PMHNP certification Exam KM Deck Flashcards
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<div><b>Which patient is at highest risk for SI</b></div>
<div><b>A. 30y/o married AA female with previous SI attempt *1 risk factor</b></div>
<div><b>B. 35 y/o single Asian male with previous SI attempt *3 risk factors</b></div>
<div><b>C. 38 y/o single AA male who is a manager of a bank *2 risk factors</b></div>
<div><b>D. 68 y/o single white male with depression *5 risk factors (age, male, white, depression)</b></div>
<div><b>D. 68 y/o single white male with depression *5 risk factors (age, male, white, depression)</b></div>
<div></div>
<div>Count the risk factors</div>
When interview teenagers (16 y/o) that arrive with their parents what should you do?
interview them separately from parents. <br></br>-This helps Build therapeutic rapport with teens by telling them the info is confidential. Parents may be upset but remember you are advocating for the child.
Which Ethnic group has the highest rate of suicide?
Native Americans
Example A patient is being treated for schizophrenia with olanzapine. Which of the following is the most common side effect of olanzapine?<br></br>A. Increased waist circumference<br></br>B. EPS (not as common in atypical antipsychotics d/t 5HT2A)-receptor antagonism<br></br>C. Increased Lipids<br></br>D. Metabolic Syndrome
<div><b>D. Metabolic Syndrome</b> (UMBRELLA ANSWER)</div>
Which antipsychotics have the least weight gain?
<div><b>Latuda, Abilify, (also least sedating), Geodon-if patient has metabolic syndrome consider switching to one of the medications above. Or if the patient is overly sedated try switching to ABILIFY</b></div>
Which mood stabilizer have the least weight gain?
Lamictal <br></br>-But remember all mood stabilizers cause some weight gain
When presented with a question about typical vs atypical antipsychotic the answer is usually to start of a
atypical
<div><b>A client presents with complains of changes in appetite, feeling fatigued, problems with sleep-rest cycle, and changes in libido. What is the neuroanatomical area of the brain that is responsible for the normal regulation of these functions?</b></div>
<div><b>A. </b>Thalamus</div>
<div><b>B.</b> Hypothalamus</div>
<div><b>C. </b>Limbic System</div>
<div><b>D. </b>Hippocampus</div>
Hypothalamus<br></br>A, B, & D are all part of the limbic system so you can rule that out
When a patient is hesitant to participate in treatment you should encourage?
Bring a support person like a husband
Thyroid-Stimulating hormone normal level
0.5-5.0 Mu/L
When T4 and T3 are high and TSH is low what is the diagnosis
HYPERTHYROIDISM, TSH secretion decreases: TSH LOW à key symptoms HEAT INTOLERANCE
Key symptoms of Heat Intolerance
Hyperthyroidism
When T4 and T3 are Low and TSH is high what is the diagnosis
(HYPOTHYROIDISM) TSH secretion increased: TSH HIGH à COLD INTERANCE
Key symptoms of Cold Intolerance
Hypothyroidism
Hyperthyroid can mimic
Mania
Hypothyroid can mimic
Depression
A patient on depakote complains of RUQ pain and has reddish/brown urine
Hepatoxicity<br></br>-Check LFTs
Signs of Depakote toxicity
Disorientation, confusion, lethargy
You suspect depakote toxicity what do you do?
Check <br></br>-LFT<br></br>-Ammonia<br></br>-Depakote Level
What herbal supplement can cause hepatoxicity?
Kava Kava
When taking Kava Kava in combinations with other medications you should caution about
Risk of Hepatoxicity and Sedation
TCAs carry a risk of
Hepatotoxicity
Signs of Stevens-Johnson Syndrome
NAME?
two psychotropics known to cause steven johnson syndrome
lamictal and tegretol
What nationality is most suseptible of getting steven johnson?
Asians
When treating asians with tegretal screen for?
HLAB-1502 Allele
What two medications cause agranulocytosis?
