Pulmonary Flashcards

1
Q

What to assess for in regards to pulmonary history?

A
  • Baseline pulmonary function
    • Exercise tolerance, dyspnea, orthopnea, smoking, sleep apnea
  • Chronic lung disease
    • Severity, meds, recent hospitalizations, frequency of exacerbations, & effective treatment
  • Acute lung disease
    • Recent URIs, abx, current symptoms
  • Previous anesthesia experiences
    • Procedures, pulmonary complications, anesthesia technique utilized
      • Pts w/ chronic lung dx often have difficulty achieving extubation criteria. a) do you remember if you had a tube inserted into your throat to help you breathe? b) did you have to be placed in ICU after your surgery, 3) did you receive a shot into your back, your hip, or in the surgical area to numb the area and decrease pain? All of these questions provide greater insight into a patient’s medical history.
  • Medical clearance
    • Evaluation of current status & therapies
    • Availability of lab & diagnostic studies
    • “clearance” is not the preferred term. Some pts require optimization prior to sx.Input from a pulmonologist and/or cardiologist might be needed for pts w/ severe respiratory illnesses.
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2
Q

What are are some quesitons to ask in regards to obstructive pulmonary disease?

A

problem with getting air out….

  • Asthma
    • An acute reversible airway obstruction that is accompanied by chronic airway inflammation & hyperreactivity
    • Ask about SOB, chest tightness, cough, recent exacerbations, therapy (esp steroids), hospitalizations, intuation
  • Chronic Obstructive Pulmonary Disease (chronic bronchitis and emphyema)
    • Airflow obstruction – difficulty getting air out
    • Ask about smoking hx, dyspnea, cough, wheezing, sputum production, tachypnea, home o2
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3
Q

Assessment considerations in restricttive pulmonary disease?

A
  • Pulmonary fibrosis, sarcoidosis, obesity
    • Reduction in total lung capacity – reduction in all lung capacities
    • Proportional decrease in all lung volumes
    • Ask about SOB, exercise tolerance, home o2 use
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4
Q

Examples of restrictive disease?

A
  • Neg press pulm edema
  • Kyphoscoliosis
  • Pregnancy
  • Sarcoidosis
  • Pneumothorax
  • Pleural effusion
  • Ascites
  • Neurological disorders (musc dystrophy)
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5
Q

Examples of obstructive diseases?

A

Asthma

Chronic bronchitis

Emphysema

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6
Q

What to assess for on initial survey of respriation and thorax?

A

Initial survey of respiration & thorax- quickly assess for resp distress

  • Inspect color for cyanosis or pallor
  • Listen for audible sounds of breathing
  • Inspect the neck (accessory muscles)
  • Tracheal position
  • Observe respirations
  • Look directly at the thorax if poss
  • Observe the shape of the chest – is it barrel shaped?
    • The lateral diameter of the chest in the normal adult is greater than the AP diameter.
    • The ratio of its AP diameter to the lateral diameter is ~0.7 to 0.9 and increases w/ age
  • Observe for chest deformities – usually present in peds, adolescents, & young adults. Sx avail for excavatum. Both are correctable . even when no corrective action is taken, severe functional limitations rarely experienced
    • Pectus carinatum “pigeon chest”
    • pectus excavatum” funnel chest”
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7
Q

What is pectus carinatum?

A
  • Pectus carinatum “pigeon chest”
    • Protrusion of sternum.
    • Present during adolescent growth spurts ie puberty
    • Less common than excavatum
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8
Q

What is pectus exacavatum?

A
  • Causes a caved in, depressed chest, sometimes the ribs can flare out
    • Associated comorbidities & functional limitations rarely exist
  • Unbalanced costochondral hypertrophy - exact cause unknown
  • ~1 of every 150-1000 births w/ 3:1 male predominance
  • Occurs ~ 5-6x more often than pectus carinatum
  • Often displays during puberty. Might worsen til ~ 18 yo, although can be present @ birth
  • Up to 43% have a family hx of the deformity
  • May be associated scoliosis & connective tissue disorders ie Marfans, Ehlers-Danlos, & Noonan’s syndrome
    • Focus on s/s that are assoc w/ these associated dx, can help deter if need addt’l preop testing.
      • Ie cardiac manifestations – MV prolapse, AV dilation, & BBB
    • Observe for bruising & skin friability which is a sign of ED
    • Look for high arched palates –> point to associated syndrome which may need more testing
  • In addition to cosmetic considerations, severe PEX may result in cardiac compression & cardiopulmonary impairment
  • Symptoms might include:
    • Dyspnea
    • Loss of endurance
    • Chest pain
    • Progressive fatigue
    • Palpitations, tachycardia
    • Syncope
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9
Q

