase freq/sev of exacerbations

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2
Q

Premature infants

A

80% predicted FEV1 because lungs are not developed.

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3
Q

Age of exacerbation symptoms

A

50-55 years

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4
Q

Top reason for hospitalization/ambulatory care sensitive conditions

A

COPD

  • 1/3 COPD pt will revise the ED within 30 days in AB
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5
Q

COPD exercise limitation: Gas exchange efficiency

A

Gas exchange = PAO2 - PaO2

  • we want little to no pressure difference between alveolar and arterial
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6
Q

COPD exercise limitation: What determines VO2max?

A

Thick Equation
VO2 = (HR x SV) x (CaO2-CvO2)

  • stroke volume response (the bigger the larger the response)
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7
Q

COPD exercise limitation: exercise stress on gas exchange

A

PAO2-PaO2 = VO2/DLO2

  • during exercise VO2 increases
  • we need to increase diffusion capacity ignorer to keep arterial and alveolar pressure difference close
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8
Q

hyperventilation results in what in the brain?

A

hypocapnia
(ventilation is greater than metabolic demand)

  • rate of CO2 removal from blood increases
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9
Q

PETO2 (alveolar PO2/end tidal) during exercise

A

will increase as we need to breathe more (exercise = ventilation)

  • accumulate lactic results in hyper ventilation as we pass the anaerobic threshold
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10
Q

limitation: out of breath - what stops us from exercise

A

cardiovascular system (not lungs)

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11
Q

Breathing reserve

A
  • should be 30% VE reserve
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12
Q

EILV and EELV compensation for ventilation needed for exercise

A
  • EILV will increase and EELV will decrease in response to exercise
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13
Q

Tidal volume & resp response to exercise

A

Healthy FEV1 = 100% predicted

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14
Q

Ventilatory Response in obstructive disease

A
  • cannot ventilate as much, DEV1 = 40% predicted
  • dynamic hyperinflation: deep breath-in and prolonged expiration, but brain wants us to keep breathing so we take a breath in
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15
Q

Rehabilitation of lung function

A
  • pulmonary rehab does not improve lung function, rather we get adaptation through cardiac function, muscle strengthening, produce less lactic acid, ventilate less
  • maximal ability to ventilate doesn’t change
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16
Q

Exercise training - MMV pre- and post-rehab

A

exercise training decreases ventilatory response to exercise

17
Q

5 year survival in COPD based on peak VO2

A

Grade I: > 995mL/min 99% survival rate after 5 years

Grade IV: < 650mL/min 40% survival rate after 5 years

18
Q

What is pulmonary rehabilitation?

A

multidisciplinary: comprehensive intervention (optimize physiological, psychological, and social outcome) for COPD patients followed by patient tailored therapies (behaviour change, education, exercise training, support/teach medications)

PR has been shown to ↑ health outcomes in: • Asthma
• Bronchiectasis
• Cystic Fibrosis
• Interstitial Lung Disease • Lung Cancer

19
Q

Goals of COPD management

A
  1. Prevent disease progression (smoking cessation)
  2. Reduce the frequency and severity of exacerbations
  3. Alleviate breathlessness & symptoms
  4. Improve exercise tolerance
  5. Improve quality of life
  6. Reduce mortality
20
Q

COPD: downward spiral

A

lose 50% of lung function during first hospital visit

- COPD, dyspnea, inactivity, decondtioning, psychological, QOL

21
Q

Pulmonary rehab: upward spiral

A

COPD - Dyspnea - Rehab program - improve activity/conditioning - decrease dyspnea and psychological conditions

22
Q

Goal of pulmonary rehab

A

increase their capacity to self-manage

23
Q

COPD: Exercise training goals

A
  1. Flexibility exercises
  2. Strengthening exercises
  3. Endurance exercises
  4. Breathing exercises
24
Q

Exercise Action Plan: Individual Session

A

Utilizes FITT (Frequency, Intensity, Time, Type) to set targets/goals for long term maintenance

  • Endurance, Strength, Breathing, Flexibility
  • Create a “Good day plan”, “Bad day plan” and a “Re-integration plan”
  • Must include location of exercise
25
Q

Exercise Action Plan: Address barriers of exercise

A
  • Motivation (goal setting, motivational interviewing, social support)
  • Financial (home exercise, low income programs, social work referral)
  • Self Efficacy (reassurance, refer to supervised exercise programs)
  • Lack of Time (scheduling, fitness calendar, activity vs. exercise)
26
Q

COPD: aerobic exercise goal

A

Frequency:

  • 3-5 days/week
  • > 20min (interval or continuous)

Intensity:
– 40-85% of VO2peak reserve
– 60-80% VO2peak
– Based around Anaerobic Threshold – Based on Dyspnea

↑intensity/volume→↑physiological benefit

27
Q

COPD Strength training goals:

A

Strength training which focuses on maintaining activities of daily living necessary

  • increase strength, muscle mass
  • no change to exercise capacity
28
Q

Oxygen supplementation: Issues

A

2 Issues:

  1. O2 to prevent hypoxemia
  2. O2 added to non-hypoxemic patients
29
Q

Oxygen supplementation: goals

A
  1. ↓ Ventilation/dyspnea
  2. ↑ O2 delivery to muscle
  3. Increase exercise workload
30
Q

Oxygen supplementation: who?

A

Supplemental O2 should be used w/ hypoxemia (<90% SpO2)

Supplemental O2 in non-hypoxemia patients?

31
Q

Long-term goal of pulmonary rehabilitation

A

Behaviour change: Increase (and maintenance) of physical activity key

32
Q

When should a COPD have an ICH

A

when FEV1 = <60% predicted

33
Q

Effect of PR on exacerbating prevention

A

PR done immediately after a COPD hospitalization reduces chance of future exacerbations

34
Q

PR vs. medication

A

PR improves quality of Iife and functional capacity better than traditional pharmacological management’

  • RCT are no longer wanted because confirmed that PR is so effective
35
Q

Final question about PR:

A

question is no longer should patients with COPD receive PR, but rather how should PR be delivered to patients and which components form the basis of the success of PR programs’