Psychological Problems Flashcards

1
Q

Mental health problems

A

Difficulties in the way a person thinks/feels/behaves

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2
Q

Characteristics of ideal mental health

A
  • self-attitude
  • personal growth
  • integration - coping with stressful situations
  • autonomy
  • accurate perception of reality
  • mastery of environment - love/relationships/function at work
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3
Q

Cultural variations in beliefs about mental health

A
  • England - hearing voices seen as symptom of schizophrenia, positive in other countries
  • North Korea doesn’t recognise depression
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4
Q

How have incidences of mental health problems changed over time

A
  • number of mental illnesses in UK increased - 1 in 2
  • 24% adults accessing treatment in 2007, 37% in 2014
  • Mind estimates there will be 2 million more by 2030
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5
Q

Increased challenges in modern living

A
  • poverty/financial issues
  • COVID-19
  • getting older
  • condense urban areas
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6
Q

How have poverty/financial issues influenced mental health problems

A

27% men report mental health problems in low income, 15% in high income

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7
Q

How has COVID-19 influenced mental health probems

A
  • increased mental health problems as:
  • less able to socialise - becoming isolated
  • increased death of loved ones
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8
Q

How do condense urban areas influence mental health problems

A
  • increased mental health problems as:
  • less socialisation
  • surrounded by people in same social cycle
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9
Q

Social stigma of mental health

A

Automatic negative response about mental health

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10
Q

How has lessening social stigma affected mental health problems

A

People may be more confident in seeking treatment to address issues

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11
Q

Individual effects of mental health problems

A
  • damage to relationships
  • difficulties coping with day to day life
  • negative impact on physical wellbeing
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12
Q

Damage to relationships

A
  • key element of relationship is communication - MHPs affect this ability
  • can’t talk / understand others points
  • fear judgment so avoid/isolate - can be misinterpreted by others
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13
Q

Difficulties coping with day to day life

A
  • difficult to look after self - get dressed, clean, work
  • may not distress patient but will others
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14
Q

Negative impact on physical wellbeing

A
  • stress = cortisol
  • cortisol prevents immune system working as well
  • stressed people get more colds ect.
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15
Q

Social effects of mental health problems

A
  • need for more social care
  • increased crime rates
  • implications for economy
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16
Q

Need for more social care

A
  • social care - food, company, ect.
  • taxes increase or other areas of tax use increase
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17
Q

Increased crime rates

A
  • research shows MHPs means 4x more likely to commit crime
  • also other factors affecting this like substance use
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18
Q

Implications for economy

A
  • same amount spent on MH as education
  • drug treatment reported to be cheaper than psychological therapy
  • increase in dementia in aging population - more costly as time goes on
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19
Q

Depressioin

A
  • abnormal state with prolonged sadness
  • physical changs - smaller hippocampus + retracted frontal lobe
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20
Q

Types of depression

A
  • unipolar
  • bipolar
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21
Q

Unipolar depression

A

Depression where person only experiences 1 emotional state

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22
Q

Bipolar depression

A

Depression where person fluctuates between 2 states - depression and mania

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23
Q

Sadness

A

Normal human emotion to certain situations

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24
Q

Key symptoms of unipolar depression

A
  • low mood or persistent sadness
  • loss of interests/pleasure
  • fatigue or low energy
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25
Q

Other symptoms of unipolar depression

A
  • disturbed sleep
  • poor concentration or indecisiveness
  • low self-confidence
  • poor or increased appetite
  • suicidal thoughts/acts
  • agitation or slowing of movements
  • guilt or self-blame
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26
Q

How long must symptoms last for unipolar depression diagnosis

A

At least 2 weeks

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27
Q

How many symptoms are needed for mild unipolar depression

A

4

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28
Q

How many symptoms are needed for moderate unipolar depression

A

5-6

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29
Q

How many symptoms are needed for severe unipolar depression

A

7

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30
Q

Unipolar depression biological explanation PARTS

A
  • neurotransmitters
  • serotonin
  • other affects of serotonin
  • reasons for low serotonin levels
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31
Q

Unipolar depression biological explanation NEUROTRANSMITTERS

A

Messages that travel along neuron electrically + transmitted across synapse chemically

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32
Q

Unipolar depression biological explanation SEROTONIN

A
  • high levels of serotonin in synaptic cleft = postsynaptic neuron stimulated more, improving mood
  • low levels of serotonin in synaptic cleft = less stimulation of postsynaptic neuron, results in low mood
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33
Q

Unipolar depression biological explanation OTHER AFFECTS OF SEROTONIN

A
  • affects memory, sleep, appetite
  • links to characteristics of depression - lack of concentration, disturbed sleep, reduced appetite
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34
Q