What should be considered when evaluating pectus exacavatum

A

Psychological issues can be SIGNIFICANT, esp in adolescents & young adults

  • Don’t focus too much attention at the defect. Don’t stare and ask direct questions about it, espeically if a teen or young adult; especially if surgery is unrelated to the defect!
  • body image very important at this age
  • However, don’t overlook the disorder. If pediatric, ask questions to patient and parent. Do you have pediatrician that you see regularly? Do you play sports? are you able to run around with your friends? have you ever passed out or had chest pain?
  • Sx - Nuss procedure & cosmetic procedures commonly used to correct.
  • Evaluation:
    • Individually tailored to pt’s symptoms & responses to aforementioned questions
    • Noncontrast CT–> extent of deformity
    • EKG –> arrhythmias
    • ECHO –>cardiac compression, MVP
    • PFTs
    • Exercise stress testing
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10
Q

What to assess for on palpation during pulmonary exam?

A
  • Areas of tenderness
  • Crepitus
  • Tactile/ vocal fremitus – palpable vibrations that are transmitted through the bronchopulmonary tree to the chest wall as the pt is speaking. Vibrations should be symmetrical
    • Tactile frem- place palms or ulnar surfaces of hands on chest while pt says 99 repeteadly. Alterations in vocal fremitus could suggest PNA, pleural effusion, COPD, etc
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11
Q

What to assess for during percussion portion of pulmonary asessment?

A
  • Helps you to establish whether the underlying tissues are air filled, fluid filled, or consolidated & ID the pathology to them
  • Healthy lungs are resonant
  • Fluid filled cavities and solid tissues percuss dullness & can indicate PNA, tumor, or pleural effusion
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12
Q

How do you auscultate the lungs?

A
  • Breathing normally, mouth open
  • 1) apply Diaphragm of stethoscope – directly to pts skin bc over clothes or gowns can alter breath sounds
  • 2) listen to ant & post chests
  • 3) listen above the clavicles bc the lung apices extend above them
    • Apices, middle & lower lung fields, posterior, lateral, & anterior
  • 4) Alternate & compare sides- ie if listen to R clavicle, 2nd pt of ausc should be above the L clav & continue stair step technique for all fields
  • Quiet respirations first, then deep breaths
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13
Q

What are normal breath sounds heard during auscultation?

A
  • Vesicular – heard over most lung fields
    • Quality – lower pitch & softer
    • Duration – inspiratory sounds last longer than expiratory sounds
  • Bronchovesicular- heart best @ 1st & 2nd ICS anteriorly & between scapulae
    • Quality- intermediate pitch & sound
    • Duration – expiratory & inspiratory sounds almost equal
  • Bronchial- heard best over the manubrium (larger prox airways)
    • Quality – higher, loud, hollow pitch
    • Duration – expiatory sounds last longer than inspiratory sounds
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14
Q

What are vesicular breath sounds?

A
  • heard over most lung fields
  • Quality – lower pitch & softer
  • Duration – inspiratory sounds last longer than expiratory sounds
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15
Q

What are bronchovesicular breath sounds?

A
  • heart best @ 1st & 2nd ICS anteriorly & between scapulae
    • Quality- intermediate pitch & sound
    • Duration – expiratory & inspiratory sounds almost equal
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16
Q

What are bronchial breath sounds?

A
  • heard best over the manubrium (larger prox airways)
    • Quality – higher, loud, hollow pitch
    • Duration – expiatory sounds last longer than inspiratory sounds
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17
Q

What are adventitious breath sounds?

A

Abnormal breath sounds

  • If bronchial breath sounds are auscultated anywhere other than expected (manubrium is expected)
    • Suspect replacement of air-filled lung tissue by consolidation - ex PNA

Adventitious sounds – umbrella term for abnormal breath sounds

  • Wheeze (high pitched), rhonchi (low pitched)
    • Musical, usually on expiration but may be heard on inspiration
  • Stridor
    • High pitched, usually on inspiration
  • Crackles/ rales – less distinct
    • Tearing Velcro open
    • Crackles of HF are best heard in the posterior lung fields
    • Pulm edema- crackles
    • Clearing of crackles, wheezes, or rhonchi after coughing or position change –> inspissated (mucoid) secretions –> present in bronchitis, atelectasis
  • Beware of the silent chest (no air mvmt) –> clinical emergency
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18
Q

What is a wheeze? rhonchi?

