Psyh Exam 2 Flashcards

1
Q

perception

A

process by which the brain organizes and interprets sensory information.

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2
Q

Doctrine of specific nerve energies

A

principle that different sensory modalities exist because signals received by the sense organs stimulate different nerve pathways, leading to different areas of the brain. (anatomical code) certain sensory organs are connected to our brains in certain paths
-we see and hear with our brain, not our eyes and ears

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3
Q

Synesthesia

A

condition that a sensation in one modality consistently evokes a sensation in another (ex. smelling colours)
-possibly due to increased neural connections between those sensory areas (or inhibition)

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4
Q

anatomical encoding:

A

doesn’t completely explain why for most ppl our senses are separate, or explain variation of experiences within a particular sense (doesn’t tell us why)

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5
Q

Functional code

A

used to figure out what our sensory receptros are sensing
-info about which cells are firing, how many and at which rate, also the pattern forms a functional code

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6
Q

Absolute theshold

A

minimum amount of energy (or quantity) of a stimulus required for it to be reliably detected (at least 50% of the time it’s presented)- works for all senses -for everybody it’s different (especially between different species, as we age etc)

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7
Q

Difference threshold:

A

smallest difference between two stimuli that a subject can detect
-also known as the “just noticable difference”

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8
Q

difference threshold expressed by?

A

Weber’s fractions (difference threshold is the same percentage for everybody, for weight it is 2% of the weight you are holding (for 10 pound you only need 0.2 pouds added, 100 pounds you need to add 2 pounds) as the stimulus gets larger you need to add a bigger difference)

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9
Q

Signal detection theory

A

psychophysical theory that divides the detection of a sensory signal into a sensory process and a decision process
-for you to actually percieve anything you need to do both processes
-sensory is actual stimuli
-decision is the observers bias
- for example being scared that theres a monster under your bed even though you’re not actually sensing something

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10
Q

hit

A

sensory and decision are both right

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11
Q

false alarm

A

didn’t sense anything but imagined it

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12
Q

miss wrong

A

didn’t sense or decide but it was there

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13
Q

correct rejection

A

didn’t sense and didn’t decide it was there and were right

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14
Q

sensory adaptation

A

resulting decline in sensory responsiveness when a stimulus is uncahgngin or repetitious (your tongue is awkward in your mouth but you forget about that, wearing clothes and not feeling them on your body)
-not always beneficial (smelling gas in your house, and it going away because you got used to it)

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15
Q

selective attention

A

-is the process of focusing on selected aspects of the environment while blocking out others

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16
Q

inattentional

A

is the failure to consciously perceive something you are looking at because you are not attending to it

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17
Q

what is the stimulus for vision?

A

light

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18
Q

hue

A

dimension of visual epxerience specified by color names (related to wavelength)

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19
Q

saturation

A

(colorfulness) dimension of visual experience related to the complexity of light (how wide of narrow the range of wavelengths are)

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20
Q

brightness

A

dimension of visual experience related to the amount/intensity of the light an object emits/relfects (amplitude (max height) of the light wave)

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21
Q

sclera

A

white outer layer of eye

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22
Q

cornea

A

clear layer that covers the front of the eye

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23
Q

pupil

A

regulates amount of light let into the eye (essentially how light gets into your eye, it dialates or constricts based on the iris but it is the thing that lets in light)

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24
Q

iris

A

round muscle that adjusts the size of the pupil

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25
Q

lens

A

clear structure that focuses light onto back of eye

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26
Q

retina (fovea)

A

neural tissue lining the back of eyeballs interior that contains the receptors for vision

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27
Q

cones

A

concentrated on fovea to produce high-detail, colour vision at our point of visual focus (anywhere from 5 and 7 million in the eye) cones are how we see colour and really high detail (more focused in the center of the retina

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28
Q

rods

A

on outer regions of retina provude peripheral vision and are specialized for low light conditions (80+ million) (more focused on the peripheral of the retina) (don’t see colour)
- why it’s harder to see colour in the dark (cones aren’t well adapted to low light but rods are)