Unipolar depression biological explanation REASONS FOR LOW SEROTONIN LEVELS

A
  • genes - may inherit poor ability to produce serotonin
  • diet - may have low levels of tryptophan (key ingredient of serotonin), high protein foods + carbohydrates contain tryptophan
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35
Q

Unipolar depression biological explanation STRENGTH

A
  • supporting research
  • McNeal + Cimbolic (1986) found lower serotonin levels in people with depression
  • suggests link between low serotonin levels and depression
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36
Q

Unipolar depression biological explanation WEAKNESSES

A
  • low serotonin levels could be caused by being depressed
  • difficult experiences can cause low levels, result of psychological experiences
  • reductionist - some with low serotonin levels don’t have depression, neurotransmitter explanation not enough
    +
  • depression may have other causes as well
  • unlikely neurotransmitters alone explain depression, ‘diathesis-stress’ explanation suggests deppression can be caused by inheriting low serotonin levels (diathesis), also have stressful experiences like war (stress)
  • combination may lead to depression
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37
Q

Unipolar depression psychological explanation PARTS

A
  • faulty thinking
  • negative schemas
  • attributions
  • leaned helplessness
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38
Q

Unipolar depression psychological explanation FAULTY THINKING

A
  • focus on negatives, ignore positives
  • causes feelings of hopelessness + depression
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39
Q

Unipolar depression psychological explanation NEGATIVE SCHEMAS

A

Negative self schema = likely to interpret all info about themselves negatively

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40
Q

Unipolar depression psychological explanation ATTRIBUTIONS

A
  • process of explaining cause of behaviour
  • Seligman suggested some people have negative attributional style
  • internal - blame self for negative events
  • stable - believe cause of failiure unchangeable
  • global - apply negative outcome of 1 situation to all aspects of life
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41
Q

Unipolar depression psychological explanation LEARNED HELPLESSNESS

A
  • Seligman suggested negative attributional style learnt (nurture)
  • e.g- negative experience makes you want to escape, failing will make you stop trying
42
Q

Unipolar depression psychological explanation STRENGTHS

A
  • Seligman demonstrated process of learnt helplessness
  • dog learnt to react to electric shock after bell rang by giving up, not trying to escape shocking floor anymore
  • research supports explanation of depression due to negative attributions
    +
  • leads to ways of treating depression
  • depression can be treated with CBT improving people’s thinking with thought diaries + disputing
  • explanation can improve people’s mental health
43
Q

Unipolar depression psychological explanation WEAKNESS

A
  • opposing research
  • Alloy + Abraham (1979) found depressed people gave more accurate estimate of likelihood of disaster than ‘normal people’ - sadder but wiser
  • opposes negative attributional style ideas
44
Q

Methods of treating depression

A
  • antidepressant medications
  • Cognitive Behaviour Therapy
45
Q

How does serotonin release happen normally with no drugs

A
  • serotonin stored in vesicles
  • electrical signal opens vesicles
  • serotonin released into synaptic cleft
  • serotonin binds to postsynaptic receptors
  • unused serotonin reabsorbed by presynaptic neuron
  • serotonin broken down + reused
46
Q

How do antidepressant meds work

A

Block reuptake to reduce symptoms of depression

47
Q

SSRIs

A

Selective Serotonin Reuptake Inhibitor

48
Q

SSRIs WEAKNESSES

A
  • reductionist approach
  • idea depression only caused by biological factor to action of neurotransmitters
  • more holistic approach would look at psychological + biological factors
    +
  • questionable evidence for effectiveness
  • takes 3-4 months for SSRIs to impact symptoms, should impact serotonin instantly so symptoms instantly, Asbert (1976) found serotonin levels may not be that different between depressed/normal anyway
    +
  • negative side effects
  • dizziness, dry mouth, erectile dysfunction, may outweigh good
  • people may stop taking them
49
Q

CBT

A

Structured therapy that helps identify + change negative thought patterns/behaviours to improve mental health

50
Q

Aims of CBT

A
  • help patient change way they think
  • help patient change way they act to improve symptoms
51
Q

How does CBT change behaviour

A
  • changes thinking through changing behaviour
  • behavioural activation - plan pleasant activity everyday (making meal) leading to feeling of accomplishment, positive emotions lead to positive mood
52
Q

CBT methods

A
  • disputing
  • thought diary
53
Q

CBT - disputing

A

Challenging clients’ irrational thoughts (asking for proof) to rationalise + increase self belief

