Post Surgical Management Flashcards
Coagulation phase
- Begins immediately and lasts minutes
- Vasoconstriction, platelet aggregation, clot formation
Inflammatory phase
- Begins immediately and within 48 hours wound is epithelialized
- Vasodilation, hematoma formation to bridge defect, increased capillary permeability, migration of leukocytes, influx of extrinsic/intrinsic inflammatory cells.
Granulation phase (reparative phase)
- Fibroblasts appear in 2-3 days and are dominant by day 10
- Fibroblasts are dominant cell types, collagen synthesis
Scar formation (maturation phase)
- Lasts weeks to months, can last up to a year.
- Collagen synthesis, rarely regain full elasticity and strength.
Skeletal ligament injury grading
- Grade I: General structural integrity is intact though there may be bleeding, swelling, and edema present
- Grade II: partial tear, more bleeding and edema
- Grade III: complete tear
Skeletal muscle healing phases
- Inflammatory: immediately and lasts for a few hours
- Proliferative: 1-6 weeks post op
- Remodeling: 7 weeks-1 year
Sheathed tendon difference to be aware of
- Sheathed tendons have limited vascular supply and so rely more on diffusion of nutrients through synovial fluid.
Skeletal muscle injuries
- DOMS: 12-48 hours, microtrauma
- Muscular contusion: local hematoma
- Muscular strain: tearing of muscle (2 joint muscles are most susceptible)
- muscle laceration: sliced, can cause significant limitations
Bone healing inflammatory phase
- Hemorrhage, necrotic cells, hematoma and fibrin to bridge the gap
- Begins immediately
Bone healing soft callus phase
- Fibrous and cartilagenouse tissu forms between fx ends, increased vascularity and increased growth of capillaries into fracture callus. Increase in cellular proliferation, osteoclasts remove dead bone fragments
- 1-6 weeks post injury
Bone healing hard callus phase
- Woven bone develops when the callus converts from fibrocartilagenous, osteoclasts continue removing dead bone, osteoblast activity is abundant
- 4-6 weeks post injury
Bone healing remodeling phase
- Woven bone changes to lamellar bone, medullary canal is then reconstituted, fracture diameter decreases to original width
- 6 weeks up to several months or years
Neuropraxia
- Complete block of nerve transmission with intact nerve fibers, typically d/t stretch injury, blunt force trauma, or prolonged compression.
Axonotmesis
- Nerve sheath remains intact but the axons become divided resulting in complete loss of motor function, sensation, and autonomic functions
Neurotomesis
- A partial or complete severance of the axons and sheath. Most severe form of nerve injury
Wallerian degeneration
- Axon distal to lesion becomes swollen day 1. Muscle innervated by the nerve ceases to contract 2-3 days after injury. Degeneration of nerve distal to injury via phagocytosis
- Immediately to about 3 days post injury
Axonal regeneration
- Begins at proximal end of nerve at injury site. Axons and schwann cells invade the region. Axons create a “growth cone” to traverse the site of the transection. Schwann cells create myelin sheaths
- Begins 4th day. 1 mm/day growth rate, total time depends on the distance to be covered.
Articular cartilage zones
- superficial: can heal independently
- intermediate: unable to repair
- deep zone
- Calcified zone: fibrocartilage typically fills defect but it is not as durable and can lead to eventual degeneration
Red blood cell count normal values
- men: 4.32-5.72 trillion cells/L, women: 3.90-5.00 trillion cells/L
Hemoglobin
- men: 13.5-17.5 g/deciliter, women: 12.0-15.5 g/deciliter
- low: anemia
- high: polycythemia vera, lung disease, dehydration, living at high altitude, heavy smoking, burns, excessive vomiting, extreme physical exertion
Hematocrit
- men: 39-51, women: 34-46
Creatine kinase
- men: 171 U/L, women 145 U/L
- > 2.5-3% = heart damage
- <2.5-3% = skeletal muscle injury
Blood glucose
- 80-110 mg/deciliter
- <70 = hypoglycemic
- 100-125 = prediabetic
- > 126 = diabetic
Effects of immobilization
- 6 weeks of immobilization can lead to a 40% decrease in strength
- Muscle atrophy begins in the first 24 hours
- Training prior to immobilization can limit atrophy
- Slow twitch muscle fibers are more adversely affected
Cervical discectomy pt presentation prior to surgery
- degenerative disc disease
- radiculopathy
- spondylitic myelopathy (gait abnormalities, and positive hoffman and babinski)
Muscles involved in cervical discectomy
- SCM, platysmus, anterior and middle scalene, longus coli muscles
Cervical discectomy reparative phase
- 0-3 weeks
- Focus on promoting dense connective tissue and woven bone, protect surgical site, promote nerve mobility and healing
- most of this is done at home
Cervical discectomy remodeling phase
- 4-52 weeks
- Focus on preventing scar formation, increase strength of affected muscles, provide stress to bone to encourage strengthening and remodeling, neural gliding, increased thoracic mobility.
4-8 weeks following cervical discectomy
- joint mobility for thoracic spine (do not asses cervical joint mobility)
- avoid neural stretching techniques, Do use glides, pt should perform neural glides while minimizing neck movement
- Proprioception/sensorimotor assessment
9-12 weeks following cervical discectomy
- cervical isometrics can be started
- can initiate UE exercises above 90 degrees
- continue to improve scapulothoracic mechanics
- use AROM to help manage pain and stiffness
- can use UBE now
13-52 weeks following cervical discectomy
- goal is to return to PLOF