A

Wheeze= high pitched

rhonchi= low pitch

  • musical, usually on expiraiton but may be heard on inspiration
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19
Q

What is stridor?

A

high pitched, usually on inspiration

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20
Q

What are crackles/rales?

A
  • Tearing Velcro open
  • Crackles of HF are best heard in the posterior lung fields
  • Pulm edema- crackles
  • Clearing of crackles, wheezes, or rhonchi after coughing or position change à inspissated (mucoid) secretions à present in bronchitis, atelectasis
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21
Q

What additional testing may be required in obstructive or restrictive disease?

A
  • O2 saturation w/ pulse oximetry
  • Chest radiograph
  • Arterial blood gases
  • Pulmonary function testing
  • Chest computed tomography
  • Lab testing – CBC, CMP
  • EKG
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22
Q

What is purpose of PFT? Indications?

A
  • Purpose
    • Standardized
    • Objective
    • Req pt cooperation
  • Indication
    • Assist in diagnosis
    • Evaluate treatment
    • Disease progression
    • Risk assessment – ie postop risk
23
Q

Who are appropriate candidates for PFT?

A

Appropriate candidates for testing

  • Patients with evidence of COPD
  • Smokers with persistent cough
  • Wheezing or dyspnea on exertion
  • Morbid obesity
  • Thoracic surgery patients
  • Open upper abdominal procedures
  • Patients > 70 years of age

Routine PFTs are NOT recommended

24
Q

Who is at risk for postop pulmonary complications?

A

Categorized as:

  • patient related
    • age >60
    • ASA 3-4
    • CHF
    • COPD
    • Cigarette smoking
  • procedure related
    • aortic >thoracic> upper abdominal- neuro-peripheral vascular> emergency
    • duration of anesthesia >2.5 hours
    • general anesthesia
  • diagnostic testing related
    • only albumin <3.5 is at increase risk for increased pulmonary risks.
25
Q

What do pulmonary function tests measure?

A
  1. Dynamic lung vol—how quickly air can be moved in and out of the lungs over a given time
  2. Static lung vol—how much air the lungs can hold at a specific time
  3. Diffusing capacity–how well the lungs can transfer gas across the alveolar-capillary membrane
26
Q

What are static lung volumes?

A
  • How much air the lungs can hold at a given time
  • These vols include insp reserve vol, exp reserve vol, TV, & residual vol.
  • Spirometry cant mesaure RV bc RV cant be exhaled. and therefore can’t measure TLC
  • FRC isn’t capable of being measured by spirometry bc RV is a component of FRC.
  • RV + expiratory reserve vol = FRC.
  • Look in the book to see correlation b/w various vols & capacities. Nagelhout 5th edition p 598
27
Q

What is FRC? How do we measure it?

A
  • Functional Residual Capacity- Volume of gas in the lungs after passive exhalation
    • Residual volume is the volume of gas in the lungs after forced max expiration
  • FRC can assist in quantifying the degree of pulmonary restriction
  • Measured
    • Indirectly
      • By Nitrogen wash-out
      • Nitrogen analyzer attached to spirometer & nitrogen end pt estab.
      • End point= alveolar nitrogen concentration falls below 7%, usually takes about 7-10 minutes
28
Q

What is spirometry and lung volumes measured? What are predicted values based on?

A
  • Dynamic lung volumes can easily be eval w/ spirometry Spirometry = measure of air moving in & out of lungs during various resp maneuvers. Deters how much air can be inhaled & exhaled & how fast.
  • The pt is asked to sit up straight, inhale maximally, estab a good seal around mouthpiece, & exhale as hard as possible & count for at least 6 sec. this is repeated 3 x & the best of the 3 trials is recorded.
  • Spirometry can record the measures listed here.
  • Spirometry is based on forced vital capacity (FVC). The FVC is divided into time intervals
    • FEV1
    • FEV1/FVC ratio
    • FEV 25-75%
  • Predicted values based on
    • Age
    • Height, weight
    • gender
29
Q

What is forced vital capacity?

A
  • Max inspiration (taking deepest breath possible) & total amt of air expired as quickly as poss with forced expiration
    • Majority of FVC can be exhaled in <3s in normal people, but more prolonged in obsx dx.
  • Function of vol/time
  • Measures resistance to flow
  • Effort and cooperation dependent
  • Interpretation % predicted
    • 80-120% normal
    • 70-79% mild
    • 50-69% moderate
    • <50% severe
30
Q

What is FEV1?