Clozaril & Tegretal
Agranulocytosis when to discontinue medication
Less than 1000
When monitoring for agranulocytosis in patients look for s/s of what?
Infection<br></br>-Fever, sore throat, fatigue, chills
Before starting any mood stabilizer in a female of childbearing age be sure to check?
HCG
Which two medications may decrease the risk of suicide?
clozaril and lithium
Medications that increase lithium level
NSAID-ibuprofen, INDOCIN<br></br>THIAZIDES-hydrochlorithiazide ACE INHIBITORS-lisinopril
Ace inhibitors are treatment of choice for?
Heart Failure
Certain medications are known to increase lithium level, but HOW?
by reducing renal clearance
When educating a patient about lithium teach them about
Hyponatremia<br></br>Dehydration-hot days, exercise
Normal Lithium Level
0.6-1.2
Lithium Toxicity
1.5 or above<br></br>Discontinue and re-order lithium level
Lithium level of 1.4
Monitor for toxicity
Labs before starting lithium
<div><b>TSH, BUN, CREATININE, HCG, U/A to check for presence of protein in the urine (4+ protein is concerning for renal impairment)à4+ protein in urine=MONITOR FOR TOXICITY</b></div>
4+ protein in the urine of a patient on lithium
<div><b>4+ protein is concerning for renal impairment</b></div>
<div><b>4+ protein in urine=MONITOR FOR TOXICITY</b></div>
Lithium side effects
<div><b>hypothyroid, leukocytosis, maculopapular rash, t-wave inversion, Coarse Hand Tremor, GI upset (nausea, vomiting, anorexia) </b></div>
<div></div>
<div>-Some of these are also signs of toxicity</div>
Signs of lithium toxicity
confusion, ataxia, GI upset, palpitation, tremor
NMS
<div><b>muscle rigidity, mutism (because of muscle rigidity), increased CPK (caused by muscle contraction and muscle destruction), increase WBC, increased WBC, myoglobinuria (also from muscle destruction)</b></div>
Cherry colored urine in a patient that exercises a lot
test for myoglobinuria may be a sign of rhabdo
Serotonin Syndrome
With any drug that increases 5-HT (e.g., MAO inhibitors, SNRIs, TCAs) hyperthermia, confusion, myoclonus, cardiovascular instability, flushing, diarrhea, seizures. <br></br>-Treatment: cyproheptadine (5-HT2 receptor antagonist).
Treatment for NMS
<div><b>Stop Offending Medication</b></div>
<div><b>-Dantrolene (muscle relaxer)</b></div>
<div><b>-Bromocriptine (Dopamine D2 agonist). </b></div>
<div><b>*In question focus on what they are asking for….dopamine agonist vs muscle relaxer</b></div>
Treatment for Serotonin Syndrome
<div><b>Stop Med (1 or more SSRI, SSNRI, TCA, MOAI)</b></div>
<div><b>-Cyproheptadine</b></div>
Triptans
<div><b>Used for MIGRAINES</b></div>
<div><span><b>-These meds increase serotonin</b></span><b> </b></div>
<div><b>example SUMATRIPTAN </b></div>
<div><b>patient taking Prozac and started on sumatriptan</b></div>
<div><b>-call PCP to ask them to switch the migraine med if patient already on SUMATRIPTAN do not start antidepressant without talking to PCP</b></div>
How long do you wait when switching between an SSRI to an MAOI?
2 weeks
How long do you wait when switching between Prozac and MAOI?
5-6 weeks wash out period
What is the first line treatment for depression and why?
<div><b>SSRI-First line treatment for depression due to less risk of injury from OVERDOSE</b></div>
If a cancer patient has depression what should you consider?
Treating with a medication with minimal drug/drug side effects like Lexapro
Patient with depression worries about sexual dysfunction what would be the medication of choice?
Wellbutrin
Primary symptoms of depression include fatigue and low energy what med would you chose?
Wellbutrin
Wellbutrin is contraindicated in patients with
Seizures and anorexia
Which medications are best for neuropathic pain?