54
Q

CBT - thought diary

A
  • client records unpleasant emotions + thoughts
  • rate them on scale (1-100%) of how much they believe them
  • client records more rational version of emotions/feelings, rate them
55
Q

CBT STRENGTHS

A
  • holistic approach
  • focuses on treating whole person, not just constituents of depression (neurotransmitters), adresses problems on deeper psychological level
  • likely more effective than antidepressants alone, deal with core symptoms
    +
  • long-lasting effectiveness
  • long term aim of giving patient tools to deal with depression themselves, learn techniques to use again and again
  • more useful, doesn’t just deal with current problems
56
Q

CBT WEAKNESS

A
  • not everyone willing to spend long-time for success
  • clients often meet therapist every week for months + do homework between sessions, takes effort (SSRIs passive)
  • many drop out, fail to benefit
57
Q

Wiles DATE

58
Q

Wiles AIM

A

See if more holistic approach is better at treating depression - antidepressants only work for 30%

59
Q

Wiles METHOD

A
  • 469 Ps with treatment resistant depression (drugs not working after 6 weeks) from 73 different GPs with BDI score 14+
  • independent groups - antidepressants only + antidpressents with CBT (12-18 sessions)
  • randomly assigned to condition of IV with computer
  • symptom changes measured with BDI - higher score = higher depression levels
60
Q

Wiles RESULTS

A
  • after 6 months, 90% of Ps remained
  • antidepressants only - 21.6% had over 50% symptom reduction
  • antidepressants + CBT - 41.6%
  • results consistent after 12 months
61
Q

Wiles CONCLUSION

A
  • CBT useful addition to drugs
  • should look at holistic biological + psychological treatment
62
Q

Wiles STRENGTHS

A
  • well designed to avoid EVs
  • independent groups may have caused on group to have sadder Ps, group assignment random from computer + groups checked for similar BDI scores
  • confident DV not interfered with by EVs
    +
  • ecological application
  • focused on developing useful therapy, demonstrated holistic treatment can work, more successful than just drugs, therapy also cheaper (avg £343 / year)
  • good reason for research
63
Q

Wiles WEAKNESS

A
  • ineffective self-report method
  • subjective, Ps may not answer truthfully, some may under/overestimate how sad they feel
  • reduced validity - not objective results
64
Q

Addiction

A
  • individual taking substance or doing behaviour that’s pleasurable, then becomes compulsive with harmful psychological/physical consequences - dependence + tolerance + withdrawals
  • needing to have/do something regularly to avoid negative feelings + go about normal routine, take over interest in other activities + friends/family
65
Q

Dependence

A
  • compulsion to keep taking substance
  • can still lead normal life
  • signaled by physical/psychological withdrawals
66
Q

Substance misuse

A

Using drug in wrong way

67
Q

Substance abuse

A

Using drug for bad purpose - e.g- getting high instead of medicinal benefits

68
Q

Clinical characteristics of addiction

A
  • strong desire to use substance despite harmful consequences
  • difficulty in controlling use
  • higher priority given to the substance than other activities/obligations
  • withdrawal state - tired/headache/anxiety
  • evidence of tolerance
69
Q

Hereditary factors

A

Information passed from 1 generation to the next, can lead to genetic vulnerability for a disorder if inherit certain genes

70
Q

Genetic vulnerability

A

Genes do not determine disorder, increase risk of individual having disorder

71
Q

Types of twin

A
  • monozygotic
  • dizygotic
72
Q

Monozygotic twins

A

Share 100% of DNA

73
Q

Dizygotic twins

A

Share 50% of DNA

74
Q

Kaji’s twin study DATE

75
Q

Kaji’s twin study AIM

A

See if children inherit genetic vulnerability to become alcoholic

76
Q

Kaji’s twin study METHOD

A
  • 310 male Swedish twins - at least 1 registered on Swedish temperance board
  • interviewed twins (sometimes relatives) - info about drinking habits + to see if twins were MZ/DZ
  • 48 (mz), 126 (dz)
77
Q

Kaji’s twin study RESULTS

A
  • 61% identical twins both alcoholic
  • 39% non-identical twins both alcoholics
78
Q

Kaji’s twin study CONCLUSION

A
  • suggests alcoholism related to hereditary factors
  • results don’t 100% support idea of genetic vulnerability - must be environmental factors also
79
Q

Kaji’s twin study STRENGTH

A
  • supported by later research
  • Kendler (1997) did better controlled study (2,516 twins), found if 1 co-twin alcoholic, other more likely to be also, 48% if MZ, 33% if DZ
  • supports Kaji’s conclusions drawn, results consistent over time
80
Q