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29
Q

bipolar neurons

A

synapse onto photoreceptors

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30
Q

ganglion cells

A

transmits signals from bipolar neurons to brain

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31
Q

optic disc

A

area on retina lacking rods and cones

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32
Q

optic nerve

A

axon tracts from ganglion cells leading to brain

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33
Q

feature-detector cells

A

-specific group of cells jusst above the cerebellum, responding specifically to faces
-cells by the hippocampus help to perceive the environment
-region of cells by the occipital cortex respond stronger to body parts

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34
Q

Trichromatic theory

A

colour vision is determined by 3 different cone types that are sensitive to short, medium and long wavelength light. Also known as Young-Helmholtz Theory
- combined activity of all 3 cone types generates a unique signature associated with each perceivec colour (even colours without corresponding wavelengths)

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35
Q

Color blindness and deficiency

A

normal cone distribution is trichromatic, those with color deficiency are dichromatic and typically don’t have red cones

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36
Q

Opponent-process theory

A

we perceive colour in terms of opposite ends of the spectrum (red to green, yellow to blue, white black)

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37
Q

Opponent-process cells

A

in the retina and the thalamus respond in opposite fashion to short and long wavelengths of light
-are inhibited by a particular color produce a burst of firing when the color is removed, just as they would if the opposing color were present
-similarly, cells that fire in response to a color stop firing when the color is removed, just as they would if the opposing color were present

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38
Q

Negative afterimages

A

the facts above explain why we are susceptible to it when we stare at a particular hue

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39
Q

gestalt psychologists

A

among the first to study how ppl organize the world visually into meaningful units and patterns

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40
Q

gestalt principles

A

desrcibe strategies used by the visual system to group sensory building blocks into perceptual units

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41
Q

Figure and ground

A

figures in our envrionment tend to stand out against a background, issues arise when we have a tough time distinguishing between what is the figure and what is the background

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42
Q

proximity

A

group them together if they are close

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43
Q

similarity

A

if things are alike in someway (color, shape, size) they are perceived to belong together

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44
Q

continuity

A

perceptual rule that lines and other objects tend to be continous, rather than abruptly changing direction

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45
Q

principle of closure

A

ppl tend to fill in the blanks to percieve a complete object

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46
Q

consciousness

A

the awareness of oneself and the envrionment

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47
Q

biological rhythms

A

are periodic, more or less regular fluctuation in a biological system, often having psychological implications (rhythms are based both on external and ENDOGENOUS cues- either inside or things going on outside of the body)

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48
Q

Circadian rhythms

A

biological rhythms with a ~24 hour periodicity
-ex. sleep cycle, body temp

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49
Q

entrainment

A

synchronization between biological rhythms and external cues (zeitgebers)
-ex. noisey cat that likes to be fed at a certain time and wakes you up (your biological rythmn is being trained by an outside thing)
-alarm clock is also an example

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50
Q

light entrainment

A

-slowly changing levels of background illumination detected by specialized ganglion cells of the retina
-light signals communicated to the brain via optic nerve to the optic chiasm
-suprachiasmatic nucleus (SCN) above the optic ciasm recieves light information
-SCN tells the pineal gland to stimulate the releasae of melatonin
-melatonin helps you sleep
ex when it’s getting dark out you start to get sleepy

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51
Q

internal desynchronization

A

when our normal routine changes
-sleep and wake patterns adjust quickly but temperature and hormones may take a few days
-affects energy level, mental skills, motor cooridination
ex. jet lag, shift work
-it’s easier to stay up at night compared to going to bed early, going to the east of the world (towards europe) it’s harder to adjust

it is real that some ppl are early risers or night owls

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52
Q

seasonal affective disorder (SAD)

A

experiencing depression during particular seasons (usually winter)
symptomns include sadness, lethargy, drowsiness, cravings (light therapy helps)

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53
Q

waves when awake

A

beta waves

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54
Q

waves during Stage 1 NREM

A

begins as alpha and moves to theta

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55
Q

Stage 1 NREM

A

-light sleep (on the edge of consciousness) very easy to wake up, highly sensitive to noises

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56
Q

waves during stage 2 NREM

A

Theta waves (sleep spindles to k-complexes)-different types of spikes in our brain waves (no one’s really sure of their purpose, we think they help maintain our sleep?)
-short bursts of rapid waves

