Pulmonary Disorders Flashcards
Intrapulmonary Shunting
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The amnt of blood tht flows to the lungs w/out being oxygenated bc of nonfunctioning alveoli
> blood not going where its suppose to
> alveolar collapse secondary to ateletctasis
> alveolar flooding w/ pus, blood, or fluid
PaO2/FiO2
aka P/F ratio
- Measurement of efficiency of oxygenation
- Measurement of intrapulm shunting
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Normal: greater than 286
> the lower the value, the worse the lung function
Acute Lung Failure (ALF)
- A clinical condition in which the pulmonary system fails to maintain adequate gas exchange
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Usually occurs secondary to another disorder tht has altered the normal function of pulm system
> conditions tht dcr func: vent drive, muscle strength, chest wall elasticity, the lung’s capacity for gas exchange
> conditions tht inr func: airway resistance, metabolic oxygen requirements
Acute Lung Failure (ALF) - CMs
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Hypoxemia
> low oxygen in blood -
Hypercapnia
> high CO2 - Acidosis
Acute Lung Failure (ALF) - Medical Management
- Treat underlying cause
-
Promote adequate gas exchange
> supplemental oxygen
> if an intrapulmonary shunt (low I/F ratio) is present, pt will require positive pressure - Correct acidosis
- Initiate nutrition support
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Prevent complications
> ischemic-anoxic encephalopathy
> cardiac dysrhythmias
> VTE
> gastrointestinal bleeding
Acute Lung Failure (ALF) - Nursing Management
- Optimize oxygenation & ventilation
-
Position
> healthy lung down
> elevate HOB
> reposition -
Prevent desat
> perform only necessary procedures
> hyperoxygenate before suctioning
> provide adequate rest & recovery time btwn procedures
> minimize oxygen consumption: limit physical activity, admin sedation to control anxiety & maintain normothermia -
Promote secretions clearance
> humidify
> suction - Maintain surveillance for comps
- Educate pt & fam
Acute Respiratory Distress Syndrome (ARDS)
-
Noncardiac pulmonary edema & disruption of the alveolar-capillary membrane as a result of injury to the pulm vasculature or airways
> the hallmark of ARDS is refractory hypoxemia
> w/in a week of no clinical injury
> bilateral opacities not fully explained by effusions, lobar/lung collapse, or nodules
> origin of edema: lungs (not from HF)
Acute Respiratory Distress Syndrome (ARDS) - Oxygenation
- Mild: P/F ratio: 200-300 mmHg WITH PEEP or CPAP greater than, equal 5 cm H2O
- Moderate: P/F ratio: 100-200 mmHg WITH PEEP greater than, equal to 5 cm H2O
- Severe: P/F ratio: less than 100 mmHg WITH PEEP greater than, equal to 5 cm H2O
Acute Respiratory Distress Syndrome (ARDS) - CMs
- Exudative Phase: tachypnea, restlessness, apprehension, & moderate incr in accessory muscle use
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FIBROPROLIFERATIVE PHASE: pt’s s/s progress to agitation, dyspnea, fatigue, excessive accessory muscle use, & fine cracles as resp failure develops
> poor end perfusion - ABGs: low PaO2, despite incrs in supp ox admin (refractory hypoxemia)
Acute Respiratory Distress Syndrome (ARDS) - why is PaCO2 initially low?
Fast breathing early on; breathing off CO2 gets fatigued; retaining CO2 (incrs lvls)
Acute Respiratory Distress Syndrome (ARDS) - why is the pH high?
Acidotic
Acute Respiratory Distress Syndrome (ARDS) - Fibroproliferative Phase
- Destruction of type II alveolar cells
- Gas exchange compromised
- Incrd peak inspiratory pressure
-
Dcrd compliance
> static & dynamic -
Refractory hypoxemia
> intraalveolar atelectasis
> incrd shunt fraction
> dcrd diffusion - Dcrd functional residual capacity
- Interstitial fibrosis
- Incrd dead space ventilation
Acute Respiratory Distress Syndrome (ARDS) - Fibroproliferative Phase Physical Examination
- Elevated pulm artery pressures
- Incrd workload on right ventricle
- Incrd use of accessory muscles
- Fine crackles
- Incring agitation r/t hypoxia
- CXR: interstitial or alveolar infiltrates; elevated diaphragm
- Hyperventilation; hypercapnia
- Widening alveolar-arterial gradient
- Incrd work of breathing
- Worsening hypercarbia & hypoxemia
- Lactic acidosis (r/t aerobic metabolism)
Acute Respiratory Distress Syndrome (ARDS) - Fibroproliferative Phase Physical Examination: Alteration in Perfusion
- Incrd HR
- Dcrd BP
- Change in skin temp & color
- Dcrd cap filling
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End-organ dysfunction
> Brain: change in mentation, agitation, hallucinations
> Heart: dcrd CO -> angina, HF, papillary muscle dysfunc, dysrhythmias, MI
> Renal: dcrd urinary or GFR
> Skin: mottled, ischemic
> Liver: elevated SGOT, bilirubin, alkaline phosphatase, PT/PTT; dcrd albumin
Acute Respiratory Distress Syndrome (ARDS) - Medical Management
- Treat underlying cause
-
Ventilation
> Mechanical Ventilation modes: pressure control ventilation & inverse ratio ventilation
> complications to assess for: pneumothorax -
Oxygenation therapy
> admin O2 at lowest lvl to support tissue oxygenation
> PEEP > 5 mmHg
What is the purpose of adding PEEP to vent settings
Open alveoli space for lung oxygen exchange
When would ECMO be considered?
High PEEP, not perfusing
Acute Respiratory Distress Syndrome (ARDS) - Nursing Management
-
Optimize oxygenation & ventilation
> position, prevent desat, promote secretion clearance -
Prone position
> ARDS: to promote posterior surface of lungs - Comfort & emotional support
- Maintain surveillance for comps
Pneumonia
- An acute inflammation of the lung parenchyma caused by an infectious agent tht can lead to alveolar consolidation & can be classified as community acquired or hosp acquired
- Dyspnea, fever, cough (productive or non)
Pneumonia - Medical Management
- Focus on initiation of antibiotic therapy
- Admin oxygen
- Mech vent
- Management of fluids & nutrition support
- Treat comps
Pneumonia - Nursing Actions
- Optimize oxygenation & ventilation
- Prevent spread of infection
- Provide comfort & emotional support
- Maintain surveillance for comps