Kaji’s twin study WEAKNESSES

A
  • flawed design
  • temperance board only includes some kinds of alcohol problems (drink driving + public drunkness), info MZ or DZ self-reported or from informant
  • conclusions may not have been accurate
    +
  • biological addiction explanation may be misleading
  • many assume inheriting certain genes makes addiction inevitable, genes increase risk but also nurture/environmental factors
  • explanation implies genes more influential than they are
81
Q

Psychological explanations of addiction

A
  • peer influence
    -social learning theory
    -social norms
    -social identity theory
    -opportunities for addictive behaviour
82
Q

Peer

A

People who are your equal - e.g- same age

83
Q

Peer influence

A

Effect peers have on us, strongest in adolescence when spending more time with peers than family

84
Q

Social learning theory

A
  • Albert Bandura
  • learn by observing + imitating behaviour of others
  • may happen unconsciously
  • may be done as we see someone respected/rewarded for a behaviour
  • more likely to imitate someone we admire + identify with
85
Q

Social norms

A

Rules about behaviour that you learn from people around you, look at behaviour of others to decide how you should act

86
Q

Social identity theory

A

Each of use belong to many groups, to be accepted by peers in group, must behave + think like them

87
Q

Opportunities for addictive behaviour

A
  • peers provide opportunity for something like smoking/drinking
  • peers may also direct/instruct on what to do in such situations
88
Q

Psychological explanation of addiction STRENGTH

A
  • research support
  • Simons-Morton + Farhat (2010) reviewed 40 studies into relationship between peers + smoking, found positive correlation
  • doesn’t mean peer influence causes addiction but likely risk factor
89
Q

Psychological explanation of addiction WEAKNESSES

A
  • peer influence may work in other direction
  • people may pick groups of peers who behave as they are
  • shared addictive behaviours within group may be consequence of original addiction
    +
  • reductionist
  • likely not peer influence alone influencing someone’s use of substances, also genetics, mental health issues, personality, past trauma, found to play role
  • shouldn’t focus on single type of factor for accurate theory
90
Q

Purpose of aversion therapy

A

Recondition brain to fight cravings to fight addiction

91
Q

Aversion therapy method

A
  • UCS - patient given something to cause negative feeling/reaction to NS - UCR
  • repeat for few times
  • CS causes negative feeling/reaction on its own - CR
92
Q

Aversion therapy STRENGTH

A
  • can be combined with CBT for greater effectiveness
  • holistic approach can deal with addictive behaviour + underlying cognitive factors, therapist can focus on coping strategies for relapse
  • can get rid of immediate urge to return whilst providing long-lasting support
93
Q

Aversion therapy WEAKNESSES

A
  • many addicts abandon therapy
  • won’t work unless unpleasant, many drop out before completing treatment, difficult to assess effectiveness as those who stay more determined to overcome addiction
  • difficult for research to come to generalisable conclusions
    +
  • benefits may be short-term
  • McConaghy (1991) found aversion therapy effective in reducing gambling behaviour after a year, follow up after 9 years found therapy no more effective than placebo
  • reduced validity
94
Q

Self-management programmes

A

Therapy not needing licensed therapist to be carried out, patient control recovery individually, benefiting members also direct activities

95
Q

Self-help groups

A

Peer sharing model when members support each other, contain/avoid religious ‘higher power’ aspects

96
Q

12-step recovery programmes PARTS

A
  • the ‘higher power’
  • admitting and sharing guilt
  • lifelong process
97
Q

12 step recovery programmes HIGHER POWER

A
  • 2,3,6,7
  • addict surrenders their control to higher power
  • even if non-religious, still letting go of free will
98
Q

12 step recovery programmes ADMITTING AND SHARING GUILT

A
  • 4,5,6,7,8,9
  • members of group + ‘higher power’ hear confessions but still accept ‘sinner’
99
Q

12 step recovery programmes LIFELONG PROCESS

A
  • 10,11,12
  • members of group continue to support each other
  • keep book of names + phone numbers if feel they will relapse and need help
  • carry message of 12 steps to others
100
Q

12 step recovery programmes STREGNGTH

A
  • holistic approach
  • many steps deal with emotions, groups provide social support
  • better than reductionist aversion therapy, only targets stimulus-response links
101
Q

12 step recovery programmes WEAKNESSES

A
  • lack of evidence indicating effectiveness
  • Cochrane review systematically reviewed Ferri et al (2006), reported no significant differences between AA and other treatments, Gray (2012) found 81% left within first year
  • difficult to draw conclusions
    +
  • may only be effective for certain types of people
  • high dropout rates suggest it is demanding, continued attendance requires high motivation, some people may also not want to share personal experiences with group
  • less useful, limited to particular people