SNRI<br></br>Gabapentin<br></br>TCA
Secondary to the black box warning providers caring for patients on antidepressants should assess for?
Suicidality, frequency, and severity at EVERY appointment
Which meds have the worse serotonin discontinuation syndrome
Those with short half lives<br></br>such as zoloft
Symptoms of serotonin withdrawal syndrome
<div><b>Fever, achiness, soreness, lethargy, fatigue, impaired memory, decreased concentration, GI UPSET</b></div>
<div></div>
<div><span>Shits and Shivers</span></div>
Ages of onset for schizophrenia in males vs females
<div><b>-MALES 18-25 years</b></div>
<div></div>
<div><b>-FEMALE 25-35 years</b></div>
Schizophrenia increases the risk for
<div>SUICIDE</div>
<div><span><b>*HIGH RISK OF SI in SCHIZOPHRENIA*</b></span><b> </b></div>
<div><b>Just having schizophrenia increases your risk of suicide. </b></div>
<div><b>MUST ASK ABOUT SI, EVERYTIME (frequency, severity of thoughts)</b></div>
What increases the causes or increases the risk or schizophrenia
<div><b>excessive pruning of synapses</b></div>
<div><b>-inadequate synapse formation, </b></div>
<div><b>-intrauterine insults such as maternal exposure to toxins, viral agents, maternal substance use, maternal illness, maternal malnutrition, fetal oxygen deprivation, </b></div>
<div><b>-first order relative (mom/dad)</b></div>
MRI or PET scan what is seen in schizophrenia
<div><b>EVERYTHING DECREASES EXCEPT </b><u><b>VENTRICLES</b></u></div>
<div></div>
<div><span><u><b>-You will see VENTRICULAR ENLARGEMENT</b></u></span></div>
Stimulants can potentiate the release of what neurotransmitter?
Dopamine which can worsen symptoms of schizophrenia
Assertive Community Treatment (ACT)
<div><b>a form of rehabilitation post hospitalization, in home treatment</b></div>
What level of prevention is ACT?
Tertiary
What adjunctive treatment is important in schizophrenia
NAME?
Exercise for mental health patients can promote
Cognition<br></br>Quality of Life<br></br>Long-term health
ACT is ideal for patients with a history of
Treatment non-compliance<br></br><br></br>-Think about making the treatment convenient for them–>bringing it to their home
What diagnosis has the highest risk of Homicidality
Antisocial
In the MMSE how do you test for abstraction?
<div><b>proverb interpretation (everyone that lives in glass houses shouldn’t throw stones) Are they able to think abstractly</b></div>
Thought Process-Tangential
<div><b>means that their response has nothing to do with the question</b></div>
Circumstantial
<div><b>means that their response goes in circles instead of getting to the point of the question</b></div>
Mental Status-Thought Content includes
<div><b>SI/HI/AH/VH</b></div>
Another name for MMSE
Folstein Scale
How to assess concentration on MMSE
Serial 7s or perform an activity backwards i.e list the days of the week backwards
Assess ability to learn new material
repeat 3 words after me
Assess ability to recall
repeat 3 words after 5 minutes
Assess fund of knowledge
Who is the president
What is a quick and easy way to assess for neurological issues
Clock drawing test
If patient is unable to draw a clock this indicates
Problem with the right hemisphere, cerebrum, or parietal lobe
mesolimbic pathway
<div><b>Hyperactivity of dopamine in the this pathway mediates positive psychotic symptoms</b></div>
