Pt w/ Headache / Head Injury Flashcards

1
Q

What signs and symptoms might there be if someone has a head injury?

A

In the absence of OBVIOUS TRAUMA and/or a HISTORY people might present in the following way:

  • Nausea and Vomiting
  • Impaired consciousness
  • Headache
  • Rhinorrhoea
  • Otorrhoea
  • Diplopia
  • Pupil changes
  • Cushing’s response
  • ABG changes
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2
Q

Why is it important to get an ABG in someone who has had a head injury?

A

Close monitoring of their PaCO2 is really important. Even small increase will lead to vasodilation and marked vasodilation increasing the ICP

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3
Q

What is the cerebral perfusion pressure equation?

A

CPP = MAP - ICP

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4
Q

Was does the Monroe-Kellie doctrine state and what does this mean in the context of head injuries?

A

Cerebral/skull volume is fixed meaning that if ICP increases for any reason (due to a bleed or a SOL) this will have the affect of decreasing the CPP.
Even small increases in ICP will lead to clinical signs and larger increases may even lead to brain herniation

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5
Q

What are the early symptoms of raised ICP?

A

Altered consciousness
Confusion
Drowsiness

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6
Q

When ICP increase considerably where does the herniation occur and what affect does this have?

A

Into the tentorial hiatus which leads to compression of the parasympathetic portion of the oculomotor nerve (which runs in this tract)
Clinically this leads to FIXED DILATED PUPILS

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7
Q

What if the ICP continues to rise despite tentorial hiatus herniation?

A

Brain continues to herniate out of the skull
Initially this will cause CONTRALATERAL HEMIPARESIS and then as the brainstem becomes compressed it can lead to CARDIOPULMONARY ARREST

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8
Q

What affect can increasing ICP have on haemodynamics?

A

CUSHING’S RESPONSE
Hypertension
Bradycardia
Irregular breathing (triad)

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9
Q

What are some more specific symptoms of a basal skull fracture?

A
Haemotympanum 
Otorrhoea (CSF leak)
Rhinorrhoea (CSF leak)
Battle's sign (bruising over mastoids)
Panda eyes (bilateral orbital bruising)
Subconjunctival haemorrhage
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10
Q

What are the two types of head injury?

A

PRIMARY (occurs at the time of accident) - axonal shearing or haemorrhage

SECONDARY (due to other problems such as hypoxia, hypovolaemia, seizures, infection, haematoma)

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11
Q

What is the CPP at which we would start to become concerned?

A

70mmHg or below

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12
Q

What investigation should be done in a patient with a head injury?

A
Head and neck CT 
Pupil responses 
Limb power 
Monitor obs closely 
AVPU, GCS and AMTS
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13
Q

What factors are really important to ask about in the history of someone who’s had a head injury?

A

When it happened
Mechanism of injury (MoI)
Did they lose consciousness? Before or after hitting their head? Did they have a seizure?
Do they remember the event or do they have AMNESIA?
Ask about symptoms above (rhinorrhoea, pyorrhoea etc.)
PMH (cardiac arrhythmias, diabetes, epilepsy, prev HI)
DH: alcohol, other drugs, regular meds
SH: is their home situation suitable for discharge?

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14
Q

What should the examinations and assessment of someone with a head injury involve?

A
Full CNS and PNS 
C-SPINE assessment - immobilise until happy no fracture (Canadian C-Spine rules)
GCS 
Regular obs 
ECG 
FBC, U&E, Glucose, Clotting
Pupil examination
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15
Q

What are some signs of cerebellar damage?

A
DANISH 
Dysdiadochokinesia 
Ataxia 
Nystagmus 
Intention tremor 
Slurred/Staccato speech
Hypotonia
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16
Q

How should a head injury patient be managed initially?

A

A-E assessment (if GCS<8 call anaesthetist)
Check blood pressure and treat hypoglycaemia if necessary
Correct hypovolaemia
Make ICU, anaesthetics and neurosurgery aware
Arrange CT and make sure radiologist is there to interpret
IV abx for compound skull fracture (IV cefuroxime)

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17
Q

What are the criteria for CT scan in the ED in patients with head injury?

A

GCS <13 at presentation
GCS <15 at 2 hours post injury after assessment in department
Suspected open or depressed skull fracture
Any signs of basal skull fracture
Post-traumatic seizure
Any focal neurological deficit
More than one episode on vomiting post injury

PERFORM WITHIN 1 HOUR

Other RFx that should warrant scan within 8 hours include (on warfarin, >65, dangerous MoI, hx of bleeding or clotting disorder or more than 30m retrograde amnesia)

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18
Q

What are some presenting clinical features of meningitis?

A
Headache 
Fever 
Photophobia 
Neck stiffness 
Reduced consciousness level 
Irritability 
Drowsiness 
Vomiting 
Myalgia, anorexia

NON-BLANCHING SKIN RASH - meningococcal septicaemia (can present with or without meningitis)

19
Q

What are some common bacterial organisms that cause sepsis?

A

Neisseria meningitides
Haemophilus influenza
Listeria

20
Q

What investigations should be done in a patient with suspected meningitis?

A

FBC, U&E, CRP, Cultures, Glucose, clotting screen
ECG
Consider LP
CT scan if suspicion of raised ICP

21
Q

How should suspected cases of meningitis be managed initially?