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57
Q

waves during stage 3/4 NREM

A

delta (very slow waves with high peaks) deepest sleep stage

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58
Q

REM (rapid eye movement)

A

when we start dreaming (typically), brain waves are very active again, most of our skeletal muscles are limp, vivid dreams are reported most often during REM
-even though your brain is active it’s very restfu

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59
Q

1->2->3->2->1->REM->1->2->3->2->1->REM (repeats)
the more cycles we go through the shorter our 3 stage is and the longer the REM

A

1->2->3->2->1->REM->1->2->3->2->1->REM (repeats)
the more cycles we go through the shorter our 3 stage is and the longer the REM

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60
Q

2 reasons why we sleep

A

Restore and Repair Hypothesis: the idea that the body needs to restore energy levels and repair any wear and tear experienced during the day’s activities
Preserve and Protect Hypothesis: suggests that two or more adaptive functions of sleep are preserving energy and protecting the organism from harm

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61
Q

signs of sleep deprivation

A

-more readily falls asleep
-irritability/emotional deficits
-attention/vigilance deficits
-maintenance of energy is hard
-reading/studying next to impossible
-after 2-3 days microsleeps begin to occur

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62
Q

types of insomnia

A

onset
maintenance
terminal
-primary and secondary

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63
Q

onset insomnia

A

difficulty falling asleep

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64
Q

maintenance insomnia

A

difficulty returning to sleep

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65
Q

terminal insomnia

A

waking up too early

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66
Q

primary insomnia

A

due to internal source (worrying) its the issue itself

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67
Q

secondary insomnia

A

results of other disorders, symptom of other things

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68
Q

Restless legs syndrome:

A

: persistent discomfort in legs and urge to continously shift them into different positions

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69
Q

REM behviour disorder

A

only occurs during REM sleep, act out dreams due to failure to inhibit motor signals

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70
Q

Somnambulism (sleepwalking)

A

disorder that involves wandering and performing other activities while asleep

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71
Q

Sleep apnea

A

temporary inability to breathe during sleep as airway becomes obstructed
-obesity
-damage to medulla

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72
Q

Narcolepsy

A

extreme daytime sleepiness and even sleep attacks
-few seconds to minutes
-immediately into REM sleep

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73
Q

sleep paralysis

A

between REM and awake, your muscles can’t move but your brain is awake

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74
Q

which stage of sleep facilitates learning

A

REM

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75
Q

which are important for locking in learning

A

late REM

76
Q

slow-wave sleep (3 and 4) are important for what?

A

learning autobiographical memories

77
Q

neural replay

A

fast-forward playback in which pattern of neural activity that occured while awake is repeated during sleep

78
Q

manifest content

A

images and storylines we dream about

79
Q

latent content

A

symoblic meaning of dream built on supressed sexual of aggressive urges

80
Q

two different explanations to dreaming

A

efforts to deal with problems and dreams as thinking

81
Q

explain dreams as efforts to deal with problems

A

-dreams reflect conscious preoccupations of waking life (ex concerns over relationships, work, sex or health)

82
Q

describe the problem focused approach to dreaming

A

-symbols and metaphors in a dream do not disguise its true meaning (they convey it)
-supported by findings that dreams are more likely to contain material related to a person’s current problems/concerns

83
Q

describe cognitive approach

A

-emphasizes current concerns, makes no claim about problem-solving during sleep
-dreams include thoughts and scenarios that may or may not relate to our daily problems
-brain does same kind of work during dreams as when awake

84
Q

the 3 main differences when we sleep compared to awake

A

-we are cut off from sensory input of the world
-our thoughts are less focused (all over the place)
-the only input to the brain si its own output

85
Q

what is now the leading contender for dream theory

A

dreams as interpreted brain activity

86
Q

what is the activation-synthesis theory

A

dreams occur when the cortex tries to make sense of or interpret spontaneous neural firing initiated in the pons
-signals from the pons
-existing knowledge
-memories

87
Q

why is the theory of dreams as interpreted brain activity criticized

A

criticized because it doesn’t explain coherent, story-like dreams and non-REM dreams