<div></div>
<div><b>-Antagonism of D2 receptors in this pathway treats positive psychotic symptoms</b></div>
mesocortical pathway
<div><b>-Decreased dopamine in the this projection to the dorsolateral prefrontal cortex is postulated to be responsible for negative and depressive symptoms of schizophrenia</b></div>
Nigrostriatal Pathway
<div><b>-This pathway mediates motor movements</b></div>
<div><b>-Dopamine blockade in this pathway can lead to </b><u><b>increase acetylcholine levels</b></u></div>
<div></div>
<div><b>-Blockade of dopamine (D2) receptors in this pathway can lead to EPS, i.e dystonia, parkinsonian symptoms and akathisia</b></div>
<div></div>
Low Dopamine in the nigrostriatal pathway increases which neurotransmitter
<div><b>-Dopamine has a reciprocal relationship with acetylcholine (Ach) (LOW DOPAMINE INCREASE Ach)</b></div>
<div><b>Long-standing D2 blockade in the nigrostriatal pathway can lead to </b></div>
<div><b>tardrive dyskinesia</b></div>
Tuberoinfundibular pathway
<div><b>-Blockade of D2 receptors in this pathway can lead to increase prolactin levels leading to hyperprolactinemia which clinically manifests as amenorrhea, galactorrhea, and sexual dysfunction, gynecomastia</b></div>
<div></div>
<div><b>-DECREASE DOPAMINE INCREASED PROLACTIN</b></div>
<div><b>Long-term hyperprolactinemia can be associated with what condition</b></div>
<div><b>osteoporosis</b></div>
Normal Prolactin Level in Men
<div><b>level less than 20ng/ml</b></div>
Normal Prolactin Level in Women
<div><b>less than 25ng/ml</b></div>
Which medication is the highest offender for increasing prolactin
Risperdal
Acute Dystonia + Treatment
<div><b>neck stiffness, muscle spasm of upper body especially neck/face/tongue</b></div>
<div></div>
<div><b>-Treatment is IM COGENTIN + continue PO COGENTIN for several days</b></div>
Akathisia + Treatment
<div><b>may mimic anxiety, restlessness, can’t sit still, rocking, pacing</b></div>
<div></div>
<div><b>-</b><span><b>First line Treatment</b></span><b> is BETA-BLOCKERS like PROPANOLOL (Inderal)</b></div>
<div><b>-</b><span><b>Second line treatment</b></span><b> is COGENTIN</b></div>
<div><span><b>-Third line treatment</b></span><b> is benzos</b></div>
Beta-Blockers such as Inderal are contraindicated with what type of asthma medication
<div><b>-DO NOT GIVE WITH BROCHODIALATOR such as ALBUTERAL this combination can cause bronchospasm</b></div>
akinesia/bradykinesia + treatment
A. difficulty initiating movement; slowness of movement<br></br>-Treatment Cogentin
PSEUDOPARKINSON or PARKINSONIAN + Treatment
<div><b>caused by dopamine blockade, results in muscle rigidity, mask like facial expression, may look blunted, pill rolling tremors in fingers, shuffling gait, motor slowing</b></div>
<div><b>-Treatment COGENTIN</b></div>
tardive dyskinesia + Treatment
<div><b>abnormal facial movements, grinding teeth, lip smacking, protruding tongue</b></div>
<div></div>
<div><b>-Treatment DECREASE DOSE OF MED, DISCONTINUE MED, Switch to CLOZARIL, Switch to different med, VINPAT</b></div>
Does Cogentin Treat TD
<div><b>COGENTIN MAKES TD WORSE</b></div>
Typical onset of TD
<div><b>OCCURS 1-2 years TYPICALLY, but can be </b><u><b>ACUTE ONSET ALSO</b></u></div>
What non-psych med can cause TD?
