Postural control lecture Flashcards

1
Q

what is the relationship of COM and BOS?

A

the relationship describes where the individuals mass falls in relation to the base of stability they have created by their stance, feet positioning ect. a symmetrical relationship models 50% of the mass falling in each leg assemetrical resembles leaning more on one leg creating the center of mass to shift to one side. causing more challagnge for the bus to support the com.

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2
Q

what are the common patterns of postural sway

A

moren anterioposterior than medial lateral

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2
Q

when it comes to multi system postural sway what factors impact biomechanics constraints?

A

degrees of freedom
strength
limits of stability

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3
Q

when it comes to multi system postural sway what factors play into movement strategies?

A

reactive
anticipatory
voluntary

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4
Q

when it comes to multi system postural sway what factors play into sensory strategies?

A

sensory integration
sensory reweighing

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5
Q

when it comes to multi system postural sway what factors play into orientation in space?

A

perception
gravity, surfaces, vision
vertically

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6
Q

when it comes to multi system postural sway what factors play into control of dynamics?

A

gait
proactive

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7
Q

when it comes to multi system postural sway what factors play into cognitive processing?

A

attention
learning

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8
Q

explain the process of sensory weighting and sensory integration? what processes are being used?

A

sensory weighting is the idea of prices coming

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9
Q

describe the single flow as it pertains to postural control?

A

sensory singnals:
go to the spinal cord as proprioceptive and skin afferent and will also lead to the cerebral cortex and limbic systems.
these signals are
visual
vestibular
auditory
proproceptive
touch
visceral

The cerebral cortex will receive information from the sensory singals and the thalamus. the cerebral cortex will provide information to the basal ganglia and the brainstem and spinal cord
the cerebral cortex will create anticipatory reactions and can be trained
these are more cognitive processes and voluntary movements

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10
Q

Postural control in stokes

A

83% of pts. 2-4 wks post stroke balance disability
Motor control impairments (caused by reduction in # and firing rate of motor units): slow movements, weakness, fatigue, incoordination, decreased force production, co-contractions

See chart slide 9

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11
Q

what does the research show about stroke patients and traction times of muscles

A

lagged time and less amplitude

tib anterior we see over exaggeration
Ham and Quads- delayed reaction times and less amplitude of contraction

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12
Q

what are the three global impairments in balance when it comes to a person with stroke?

A

sensation (to detect or anticipate postural disturbance),
neural processing (to select appropriate feedback/feedforward postural control ),
effective motor output

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13
Q

what is the major impairment in people with strokes that the research shows

A

Impairment to the timing, magnitude and sequencing of muscle activation

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14
Q

what does postural control look like in patients with PD

A

With ds progression - loss in postural stability, gait dysfunction, frequent falls
Postural instability less responsive to drug therapy
Up to 68% falls in later stages of ds
Lack of balance reaction, flexed posture, decreased trunk rotation, difficulty executing simultaneous movements/sequential movements

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15
Q

what is the roles of the basal ganglia?

A

controlling the flexibility of postural tone
scaling up the magnitude of postural movements
selecting postural strategies for environmental context
automatizing postural responses and gait

16
Q

what are the two cardinal signs of pd?

A

bradykinesia and rigidity

17
Q

when talking about parkensons disease and gait what plays into Bradykinesia?

A

control of dynamics:
gait and proactiveness

movement strategies:
reactive, anticipatory, and voluntary

sensory strategies:
stratagies integration
stratagies reweighing

18
Q

when talking about parkensons disease and gait what plays into Rigidity?

A

degrees of freedom, strength, and limits of stability

19
Q

when talking about parkensons disease and gait what plays into Proprioception deficits?

A

orientation in space:
perception, gravity, surfaces, vision, and verticality

20
Q

when talking about parkensons disease and gait what plays into Non-motor symptoms?

A

Cognitive processing:
attention and learning

21
Q

when looking at the stability margin of patients with parkensons where is limited margins?

A

backwards

22
Q

when looking a patients with parkensons LOPADOPA drugs have shown what?

A

they put patients at an increased risk with balance related issues.
Lopadopa can be counterproductive for balance.
compared to treatment with deep brain stimulation we see a greater impact of instability on lopadopa

23
Q
A