88
Q

Hypnosis

A

procedue in which the practitioner auggests changes in a subjects
-sensations
-thoughts
-feelings
-behaviour
the person tries to alter their cognitive processes to comply with the hypnotists suggestions

89
Q

Dissociation

A

a split in consciousness
(split is between part of conciousness that is hypnotized, a hidden observer that watches but does not participate)
(executive-control system in the brain, other brain systems responsible for thinking and acting, executive system turns off and hands its function over to the hypnotist, they can suggest how we should interpret the world and respond to it)

90
Q

Sociocognitive explanation

A

product of normal social and cognitive processes
-combination of the hypnotized persons expectations and beliefs
-desire to comply with the hypnotists suggestions

91
Q

Biological theories for hypnosis

A

-alpha waves
-changes in the control of attention, the central executive network
-it can also reduce conflict between 2 mental tasks
(stroop effect, name the color when the word says a different color, when youre hypnotized you are very able to seperate the color and the word)

92
Q

what is the scale of consciousness measured on?

A

glasgow coma scale
-monitors eye response, motor response and verbal response

93
Q

brain death

A

brain stem no longer functions, no potential for recovery

94
Q

coma

A

complete loss of consciouness, no response from pupils

95
Q

Persistent vegetative state (PVS)

A

state of minimal to no consciousness
-eyes open but do not track movement
-normal sleep cycles
-best hope for recovery is before 6 months
(terry shiavo, kick started a lot of ethical dilemas, husband and family wanted to do two different things, massive fight over who had power of attourny if they should pull the plug)
- some patients show signs of consciousness
-fMRI sugggests comprehension of comands and mental imagery

96
Q

mnimally conscious state

A

ability to show some behaviours that suggests partial conciousness (beyond reflexes)

97
Q

locked-in syndrome

A

patient is aware and awake but often appears unconcious

98
Q

short-term drug effects

A

affect neurotransmitter activity through various agonistic and antagonistic effects

99
Q

Nucleus accumbens

A

prime area of activation when a person engages in rewarding behaviour
-lots of drugs target this area of the brain
-lots of dopamine release
-the first time is always the best and you are always trying to chasae that same high, continue to use more and more and your dopamine system gets messed up so you don’t get it from other places

100
Q

Pysical dependence

A

withdrawl physical systems

101
Q

psychological dependence

A

need to take a drug to ward off negative emotions (bad coping mechanism)

102
Q

context-dependent overdose

A

our body anticipates the drug response, conditioned to using the drug, if youre always using a drug in a certain place your body prepares

103
Q

what do stimulants do?

A

speed up nervous system, enhancing wakefulness and alertness

104
Q

symptoms of stimulants

A

-euphoria, increased energy, lowered inhibitions
-increased dopamine, serotonin, norepinephrine
-tolerance develops quickly
-high risk of dependence

105
Q

examples of stimulants

A

cocaine, ecstasy

106
Q

examples of physical deterioration due to stimulants

A

-hygiene neglected
-drug cocktails often include (farm fertilizer and hydrochloric acid)

107
Q

examples of cognitive deterioration from stimulants

A

-structural abnormalities in cells of the frontal lobes
-users develop difficulties ignoring irrelevant thoughts

108
Q

symptoms of sedatives

A

-drowsiness, relaxation, sleep
-increase GABA activity
-tolerance develops quickly
-high risk of dependence

109
Q

examples of sedatives

A

xanax and valium

110
Q

symptoms of hallucinogens

A

perceptual distortions
-range from euphoria to fear, panic, paranoia
increased serotonin, blocks glutamate receptros
-tolerence develops slowly
-low risk of dependence

111
Q

3 examples of hallucinogens

A

LSD, ketamine, DMT

112
Q

symptoms of opiates

A

reduce pain and induce extremely intense feelings of euphoria
-intense euphoria, pain relief
-stimulate endorphin receptors
-tolerance develops very quickly

113
Q

6 examples of opiates

A

morphine, heroin, fentanyl, oxycodone, oxycontin, codeine

114
Q

what is classical conditioning?

