Practise viva Flashcards
Budd-Chairi
Nodular liver contour Idiopathic, congenital (venous web), thrombosis (pregnant, OC, polycythaemia, antiphospholipid, sickle cell disease, other) Phlebitis - BMT, chemo Autoimmune Tumour invasion - RCC, HCC, adrenal Ca Leiomyosarc of IVC
Calcific mets
BOTOM Breast Osteosarc Papillary thyroid Ovary Mucinous adenocarcinoma (esp colorectal)
Random points
Second unrelated sign
Common condition on uncommon exam
Talk about findings being persistent or not with fluoroscopy
Random cases
Aspiration on barium swallow
Double cystic duct and other anatomical variants
Renal scarring
Reflux nephropathy
Infarct
Chronic infection
Sigmoid volvulus
Large bowel obstruction
Ahaustral wall, lower end points to pelvis
More common in elderly - associated with chronic neuro conditions, meds for psych conditions, Chagas, chronic laxatives or constipation, fibre rich diet
Rectal tube insertion succesful in treating 90%
Caecal volvulus
Younger patients than sigmoid
Laparotomy for reduction, may need caecoplexy, or hemicolectomy if ischaemic
Emphysematous pyelonephritis
Diabetes E coli, klebsiella, proteus. Retroperitoneal air or air over renal shadow High mortality Drain collections and IV antibiotics
Emphysematous cholecystitis
Diabetes
More common in men
50% acalculous
High rate of perforation
Biliary gas v portal venous gas
Biliary central, portal venous peripheral
Small bowel obstruction radiograph
Herniae
Appendicoliths
Gallstones
Staghorn calculi
Struvite stone
Setting of recurrent infection with urease producing organisms
Proteus, klebsiella, pseudomonas, enterobacter
More common in women, cord injury, renal tract anamoly, reflux
May progress to xanthogranulomatous pyelonephritis
Xanthogranulomatous pyelonephritis
Associated with staghorn calculi
Chronic granulomatous pyelonephritis
E coli, proteus
Surgical nephrectomy
Sclerosing encapsulating peritonitis
Rare benign cause of acute or subacute bowel obstruction
Total or partial encasement of small bowel by thick fibrocollagenous membrane
Idiopathic or secondary to peritoneal dialysis, shunts e.g. VP
And rarely other causes e.g. TB, sarcoid
Wall may calcify
Relative washout
PV - delayed / PV
>40% suggests adenoma
Absolute washout
PV - delayed / PV - unenhanced
>60% suggests adenoma
Abdominal calcification
Don’t forger aneurysm as possible cause
Hypervascular liver mets
Renal, thyroid, breast, lung, melanoma, carcinoid, chodiocarcinoma, other
Central scar
FNH, large haemangioma, fibrolamellar HCC
May also be seen in normal HCC, some mets, cholangiocarc
Hypervascular liver lesion
HCC, haemangioma, FNH, adenoma, mets
FNH
T2 hyperintense scar Delayed enhancement of central scar Takes up hepatobiliary contrast agent - iso cf adenoma hypo Spoke wheel on doppler, DSA Usually soitary, subcapsular mass
Fibrolamellar HCC
May resemble FNH
Will not take up sulphur colloid (Kuppfer cells)
Young adults without cirrhosis
FNH has T2 scar and has less calcs and is less heterogeneous
Better prognosis - 60% 5yr