Psychological Explanations: Cognition Flashcards
What biological evidence have we already covered that would suggest cognition is impaired in schizophrenia
- reduced processing in the ventral striatum is associated with negative symptoms
- reduced processing of information in the temporal and cingulate gyri are associated with hallucinations
- lower than usual levels of information processing suggests that cognition is likely to be impaired
how does Frith (1979) suggest that attention plays a role in cognitive deficits in SZ
- “attention deficit theory” = SZ is due to a faulty attention system unable to filter preconscious thought and gives too much significance to the information that would usually be filtered, overloading the mind
- accounts for +ve symptoms
frith identified 2 kinds of dysfunctional thought processing that could underlie some symptoms. What are they
- metarepresentation
- central control
metarepresentation = …
- the cognitive ability to reflect on thoughts and behaviour
- disrupts our ability to recognise our thoughts as our own
metarepresentation: egocentricity bias = ….
the patient wrongly believes that they are central to any event or situation
central control = …
- cognitive ability to suppress automatic responses
- been identified as a way to explain speech poverty
- people with SZ experience derailment of thoughts and spoken sentences because each word triggers automatic associations that they cannot suppress
What evidence did Frith provide to support his theory
- cognitive neuroscience studies
- 30 SZ patients with various symptoms had PET scans
- indicated a reduction in blood flow in the frontal cortex with patients with negative symptoms
- showed an increase in activity in the area temporal lobe responsible for the retrieval for the of memories with patients with the reality distortion
- suggests biological differences in SZ brain regions associated with the theorised cognitive processes
What evidence did Stirling et al (2006) provide of dysfunctional central control
- Stroop test conducted on 30 patients with SZ and 18 control patients
- involved naming the ink colours on words without saying the word
- found that patients with SZ took 2x as long to name the colour as the controls
- suggests SZ patients have dysfunctional thought processing in case with faulty central control
What does Frith suggest the role of meta-representation does and relate to symptoms
- dysfunction disrupts our ability to recognise out thoughts as our own - could lead to hearing voices
- patient wrongly believes that they are central to any event or situation
What is central control and how does Frith suggest a dysfunction relates to symptoms
- people with SZ experience derailment of thoughts and spoken sentences because each word triggers automatic associations that they cannot suppress
- been identified as a way to explain speech poverty
How do ‘Jumping to Conclusions Bias’ and ‘Reality Monitoring’ tasks relate to symptoms, according to Sarin & Wallin (2014)
- research relating to the role of cognitive biases and found supporting evidence for the claim that the positive symptoms of SZ have their origin in faulty cognition
- Delusional patients were found to show various biases in their informational processing, such as jumping to conclusions and lack of reality testing
- patients with hallucinations were found to have impaired self-monitoring and tended to experience their own thoughts as voices
what can be said about cause and effect of the relationship between cognitive deficits and SZ symtoms
- cognitive approaches DO NOT explain the causes
- links between symptoms and faulty cognitions are however clear
what does CBTp stand for
cognitive behavioural therapy for psychosis
how does efficacy of CBT support the cognitive approach to SZ
- reinforced by the success of CBTp as a treatment for SZ
- NICE review 2014 found CBTp to be a more effective treatment than antipsychotics in reducing symptom severity and social functioning
explain Howes & Murray’s 2014 Intergrative Model
- early vulnerability factors
- biased cognitive processing of this increased dopamine activity results in paranoia, hallucinations and eventually a development of psychosis
- increased the stress of the individual leading to more dopamine release and the cycle continues