A

process in a previously neutral stimulus becomes associated with a stimulus that already elicits a response and in turn acquires the capacity to elicit a similar or related response

115
Q

another word for classical conditioning

A

pavlovian conditioning

116
Q

what is an unconditioned stimulus

A
  • stimulus that elicits a reflexive response without learning (food water pain)
117
Q

what is an unconditioned response

A

a relflexive, unlearned reaction to an uncondtioned stimulus (hunger, drooling, expression of pain)

118
Q

what is a conditioned stimulus

A
  • a stimulus that doesn’t elicit a specific response ex. a bell
119
Q

what is a conditioned response

A

learned response that occurs to the conditioned stimulus

120
Q

what is extinction

A

conditioned stimulus is repeatedly presented without the unconditioned stimulus. the conditioned response eventually disappears although later it may reappear (sponateous recovery). reappearance shows why more than one extinction session is often required.

121
Q

what is higher order conditioning

A

a neutral stimulus becomes conditioned stimulus by being paired with an already-established condtioned sitmulus. this may explain why some words trigger emotional lresponse (anger, sentimental feelings) why a child may learn positive word associations (birthdays=gifts) or negative ones to ethnic or demographic labels paired with disagreeable words (lazy, violent) and this may contribute to the formation of predjudice

122
Q

stimulus generalization

A

a process where a response that originally occurred for a specific stimulus also occurs for different thought similar stimuli (reaching for your phone when you hear someone else’s phone buzz)

123
Q

stimulus discrimination

A

occurs when an organism learns to respond to one original conditioned stimulus but not to new stimuli that may be like the original stimulus. the opposite of stimulus generalization

124
Q

behaviourism

A

approach that empahsizes the study of observable behaviour and the role of the envrionment and prior experiences as determinants of behviour. positive emotional responses to objects and events through the paring of a neutral stimulus with a pleasureable one (advertising). if something good happened after doing something you will be more likely to do that activity again

125
Q

fear conditioning

A

a process by which an organism learns that a previously neutral stimulus reliably predicts an unconditioned stimulus, and the conditoined stimulus provokes a conditioned fear response

126
Q

fear extinction

A

teaching the subject that they do not have to fear the conditioned stimulus. the conditioned fear response declines

127
Q

counter-conditioning-

A

process of associating a condiioned stimulus with a stimulus that elictis a response that is incompatible with an unwanted conditioned response. basically, every time you give someone something that they fear you alsogive them something that makes them happy. systematic desensitization and exposure therapies now used to treat phobias and other anxiety disorders

128
Q

how can classical conditioning explain learned reactions to food taste

A

behavioural scientisits taught animals to dislike foods or odoros associating them with drugs that cause nausea or othe unpleasent symptomns. due to evolutionary adaptations we are biologically primed to acquire some classically conditioned responses easily (garcia effect) often a single tiral pairing a stimulus with an unpleasent outcome can produce aversive conditioning

129
Q

operant conditioning

A

behaviour becomes more or less likely to occur depending on its consequences
-responses are generally not reflexive
-more complex then classical conditioning (seeing that a particular behaviour leads to different outcomes)
-relies on contingency (consequence depends on action)
-reinforcement(any action following that behaviour increases the lieklihood of that behaviours occuring again)
-punishment( any action following that behaviour decreases the likelihood of it happening again)

130
Q

what is a primary reinforcer

A

naturally reinforcing (satisfy a biological need) ex. food

131
Q

what is a secondary reinforcer

A

when they have aquired their ability to strengthen a response through association with other reinforcers (ex money can get you the primary things like food because of its value) similar for punishemnet

132
Q

positive reinforcement

A

pleasant follows response

133
Q

negative reinforcement

A

unpleased is removed

134
Q

positive punishment

A

unpleasent follows response

135
Q

negative punishment

A

pleasant is removed

136
Q

positive

A

add stimulus to increase behaviour

137
Q

negative

A

take away to decrease behaviour

138
Q

avoidance learning

A

type of negative reinforcement that removes the possibility that a stimulus would occur ex. paying your bill early to avoid late fees

139
Q

escape learning

A

negative reinforcement that occurs if a response removes a stimulus that is already present ex. covering your ears if it’s loud

140
Q

stimulus generatization

A

responses may generlize to similar stimuli not originally present during the original learning situation but resemble the original in some way (different loud noises and being scared)