A
  • Forced Expiratory Volume in 1 sec: FEV1
    • vol of air forcefully expired from full inspiration in the first second
    • Normal is 75-80% of FVC
    • Effort and cooperation dependent
    • Interpretation of FEV1/FVC
      • >80% normal
      • 60-79% mild
      • 50-59% moderate
      • <49% severe obstruction
31
Q

What is FEF25-75%

A

Forced expiratory flow 25-75% (FEF25-75)

  • mean forced expiratory flow during middle of FVC
  • Most sensitive indicator of small airway disease
  • More reliable that FEV1-FVC
  • Interpretation of % predicted:
    • >60% normal
    • 40-60% mild
    • 20-40% moderate
    • <10% severe obstruction
32
Q

What is seen in obstructive vs restrictive dx?

FVC?

FEV1?

FEV1/FVC?

FEF25-75%

RV

FRC

TLC?

A
33
Q

What are flow volume loops?

A
  • FVL = plots of insp & exp flow on the Y axis against vol on X axis, during the performance of max forced insp & exp maneuvers.
  • The contour of the loops assist in dx & localization of airway obsxn bc diff lung disorders produce easily recognized patterns
  • Events occurring in the loop:
    • 1.) The pt inhales from RV to TLC
    • 2.) Then the pt exhales back to RV as fast as possible
34
Q

What does a normal flow volume loop look like?

A

upside down ice cream cone

35
Q

What does an obstructive flow volume loop look like?

A
  • In obstructive ds the expiratory limb has a concave shape
  • Ex: COPD
  • “Someone took a bite of my ice cream cone”
36
Q

What is seen in flow volume loop in restrictive leasions?

A
  • In restrictive disease the shape of the loop is similar to the normal loop, just smaller and right shifted
  • Ex: Pregnancy
  • ”on a restrictive diet you’ll have to eat a smaller ice cream cone”
37
Q

What is seen in flow volume loop with fixed leasions?

A
  • With a fixed lesion, the inspiratory and expiratory limbs are flat
  • Ex: tracheal stenosis
  • “Someone smashed my ice cream cone and it needs to be fixed”
38
Q

How do we maximize pulmonary function?

A

Goal:

  • Reduce intraoperative and postoperative morbidity and mortality

Methods:to maximize pulm fxn

  • smoking cessation
  • mobilization of secretions/treat infections
  • bronchospasm alleviation
  • motivation and stamina improvement
39
Q

What happens during smoking cessation for surgery?

12-24 hours?

2-3 weeks

6 weeks?

8 weeks?

A
  • 12-24 hours= reduction of carboxyhemoglobin/carbon monoxide levels to normal
    • d/c smoking for even 1 night b4 sx can dec HR, BP, circulating catechol lvls & allows carboxyhgb to return to normal lvls
  • 2-3 week mucociliary function returns –> increased secretions
    • Used to think that this would inc r/f postp comps but not tru. Longer per of cessation more beneficial but cessation of any time frame recommended.
  • 6 weeks= reduction in secretions
  • 8 weeks=rate of postoperative pulmonary complications decreases
40
Q

How can someone treat pulmonary infeciton and mobilize secretions prior to sx?

A
  • Antibiotic therapy in the presence of chronic bronchitis – may need abx as well as measures to mobilize secretions. Doing these can help optimize pt preop
  • Mobilizations of secretions
    • mucolytic agents
    • hydration
    • aerosol therapy
    • mechanical therapy
41
Q

Treatment of bronchospasms?

A
  • know specific drugs, MOA, & classification
  • beta 2 agonists
  • anticholinergics
  • methylxanthines - theophylline
  • corticosteroids - triamcinolone
42
Q

What can be done to improve motivation and stamina and maximize pulmonary function?

A

education & practice w/ IS – for cardiac & majorsx. Breathe in as slowly and deeply as poss. Hold breath for 2-6 s. recruits alveoli. Several x a day

43
Q

Keys to preanesthetic managment of pt with pulmonary disease?

A
  • don’t give drugs that can cause resp dep
  • Sedation (benzodiazepines)
    • Titrate carefully due to risk respiratory events & give sm amts only
    • Alpha 2 agonists–better suited if premedication is needed
  • Albuterol-recommended 20-30 minutes before induction
  • Anticholinergics
    • Dries secretions and decreases airway vagal response
    • Need to consider increase in HR
44
Q

What is distribution of blood flow in the lungs dependent on?

