Pulmonary Disorders Flashcards

1
Q

What is Acute Respiratory failure (ARF)?

A

Inadequate gas exchange marked by HYPOXEMIA (low O2 in blood)

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2
Q

What are the three main causes of ARF?

A

Intrapulmonary shunting
V/Q mismatch
Alveolar hypoventilation

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3
Q

S/S of ARF

A

Tachycardia, tachypnea, use of accessory muscles, nasal flaring, grunting
-any pt who exhibits confusion, agitation or restlessness must be assessed for HYPOXEMIA FIRST

PaO2 <60 / SaO2 <90% OR paCO2 >50 with acidosis (low pH)

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4
Q

Management of ARF

A

Supplemental O2 therapy- goal is SaO2 >90%

  • supplemental O2 effective with alveolar hypoventilation or V/Q mismatch but NOT in shunt
    -Intrapulmonary shunt requires positive pressure (CPAP, BiPAP, or Ventilator)
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5
Q

Alveolar hypoventilation, causes, and interventions

A

Same as Resp. Acidosis with hypoxemia

Causes:
- anything that limits ventilation (neuro trauma, pneumothorax, lung trauma, oversedation, pain)

Interventions:
- optimize ventilation (positioning, pain relief, secretion removal, correct underlying cause)
-if on ventilator, increase rate and/or tidal volume to breathe off CO2

PaCO2 is a direct reflection of alveolar hypoventilation

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6
Q

How does patient positioning affect ventilation (V) and perfusion (Q)?

A

When sitting upright,
- upper zone of the lungs has more ventilation and less perfusion (V>Q)
-middle zone has equal ventilation and perfusion
-lower zone has more perfusion than ventilation

More perfusion will go to the alveoli that are closer to the ground (affected by gravity)

When laying down, the weight of the heart and diaphragm compress the dependent alveoli and secretions pool in posterior lungs where perfusion is higher
-why we prone

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7
Q

What are the benefits of heated and humidified air?

A

Heated and humidified O2:
- decrease airway inflammation
- maintain mucociliary function
-increase mucous clearance
- reduce caloric expenditure (decrease work on lungs)

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8
Q

What is the benefit for the patient when using a high-flow rate?

A

-increases FiO2
- decreases dead space
- provides low level positive pressure (PEEP)
- decreases the work of breathing

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9
Q

What are the benefits of using a High-flow nasal cannula (HFNC)?

A
  • reduces the need for intubation
  • decreases the work of breathing
  • provides a low level of positive pressure
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10
Q

What is proning and why is it used?

A

Proning positions the patient on their stomach in a “swimmer” position or places them with their good lung down to improve expansion in the bad lung
—improved V/Q matching

Reduces alveolar compression by the heart and diaphragm, allowing for better expansion of the dorsal lung, more surface area = alveolar recruitment

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11
Q

Nursing management for ARF

A

-positioning that is optimal for oxygenation
-pulmonary exercises
—deep breath hold 3 seconds; IS
—no dry coughing
-hydrate and suction secretions
-increase movement when possible
-don’t cluster care to allow for rest

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12
Q

Medications for ARF

A

Medication treatment depends on the cause of the respiratory failure and the patient’s condition

-steroids
-sedation
-neuromuscular blockers
-analgesia

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13
Q

What is pneumonia?

A

Acute inflammation of the lung parenchyma caused by an infectious agent

-often related to dental plaque and aspiration of plaque organisms
—BASIC MOUTH CARE is so important

HAP= hospital acquired pneumonia
-occurs after 48 hours in the hospital

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14
Q

Pneumonia management

A

-O2 therapy (non-invasive but mechanical ventilation if necessary)
-nursing same as ARF
-Drugs including antibiotics and bronchodilators

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15
Q

What happens when a patient aspirates acid?

A

-bronchospasm and atelectasis
-may cause an inflammatory response with severe hypoxemia, V/Q mismatch, and shunt
- frequently progresses into ARDS

If aspirated high pH gastric contents, bacterial pneumonia may develop

**patients with aspiration are at high risk of developing ARDS

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16
Q

Management of aspiration

A

-supportive care first
-O2 therapy
-Nursing: HOB up, good lung down, deep breathing, IS
-possible bronchoscopy

NO steroids
Antibiotics after 48 hours

17
Q

Best practices for preventing aspiration in adults

A
  • maintain HOB 30-45 degrees
  • use sedatives sparingly
  • maintain endotracheal cuff pressures at an appropriate level
18
Q

What is acute respiratory distress syndrome (ARDS)?

A

Inflammatory syndrome marked by:
- disruption of the alveolar-capillary membrane
- non-Cardiogenic pulmonary edema

In ARDS, the alveolar-capillary membrane leaks fluid out of capillaries and into the alveolar space AND alveoli leaks into the interstitial space
—causes ‘junk’ in the alveoli
—CO2 is smaller so it can still pass through, but O2 is larger and has a harder time entering the capillary
THEREFORE, O2 is a more sensitive indicator of ARDS

19
Q

What are the causes and major risk factors for ARDS?

A

May be caused by direct injury to pulmonary epithelium or secondary to non-pulmonary insult
- sepsis (non-pulm.)
-aspiration (pulm. Or non-pulm.)
- pneumonia (non-pulm.)
- trauma (pulm.)

20
Q

What clinical symptoms would you expect to see with ARDS?

A

Persistent hypoxemia despite supplemental oxygen

Diagnostic criteria: P/F ratio < 300

21
Q

Care for ARDS

A

Most effective treatment is supportive care

Ventilator
-maintain pH 7.2 or higher

Proning
-at least 16 hours/day to improve V/Q matching and alveolar recruitment

Maintain C.O.
-but keep the patient dry (adequate preload but not high)

22
Q

Outcomes for ARDS

A

Mortality about 40%

Pulmonary fibrosis is common afterwards

Potential for permanent neurologic impairment with impaired quality of life

Communicate that recovery will take time and there will be residual pulmonary damage