141
Q

stimulus discrimination

A

signals when a response is likely to be followed by a certain type of consequence (ex doing things infront of your friends that you wouldn’t infront of your parents)

142
Q

shaping

A

process of reinforcing successive approximations of a specific operant response

143
Q

chaining

A

involves linking together 2+ shaped behaviors into a sequence of actions (atomic habits like)

144
Q

latent learning

A

not immediatly expressed in an overt response and occurs without obvious reinforcement. ex tolman and honziks rat box study (not a specific response but the knowledge about responses and their consequences)

145
Q

Social-cognitive learning theories

A

1960-70s increased influence
-beliefs, interpretations andd other cognitions determining behaviour
-two people come away with entirely different lessons from the same event

146
Q

in explicit memory is recognition or recall better?

A

recognition

147
Q

recognition

A

ability to identify information previously seen

148
Q

example of recognition

A

true or false

149
Q

recall

A

ability to retrieve and reproduce information previously seen

150
Q

examples of recall

A

short answer/essay questions

151
Q

information processing models 4 steps

A

input
output
accessing and
retrieval of information

152
Q

metaphor for the information processing model

A

computer with codes

153
Q

three box model 3 interacting systems

A

sensory register
working memory and
long-term memory

154
Q

the connectionist model is also known as

A

parallel distributed processing model

155
Q

connectionist model is similar to what

A

neurons in the brain

156
Q

sensory register

A

memory store that holds perceptual information for a brief amount of time

157
Q

iconic memory

A

visual form, held for about 1 second

158
Q

echoic memory

A

audio form, held for 5-10 seconds

159
Q

full report

A
160
Q

half report

A
161
Q

short term/working memory

A

actively retains info for brief period and keeps it availble for use

162
Q

magic number

A

7-/+ 2

163
Q

how do we store things in our long term memory

A

into semantic categories ex. cat would be closer to dog and not tiger because they are both pets
-also organize info based on the sound or how the word looks (leads to tip of the tongue phenomenon)

164
Q

two sections of explicit memory

A

semantic and episodic

165
Q

tow section of implicit memory

A

procedural and conditioning

166
Q

semantic

A

carry facts about the world
ex. what a bike is and how the gears work

167
Q

episodic

A

personal experience in “episodes”, first person perspective
ex. a specific memory of you riding a bike

168
Q

procedural

A

muscle memory
ex. how to ride a bike

169
Q

priming

A

exposure to a stimulus unconciously changes our behaviour at a later point

170
Q

serial position effect

A

you remember stuff at the beginning and end of a list

171
Q

primary effect

A

you remember stuff at the beginning

172
Q

recency effect

A

remember stuff at the end of the list and what you heard most recently

173
Q

proactive interference

A

makes it difficult to remember additional info

174
Q

retroactive interference

A

makes it difficult to remember preceding words

175
Q

does long term memory have strucutral changes on the brain?

A

yes

176
Q

long term potentiation

A

a long-lasting increase in the strength of sunaptic responsiveness, long term memory, likely underlies many and perhaps all forms of learning and memory. (basically using neurons makes them stronger)

177
Q

role of the amygdala

A

formation, consolidation and retrieval of memories (especially emotionally arousing events)

178
Q

role of the frontal lobe

A

especially active during working-memroy tasks

179
Q

prefrontal cortex/temporal lobe

A

encoding words and picutres

180
Q

hippocampus

A

formation and retrieval of long term memories, recalling past events, general ability to percieve the passage of time

181
Q

cerebellum/striatum

A

crucial for formation of implicit memories

182
Q

memory is a cluster of info

A
183
Q

flashbulb memories

A

emotional memories that are often intense and vivid, they still become less accurate overtime

184
Q

what releases hormones

A

adrenal glands

185
Q

2 hormones

A

epinephrine and norepinephrine

186
Q

how can hormones enhance memory

A

-cause the level of glucose to rise in blood stream
-glucose may enhance memorydirectly or by altering the effects of neurotransmitters
** the effective dose is narrow tho, too much can impair instead of aide

187
Q

mnemonic

A

formal strategy for improving memory
ex. memory palace
-studying is basically using mnemonics