<div><b>REGLAN (Metoclopramide) can CAUSE Tardive Dyskinesia must educate patient that this med or the combination of this PLUS antipsychotic can increase risk of TD*** encourage them to discontinue reglan if TD develops</b></div>
<div>In<u><b>D</b></u>ucers CYP450</div>
<div>DECREASE</div>
<div></div>
<div><b>C</b>arbamazepine</div>
<div><b>R</b>ifampin</div>
<div><b>A</b>lcoholics (chronic)</div>
<div><b>P</b>henytoin</div>
<div></div>
<div><b>G</b>risiofulvin</div>
<div><b>P</b>henobarb</div>
<div><b>S</b>ulphonylureas </div>
<div></div>
<div>Crap GPS Induces me to Madness! </div>
<div>Inh<u><b>I</b></u>bitors of CYP450 </div>
INCREASE<br></br><br></br>Ciprofloxacin<br></br>Ritonavir<br></br>Amiodarone<br></br>Cimetidine<br></br>Ketoconazole <br></br><br></br>Acute Etoh<br></br>Macrolides<br></br>INH<br></br>Grapefruit Juice<br></br>Omeprazole<br></br><br></br>Crack Amigos
Erythromycin and Clarithromycin can cause
Increased tegretol levels
<div><b>Patient started on Clozaril or Zyprexa and two months later starts smoking</b></div>
<div><b>as a provider you know that the smoking can decrease the medication effectiveness</b></div>
<div><b>-Increase medication dose</b></div>
Patient has been a chronic smoker and has been stable on Zyrexa but tells you that he recently quit smoking cold turkey
<div><b>as a provider you know that you must now decrease the dose of the antipyshcotic</b></div>
Medications that cause mania
<div><b>Steroids, Disulfiram (Antabuse), Isoniazid (INH), Antidepressants in persons with bipolar</b></div>
<div></div>
<div>-If a patient must take steroids, the provider should <u>increase</u> the mood stabilizer</div>
Medications that cause depression
<div><b>steroids, beta blockers, interferon, Accutane (isotrentinoin), some retroviral drugs, antineoplastic drugs, benzodiazepines, progesterone</b></div>
<div></div>
<div><b>-may need to increase antidepressant</b></div>
Accutane (isotretinoin)
Can cause depression and birth defects
Flonase
As a provider you know that flonase is a STEROID so it may exacerbate mood symptoms<br></br><br></br>Increase mood stabilizer to maintain stability, steroids can also trigger depression
Flonase can trigger mood instability but it can also cause an increase in
<div>Psychosis</div>
<div><b>patient is taking flonase while on antipsychotic but you find that the antipsychotic is ineffective it is likely because the flonase is exacerbating psychosis</b></div>
<div><b>-increase the dose of antipsychotic</b></div>
Neurotransmitters involved in Addiction
Dopamine and GABA
Symptoms of Stimulant Abuse
- agitation/aggression<br></br>2. impaired judgment<br></br>3. euphoria<br></br>4. elevated BP<br></br>5. tachycardia<br></br>6. dilated pupils<br></br>7. hallucinations<br></br>8. TREMORS<br></br>9. IMSOMNIA
If an anorexic patient complains of pain or bloating after eating this may indicate
delayed gastric emptying
Medications that delay gastric emptying
<div><b>Omeprazole, ranitidine, famotidine</b></div>
Proton Pump Inhibitors (omeprazole & Protonix)
Decrease absorption of antipsychotics & SSRI<br></br><br></br>-MUST WAIT TWO HOURS BEFORE TAKING ANTIPSYCHOTIC OR SSRI
When initiating an SSRI on an elderly patient you should advise about
increased anxiety
Paradoxical effect
when meds cause the opposite effect than expected
Apoptosis
programmed cell death/neuronal loss
<div><b>At age 45 and above the patient displays mania for first time what should be ruled out </b></div>
MEDICAL CONDITION
<div><b>Patient with bipolar disorder presents with depressed mood & emotional lability</b></div>
Give Depakote
Hallmark sx of Borderline Personality
Recurrent self harm
Treatment for Borderline Personality
DBT
Creator of DBT
Marsha Linehan
What activity is helpful in making a diagnosis of borderline personality