A
  • Blood flow in lungs(perfusion) largely gravity dependent
    • Blood flow dependent on relationship between:
      • Pulmonary artery pressure (Pa)
      • Alveolar pressure (PA)
      • Pulmonary venous pressure (Pv)
45
Q

What happens in Zone 1 of West Lung Zones?

A
  • PA>Pa>Pv
  • There is ventilation, but no perfusion= dead space
  • Zone 1 does not occur in normal lung
  • Is increased by hypotension, PE, or PPV
  • To combat Zone 1, the bronchioles of unperfused alveoli constrict to reduce dead space
46
Q

What is Zone 2 of West’s Lung Zones?

A
  • Pa>PA>Pv
  • There is ventilation and perfusion

“waterfall”

  • Think of a waterfall flowing over a dam.
    • Pa= height of upstream river
    • PA= height of dam
    • A greater height of the upstream river = higher rate of water flow over the dam. Like waterfall, BF = directly proportional to differences in Pa – PA. the greater the diff b/w art & alv prs, the greater the BF
47
Q

What is Zone 3 of West’s Lung Zone?

A
  • Pa>Pv>PA
  • Venous pressure now exceeds alveolar pressure
  • Perfusion is greater than ventilation
  • Physiologic shunt
  • To combat Zone 3, hypoxic pulm vasoconstriction reduces pulm blood flow to underventilated units
48
Q

What happens with perfusion/ventilation in lungs when patient is awake, sitting up, and spontaneously breathing?

Where is perfusion the least? greatest?

which area is most compliant?

Where is perfusion and ventilation the greatest?

A
  • When sitting up, awake, spontaneously breathing:
    • Perfusion is least at the highest portion of the lung
    • Perfusion is greatest in the dependent parts of the lung due to gravity (the bases)
    • Alveoli in bases are most compliant
    • Good alveolar compliance promotes ventilation at the lung bases bc the most compliant alv are in the dep portion of the lungs
    • To summarize: perfusion and ventilation are greatest at the lung bases
49
Q

How does lung perfsuion change when patient lies supine?

A
  • Upright position
    • GRAVITY
  • Supine position – awake, supine, spont br pt- perfusion & ventilation follow the same rules but the dep & non dep areas of the lungs change.
    • Uniform blood flow from apices to bases
    • Anterior vs posterior – supine, the ant surfaces of the lungs are the nondeep & the posterior are the dep
50
Q

Anesthetic managmenet in restrictive disease?

A
  • FRC is reduced –> susceptible to rapid arterial desaturation during periods of apnea
  • The best ventilatory strategy aims to minimize barotrauma:
    • Smaller TVs (6-8 mL/kg) IBW
    • Faster RR (14-18 breaths/min)
    • Peak pressures < 30cm/H20
    • Prolong inspiratory time (I:E ratio 1:1- 2:1)
51
Q

Anesthetic management in obsturctive dx?

A
  • Reducing airway reactivity
    • Aggressive bronchodilator therapy
    • Lidocaine prior to airway manipulation
    • Desflurane–> airway irritant, Sevo better option
  • All volatiles are bronchodilators–> decrease airway resistance
  • Nitrous oxide questionable & prob should be avoided–> rupture of blebs–> pneumothorax
  • Emergence might be prolonged d/t exhaled agent trapped in alveoli
  • Regional anesthesia–> should be considered esp for sx of extrems & or lower abd
    • If sensory blockade @ T6 or higher needed, regional shouldn’t be used bc T6 level= loss of accessory respiratory muscles & interfere w/ pts abil to spont vent
    • If do regional, carefully titrate anesthesia adjuncts & be careful not to oversedate. COPD pts can be v sensitive to the vent dep effects of sedating drugs
52
Q

Tips for PPV managmenet in obsturctive dx?

A
  • Positive pressure ventilation
    • Tidal volume (6-8 ml/kg) w/slow inspiratory flow rates
    • Slow respiratory rates (6-10)
    • Keep peak pressure below 40 cm/H20
    • Increase expiratory time (I:E ratio) to minimize air trapping and auto-peep (dynamic hyperinflation)
53
Q
  • A patient is mechanically ventilated. Which interventions will improve the patient’s condition? (Select 2)
  • A) disconnect the circuit
  • B) decrease respiratory rate
  • C) increase inspiratory time
  • D) increase inspiratory flow
A
  • A & B
  • The waveform indicates “breath stacking” (auto-peep). Pts who have obstructive disease require prolonged time to exhale. So this condition could be improved by decreasing RR. If peep rises to dangerous levels. The patient should be disconnected from the ventilator.
  • Increasing inspiratory time is another way of saying reducing expiratory timeà would not be beneficial