Pulmonary Disorders Flashcards
Intrapulmonary Shunting
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The amnt of blood tht flows to the lungs w/out being oxygenated bc of nonfunctioning alveoli
> blood not going where its suppose to
> alveolar collapse secondary to ateletctasis
> alveolar flooding w/ pus, blood, or fluid
PaO2/FiO2
aka P/F ratio
- Measurement of efficiency of oxygenation
- Measurement of intrapulm shunting
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Normal: greater than 286
> the lower the value, the worse the lung function
Acute Lung Failure (ALF)
- A clinical condition in which the pulmonary system fails to maintain adequate gas exchange
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Usually occurs secondary to another disorder tht has altered the normal function of pulm system
> conditions tht dcr func: vent drive, muscle strength, chest wall elasticity, the lung’s capacity for gas exchange
> conditions tht inr func: airway resistance, metabolic oxygen requirements
Acute Lung Failure (ALF) - CMs
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Hypoxemia
> low oxygen in blood -
Hypercapnia
> high CO2 - Acidosis
Acute Lung Failure (ALF) - Medical Management
- Treat underlying cause
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Promote adequate gas exchange
> supplemental oxygen
> if an intrapulmonary shunt (low I/F ratio) is present, pt will require positive pressure - Correct acidosis
- Initiate nutrition support
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Prevent complications
> ischemic-anoxic encephalopathy
> cardiac dysrhythmias
> VTE
> gastrointestinal bleeding
Acute Lung Failure (ALF) - Nursing Management
- Optimize oxygenation & ventilation
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Position
> healthy lung down
> elevate HOB
> reposition -
Prevent desat
> perform only necessary procedures
> hyperoxygenate before suctioning
> provide adequate rest & recovery time btwn procedures
> minimize oxygen consumption: limit physical activity, admin sedation to control anxiety & maintain normothermia -
Promote secretions clearance
> humidify
> suction - Maintain surveillance for comps
- Educate pt & fam
Acute Respiratory Distress Syndrome (ARDS)
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Noncardiac pulmonary edema & disruption of the alveolar-capillary membrane as a result of injury to the pulm vasculature or airways
> the hallmark of ARDS is refractory hypoxemia
> w/in a week of no clinical injury
> bilateral opacities not fully explained by effusions, lobar/lung collapse, or nodules
> origin of edema: lungs (not from HF)
Acute Respiratory Distress Syndrome (ARDS) - Oxygenation
- Mild: P/F ratio: 200-300 mmHg WITH PEEP or CPAP greater than, equal 5 cm H2O
- Moderate: P/F ratio: 100-200 mmHg WITH PEEP greater than, equal to 5 cm H2O
- Severe: P/F ratio: less than 100 mmHg WITH PEEP greater than, equal to 5 cm H2O
Acute Respiratory Distress Syndrome (ARDS) - CMs
- Exudative Phase: tachypnea, restlessness, apprehension, & moderate incr in accessory muscle use
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FIBROPROLIFERATIVE PHASE: pt’s s/s progress to agitation, dyspnea, fatigue, excessive accessory muscle use, & fine cracles as resp failure develops
> poor end perfusion - ABGs: low PaO2, despite incrs in supp ox admin (refractory hypoxemia)
Acute Respiratory Distress Syndrome (ARDS) - why is PaCO2 initially low?
Fast breathing early on; breathing off CO2 gets fatigued; retaining CO2 (incrs lvls)
Acute Respiratory Distress Syndrome (ARDS) - why is the pH high?
Acidotic
Acute Respiratory Distress Syndrome (ARDS) - Fibroproliferative Phase
- Destruction of type II alveolar cells
- Gas exchange compromised
- Incrd peak inspiratory pressure
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Dcrd compliance
> static & dynamic -
Refractory hypoxemia
> intraalveolar atelectasis
> incrd shunt fraction
> dcrd diffusion - Dcrd functional residual capacity
- Interstitial fibrosis
- Incrd dead space ventilation
Acute Respiratory Distress Syndrome (ARDS) - Fibroproliferative Phase Physical Examination
- Elevated pulm artery pressures
- Incrd workload on right ventricle
- Incrd use of accessory muscles
- Fine crackles
- Incring agitation r/t hypoxia
- CXR: interstitial or alveolar infiltrates; elevated diaphragm
- Hyperventilation; hypercapnia
- Widening alveolar-arterial gradient
- Incrd work of breathing
- Worsening hypercarbia & hypoxemia
- Lactic acidosis (r/t aerobic metabolism)
Acute Respiratory Distress Syndrome (ARDS) - Fibroproliferative Phase Physical Examination: Alteration in Perfusion
- Incrd HR
- Dcrd BP
- Change in skin temp & color
- Dcrd cap filling
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End-organ dysfunction
> Brain: change in mentation, agitation, hallucinations
> Heart: dcrd CO -> angina, HF, papillary muscle dysfunc, dysrhythmias, MI
> Renal: dcrd urinary or GFR
> Skin: mottled, ischemic
> Liver: elevated SGOT, bilirubin, alkaline phosphatase, PT/PTT; dcrd albumin
Acute Respiratory Distress Syndrome (ARDS) - Medical Management
- Treat underlying cause
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Ventilation
> Mechanical Ventilation modes: pressure control ventilation & inverse ratio ventilation
> complications to assess for: pneumothorax -
Oxygenation therapy
> admin O2 at lowest lvl to support tissue oxygenation
> PEEP > 5 mmHg