Journaling or diary keeping
Conversion Disorder
<div><b>STRESS leads to neurological symptoms such as seizures, paresthesia, blindness, mutism</b></div>
Adjustment Disorder
<div><b>adjusting to a situation resulting in depression or anxiety or both or mixed disturbance of emotions and conduct (this type is more common in children: insomnia, peer conflict, verbal altercations, truancy, crying)</b></div>
<div></div>
<div><span><b>-Symptoms occur within 3 months of the stressor</b></span></div>
<div><b>If question states recently moved, recent death….THINK ADJUSTMENT</b></div>
factitious disorder
<div><b>when patients introduce foreign substances into their body or contaminate their food</b></div>
<div><b>-Faking illness but NO MOTIVE BEHIND IT</b></div>
Malingering
<div><b>Faking illness for financial gain</b></div>
Reactive Attachment
<div><b>common in children in foster care, abuse from parents</b></div>
<div><b>-Withdrawn and shows no emotion towards caregiver</b></div>
ODD
<div><b>They deliberately annoy others, no aggression, defiance of authority</b></div>
<div>-Family Therapy is mainstay</div>
<div>-Child management /Parent management skills is the focus in therapy</div>
<div>-Positive reinforcement</div>
<div>-Boundary Setting</div>
Conduct Disorder
<div><b>violence, criminal, fire setting, killing animals, gang activity, +AGGRESSION, NO REMORSE</b></div>
<div><b>-May need meds and therapy</b></div>
<div><b>-Goal of therapy is to target MOOD & AGGRESSSION (mood stabilizers, antipsychotics, alpha agonists/alpha 2 adrenergic receptor blockers such as guanfacine and clonidine)</b></div>
<div><b>-Monitor BP with guanfacine and clonidine</b></div>
Acute Stress Disorder
<div><b>similar to PTSD but the timeline differs</b></div>
<div><b>-heightened arousal, nightmares, flashbacks</b></div>
<div><b>-LESS THAN ONE MONTH</b></div>
PTSD
<div><b>-OVER ONE MONTH</b></div>
<div><b>-3 HALLMARK SXS: intrusive re-experiencing of trauma, increased arousal, avoidance</b></div>
<div><b>-May also have NIGHTMARESà GIVE PRAZOSIN</b></div>
<div><b>-Non-pharm tx of PTSD- EMDR, CBT</b></div>
Panic attack vs Panic disorder (treatment)
Panic attack = BZ<br></br>Panic disorder = SSRI<br></br><br></br>Panic Attack is ACUTE<br></br>Panic Disorder is CHRONIC <br></br><br></br>Feels like impending doom
Tourette’s Syndrome
<div>Criteria for diagnosis</div>
<div>-TWO moto tics and ONE vocal tics</div>
<div>-LASTS more than ONE YEAR</div>
<div>-By age 18</div>
<div><b>**CHILDREN MAY NORMALLY HAVE TICS so if they have one tic only THIS IS NORMAL**</b></div>
Child presents with one tic and the parent is worried
<div><b>**CHILDREN MAY NORMALLY HAVE TICS so if they have one tic only THIS IS NORMAL**</b></div>
Neurotransmitters involved in Tourettes
DNS: Dopamine, Norepinephrine, Serotonin
Treatment for tourettes
<div><b>Treatment: Haldol, Pimozide, Abilify, Guanfacine, clonidine</b></div>
What type of medication can cause tics or exacerbate them
Stimulants
Neurotransmitters involved in mood disorders
DNS: Dopamine, Norepinephrine, Serotonin + GABA
Neurotransmitters involved in ADHD
DNS: Dopamine, Norepinephrine, Serotonin
part of brain implicated in ADHD
prefrontal cortex<br></br>basal ganglia <br></br>reticular activating system
ADHD inattentive type is caused in what part of the brain
Prefrontal Cortex which is known to regulate ATTENTION and EXECUTIVE FUNCTION
dorsolateral prefrontal cortex
Attention<br></br>Executive Function<br></br>Cognition<br></br>Processing<br></br>Working Memory<br></br>Problem Solving
Deficit in the _____ can lead to ADHD inattentive type
Prefrontal Cortex
<div><b>Teacher reports that the stimulant only works for first few hours of class</b></div>
<div><b>medication has worn off too fast. Order multiple dosing throughout the day</b></div>