A

Resuscitative treatment where needed (fluids, O2)
Start abx immediately (do not wait for test results)
IV CEFTRIAXONE OF CEFOTAXIME
If >55 add AMPICILLIN (listeria)
IV Dexamethasone with starting dose of abx
CONSULT MICROBIOLOGY AND REFER TO TRUST GUIDELINES

22
Q

How does a sub-arachnoid haemorrhage classically present and what are some other ket symptoms?

A

SUDDEN SEVERE HEADACHE (THUNDERCLAP/’KICKED IN THE HEAD’)
LOC might occur - may make diagnosis more difficult
Neck pain
Photophobia
Vomiting
Drowsiness and confusion
Unilateral eye pain

23
Q

What is the aetiology of an SAH?

A

Most commonly is rupture of Berry Aneurysm in the circle of Willis
Ask about family history because aneurysms have a considerably familial link

24
Q

What investigations should be done for a patient with suspected SAH?

A

Get regular obs (monitor for Cushing’s response: hypertension, bradycardia and irregular breathing) - due to increased ICP
Full CNS and PNS - if there is oculomotor never palsy this is a key signs of berry aneurysm in the posterior communication circulation
CT head
LP is informative up to 2 weeks after (xanthochromia) but can’t do for 12 hours

25
Q

How should a patient with a suspected SAH be managed initially?

A
A-E
Get IV access and check glucose, FBC, Clotting and U&amp;E
ECG might show ischaemia 
Get urgent CT 
LP
Involve neurosurgery 
Maintain good o2 sats 
Provide analgesia and anti-emetic (codeine / paracetamol)
26
Q

What drug can be used to reduce ICP?

A

MANNITOL (200mL 10%)

27
Q

What clinical features would make you begin to consider SOL?

A
Long-term headache and focal neurological pathology:
- Vision changes 
- Motor / sensory changes 
- ICP (vomiting, papilloedema, seizures)
Personality changes
28
Q

How do patients with SOL describe the headache?

A

Dull, achey and made worse by lying down or straining (due to increase in ICP)
- this sort of narrative of pain could also fit with an artery-venous fistula

29
Q

What investigations should be done in a patient who has a suspect SOL?

A

CT head
FBC, U&E, Glucose
ECG

30
Q

How should a patient with SOL be managed in ED?

A

NOT MANAGED IN ACUTE SETTING - refer to neurology
Dexamethasone can be useful in treating cerebral oedema
Symptom control (analgesics for headache)
Temozolomide (new agent used for glioblastomas)

31
Q

How does a patient with temporal arteritis present?

A
Weight loss 
Night sweats
Low grade fever 
Jaw claudication 
Scalp tenderness (tender when shampooing or brushing hair)
Reduced or lost vision (must be dealt with soon because can quickly become irreversible)
Shoulder girdle stiffness 
Muscle aches 

o/e: Area over temporal artery might be red, tender and pulseless

32
Q

What are some common causes / associations with temporal arteritis?

A

AGE (unheard of in under 55)
Women more likely
More likely if you’ve got polymyalgia rheumatica

33
Q

What investigations should be done in someone with temporal arteritis?

A

FUNDOSCOPY - really important
FBC, ESR (ESR will be really high - although if normal does NOT exclude Temporal arteritis)
Biopsy is definitive

34
Q

How should temporal arteritis be managed initially?

A

200mg IV hydrocortisone or 40mg PO prednisolone (assess severity and risk to vision)
Refer to neurology or ophthalmology

35
Q

How will a dural venous sinus thrombosis present?

A

DEPENDS IF IT IS A CORTICAL VENOUS THROMBOSIS OR SINUS THROMBOSIS

CORTICAL: Sudden onset headache, Nausea and vomiting (similar to SAH)

SINUS: headache, N&V, visual losses and papilloedema - more gradual onset

36
Q

Where is the most common sinus for venous thrombosis to form?

A

Saggital sinus

37
Q

What are some important causes of venous sinus thrombosis?

A
Sinus infections
Pregnancy
Post part period 
Head injury 
Recent LP
Malignancy 
Infliximab
38
Q

What investigations is it important to do in people who have a suspected venous sinus thrombosis?

A

LP (raised ICP)
CT
Check for meningitic symptoms
MRI T2 weighted might be useful for visualising thrombus

39
Q

How should venous sinus thrombi be managed initially?

A

Neuro referral

Heparins can improve outcomes

40
Q

What is the difference in onset between sub-dural and extradural haematoma?

A

Sub-dural haematoma is SLOWER (Subdural Slow)
Extra-dural is faster (hours) (Extradural Emergency) and usually it has a period of lucidity, after the trauma, before the onset of symptoms

41
Q

How might we treat a raised ICP?

A

MANNITOL - increases the osmotic pressure of the blood drawing fluid out of the tissues 0.25-1g/kg every 2-6hrs
HYPERTONIC SALINE can be used for the same reasons - increasing osmotic pressure of blood
POSITIONING - elevate the head by 15-30 degrees

42
Q

What are some symptoms of concussion?

A

These symptoms are very common but can cause anxiety in patients and relatives. Common symptoms that are not specifically concerning include:

  • Headache - occurs in most patients and can persist for up to 2 months (intermittent and worse on exertion)
  • Dizziness - non specific
  • Lethargy
  • Inability to concentrate
43
Q

When can we make a diagnosis of concussion?

A

When other symptoms e.g. rhinorrhoea, otorrhoea, bruising, neck pain, photophobia, vomiting have been RULED OUT

Always make sure to safety net about symptoms getting worse or coming back unusually.