When does the aftercare plan start
on admission
If parents become anxious while you are educating about a new diagnosis what should you do
<div>-Provide patient and parents information immediately don’t wait till discharge</div>
<div>-Parents may become anxious after a diagnosis of mental illness such as ADHD, <b>stop teaching</b> offer support because they will not absorb the education. <b>Provide supportive therapy</b></div>
Neurotransmitters involved in OCD
serotonin, dopamine, glutamate & GABA
A tic may also be a ___
Compulsion
Facts about OCD
<div>Obsession/Compulsion</div>
<div>-A tic may be a compulsion</div>
<div>-If first order relative has OCD the child’s risk of developing OCD is increased</div>
<div>-Streptococcal infections increase risk of OCD</div>
<div><b>-Treatment SSRI-prozac, Zoloft, if adult you may also use TCA such as clomipramine</b></div>
DMDD
<div><b>6-17 years ONLY</b></div>
<div><b>-Irritability for no reason, sad, depressed mood, tantrums, crying, moody, always mad</b></div>
If patient presents with irritability or labile mood and you need help further delineating symptoms
Administer MOOD QUESTIONAIRE<br></br>7/13 Bipolar Diagnosis Likely
Sleep Disorders are often _____ <br></br>So what should you assess if a parent reports that a child is having nightmares
<div>GENETIC </div>
<div><b>ask if someone in the family has a similar issue with sleep…look for family patterns of sleep problems</b></div>
GAD
<div><b>Worry, apprehension, fear must LAST ATLEAST 6 MONTHS</b></div>
Delirium
<div><b>-ACUTE (within hours to days) onset of disturbance of LOC, COGNITION, </b></div>
<div><b>inattention</b></div>
<div><b>-Urinary Tract Infections are common cause for DELIRIUM always check UA</b></div>
<div><b>-Treatment is antipsychotics like HALDOL</b></div>
Dementia
<div><b>-Chronic and slow onset (months to years to develop)</b></div>
<div><b>-Mental decline in cognition, irritability, personality changes</b></div>
<div>-<b>When asked questions they may try to answer or MAKE UP ANSWERS (confabulate)</b></div>
Low levels of what labs may mimic dementia
Vit B12 and Folic Acid
Cortical Dementia
Language and memory (aphasia and amnesia)
Subcortical Dementia
Motor abnormalities/Mood issues like apathy, depression, irritability<br></br><br></br>HIV Dementia is a type of subcortical dementia
Early signs of HIV dementia
<div>subcortical form of dementia</div>
<div><b>COGNITIVE, MOTOR, BEHEAVIOR for example a patient with lack of coordination, unsteady gait</b></div>
Treatment for HIV dementia
Antivirals
Pseudo Dementia
<div><b>Depression causes the memory issues, common in older adults</b></div>
<div></div>
<div><b>-Also assess onset of symptoms, pseudo dementia is more acute onset</b></div>
<div><b>-When asked questions they often say “I DON’T KNOW”</b></div>
<div></div>
Instruments to use to differentiate between dementia and pseudo dementia
<div><b>-Use instrument to further screen out cognitive issues such as SLUMS, MOCHA, MMSE</b></div>
hallmark of lewy body dementia
visual hallucinations
Frontotemporal lobe Dementia
<div>PICKs Disease </div>
<div><span><b>-Hallmark is personality changes, language difficulties, poor impulse control, and behavioral changes</b></span></div>
<div>-May see slurred speech or difficulty getting words out </div>
What lobe is associated with ability to understand what others are saying (comprehending speech)
Temporal Lobe
Neurotransmitters involved in Autism
GABA, Glutamate, Serotonin
Autism
<div>a disorder that appears in childhood and is marked by deficient communication, social interaction, Poor eye contact, May not respond when you call their name, Stereotypical movement</div>
<div><b>When play they often like to line up their toys, stack them in tidy rows</b></div>